Search results for "Transcription"

showing 10 items of 2278 documents

Long-term expression of differentiated functions in hepatocytes cultured in three-dimensional collagen matrix.

1998

Hepatocytes entrapped in collagen gel and cultured in serum-free conditions survived longer than cells cultured on plastic (5 days vs. 3 weeks), showed fewer signs of early cell senescence (no increase in c-fos oncoprotein expression), and maintained the expression of differentiated hepatic metabolic functions over a longer period of time. Cells cultured in collagen gels retained their ability to respond to hormones. The insulin-stimulated glycogen synthesis rate remained fairly constant during 18 days in culture (between 5.4 +/- 0.37 and 9 +/- 2.7 nmol glucose/h/microg DNA). Collagen-cultured hepatocytes recovered glycogen stores to levels similar to those found in liver, or in hepatocytes…

Malemedicine.medical_specialtyPhysiologyCellular differentiationClinical BiochemistryCell Culture TechniquesIsozymeCulture Media Serum-FreeRats Sprague-Dawleychemistry.chemical_compoundCytochrome P-450 Enzyme SystemInternal medicinemedicineAnimalsInsulinUreaRNA MessengerEnzyme inducerGlycogen synthaseBiotransformationCells CulturedbiologyGlycogenReverse Transcriptase Polymerase Chain ReactionGenes fosCell DifferentiationCell BiologyGlutathioneMolecular biologyExtracellular MatrixLiver GlycogenRatsIsoenzymesEndocrinologychemistryGene Expression RegulationLiverPharmaceutical PreparationsCell cultureEnzyme InductionMethylcholanthrenebiology.proteinMicrosomes LiverHepatocytesCollagenProto-Oncogene Proteins c-fosTranscription Factors
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The Adult Pituitary Shows Stem/Progenitor Cell Activation in Response to Injury and Is Capable of Regeneration

2012

The pituitary gland constitutes, together with the hypothalamus, the regulatory core of the endocrine system. Whether the gland is capable of cell regeneration after injury, in particular when suffered at adult age, is unknown. To investigate the adult pituitary's regenerative capacity and the response of its stem/progenitor cell compartment to damage, we constructed a transgenic mouse model to conditionally destroy pituitary cells. GHCre/iDTR mice express diphtheria toxin (DT) receptor after transcriptional activation by Cre recombinase, which is driven by the GH promoter. Treatment with DT for 3 d leads to gradual GH+ (somatotrope) cell obliteration with a final ablation grade of 80–90% 1…

Malemedicine.medical_specialtyPituitary glandTime FactorsSomatotropic cellMice TransgenicBiologyMiceEndocrinologySOX2Internal medicinemedicineAnimalsRegenerationCell LineageProgenitor cellPromoter Regions GeneticSOXB1 Transcription FactorsStem CellsDeoxyuridineEndothelial stem cellEndocrinologymedicine.anatomical_structureGrowth HormonePituitary GlandIntercellular Signaling Peptides and ProteinsFemaleStem cellHeparin-binding EGF-like Growth FactorAdult stem cellEndocrine glandEndocrinology
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La concentración sanguínea de PGC-1a predice miocardio salvado y remodelado ventricular tras infarto agudo de miocardio con elevación del segmento ST

2015

et al.

Malemedicine.medical_specialtyST-segment elevation acute myocardial infarctionMyocardial InfarctionIschemiaPGC-1αMagnetic Resonance Imaging CineInfarctionRemodelado ventricularElectrocardiographyCardiac magnetic resonance imagingVentricular remodelingInternal medicineEdemamedicineHumansST segmentProspective Studiescardiovascular diseasesMyocardial infarctionVentricular remodelingHeat-Shock ProteinsVentricular Remodelingmedicine.diagnostic_testbusiness.industryMyocardiumEstrés oxidativoStroke VolumeMagnetic resonance imagingGeneral MedicineMiddle AgedPrognosismedicine.diseasePeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaInfarto agudo de miocardio con elevación del segmento STMetabolismo oxidativoOxidative stresscardiovascular systemCardiologyOxidative metabolismFemalemedicine.symptombusinessFollow-Up StudiesTranscription FactorsRevista Española de Cardiología (English Edition)
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Assessment of hepatitis C virus-RNA clearance under combination therapy for hepatitis C virus genotype 1: performance of transcription-mediated ampli…

2007

Monitoring of HCV-RNA in blood during antiviral therapy is performed mostly by commercially available reverse transcription polymerase chain reaction-based (RT-PCR) assays, with a lower detection limit of 30-50 IU/mL of HCV-RNA. Use of different tests in the pivotal trials of combination therapy has generated some discordance, in terms of predictive value of the early virological response (EVR). To evaluate whether the use of a more sensitive test, as a qualitative assay based on transcription mediated amplification (TMA) with a lower detection limit of 5-10 IU/mL of HCV-RNA, may obtain a better prediction of EVR and of the ultimate virological outcome, we retrospectively evaluated serial s…

Malemedicine.medical_specialtySettore MED/07 - Microbiologia E Microbiologia ClinicaSettore MED/09 - Medicina InternaCombination therapyGenotypeTranscription GeneticTranscription-mediated amplificationHepacivirusAlpha interferonHepacivirusInterferon alpha-2GastroenterologyAntiviral AgentsSensitivity and Specificityantiviral therapy EVR HCV chronic hepatitis HCV-RNA RT-PCR TMAPolyethylene Glycolschemistry.chemical_compoundInterferonPredictive Value of TestsVirologyInternal medicineRibavirinmedicineHumansRetrospective StudiesSettore MED/12 - GastroenterologiaHepatologybiologybusiness.industryReverse Transcriptase Polymerase Chain ReactionRibavirinInterferon-alphaNucleic acid amplification techniqueHepatitis C Chronicbiology.organism_classificationVirologydigestive system diseasesRecombinant ProteinsInfectious DiseasesReal-time polymerase chain reactionTreatment OutcomechemistryRNA ViralDrug Therapy CombinationFemalebusinessNucleic Acid Amplification Techniquesmedicine.drug
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The impact of ovarian stimulation on the expression of candidate reprogramming genes in mouse preimplantation embryos.

2012

Ovarian stimulation with gonadotrophins is an integral part of assisted reproductive technologies in human subfertility/infertility treatment. Recent findings have associated ovarian stimulation with the increased incidence of imprinting disorders in humans as well as defects in genome-wide methylation reprogramming and, in particular, imprinting in mice. Here, we present the first study that determined the impact of ovarian stimulation on the expression of developmentally important reprogramming genes <i>(Apex1, Lig1, Lig3, Mbd2, Mbd3, Mbd4, </i>and<i> Polb)</i> in single early mouse morula embryos (16-cell stage). Using absolute quantification of mRNA by quantitati…

Malemedicine.medical_specialtyTime FactorsGonadotropins EquineDown-RegulationStimulationReproductive technologyBiologyChorionic GonadotropinMBD4AndrologyMiceOogenesisOvulation InductionInternal medicineGeneticsmedicineDNA-(Apurinic or Apyrimidinic Site) LyaseAnimalsHumansHorsesRNA MessengerMolecular BiologyGenetics (clinical)GametogenesisDNA Polymerase betaRegulation of gene expressionReverse Transcriptase Polymerase Chain ReactionEmbryogenesisGene Expression Regulation DevelopmentalEmbryoDNA-Binding ProteinsMice Inbred C57BLEndocrinologyBlastocystMicroscopy Fluorescenceembryonic structuresFemaleReprogrammingTranscription FactorsCytogenetic and genome research
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Hepatocyte growth factor/hepatopoietin A is expressed in fat-storing cells from rat liver but not myofibroblast-like cells derived from fat-storing c…

1992

Hepatocyte growth factor/hepatopoietin A is a complete mitogen for parenchymal liver cells, and its expression is increased as an early response to acute liver injury. To identify the liver cell population responsible for hepatocyte growth factor gene expression, we investigated tissue sections and isolated and purified cell fractions from normal rat liver by in situ and Northern blot hybridization. Hepatocyte growth factor transcripts were present in sinusoidal liver cells, which were preferentially located in the periportal parenchyma. Northern hybridization analysis of RNA isolated from purified liver cell fractions demonstrated that HGF messenger RNA is present only in fat-storing cells…

Malemedicine.medical_specialtyTranscription Geneticmedicine.medical_treatmentGene ExpressionMESSENGER-RNA; MOLECULAR-CLONING; MARKED INCREASE; VITAMIN-A; PURIFICATION; COLLAGEN; SEQUENCE; STORAGE; GENESBiologyCell LineParacrine signallingInternal medicinemedicineAnimalsNorthern blotRNA MessengerGrowth SubstancesHepatologyHepatocyte Growth FactorGrowth factorLiver cellNucleic Acid HybridizationMuscle SmoothRats Inbred StrainsFibroblastsBlotting NorthernLipid MetabolismCell biologyRatsEndocrinologymedicine.anatomical_structureHepatocyte nuclear factor 4LiverCell cultureHepatocyteHepatocyte growth factormedicine.drug
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Mild phenotypes in a series of patients with Opitz GBBB syndrome with MID1 mutations

2004

Contains fulltext : 48815.pdf (Publisher’s version ) (Closed access) Opitz syndrome (OS; MIM 145410 and MIM 300000) is a congenital midline malformation syndrome characterized by hypertelorism, hypospadias, cleft lip/palate, laryngotracheoesophageal (LTE) abnormalities, imperforate anus, developmental delay, and cardiac defects. The X-linked form (XLOS) is caused by mutations in the MID1 gene, which encodes a microtubule-associated RBCC protein. In this study, phenotypic manifestations of patients with and without MID1 mutations were compared to determine genotype-phenotype correlations. We detected 10 novel mutations, 5 in familial cases, 2 in sporadic cases, and 3 in families for whom it …

Malemedicine.medical_specialtyUbiquitin-Protein LigasesBiologymedicine.disease_causeGastroenterologyG/BBB SYNDROMEFAMILIESGenomic disorders and inherited multi-system disorders [IGMD 3]Genotype-phenotype distinctionInternal medicineGeneticsmedicineHumansHypertelorismGeneGenetics (clinical)GeneticsFamily HealthX-linked Opitz syndromeMutationMID1Nuclear ProteinsGenetic Diseases X-LinkedExonsOpitz G/BBB Syndromemedicine.diseasePhenotypeGENEPedigreeSmith-Lemli-Opitz SyndromePhenotypeGenetic defects of metabolism [UMCN 5.1]HypospadiasMutationMicrotubule ProteinsFemalephenotypic variabilityXP22medicine.symptomImperforate anusFunctional Neurogenomics [DCN 2]BBBTranscription FactorsAmerican Journal of Medical Genetics. Part A
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Fludarabine prevents smooth muscle proliferation in vitro and neointimal hyperplasia in vivo through specific inhibition of STAT-1 activation.

2007

Drug-eluting stents are increasingly used to reduce in-stent restenosis and adverse cardiac events after percutaneous coronary interventions. However, the race for the ideal drug-eluting stent is still on, with special regard to the best stent-coating system and the most effective and less toxic drug. Fludarabine, a nucleoside analog, has both anti-inflammatory and antiproliferative cellular effects. The aim of the present study was to assess the cellular and molecular effects of fludarabine on vascular smooth muscle cell (VSMC) growth in vitro and in vivo and the feasibility and efficacy of a fludarabine-eluting stent. To study the biomolecular effects of fludarabine on VSMC proliferation…

Malemedicine.medical_specialtyVascular smooth muscleTime FactorsPhysiologyMyocytes Smooth MusclePharmacologyProsthesis DesignTransfectionMuscle Smooth VascularRestenosisIn vivoPhysiology (medical)medicineAnimalsCarotid StenosisRNA AntisensePhosphorylationRats WistarAortaCells CulturedCell ProliferationNeointimal hyperplasiaHyperplasiaDose-Response Relationship Drugbusiness.industryCardiovascular AgentsHyperplasiaJanus Kinase 2medicine.diseaseFludarabineSurgeryRatsDisease Models AnimalSTAT1 Transcription FactorCardiovascular agentSTAT proteinFeasibility StudiesStentsRabbitsCardiology and Cardiovascular MedicinebusinessCarotid Artery InjuriesTunica IntimaAngioplasty BalloonVidarabinemedicine.drug
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A non-redundant role for OX40 in the competitive fitness of Treg in response to IL-2.

2010

OX40 stimulation is known to enhance activation of effector T cells and to inhibit induction and suppressive function of Treg. Here we uncovered a novel role of OX40 in sustaining Treg competitive fitness in vivo, during repopulation of lymphopenic hosts and reconstitution of BM chimeras. Defective expansion of OX40-null Treg diminished their ability to suppress inflammation in a model of lymphopenia-driven colitis. OX40-mediated promotion of Treg fitness spanned beyond lymphopenic environments, as endogenous Treg in OX40-null mice showed decreased accumulation during thymic development, enhanced susceptibility to antibody-mediated depletion and defective turnover following thymectomy. In v…

Malemedicine.medical_treatmentImmunologyBlotting Westernchemical and pharmacologic phenomenaEndogenyInflammationSuppressor of Cytokine Signaling ProteinsT-Lymphocytes RegulatoryMiceSuppressor of Cytokine Signaling 1 ProteinLymphopeniaOX40; Treg; IL-2.medicineSTAT5 Transcription FactorImmunology and AllergyAnimalsOX40PhosphorylationReceptorSTAT5Cell ProliferationMice KnockoutbiologyEffectorCell growthSuppressor of cytokine signaling 1hemic and immune systemsReceptors OX40IL-2.ColitisFlow Cytometrycytokinescompetitive fitnessSpecific Pathogen-Free OrganismsThymectomyMice Inbred C57BLTregRadiation ChimeraImmunologybiology.proteinInterleukin-2costimulatory moleculesmedicine.symptomcompetitive fitness; costimulatory molecules; cytokines; treg
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Genome-Wide Expression Profiles in Very Low Birth Weight Infants With Neonatal Sepsis

2014

BACKGROUND: Bacterial sepsis is associated with high morbidity and mortality in preterm infants. However, diagnosis of sepsis and identification of the causative agent remains challenging. Our aim was to determine genome-wide expression profiles of very low birth weight (VLBW) infants with and without bacterial sepsis and assess differences. METHODS: This was a prospective observational double-cohort study conducted in VLBW (<1500 g) infants with culture-positive bacterial sepsis and non-septic matched controls. Blood samples were collected as soon as clinical signs of sepsis were identified and before antibiotics were initiated. Total RNA was processed for genome-wide expression an…

Malemedicine.medical_treatmentInfant Premature DiseasesCohort StudiesSepsisSepsisGene expressionHumansInfant Very Low Birth WeightMedicineProspective StudiesGeneGram-Positive Bacterial InfectionsPrincipal Component AnalysisNeonatal sepsisTumor Necrosis Factor-alphabusiness.industryInfant NewbornBacterial Infectionsmedicine.diseaseImmunity InnateReverse transcription polymerase chain reactionLow birth weightEarly DiagnosisCytokinePediatrics Perinatology and Child HealthImmunologyCytokinesFemaleTumor necrosis factor alphamedicine.symptomGram-Negative Bacterial InfectionsTranscriptomebusinessGenome-Wide Association StudySignal TransductionPediatrics
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