Search results for "Transgenic"

showing 10 items of 552 documents

Virus replication and virion export in X-deficient hepatitis B virus transgenic mice

2002

The function of the X protein (pX) in the replication cycle of mammalian hepadnaviruses is enigmatic. Using tissue culture experiments it has been shown that the X gene product is not central to hepatitis B virus (HBV) replication and virion export. However, at present it is still unclear whether this also applies to the in vivo situation. Using a terminally redundant X-deficient HBV DNA construct, transgenic mice were established that exhibited high-level expression of the viral core protein in liver and kidneys. Importantly, replicative DNA intermediates and mature viral genomes could be detected in the liver and serum of these mice, respectively. These findings indicate that, in the in v…

Hepatitis B virusHepatitis B virus DNA polymerasevirusesTransgeneMice TransgenicBiologyVirus Replicationmedicine.disease_causeHepatitis B virus PRE betaGene productMicechemistry.chemical_compoundVirologymedicineAnimalsViral Regulatory and Accessory ProteinsHepatitis B virusVirionVirologyMolecular biologydigestive system diseasesMice Inbred C57BLHBxViral replicationchemistryMice Inbred DBATrans-ActivatorsDNAJournal of General Virology
researchProduct

Optimized recombinant dense bodies of human cytomegalovirus efficiently prime virus specific lymphocytes and neutralizing antibodies without the addi…

2010

Control of human cytomegalovirus (HCMV) infection correlates with the reconstitution of antiviral T lymphocytes in haematopoietic stem cell transplant recipients. A vaccine to foster this reconstitution and to ameliorate the severe consequences of HCMV reactivation is yet unavailable. This work focused on providing a rationale for the amendment of the yields and the antigenic composition of a vaccine, based on subviral dense bodies (DB) of HCMV. Modified DB were generated that contained the HLA-A2 presented IE1 model peptide TMYGGISLL, integrated at different positions in the major DB protein pp65. Insertion at position W175 of pp65 allowed efficient formation of recDB in the cytoplasm of i…

Human cytomegalovirusCD4-Positive T-Lymphocytesvirusesmedicine.medical_treatmentCongenital cytomegalovirus infectionCytomegalovirusMice TransgenicBiologyCD8-Positive T-LymphocytesAntibodies ViralVirusCell LineViral Matrix ProteinsCytomegalovirus VaccinesMiceAntigenmedicineCytotoxic T cellAnimalsHumansNeutralizing antibodyAntigens ViralMice Inbred BALB CGeneral VeterinaryGeneral Immunology and MicrobiologyPublic Health Environmental and Occupational Healthvirus diseasesmedicine.diseasePhosphoproteinsVirologyAntibodies NeutralizingMutagenesis InsertionalInfectious DiseasesCytomegalovirus InfectionsDNA Viralbiology.proteinMolecular MedicineAdjuvantCD8Vaccine
researchProduct

Development of novel vaccine strategies against human cytomegalovirus infection based on subviral particles.

2002

Abstract Background: Pre- and perinatal human cytomegalovirus (HCMV) infection remains one of the major causes of mental defects and sensineural hearing loss in children. In addition, it is a prominent infectious complication in immunosuppressed individuals such as AIDS patients or transplant recipients. Therefore, the development of an HCMV vaccine has been given top priority by health care institutions. Study design: Defective subviral particles of HCMV, termed Dense Bodies (DB) contain the dominant target antigens for humoral and cellular immune responses elicited during natural infection. These enveloped particles are released from infected culture cells and can be purified by gradient …

Human cytomegalovirusCytotoxicity ImmunologicImmunogenCytomegalovirusMice TransgenicBiologyAntibodies ViralVirusCell LineCytomegalovirus VaccinesMiceImmune systemAntigenNeutralization TestsVirologymedicineCytotoxic T cellAnimalsHumansMice Inbred BALB CVirionmedicine.diseaseVirologyCTL*Infectious DiseasesImmunizationVaccines InactivatedImmunologyCytomegalovirus InfectionsT-Lymphocytes CytotoxicJournal of clinical virology : the official publication of the Pan American Society for Clinical Virology
researchProduct

Shedding light on the elusive role of endothelial cells in cytomegalovirus dissemination.

2011

Cytomegalovirus (CMV) is frequently transmitted by solid organ transplantation and is associated with graft failure. By forming the boundary between circulation and organ parenchyma, endothelial cells (EC) are suited for bidirectional virus spread from and to the transplant. We applied Cre/loxP-mediated green-fluorescence-tagging of EC-derived murine CMV (MCMV) to quantify the role of infected EC in transplantation-associated CMV dissemination in the mouse model. Both EC- and non-EC-derived virus originating from infected Tie2-cre + heart and kidney transplants were readily transmitted to MCMV-naïve recipients by primary viremia. In contrast, when a Tie2-cre + transplant was infected by pri…

Human cytomegalovirusMuromegalovirusmedicine.medical_treatmentKidneyMicelcsh:QH301-705.5Kidney transplantationHeart transplantationbiologyvirus diseasesHeartAnimal ModelsHost-Pathogen InteractionInfectious Diseasessurgical procedures operativemedicine.anatomical_structureOncologyMedical MicrobiologyCytomegalovirus InfectionsMedicineResearch Articlelcsh:Immunologic diseases. AllergyEndotheliumImmunologyCongenital cytomegalovirus infection610ViremiaMice TransgenicMicrobiologyVirusModel OrganismsMuromegalovirusVirologyGeneticsmedicineAnimalsViremiaBiologyMolecular BiologyEndothelial Cellsmedicine.diseasebiology.organism_classificationVirologyKidney Transplantationlcsh:Biology (General)ImmunologyHeart TransplantationSurgeryParasitologyEndothelium Vascularlcsh:RC581-607PLoS pathogens
researchProduct

Evaluating Human T-Cell Therapy of Cytomegalovirus Organ Disease in HLA-Transgenic Mice

2015

Reactivation of human cytomegalovirus (HCMV) can cause severe disease in recipients of hematopoietic stem cell transplantation. Although preclinical research in murine models as well as clinical trials have provided 'proof of concept' for infection control by pre-emptive CD8 T-cell immunotherapy, there exists no predictive model to experimentally evaluate parameters that determine antiviral efficacy of human T cells in terms of virus control in functional organs, prevention of organ disease, and host survival benefit. We here introduce a novel mouse model for testing HCMV epitope-specific human T cells. The HCMV UL83/pp65-derived NLV-peptide was presented by transgenic HLA-A2.1 in the conte…

Human cytomegaloviruslcsh:Immunologic diseases. Allergymedicine.medical_treatmentT cellImmunologyCell- and Tissue-Based TherapyCytomegalovirusEpitopes T-LymphocyteMice TransgenicHematopoietic stem cell transplantationHuman leukocyte antigenMice SCIDBiologyMicrobiologyViral Matrix ProteinsMice Inbred NODVirologyHLA-A2 AntigenGeneticsmedicineCytotoxic T cellAnimalsHumansMolecular Biologylcsh:QH301-705.5ImmunotherapyViral Loadmedicine.diseaseMice Inbred C57BLDisease Models Animalmedicine.anatomical_structurelcsh:Biology (General)ImmunologyCytomegalovirus InfectionsParasitologylcsh:RC581-607Viral loadCD8Research ArticlePLoS Pathogens
researchProduct

Ubiquitin-independent function of optineurin in autophagic clearance of protein aggregates.

2013

Summary Aggregation of misfolded proteins and the associated loss of neurons are considered a hallmark of numerous neurodegenerative diseases. Optineurin is present in protein inclusions observed in various neurodegenerative diseases including amyotrophic lateral sclerosis (ALS), Huntington's disease, Alzheimer's disease, Parkinson's disease, Creutzfeld-Jacob disease and Pick's disease. Optineurin deletion mutations have also been described in ALS patients. However, the role of optineurin in mechanisms of protein aggregation remains unclear. In this report, we demonstrate that optineurin recognizes various protein aggregates via its C-terminal coiled-coil domain in a ubiquitin-independent m…

HuntingtinSOD1AggrephagyCell Cycle ProteinsMice TransgenicProtein aggregationBiologyArticle03 medical and health sciencesMice0302 clinical medicineTANK-binding kinase 1UbiquitinTranscription Factor TFIIIAAutophagyAnimalsHumansPhosphorylationZebrafishZebrafish030304 developmental biologyOptineurin0303 health sciencesUbiquitinamyotrophic lateral sclerosis; Huntington disease; Huntingtin; optineurin; phosphorylation; SOD1; TBK1; ubiquitinMembrane Transport ProteinsNeurodegenerative DiseasesCell Biologybiology.organism_classification3. Good healthMice Inbred C57BLDisease Models AnimalCancer researchbiology.protein030217 neurology & neurosurgeryHeLa CellsProtein BindingJournal of cell science
researchProduct

Expression of genes encoding the calcium signalosome in cellular and transgenic models of Huntington's disease

2013

Huntington's disease (HD) is a hereditary neurodegenerative disease caused by the expansion of a polyglutamine stretch in the huntingtin (HTT) protein and characterized by dysregulated calcium homeostasis. We investigated whether these disturbances are correlated with changes in the mRNA level of the genes that encode proteins involved in calcium homeostasis and signaling (i.e., the calciosome). Using custom-made TaqMan low-density arrays containing probes for 96 genes, we quantified mRNA in the striatum in YAC128 mice, a model of HD, and wildtype mice. HTT mutation caused the increased expression of some components of the calcium signalosome, including calretinin, presenilin 2, and calmyri…

Huntingtinhuntingtincalcium signalosomechemistry.chemical_elementtransgenic miceCalciumlcsh:RC321-571Cellular and Molecular Neurosciencehuntingtin-associated protein 1mental disordersGene expressionOriginal Research Articlelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular BiologyCalcium metabolismTaqMan low-density arraysbiologyHuntingtin-associated protein 1Calcium channelTaqMan Low Density Arraysstore-operated calcium entrycalcyclin-binding proteinHuntington's diseaseMolecular biologyStore-operated calcium entrynervous systemchemistrybiology.proteinCalretininHuntington’s diseaseNeuroscienceFrontiers in Molecular Neuroscience
researchProduct

Increased Goodpasture antigen-binding protein expression induces type IV collagen disorganization and deposit of immunoglobulin A in glomerular basem…

2007

Increased expression of Goodpasture antigen-binding protein (GPBP), a protein that binds and phosphorylates basement membrane collagen, has been associated with immune complex-mediated pathogenesis. However, recent reports have questioned this biological function and proposed that GPBP serves as a cytosolic ceramide transporter (CERTL). Thus, the role of GPBP in vivo remains unknown. New Zealand White (NZW) mice are considered healthy animals although they convey a genetic predisposition for immune complex-mediated glomerulonephritis. Here we show that NZW mice developed age-dependent lupus-prone autoimmune response and immune complex-mediated glomerulonephritis characterized by elevated GP…

Immunoglobulin ACollagen Type IVAgingMice Inbred StrainsMice TransgenicAntigen-Antibody ComplexProtein Serine-Threonine Kinasesurologic and male genital diseasesPathology and Forensic MedicinePathogenesisType IV collagenMiceGlomerulonephritisSpecies SpecificityGlomerular Basement MembranemedicineGoodpasture syndromeAnimalsHumansLupus Erythematosus SystemicAutoantibodiesAutoimmune diseaseBasement membranebiologyGlomerular basement membraneGlomerulonephritismedicine.diseaseImmunoglobulin Amedicine.anatomical_structureImmunologyCancer researchbiology.proteinRegular Articles
researchProduct

Interferon-λ and interleukin 22 act synergistically for the induction of interferon-stimulated genes and control of rotavirus infection.

2015

The epithelium is the main entry point for many viruses, but the processes that protect barrier surfaces against viral infections are incompletely understood. Here we identified interleukin 22 (IL-22) produced by innate lymphoid cell group 3 (ILC3) as an amplifier of signaling via interferon-λ (IFN-λ), a synergism needed to curtail the replication of rotavirus, the leading cause of childhood gastroenteritis. Cooperation between the receptor for IL-22 and the receptor for IFN-λ, both of which were 'preferentially' expressed by intestinal epithelial cells (IECs), was required for optimal activation of the transcription factor STAT1 and expression of interferon-stimulated genes (ISGs). These d…

ImmunologyImmunoblottingMolecular Sequence DataGene ExpressionMice Transgenicmedicine.disease_causeRotavirus InfectionsCell LineMadin Darby Canine Kidney CellsInterleukin 22DogsInterferonRotavirusChlorocebus aethiopsmedicineImmunology and AllergyAnimalsHumansSTAT1Intestinal MucosaReceptors CytokineVero CellsMice KnockoutbiologyReverse Transcriptase Polymerase Chain ReactionInterleukinsInnate lymphoid cellInterleukinDrug SynergismEpithelial CellsVirology3. Good healthIntestinesMice Inbred C57BLSTAT1 Transcription FactorViral replicationImmunologybiology.proteinVero cellCytokinesCaco-2 CellsHT29 Cellsmedicine.drugNature immunology
researchProduct

A central role for Notch in effector CD8(+) T cell differentiation.

2014

Activated CD8(+) T cells choose between terminal effector cell (TEC) or memory precursor cell (MPC) fates. We found that the signaling receptor Notch controls this 'choice'. Notch promoted the differentiation of immediately protective TECs and was correspondingly required for the clearance of acute infection with influenza virus. Notch activated a major portion of the TEC-specific gene-expression program and suppressed the MPC-specific program. Expression of Notch was induced on naive CD8(+) T cells by inflammatory mediators and interleukin 2 (IL-2) via pathways dependent on the metabolic checkpoint kinase mTOR and the transcription factor T-bet. These pathways were subsequently amplified d…

ImmunologyNotch signaling pathwayMice TransgenicCell SeparationBiologyAdaptive ImmunityCD8-Positive T-LymphocytesEffector cellLymphocyte ActivationReal-Time Polymerase Chain ReactionArticlememoryMiceOrthomyxoviridae InfectionsCell surface receptorT-Lymphocyte SubsetsTransduction GeneticPrecursor cellImmunology and AllergyCytotoxic T cellAnimalsGeneticsReceptors NotchEffectorCell DifferentiationFlow CytometryAdoptive TransferTEC3. Good healthCell biologyMice Inbred C57BLeffectorCD8 T cellMPCInfluenza A virusinflammationTranscriptomeCD8Nature immunology
researchProduct