Search results for "Transforming growth factor"

showing 10 items of 294 documents

Genotyping strategy of SMAD-3 rs3825977 gene variant for a differential management of ascending aorta aneurysm in women people: Gender oriented diagn…

2020

Abstract Background and objectives The research of opportune strategies for facilitating the management of complex pathologies, such as ascending aorta aneurysm (AAA), currently represents the principal object of clinicians, clinical pathologists included. Herein, we propose genotyping of gene variants related to TGF-β pathway as useful strategy to improve the complex AAA management, exclusively based on imaging evaluations. Precisely, we investigated four functional SNPs in SMAD and VEGF genes, encoding molecules able to modulate functions and cross-talks of TGF-β pathway. Populations and methods Our study included 92 individuals (70 men (76%) and 22 (24%) women; mean age: 71.4 ± 2.6 years…

0301 basic medicinetransforming growth factorSNPs of SMAD and VEGF genesSMADBioinformaticsAscending aorta aneurysm (AAA)03 medical and health sciences0302 clinical medicineImmune systemmedicine.arteryGenotypeGeneticsmedicineGenotypingGeneGenetics (clinical)aorta aneurysmMetalloproteinaseAortaFemale peoplebusiness.industryGenetic variantsaorta aneurysm; transforming growth factorSettore MED/23030104 developmental biology030220 oncology & carcinogenesisGender medicinebusinessGenotyping strategy
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Spray dried hyaluronic acid microparticles for adhesion controlled aggregation and potential stimulation of stem cells

2017

Spray-dried microparticles of a derivative of hyaluronic acid (HA) have been engineered to obtain a controlled aggregation with Human Mesenchymal Stem Cells (hMSCs) into 3D constructs. We demonstrated the utility of chemical functionalization of a native constituent of the extracellular matrix to improve processing performances and to control on stem cell adhesion and differentiation. Native hyaluronic acid (HA), cell adhesive peptides (RGD), transforming growth factor β3, dexamethasone are biological agents potentially suitable for chondrogenic stimulation of hMSCS. However unmodified HA suffers of drawbacks in terms of stability and versatility of processing. Functionalization strategies…

3003Hyaluronic acid0206 medical engineeringPharmaceutical Science02 engineering and technologyDexamethasoneExtracellular matrixchemistry.chemical_compoundTissue engineeringTransforming Growth Factor betaHyaluronic acidCell AdhesionHumansCell adhesionCells CulturedBottom-up approachStem cellMesenchymal Stromal CellTissue EngineeringChemistryMesenchymal stem cellMesenchymal Stem CellsCell DifferentiationAdhesion021001 nanoscience & nanotechnology020601 biomedical engineeringExtracellular MatrixBiochemistryMicroparticleSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoSurface modificationChondrogenesiStem cell0210 nano-technologyChondrogenesisHuman
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Controversies on the role of Th17 in cancer: a TGF-β-dependent immunosuppressive activity?

2012

The immune system has important roles in limiting the spread of cancer and shaping the tumor microenvironment. Although the contributions of T helper 17 (Th17) cells (a subtype of CD4(+) T lymphocytes) to autoimmunity and allergy response are well known, their roles in cancer remain ambiguous. Despite adoptive transfer studies indicating that mouse Th17 cells support anticancer immunity, the Th17 cells that naturally infiltrate experimental tumors appear to have a tumor-promoting effect. These contradictory properties can be related to the high degree of plasticity inherent in Th17 cells and their capacity to differentiate into tumoricidal Th1-like cells. Mouse Th17 cells induced by transfo…

Adoptive cell transferAngiogenesisAntigen-Presenting Cellschemical and pharmacologic phenomenaBiologymedicine.disease_causeAutoimmunityMice03 medical and health sciences0302 clinical medicineImmune systemAntigenAntigens CDTransforming Growth Factor betaImmunityNeoplasmsImmune TolerancemedicineAnimals5'-NucleotidaseMolecular Biology030304 developmental biologyImmunity Cellular0303 health sciencesTumor microenvironmentNeovascularization PathologicApyraseModels ImmunologicalCell DifferentiationTh1 Cells3. Good health030220 oncology & carcinogenesisImmunologyCancer researchTh17 CellsMolecular MedicineTransforming growth factorTrends in Molecular Medicine
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Stat3 and Gfi-1 Transcription Factors Control Th17 Cell Immunosuppressive Activity via the Regulation of Ectonucleotidase Expression

2012

International audience; Although Th17 cells are known to promote tissue inflammation and autoimmunity, their role during cancer progression remains elusive. Here, we showed that in vitro Th17 cells generated with the cytokines IL-6 and TGF-β expressed CD39 and CD73 ectonucleotidases, leading to adenosine release and the subsequent suppression of CD4(+) and CD8(+) T cell effector functions. The IL-6-mediated activation of the transcription factor Stat3 and the TGF-β-driven downregulation of Gfi-1 transcription factor were both essential for the expression of ectonucleotidases during Th17 cell differentiation. Stat3 supported whereas Gfi-1 repressed CD39 and CD73 expression by binding to thei…

Adoptive cell transferMESH : Transcription FactorsCellular differentiationMESH: Th17 CellsT-LymphocytesCellMESH : Promoter Regions GeneticMESH : RNA Small InterferingMESH: Mice KnockoutMice0302 clinical medicineTransforming Growth Factor betaMESH: RNA Small InterferingMESH : STAT3 Transcription FactorImmunology and Allergy[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyEctonucleotidaseMESH: AnimalsRNA Small InterferingSTAT3MESH: Lymphocytes Tumor-InfiltratingPromoter Regions GeneticMESH: Antigens CD5'-NucleotidaseRegulation of gene expressionMice Knockout0303 health sciencesMESH : Gene Expression RegulationApyraseMESH: STAT3 Transcription FactorMESH: Transcription FactorsMESH: Gene Expression RegulationMESH : Mice TransgenicCell biologyMESH : Lymphocytes Tumor-InfiltratingDNA-Binding ProteinsMESH : ApyraseInfectious Diseasesmedicine.anatomical_structure[SDV.IMM]Life Sciences [q-bio]/ImmunologyMESH : DNA-Binding ProteinsMESH: ApyraseSTAT3 Transcription Factor[SDV.IMM] Life Sciences [q-bio]/ImmunologyMESH : Interleukin-6MESH: Mice TransgenicT cellImmunologyMice TransgenicMESH : Mice Inbred C57BLBiology03 medical and health sciencesLymphocytes Tumor-InfiltratingMESH: Mice Inbred C57BLAntigens CDMESH: Promoter Regions GeneticMESH : 5'-NucleotidaseMESH : MicemedicineMESH : Antigens CDMESH : Th17 CellsAnimalsTranscription factorMESH: MiceMESH: Transforming Growth Factor beta030304 developmental biologyMESH : T-LymphocytesBinding SitesInterleukin-6MESH: Interleukin-6Mice Inbred C57BLMESH: T-LymphocytesMESH : Transforming Growth Factor betaMESH: Binding SitesGene Expression Regulationbiology.proteinMESH : Mice KnockoutTh17 CellsMESH : AnimalsMESH: 5'-NucleotidaseMESH: DNA-Binding ProteinsMESH : Binding Sites030215 immunologyTranscription FactorsImmunity
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Coexpression of TGF-β1 and IL-10 Enables Regulatory T Cells to Completely Suppress Airway Hyperreactivity

2008

Abstract In allergic airway disease, Treg may play an important role in the modulation of airway hyperreactivity (AHR) and inflammation. We therefore investigated the therapeutic potential of Treg in an Ag-dependent murine asthma model. We here describe that AHR can be completely suppressed by adoptive transfer of Treg overexpressing active TGF-β1. Using mice with impaired TGF-β signaling in T cells, we could demonstrate that TGF-β signaling in recipient effector T cells or transferred Treg themselves is not required for the protective effects on AHR. However, the expression of IL-10 by Treg was found to be essential for the suppression of AHR, since Treg overexpressing active TGF-β1 but de…

Adoptive cell transferTransgeneImmunologyGene ExpressionMice Transgenicchemical and pharmacologic phenomenaInflammationT-Lymphocytes RegulatoryTransforming Growth Factor beta1MiceRespiratory HypersensitivitymedicineAnimalsImmunology and AllergyAsthmaInflammationbusiness.industryEffectorhemic and immune systemsrespiratory systemmedicine.diseaseAdoptive TransferAirway hyperreactivityInterleukin-10respiratory tract diseasesDisease Models AnimalInterleukin 10Immunologymedicine.symptombusinessSignal TransductionTransforming growth factorThe Journal of Immunology
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IL-21 regulates experimental colitis by modulating the balance between Treg and Th17 cells

2007

Regulatory T (T(reg)) cells play a key role in the maintenance of the immune system homeostasis. T(reg) cells can be generated in the periphery under control of TGF-beta, a cytokine involved in the negative control of the immune system. However, TGF-beta cooperates with IL-6 in the generation of Th17 cells, a novel class of effector cells involved in numerous inflammatory diseases, including colitis. Therefore, TGF-beta emerges as a mediator of both anti-inflammatory and pro-inflammatory processes, depending on the local cytokine milieu. Here we demonstrate that IL-21, a type-1 cytokine produced by T cells and involved in the pathogenesis of immune-mediated diseases, prevents the TGF-beta-d…

Adoptive cell transfermedicine.medical_treatmentImmunologyCellGene Expressionchemical and pharmacologic phenomenaMice SCIDBiologyT-Lymphocytes RegulatoryMiceImmune systemT-Lymphocyte SubsetsTransforming Growth Factor betaFoxP3IL-21medicineAnimalsImmunology and AllergyRNA MessengerIL-2 receptorTranscription factorSettore MED/12 - GastroenterologiaMice Inbred BALB CReverse Transcriptase Polymerase Chain ReactionInterleukinsInterleukin-17FOXP3Forkhead Transcription Factorshemic and immune systemsColitisFlow CytometryAdoptive TransferCell biologyColitis; FoxP3; IL-21; Treg cells; TGF-βTGF-βDisease Models Animalmedicine.anatomical_structureCytokineImmunologyTreg cellsHomeostasisEuropean Journal of Immunology
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Epithelial contribution to the profibrotic stiff microenvironment and myofibroblast population in lung fibrosis

2017

The contribution of epithelial-to-mesenchymal transition (EMT) to the profibrotic stiff microenvironment and myofibroblast accumulation in pulmonary fibrosis remains unclear. We examined EMT-competent lung epithelial cells and lung fibroblasts from control (fibrosisfree) donors or patients with idiopathic pulmonary fibrosis (IPF), which is a very aggressive fibrotic disorder. Cells were cultured on profibrotic conditions including stiff substrata and TGF-β1, and analyzed in terms of morphology, stiffness, and expression of EMT/myofibroblast markers and fibrillar collagens. All fibroblasts acquired a robust myofibroblast phenotype on TGF-β1 stimulation. Yet IPF myofibroblasts exhibited highe…

Adult0301 basic medicineEpithelial-Mesenchymal TransitionPulmonary FibrosisPopulationmacromolecular substancesEpithelial cellsBiologyEpitheliumPulmonary fibrosisTransforming Growth Factor beta103 medical and health sciencesIdiopathic pulmonary fibrosisMechanobiology0302 clinical medicinePulmonary fibrosismedicineHumansMyofibroblastsFibroblasteducationLungMolecular BiologyCells Culturededucation.field_of_studyCèl·lules epitelialsLungEpithelial CellsFibrosi pulmonarArticlesCell BiologyFibroblastsmusculoskeletal systemmedicine.diseasePhenotype030104 developmental biologymedicine.anatomical_structureCellular MicroenvironmentCell Biology of DiseaseCase-Control Studies030220 oncology & carcinogenesisembryonic structurescardiovascular systemCancer researchMyofibroblastcirculatory and respiratory physiology
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Comparative immunoexpression of ICAM-1, TGF-?1 and ki-67 in periapical and residual cysts

2016

Background This study compared the immunohistochemical expression of ki-67, transforming growth factor beta 1 (TGF-β1) and intercellular adhesion molecule-1 (ICAM-1) in inflammatory periapical cysts and residual cysts. Material and Methods The study sample was composed by 25 periapical cysts and 25 residual cysts and immunohistochemical reactions were carried out using antibodies directed against ICAM-1, TGF-β1 and ki-67. Clinical, radiological, gross, histological and immunohistochemical data were tabulated for descriptive and comparative analysis using the SPSS software and differences were considered statistically significant when p<0.05%. Results There were no differences between the ex…

Adult0301 basic medicinePathologymedicine.medical_specialtyAdolescentLabeling indexTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineparasitic diseasesHumansMedicineChildGeneral DentistryAgedRadicular CystICAM-1Oral Medicine and Pathologybiologybusiness.industryResearch030206 dentistryTransforming growth factor betaMiddle AgedIntercellular Adhesion Molecule-1:CIENCIAS MÉDICAS [UNESCO]Ki-67 Antigen030104 developmental biologyOtorhinolaryngologyKi-67UNESCO::CIENCIAS MÉDICASbiology.proteinImmunohistochemistrySurgerybusinessTransforming growth factor
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Histone deacetylase inhibition by suberoylanilide hydroxamic acid: a therapeutic approach to treat human uterine leiomyoma.

2022

Objective To evaluate the effect of inhibition of histone deacetylases (HDACs) by suberoylanilide hydroxamic acid (SAHA) treatment of human uterine leiomyoma primary (HULP) cells in vitro on cell proliferation, cell cycle, extracellular matrix (ECM) formation, and transforming growth factor β3 (TGF-β3) signaling. Design Prospective study comparing uterine leiomyoma (UL) vs. adjacent myometrium (MM) tissue and cells with or without SAHA treatment. Setting Hospital and university laboratories. Patient(s) Women with UL without any hormone treatment. Intervention(s) Myomectomy or hysterectomy surgery in women for leiomyoma disease. Main Outcome Measure(s) HDAC activity was assessed by enzyme-li…

AdultAntineoplastic AgentsHistone Deacetylase 1MMP9Histone Deacetylase 6Histone DeacetylasesCyclin D1Transforming Growth Factor beta3Cell proliferation SAHA ULS-ß3 pathway extracellular matrix uterine leiomyomaTumor Cells CulturedHumansViability assayProspective StudiesCell ProliferationVorinostatbiologyLeiomyomaChemistryCell growthCell CycleObstetrics and GynecologyCell cycleMiddle AgedHDAC3Molecular biologyProliferating cell nuclear antigenExtracellular MatrixGene Expression Regulation NeoplasticHistone Deacetylase InhibitorsReproductive MedicineUterine Neoplasmsbiology.proteinFemaleHistone deacetylaseSignal TransductionFertility and sterility
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Differences in platelet growth factor release and leucocyte kinetics during autologous platelet gel formation.

2006

Three commercial systems for whole blood separation were compared to obtain the buffy coat composed of platelet-rich plasma (BC-PRP) and leucocytes . These samples of the buffy coat were used to make a platelet gel (PG), which was used to measure platelet growth factor (PGF) release, to perform a white blood cell (WBC) count and to measure myeloperoxidase (MPO) release from WBCs. Aliquots of whole blood obtained from ten volunteers were distributed either to a blood cell separator (The Electa Cell-Separator (TM), E-CS) or to a tabletop centrifuge (Gravitational Platelet Sequestration System (TM), GPS) to prepare the BC-PRP. The third system combines the BC-PRP production by E-CS with a micr…

AdultBlood PlateletsBuffy coatFibrin Tissue AdhesiveBlood cellTransforming Growth Factor beta1Blood Transfusion AutologousLeukocyte CountThrombinTransforming Growth Factor betaWhite blood cellmedicineLeukocytesHumansPlateletPlatelet activationWhole bloodPeroxidasePlatelet-Derived Growth FactorWound HealingChromatographybiologyChemistryHematologyPlatelet Activationmedicine.anatomical_structureBiochemistryMyeloperoxidasebiology.proteinBlood Component RemovalGelsmedicine.drugTransfusion medicine (Oxford, England)
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