Search results for "Transfection"

showing 10 items of 581 documents

CD83+ human dendritic cells transfected with tumor peptide cDNA by electroporation induce specific T-cell responses: A potential tool for gene immuno…

2000

Dendritic cells (DC) are the most potent immunostimulatory cells, with the capacity to induce primary T-cell responses. Functional autologous DC can be generated from fetal calf serum-free peripheral blood mononuclear cells in the presence of interleukin-4 and granulocyte-macrophage colony-stimulating factor and are stimulated with a defined cytokine cocktail for terminal maturation. We were able to establish a nonviral transfection protocol for these DC by electroporation. Using enhanced green fluorescent protein as a reporter gene, we achieved transfection efficiencies of up to 10%. FACScan analyses revealed a stable phenotype, and the expression of major histocompatibility complex class …

Cancer Researchanimal structuresDNA Complementaryvirusesmedicine.medical_treatmentT cellT-LymphocytesGreen Fluorescent ProteinsImmunoglobulinsTransfectionGreen fluorescent proteinAntigens CDGenes ReportermedicineHumansMolecular BiologyCells CulturedReporter geneMembrane GlycoproteinsChemistryElectroporationfungiGranulocyte-Macrophage Colony-Stimulating FactorImmunotherapyTransfectionDendritic CellsGenetic TherapyFlow CytometryMolecular biologyRecombinant ProteinsLuminescent ProteinsCytokinemedicine.anatomical_structureElectroporationembryonic structuresMolecular MedicineImmunotherapyInterleukin-4Clone (B-cell biology)Cancer gene therapy
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?(1,3)Fucosyltransferase expression in E-selectin-mediated binding of gastrointestinal tumor cells

1996

E-selectin recognizes the oncofetal antigen sialyl-Lewis X, which is highly expressed in adenocarcinoma. Five α(1,3)fucosyltransferases (FT) have been cloned that confer cell-surface expression of sialyl-Lewis X on transfected cells. We show here that 12/18 gastrointestinal-tumor cell lines bind specifically to immobilized E-selectin and that in sialyl-Lewis-X-positive cells binding is inhibited with a monoclonal antibody against sialyl-Lewis X. Using RT-PCR, we determined the expression of the α(1,3)fucosyltransferases III, IV, V, VI and VII in gastrointestinal tumor cells. Transcripts of FT IV and FT VII are abundantly expressed in all tested cells. Therefore no single fucosyltransferase …

Cancer Researchmedicine.medical_specialtyFucosyltransferasebiologyCell adhesion moleculemedicine.drug_classTransfectionMonoclonal antibodyMolecular biologycarbohydrates (lipids)FucosyltransferasesEndocrinologyOncologyCell cultureInternal medicineE-selectinmedicinebiology.proteinOncofetal antigenInternational Journal of Cancer
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Cyclooxygenase-2 inhibition induces apoptosis signaling via death receptors and mitochondria in hepatocellular carcinoma.

2006

AbstractInhibition of cyclooxygenase (COX)-2 elicits chemopreventive and therapeutic effects in solid tumors that are coupled with the induction of apoptosis in tumor cells. We investigated the mechanisms by which COX-2 inhibition induces apoptosis in hepatocellular carcinoma (HCC) cells. COX-2 inhibition triggered expression of the CD95, tumor necrosis factor (TNF)-R, and TNF-related apoptosis-inducing ligand (TRAIL)-R1 and TRAIL-R2 death receptors. Addition of the respective specific ligands further increased apoptosis, indicating that COX-2 inhibition induced the expression of functional death receptors. Overexpression of a dominant-negative Fas-associated death domain mutant reduced COX…

Cancer Researchmedicine.medical_specialtyProgrammed cell deathCarcinoma HepatocellularApoptosisMitochondria LiverBiologyTransfectionReceptors Tumor Necrosis FactorInternal medicineCell Line TumormedicineHumansfas ReceptorDeath domainInhibitor of apoptosis domainSulfonamidesCyclooxygenase 2 InhibitorsIntrinsic apoptosisLiver NeoplasmsFas receptorReceptors TNF-Related Apoptosis-Inducing LigandEndocrinologyOncologyUVB-induced apoptosisApoptosisCelecoxibCyclooxygenase 2Cancer researchPyrazolesSignal transductionSignal TransductionCancer research
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Small interfering RNA (siRNA) inhibits the expression of the Her2/neu gene, upregulates HLA class I and induces apoptosis of Her2/neu positive tumor …

2003

Silencing of a specific mRNA using double stranded RNA oligonucleotides represents one of the newest technologies for suppressing a specific gene product. Small interfering RNA (siRNA) are 21 nucleotides long, double stranded RNA fragments that are identical in sequence to the target mRNA. We designed 3 such siRNA against the Her2/neu (HER2) gene. The HER2 gene is known to play an important role in the oncogenesis of several types of cancers, such as breast, ovarian, colon and gastric cancers. Introduction of the siRNA into HER2 positive tumor lines in vitro greatly reduced the cell surface expression of the HER2 protein. Concurrently, a range of effects on cell physiology, such as growth i…

Cancer Researchmedicine.medical_specialtySmall interfering RNAApoptosisBreast NeoplasmsAntibodies Monoclonal HumanizedTransfectionHER2/neuGene productRNA interferenceInternal medicineCell Line TumormedicineGene silencingHumansGene SilencingRNA Small Interferingskin and connective tissue diseasesneoplasmsOvarian NeoplasmsMessenger RNAbiologyHistocompatibility Antigens Class IRNAAntibodies MonoclonalTransfectionGenes erbB-2TrastuzumabUp-RegulationEndocrinologyOncologyCancer researchbiology.proteinFemaleInternational journal of cancer
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Comparative antitumor effect among GM-CSF, IL-12 and GM-CSF+IL-12 genetically modified tumor cell vaccines.

2013

Genetically modified cells have been shown to be one of the most effective cancer vaccine strategies. An evaluation is made of the efficacy of both preventive and therapeutic antitumor vaccines against murine melanoma, using C57BL/6 mice and irradiated B16 tumor cells expressing granulocyte and macrophage colony-stimulating factor (GM-CSF), interleukin-12 (IL-12) or both. Tumor was transplanted by the injection of wild-type B16 cells. Tumor growth and survival were measured to evaluate the efficacy of vaccination. Specific humoral response and immunoglobulin G (IgG) switch were evaluated measuring total IgG and IgG1 and IgG2a subtypes against tumor membrane proteins of B16 cells. In prevent…

Cancer Researchmedicine.medical_treatmentMelanoma ExperimentalBiologyTransfectionCancer VaccinesImmunotherapy AdoptiveImmunoglobulin GMicemedicineMacrophageAnimalsMolecular BiologyMicroscopy ConfocalMelanomaGranulocyte-Macrophage Colony-Stimulating FactorImmunotherapymedicine.diseaseInterleukin-12Survival AnalysisGenetically modified organismVaccinationMice Inbred C57BLImmunologyInterleukin 12biology.proteinMolecular MedicineCancer vaccineCancer gene therapy
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Re-expression of C/EBP alpha induces CYP2B6, CYP2C9 and CYP2D6 genes in HepG2 cells.

1998

Cytochrome P450 (CYP) activity is very low or even absent in human hepatomas, a phenomenon that is accompanied by low levels of some liver transcription factors, notably C/EBP alpha. To investigate a possible link between this transcription factor and hepatic CYP expression, we have stably transfected HepG2 cells with a C/EBP alpha vector containing a Zn-inducible metallothionein promoter. Expression of functional C/EBP alpha up to liver levels concomitantly increased the mRNAs of several members of the CYP2 family (2B6, 2C9 and 2D6), suggesting that this transcription factor may play a relevant role in controlling the hepatic expression of CYP enzymes.

Carcinoma HepatocellularCYP2B6BiophysicsHepG2 cellTransfectionBiochemistryGene Expression Regulation EnzymologicCytochrome P-450 Enzyme SystemStructural BiologyTumor Cells CulturedGeneticsHumansMetallothioneinRNA MessengerVector (molecular biology)Molecular BiologyTranscription factorGeneCells CulturedCytochrome P-450 CYP2C9biologyChemistryNuclear ProteinsCytochrome P450Oxidoreductases N-DemethylatingCell BiologyTransfectionMolecular biologyDNA-Binding ProteinsCytochrome P-450 CYP2B6C/EBPαCytochrome P-450 CYP2D6Steroid 16-alpha-HydroxylaseHepatocyte nuclear factor 4 alphaEnzyme InductionSteroid HydroxylasesCCAAT-Enhancer-Binding Proteinsbiology.proteinAryl Hydrocarbon HydroxylasesHuman hepatocyteCytochrome P450 gene regulationTranscription Factors
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Novel cell culture systems for the hepatitis C virus.

2001

Infections with the hepatitis C virus (HCV) are a major cause of acute and chronic liver disease. The high prevalence of the virus, the insidious course of the disease and the poor prognosis for long-term persistent infection make this pathogen a serious medical and socioeconomical problem. The identification of the viral genome approximately 10 years ago rapidly led to the delineation of the genomic organization and the structural and biochemical characterization of several virus proteins. However, studies of the viral life cycle as well as the development of antiviral drugs have been difficult because of the lack of a robust and reliable cell culture system. Numerous attempts have been un…

Carcinoma HepatocellularVirus CultivationvirusesHepacivirusHepatitis C virusGenome ViralHepacivirusmedicine.disease_causeTransfectionVirus ReplicationVirusFlaviviridaeViral life cycleVirologymedicineTumor Cells CulturedAnimalsHumansCells CulturedPharmacologybiologyLiver Neoplasmsbiology.organism_classificationVirologyViral replicationCell cultureDrug DesignImmunologyRepliconViral diseaseAntiviral research
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Replication of subgenomic hepatitis C virus RNAs in a hepatoma cell line.

1999

An estimated 170 million persons worldwide are infected with hepatitis C virus (HCV), a major cause of chronic liver disease. Despite increasing knowledge of genome structure and individual viral proteins, studies on virus replication and pathogenesis have been hampered by the lack of reliable and efficient cell culture systems. A full-length consensus genome was cloned from viral RNA isolated from an infected human liver and used to construct subgenomic selectable replicons. Upon transfection into a human hepatoma cell line, these RNAs were found to replicate to high levels, permitting metabolic radiolabeling of viral RNA and proteins. This work defines the structure of HCV replicons funct…

Carcinoma HepatocellularVirus CultivationvirusesHepatitis C virusDrug ResistanceGenome ViralHepacivirusBiologyViral Nonstructural Proteinsmedicine.disease_causeTransfectionVirus ReplicationViruschemistry.chemical_compoundmedicineTumor Cells CulturedHumansCloning MolecularNS5ANS5BSubgenomic mRNAGeneticsNS3MultidisciplinaryLiver NeoplasmsVirologyHepatitis CNS2-3 proteaseViral replicationchemistryRNA ViralRepliconGentamicinsScience (New York, N.Y.)
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Can Hepatoma Cell Lines be Re-differentiated to be Used in Drug Metabolism Studies?

2013

Knowledge of metabolism, enzymes so far involved, and potential enzyme-inhibiting or enzyme-inducing properties of new compounds is a key issue in drug development. Primary cultured hepatocytes, cytochrome P450 (CYP)-engineered cells and hepatoma cell lines are currently being used for this purpose, but only primary cultures can produce a metabolic profile of a drug similar to that found in vivo and can respond to inducers. Because of their limited accessibility, alternatives to replace human hepatocytes are currently being explored, including the immortalisation of hepatocytes by using different strategies (i.e. SV40 T-large antigen, conditionally immortalised hepatocytes, transfection wi…

Carcinoma Hepatocellularbiologybusiness.industryTransgeneCellular differentiationLiver NeoplasmsCytochrome P450Cell DifferentiationGeneral MedicineTransfectionToxicologyGeneral Biochemistry Genetics and Molecular BiologyBiotechnologyCell biologyMedical Laboratory TechnologyCytochrome P-450 Enzyme SystemDrug developmentCell cultureCell Line Tumorbiology.proteinHumansbusinessTranscription factorDrug metabolismTranscription FactorsAlternatives to Laboratory Animals
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A novel cell model to study the function of the adrenoleukodystrophy-related protein

2006

X-linked adrenoleukodystrophy (X-ALD) is a neurodegenerative disorder due to mutations in the ABCD1 (ALD) gene. ALDRP, the closest homolog of ALDP, has been shown to have partial functional redundancy with ALDP and, when overexpressed, can compensate for the loss-of-function of ALDP. In order to characterize the function of ALDRP and to understand the phenomenon of gene redundancy, we have developed a novel system that allows the controlled expression of the ALDRP-EGFP fusion protein (normal or non-functional mutated ALDRP) using the Tet-On system in H4IIEC3 rat hepatoma cells. The generated stable cell lines express negligible levels of endogenous ALDRP and doxycycline dosage-dependent lev…

Carcinoma Hepatocellularendocrine system diseasesRecombinant Fusion ProteinsBiophysicsGene redundancyATP-binding cassette transporterContext (language use)BiologyATP Binding Cassette Transporter Subfamily DProtein EngineeringTransfectionBiochemistryCell Line TumormedicineAnimalsAdrenoleukodystrophyMolecular BiologyGeneCell BiologyPeroxisomemedicine.diseaseFusion proteinRatsCell biologyDisease Models AnimalBiochemistryATP-Binding Cassette TransportersAdrenoleukodystrophyFunction (biology)
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