Search results for "Tolerance induction"

showing 10 items of 38 documents

Intrathymic tolerance induction: determination of tolerance to class II major histocompatibility complex antigens in maturing T lymphocytes by a bone…

1987

Two new experimental approaches were established to analyse the influence of the thymus on tolerance induction to major histocompatibility complex (MHC) antigens: The aim of the first experiment was to perform successful transplantation of adult allogeneic thymus tissue into nude mice, an attempt that has been unsuccessful in the past. Tolerance for the MHC genotype of a prospective thymus graft recipient (A) was induced in mice of strain B by injection of (A X B) splenocytes during the neonatal period. Adult thymic tissue obtained from these allogeneic donors (B) were grafted into the nude mice of strain A. The allogeneic thymus was accepted by the nude mice and immunoreconstitution was ac…

LymphocyteT-LymphocytesImmunologyMice NudeBone Marrow CellsThymus GlandBiologyMajor histocompatibility complexImmune toleranceMiceAntigenmedicineImmune ToleranceAnimalsTransplantation HomologousMice Inbred BALB CAge FactorsHistocompatibility Antigens Class IIGeneral MedicineDendritic cellTransplantationMice Inbred C57BLThymic TissueTolerance inductionmedicine.anatomical_structureMice Inbred DBARadiation ChimeraImmunologybiology.proteinScandinavian journal of immunology
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Induction of Cerebral Ischemic Tolerance by Erythromycin Preconditioning Reprograms the Transcriptional Response to Ischemia and Suppresses Inflammat…

2007

Background A single dose of the macrolide antibiotic erythromycin can induce tolerance against cerebral ischemia in vivo (pharmacologic preconditioning). This study identified potential mechanisms of tolerance induction by assessing effects of erythromycin preconditioning on the cerebral transcriptional response to transient global cerebral ischemia. Methods Preconditioned and nonpreconditioned rats were exposed to 15 min of global cerebral ischemia, and changes in cerebral gene expression were identified by complementary DNA expression array and quantified by real-time reverse-transcription polymerase chain reaction. Results Ischemia caused a widespread up-regulation of transcription in n…

MaleDNA ComplementaryTranscription GeneticIschemiaInflammationPharmacologyNeuroprotectionBrain IschemiaProinflammatory cytokineIn vivoGene expressionmedicineAnimalsRNA MessengerRats WistarIschemic PreconditioningAntibacterial agentInflammationReverse Transcriptase Polymerase Chain Reactionbusiness.industryBrainmedicine.diseaseAnti-Bacterial AgentsErythromycinRatsDisease Models AnimalTolerance inductionAnesthesiology and Pain MedicineAnesthesiamedicine.symptombusinessAnesthesiology
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The antibiotic erythromycin induces tolerance against transient global cerebral ischemia in rats (pharmacologic preconditioning).

2006

Background Cerebral ischemic tolerance can be induced by a variety of noxious stimuli, but no clinically applicable regimen for preconditioning has been described. Therefore, the authors tested the ability of a pharmacologic preconditioning strategy using the well-known macrolide antibiotic erythromycin to induce tolerance against transient global cerebral ischemia in vivo. They also investigated whether tolerance induction by erythromycin involves transcriptional and translational changes of cerebral B-cell leukemia/lymphoma-2 (bcl-2) expression. Methods Male Wistar rats were treated with erythromycin (25 mg/kg intramuscularly) or vehicle and subjected to 15 min of transient global cerebr…

MaleIschemiaHippocampusErythromycinPharmacologyNeuroprotectionHippocampusIn vivomedicineAnimalsRNA MessengerRats WistarIschemic PreconditioningAntibacterial agentNeuronsbusiness.industrymedicine.diseaseAnti-Bacterial AgentsErythromycinRatsTolerance inductionAnesthesiology and Pain MedicineProto-Oncogene Proteins c-bcl-2Ischemic Attack TransientImmunologyReperfusionIschemic preconditioningbusinessmedicine.drugAnesthesiology
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Late-onset myasthenia gravis - CTLA4(low) genotype association and low-for-age thymic output of naïve T cells.

2014

Abstract Late-onset myasthenia gravis (LOMG) has become the largest MG subgroup, but the underlying pathogenetic mechanisms remain mysterious. Among the few etiological clues are the almost unique serologic parallels between LOMG and thymoma-associated MG (TAMG), notably autoantibodies against acetylcholine receptors, titin, ryanodine receptor, type I interferons or IL-12. This is why we checked LOMG patients for two further peculiar features of TAMG – its associations with the CTLA4 high/gain-of-function  +49A/A genotype and with increased thymic export of naive T cells into the blood, possibly after defective negative selection in AIRE-deficient thymomas. We analyzed genomic DNA from 116 …

Malemedicine.medical_specialtyGenotypeThymomaT-LymphocytesImmunologyDNA Mutational AnalysisRecent Thymic EmigrantLate onsetCell CountThymus GlandBiologyPeripheral blood mononuclear cellWhite PeopleGene FrequencyInternal medicineGenotypeMyasthenia GravismedicineImmune ToleranceImmunology and AllergyHumansCTLA-4 AntigenGenetic Predisposition to DiseaseGenetic Association StudiesAgedPeripheral tolerance inductionAged 80 and overPolymorphism GeneticThymocytesT-cell receptor excision circlesAutoantibodyCell DifferentiationThymus NeoplasmsMiddle Agedmedicine.diseaseMyasthenia gravisEndocrinologyImmunologyFemaleJournal of autoimmunity
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Early-onset tolerance in rat global cerebral ischemia induced by a mitochondrial inhibitor

2000

It was studied whether a subtoxic dose of the mitochondrial neurotoxin, 3-nitropropionic acid (3-NPA), can initiate early-onset tolerance induction for subsequent ischemic injury. Wistar rats were pretreated for 3 h by intraperitoneal 3-NPA (20 mg/kg body weight; n=13) or solvent (n=12). Fifteen minutes global cerebral ischemia was induced by bilateral carotid artery occlusion and hypobaric hypotension. rCBF and tissue hemoglobin oxygen saturation were measured by laser Doppler scanning and a microspectrophotometric method. Ischemic insult and brain temperature were identical in both groups. Body weight and neurological scores recovered in the pretreated group but further deteriorated in th…

Malemedicine.medical_specialtyTime FactorsNeurotoxinsIschemiaConvulsantsMotor ActivityHippocampal formationBrain IschemiaCentral nervous system diseaseBrain ischemiaProsencephalonInternal medicinemedicineAnimalsNeurotoxinRats WistarNeuronsNeocortexbusiness.industryGeneral NeuroscienceNitro Compoundsmedicine.diseaseMitochondriaRatsTolerance inductionNeuroprotective Agentsmedicine.anatomical_structureEndocrinologyReperfusion InjuryAnesthesiaPropionatesbusinessReperfusion injuryNeuroscience Letters
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Virally Infected Mouse Liver Endothelial Cells Trigger CD8+ T-Cell Immunity

2009

Background & Aims Dendritic cell activation through ligation of pattern recognition receptors leading to full functional maturation causes induction of CD8 + T-cell immunity through increased delivery of costimulatory signals instead of tolerance. Here we investigate whether organ-resident antigen-presenting cells, such as liver sinusoidal endothelial cells (LSECs), also switch from tolerogenic to immunogenic CD8 + T-cell activation upon such stimulation. Methods Murine LSECs were isolated by immunomagnetic separation and analyzed for functional maturation upon triggering pattern recognition receptors or viral infection employing gene expression analysis and T cell coculture assays. In vivo…

MuromegalovirusT cellCD8-Positive T-LymphocytesBiologyLigandsMiceBone MarrowImmune TolerancemedicineAnimalsCytotoxic T cellAntigen-presenting cellCells CulturedOligonucleotide Array Sequence AnalysisToll-like receptorHepatologyChimeraGastroenterologyPattern recognition receptorEndothelial CellsCell DifferentiationHerpesviridae InfectionsDendritic cellAdoptive TransferCell biologyTolerance inductionmedicine.anatomical_structureLiverOrgan SpecificityReceptors Pattern RecognitionImmunologyCD80Gastroenterology
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Dynamic regulation of CD8 T cell tolerance induction by liver sinusoidal endothelial cells.

2010

Abstract Cross-presentation of soluble Ag on MHC class I molecules to naive CD8 T cells by liver sinusoidal endothelial cells (LSECs) leads to induction of T cell tolerance that requires interaction between coinhibitory B7-H1 on LSECs and programmed cell death-1 on CD8 T cells. In this study, we investigate whether cross-presentation of high as well as low Ag concentrations allowed for LSEC-induced tolerance. Ag concentration directly correlated with the cross-presentation capacity of murine LSECs and thus strength of TCR stimulation. Although LSEC cross-presentation at low-Ag concentrations resulted in tolerance, they induced differentiation into effector T cells (CTL) at high-Ag concentra…

OvalbuminT cellImmunologychemical and pharmacologic phenomenaMice TransgenicCD8-Positive T-LymphocytesLymphocyte ActivationResting Phase Cell CycleMiceCross-PrimingAntigenMHC class ImedicineImmune ToleranceImmunology and AllergyCytotoxic T cellAnimalsCells CulturedMice KnockoutAntigen PresentationbiologyT-cell receptorEndothelial CellsCytotoxicity Tests ImmunologicCoculture TechniquesCell biologyMice Inbred C57BLTolerance inductionCTL*medicine.anatomical_structureLiverbiology.proteinCD80Journal of immunology (Baltimore, Md. : 1950)
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Crosstalk of regulatory T cells and tolerogenic dendritic cells prevents contact allergy in subjects with low zone tolerance

2012

Background Allergic contact dermatitis is one of the most common occupational diseases. A main protective mechanism in those who do not develop allergic contact dermatitis is tolerance induction by repeated exposure to low doses of contact allergen, which is termed low zone tolerance (LZT). The mechanisms that determine the tolerance induction in subjects with LZT are still elusive. Objective We performed analysis of the role of CD4 + CD25 + forkhead box protein 3 (FOXP3)–positive regulatory T (Treg) cells and dendritic cells (DCs) in mice with LZT. Methods Mechanisms of tolerance induction were analyzed in a murine model of LZT by using FOXP3 and IL-10 reporter mice, as well as mice that a…

Receptors CCR7Adoptive cell transferImmunologyMice Transgenicchemical and pharmacologic phenomenaCell CommunicationBiologyLymphocyte ActivationT-Lymphocytes RegulatoryMiceImmune ToleranceAnimalsImmunology and AllergyIL-2 receptorInterleukin-2 Receptor alpha SubunitFOXP3Forkhead Transcription Factorshemic and immune systemsDendritic CellsDendritic cellCD11c AntigenInterleukin-10Tolerance inductionInterleukin 10CTLA-4Dermatitis Allergic ContactImmunologyCD8Journal of Allergy and Clinical Immunology
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Novel Immunomodulatory Markers Expressed by Human WJ-MSC: an Updated Review in Regenerative and Reparative Medicine.

2012

Mesenchymal (stromal) stem cells (MSC) are a broad class of stromal populations which are able to differentiate towards mature cell types, and do express molecules involved in immune modulation, tolerance induction and inflammation dampening. MSC can be virtually isolated from each adult organ, as well as from foetus-associated perinatal tissues. In particular, Wharton's jelly-derived MSC (WJ-MSC) bear all of these key properties, together with their ease of sourcing and lack of ethical issues. Cellular therapy is a key technique in regenerative medicine approaches, in particular for the treatment of diseases in which physiological processes of cellular repopulation are blocked by the under…

Stromal cellCellular differentiationImmune modulationRegenerative medicineCell therapyDevelopmental NeuroscienceMedicineProgenitor cellTissue repairUmbilical cordMesenchymal stem cellInflammationbusiness.industrySettore BIO/16 - Anatomia UmanaWharton's jellyMesenchymal stem cellMatrix metalloproteinaseTolerance inductionDifferentiationHypoimmunogenicityImmunologyRegenerative medicineStem cellbusinessNeuroscienceDevelopmental Biology
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Impact on Immune Tolerance induced by Medullary Thymic Epithelial Cells and Limbal Stem Cells

Tolerance induction immunomodulation Organ bioengineering stem cellsTolerance induction Organ bioengineering Limbal Stem Cells Aire expressing cellsSettore MED/13 - Endocrinologia
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