Search results for "Transfection"

showing 10 items of 581 documents

Systematic Study of a Library of PDMAEMA-Based, Superparamagnetic Nano-Stars for the Transfection of CHO-K1 Cells.

2017

The introduction of the DNA into mammalian cells remains a challenge in gene delivery, particularly in vivo. Viral vectors are unmatched in their efficiency for gene delivery, but may trigger immune responses and cause severe side-reactions. Non-viral vectors are much less efficient. Recently, our group has suggested that a star-shaped structure improves and even transforms the gene delivery capability of synthetic polycations. In this contribution, this effect was systematically studied using a library of highly homogeneous, paramagnetic nano-star polycations with varied arm lengths and grafting densities. Gene delivery was conducted in CHO-K1 cells, using a plasmid encoding a green fluore…

magnetic nanoparticlesPDMAEMAPolymers and PlasticsEGFP02 engineering and technologyATRPPDEGMAGene delivery010402 general chemistry01 natural sciencesArticleViral vectorGreen fluorescent proteinpolycationchemistry.chemical_compoundPlasmidIn vivogene deliveryChemistryChinese hamster ovary cellcellular uptakeCHO cellsGeneral ChemistryTransfection021001 nanoscience & nanotechnologyMolecular biology0104 chemical sciencestransfectionBiophysics0210 nano-technologyEthylene glycolATRP; cellular uptake; CHO cells; EGFP; gene delivery; magnetic nanoparticles; PDMAEMA; PDEGMA; polycation; transfectionPolymers
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Geranylgeranyl as well as farnesyl moiety is transferred to Ras p21 overproduced in adrenocortical cells transformed by c-Ha-rasEJ oncogene.

1997

The ras-transformed newborn rat adrenocortical (RTAC) cells were obtained by transfection with the mutated c-Ha-rasEJ oncogene. They are proliferative and tumorigenic cells characterized by expression of the c-Ha-rasEJ oncogene and overexpression of a wild-type ras oncogene. The overproduced Ras p21 was identified here as Ki-Ras p21 by western blotting using a specific anti-Ki-Ras monoclonal antibody. Radioactivity derived from [14C]mevalonolactone was strongly incorporated into Ras p21 overproduced in RTAC cells. RTAC cells pretreated with lovastatin and labeled with either [3H]geranylgeranyl-pyrophosphate or [3H]farnesyl-pyrophosphate incorporated also radioactivity into Ras p21. These re…

medicine.drug_classChemistryBiophysicsProtein PrenylationMevalonic AcidCell BiologyTransfectionMonoclonal antibodyBiochemistryMolecular biologyRatsBlotProto-Oncogene Proteins p21(ras)GeranylgeranylationCell Transformation NeoplasticPrenylationmedicineAdrenal CortexMoietyAnimalsLovastatinMolecular Biologymedicine.drugBiochemical and biophysical research communications
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Aldosterone biosynthesis induced by ACTH and angiotensin II in newborn rat adrenocortical cells transfected by c-EJ-Ha-ras oncogene

1991

Abstract Adrenocortical cells were obtained by fractionated trypsination of newborn rat adrenal glands and transfected with a plasmid containing the EJ T24 -Ha-ras oncogene. Isolation of adhesive cells led to a proliferative cell line with an overexpression of 21 kDa ras protein. These cells incubated with corticosterone or deoxycorticosterone as the precursor produced a high level of 18-hydroxycorticosterone and aldosterone as identified by gas chromatography- mass spectrometry. ACTH and angiotensin II increased the basal production of aldosterone nineteen-fold and six-fold respectively. Under ACTH stimulation the ratio between aldosterone and 18-hydroxycorticosterone production was 1:3. T…

medicine.medical_specialtyBiophysicsBiologyPeptide hormoneTransfectionBiochemistryMass SpectrometryProto-Oncogene Proteins p21(ras)chemistry.chemical_compoundAdrenocorticotropic HormoneCorticosteroneInternal medicineAdrenal GlandsmedicineAnimals18-HydroxycorticosteroneAldosteroneMolecular BiologyChromatography High Pressure LiquidAldosteroneOncogeneAdrenal cortexCell growthAngiotensin IICell BiologyAngiotensin IIRatsEndocrinologymedicine.anatomical_structurechemistryCell cultureBiochemical and Biophysical Research Communications
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Cationic lipide mediated transfer of c-abl and bcr antisense oligonucleotides to immature normal myeloid cells: Uptake, biological effects and modula…

1996

Uptake and biochemical and biological effects of antisense oligodeoxynucleotides (ODN) specific for c-abl and bcr genes were studied in normal immature myeloid cells. CD34-positive cells were purified by positive and negative selection and cultured in liquid culture for 7 days. These cells were then incubated with ODNs, either alone or in combination with cationic lipids. The uptake of ODNs was enhanced by the use of cationic lipids. In addition, very low concentrations of ODNs in combination with cationic lipids were capable of specifically inhibiting the expression of the c-abl gene. In contrast, no effects were seen on the expression of bcr. However, despite the effective blocking of c-a…

medicine.medical_specialtyCell Membrane PermeabilityChemical PhenomenaMolecular Sequence DataRibonuclease HAntigens CD34BiologyTransfectionPolymerase Chain ReactionCationsProto-Oncogene Proteinshemic and lymphatic diseasesInternal medicineGene expressionmedicineHumansCation Exchange ResinsRNA NeoplasmProto-Oncogene Proteins c-ablGeneCells CulturedOncogene ProteinsABLHematologyBase SequenceCell-Free SystemChemistry PhysicalCell growthCationic polymerizationbreakpoint cluster regionBiological Transporthemic and immune systemsHematologyGeneral MedicineOligonucleotides AntisenseProtein-Tyrosine Kinasesrespiratory systemHematopoietic Stem CellsLipidsMolecular biologyHaematopoiesisGene Expression RegulationDepression ChemicalLiposomesProto-Oncogene Proteins c-bcrAnnals of Hematology
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529. PROPROTEIN CONVERTASE 6 PLAYS A CRITICAL ROLE IN MODULATING THE HUMAN ENDOMETRIAL EPITHELIUM FOR RECEPTIVITY AND IMPLANTATION

2009

Successful embryo implantation is an important step in establishing pregnancy, requiring a healthy embryo and a receptive endometrium. Establishment of endometrial receptivity involves morphological and physiological changes initially in the endometrial epithelium, but the underlying molecular mechanisms are not fully understood. We have previously demonstrated that proprotein convertase 5/6 (PC6), a member of the proprotein convertase (PC) family, is up-regulated in the endometrium specifically at implantation in association with epithelial differentiation, in the human and monkey. PCs convert a range of precursor proteins of important functions into their bioactive forms; they are thus r…

medicine.medical_specialtyGene knockdownReproductive immunologyEmbryo cultureEmbryoReproductive technologyTransfectionBiologyEndometriumProprotein convertaseAndrologyEndocrinologymedicine.anatomical_structureEndocrinologyReproductive MedicineInternal medicineGeneticsmedicineAnimal Science and ZoologyMolecular BiologyDevelopmental BiologyBiotechnologyReproduction, Fertility and Development
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Downregulation of alpha-galactosidase A upregulates CD77: functional impact for Fabry nephropathy.

2009

Anderson-Fabry disease, an inherited deficiency in the lysosomal enzyme alpha-galactosidase A, is characterized by the progressive accumulation of globotriaosylceramide (Gb3), also known as CD77. We sought to clarify the pathogenesis of Fabry disease by establishing a cell model of this disorder. The expression of alpha-galactosidase A was transiently silenced by RNA interference in HK2 and primary human renal epithelial cells and stably silenced in HK2 cells by retroviral transfection with small hairpin RNA. All of the silenced cells had histological similarities to cells of patients with Fabry disease. The cells had reduced viability, significant accumulation of intracellular Gb3, and a m…

medicine.medical_specialtyGlobotriaosylceramideGb3Cell LineSmall hairpin RNAchemistry.chemical_compoundRNA interferenceDownregulation and upregulationInternal medicineMedicineGene silencingHumansGene SilencingRNA Small InterferingAnderson–Fabry diseaseGlobosidesbusiness.industryTrihexosylceramidesEpithelial CellsTransfectionEnzyme replacement therapymedicine.diseaseFabry diseaseα-galactosidaseEndocrinologychemistryGene Expression RegulationNephrologyCell culturealpha-GalactosidaseCancer researchFabry DiseaseCD77businessenzyme replacement therapyKidney international
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The role of insulin-like growth factor II in the malignant transformation of rat liver oval cells

1997

Oval cells are small nonparenchymal epithelial cells that first appear in the periportal areas of the liver and thereafter invade the whole parenchyma when mice or rats are exposed to a variety of chemical carcinogens. In the present study we have analyzed the expression of insulin-like growth factor II (IGF II) in the recently established oval cell line OC/CDE 22 and its malignantly transformed counterpart (the M22 cells) and the biological consequences of the constitutive expression of IGF II in oval cells. OC/CDE 22 cells do not express the above-mentioned growth factor, whereas the M22 cells do and addition of a neutralizing anti-IGF II antibody to M22 cells resulted in an almost comple…

medicine.medical_specialtyLiver cytologymedicine.medical_treatmentBiologyCell LineMalignant transformationMiceLiver Neoplasms ExperimentalGrowth factor receptorInsulin-Like Growth Factor IINeutralization TestsInternal medicinemedicineAnimalsAutocrine signallingHepatologyGrowth factorEpithelial CellsOncogenesTransfectionMolecular biologyRatsCell Transformation NeoplasticEndocrinologyLiverCell cultureInsulin-like growth factor 2biology.proteinMitogensHepatology
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Nonsteroidal Anti-Inflammatory Drugs and Ectodomain Shedding of the Amyloid Precursor Protein

2008

<i>Background:</i> Epidemiological studies have suggested that long-term use of nonsteroidal anti-inflammatory drugs (NSAIDs) is associated with a reduced incidence of Alzheimer’s disease (AD). Several mechanisms have been proposed to explain these findings including increased shedding of the soluble ectodomain of the amyloid precursor protein (sAPP), which functions as a neurotrophic and neuroprotective factor in vitroand in vivo. <i>Objective:</i> To clarify whether NSAIDs consistently stimulate sAPP secretion. <i>Methods:</i> 293-EBNA cells with stable overexpression of an APP-alkaline phosphatase fusion protein (APP-AP), SH-SY5Y and PC12 cells or prim…

medicine.medical_specialtyMedizinische Fakultät -ohne weitere Spezifikation-IndomethacinIbuprofenStimulationCHO Cells-PC12 CellsNeuroprotectionCell LineAmyloid beta-Protein PrecursorNeuroblastomaCricetulusWestern blotDownregulation and upregulationCell Line TumorCricetinaeInternal medicinemedicineAmyloid precursor proteinAnimalsddc:610medicine.diagnostic_testbiologyChemistryAnti-Inflammatory Agents Non-SteroidalTransfectionAlkaline PhosphataseRatsKineticsEndocrinologyNeurologyEctodomainCell culturebiology.proteinTetradecanoylphorbol AcetateNeurology (clinical)
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Embryonic adhesion is not affected by endometrial leptin receptor gene silencing.

2006

Objective In rodents, evidence suggests that the leptin system is mandatory for embryonic implantation. We aimed to investigate the functional relevance of the endometrial leptin receptor (OB-R) in the adhesion phase of human implantation. Design We used an in vitro model for embryonic adhesion, composed of a human endometrial cell line (HEC1-A) and B6C3F1 mouse embryos. The OB-R gene was silenced in a stable manner by RNA interference, and embryonic adhesion rates were analyzed. Setting Research laboratory at a university-affiliated center. Intervention(s) RNA interference. Main Outcome Measure(s) Embryonic adhesion in cells treated with OB-R RNAi. Result(s) The OB-R shRNA-transfected cell…

medicine.medical_specialtyReceptors Cell SurfaceBiologyCell LineSmall hairpin RNAEndometriumMiceRNA interferencePregnancyInternal medicinemedicineCell AdhesionGene silencingAnimalsHumansBlastocystEmbryo ImplantationGene SilencingCells CulturedLeptin receptorObstetrics and GynecologyTransfectionEmbryo MammalianEmbryonic stem cellCell biologymedicine.anatomical_structureEndocrinologyReproductive MedicineReceptors LeptinNeural cell adhesion moleculeFemaleFertility and sterility
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Aryl hydrocarbon receptor-dependent cell cycle arrest in isolated mouse oval cells

2013

The aryl hydrocarbon receptor (AhR) is a ligand-activated transcription factor, which mediates toxic responses to environmental pollutants, such as 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and related compounds. Besides its well known role in induction of xenobiotic metabolizing enzymes, for instance CYP1A1, the AhR is also involved in tumor promotion in rodents although the underlying mechanisms are still poorly understood. Additionally, the AhR is known to regulate cellular proliferation, which might result in either inhibition or stimulation of proliferation depending on the cell-type studied. Potential targets in hepatocarcinogenesis are liver oval (stem/progenitor) cells. In the pres…

medicine.medical_specialtyTCDDPolychlorinated DibenzodioxinsCell cycle checkpointBlotting WesternCyclin AMice TransgenicCyclin ATransfectionToxicologyRetinoblastoma ProteinCell LineMiceCyclin D1Proliferating Cell Nuclear AntigenInternal medicinemedicineAnimalsCyclin D1RNA Small InterferingTranscription factorCell Proliferationbiologyaryl hydrocarbon receptorRetinoblastoma proteinmouse oval cellsCell Cycle CheckpointsGeneral MedicineCell cycleAryl hydrocarbon receptorCell biologyEndocrinologyLiverReceptors Aryl Hydrocarbonbiology.proteinEnvironmental PollutantsTumor promotioncell cycle
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