Search results for "Transgenic"

showing 10 items of 552 documents

Distinct 5' SCL enhancers direct transcription to developing brain, spinal cord, and endothelium: neural expression is mediated by GATA factor bindin…

1999

The SCL gene encodes a basic helix-loop-helix transcription factor with a pivotal role in the development of endothelium and of all hematopoietic lineages. SCL is also expressed in the central nervous system, although its expression pattern has not been examined in detail and its function in neural development is unknown. In this article we present the first analysis of SCL transcriptional regulation in vivo. We have identified three spatially distinct regulatory modules, each of which was both necessary and sufficient to direct reporter gene expression in vivo to three different regions within the normal SCL expression domain, namely, developing endothelium, midbrain, and hindbrain/spinal …

animal structuresEmbryo NonmammalianTranscription GeneticHindbrainMice TransgenicChick EmbryoBiologybehavioral disciplines and activities03 medical and health sciencesMice0302 clinical medicineTranscription (biology)Genes Reporterhemic and lymphatic diseasesProto-Oncogene ProteinsBasic Helix-Loop-Helix Transcription FactorsAnimalsTissue DistributionEndotheliumEnhancerMolecular BiologyTranscription factorGeneIn Situ HybridizationT-Cell Acute Lymphocytic Leukemia Protein 1Zebrafish030304 developmental biologyRegulation of gene expressionGenetics0303 health sciencesReporter geneModels GeneticfungiBrainCell BiologyZebrafish ProteinsEmbryo MammalianCell biologyDNA-Binding ProteinsLac OperonSpinal CordNeural development030217 neurology & neurosurgeryDevelopmental BiologyTranscription FactorsDevelopmental biology
researchProduct

Abnormal peripubertal development of the rat mammary gland following exposure in utero and during lactation to a mixture of genistein and the food co…

2012

The impact of early exposure to endocrine disruptor mixtures on mammary gland development is poorly known. Here, we identify the effects of a conception to weaning exposure of rats to the phytoestrogen genistein (G) and/or the antiandrogen vinclozolin (V) at 1 mg/kg-d, alone or in association. Using several approaches, we found that G- and GV-exposed rats displayed significantly greater epithelial branching and proliferation, wider terminal end buds than controls at PND35, as well as ductal hyperplasia and periductal fibrosis. Focal branching defects were present in V-exposed rats. An increased ER and AR expression was observed in G- and CV- as compared to V-exposed rats at PND35. Surprisin…

anti-androgenbreast-cancer risk[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritionreceptorgrowthsprague-dawley ratsandrogendifferentiationendocrine disruptionmammary gland developmenttransgenic mice[SDV.AEN] Life Sciences [q-bio]/Food and Nutritionbisphenol-a alterstumorigenesisgestational and lactational exposurecellsphytoestrogenfemale mice[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
researchProduct

Smad7 controls resistance of colitogenic T cells to regulatory T cell-mediated suppression.

2008

Background & Aims Foxp3-expressing regulatory T cells (Tregs) play a key role in the maintenance of the gut immune homeostasis, and an intact transforming growth factor (TGF)-β signaling is required for their function. In inflammatory bowel disease (IBD), the TGF-β signaling is impaired because of high expression of the inhibitory molecule Smad7. Although no intrinsic defects in Tregs function have been shown in IBD, it is still unknown whether colitogenic T cells are susceptible to Treg-mediated suppression. In this study, we have investigated whether IBD mucosal CD4+ T cells are resistant to Tregs and whether Smad7 is involved in this process. Methods IBD lamina propria mononuclear cells …

antisense oligonucleotideCD4-Positive T-LymphocytesAdoptive cell transferT-Lymphocytesanimal cellCell CommunicationInbred C57BLT-Lymphocytes RegulatoryTransgenicMiceregulatory T lymphocyteCrohn DiseaseTransforming Growth Factor betamononuclear cellRAG1 proteinIntestinal MucosaenteritisCells CulturedMice KnockoutSettore MED/12 - GastroenterologiaCulturedintegumentary systemmedicine.diagnostic_testarticleGastroenterologyInterleukinhemic and immune systemsT helper cellColitisRegulatoryUp-Regulationmedicine.anatomical_structurepriority journalgamma interferonSignal TransductionRegulatory T cellColonCellsKnockoutanimal experimentinterleukin 6chemical and pharmacologic phenomenaMice TransgenicBiologyinterleukin 2Recombination-activating geneFlow cytometryProinflammatory cytokineSmad7 ProteinmedicineAnimalsHumanscontrolled studyhumanlamina propriamouseCell ProliferationHomeodomain ProteinsCD4+ T lymphocytenonhumanHepatologyAnimalflow cytometryhuman cellanimal cell culturetransgenic mouseMice Inbred C57BLDisease Models Animalantisense oligonucleotide; gamma interferon; interleukin 17; interleukin 2; interleukin 6; RAG1 protein; Smad7 protein; animal cell; animal cell culture; animal experiment; article; CD4+ T lymphocyte; cell proliferation; colitis; controlled study; enteritis; flow cytometry; human; human cell; knockout mouse; lamina propria; mononuclear cell; mouse; nonhuman; priority journal; regulatory T lymphocyte; transgenic mouse; Animals; CD4-Positive T-Lymphocytes; Cell Communication; Cell Proliferation; Cells Cultured; Colitis; Colon; Crohn Disease; Disease Models Animal; Homeodomain Proteins; Humans; Intestinal Mucosa; Mice; Mice Inbred C57BL; Mice Knockout; Mice Transgenic; Signal Transduction; Smad7 Protein; T-Lymphocytes Regulatory; Transforming Growth Factor beta; Up-RegulationDisease ModelsImmunologyinterleukin 17knockout mouseTransforming growth factorGastroenterology
researchProduct

The Role of the anti-amyloidogenic secretase ADAM10 in shedding the APP-like proteins.

2011

ADAM10 (A disintegrin and metalloproteinase 10) has been demonstrated as an enzyme with protective properties in Alzheimer's disease: in mouse models it not only lowered generation of toxic A-beta peptides and formation of senile plaques but also alleviated learning deficits and enhanced synaptic density. This is due to cleavage of the amyloid precursor protein (APP) within its A-beta stretch and to the release of the extracellular domain of APP with neuroprotective function. Aside from cleaving APP, ADAM10 has been linked to over 40 putative substrates at least in cell culture. These substrates are connected with important cellular functions such as cell migration, stress response and tran…

biologyChemistryADAM10Mice TransgenicCell biologyADAM ProteinsAmyloid beta-Protein PrecursorMiceNeurologyAlpha secretaseAlzheimer DiseaseAmyloid precursor proteinbiology.proteinDisintegrinExtracellularAnimalsHumansNeurology (clinical)Senile plaquesAmyloid Precursor Protein SecretasesAmyloid precursor protein secretaseAPLP2Current Alzheimer research
researchProduct

Living Forms (of Art). Edoardo Kac's Transgenic Art between Ethics and Aesthetics

2013

Nel corso del Novecento si è assistito ad un intenso sviluppo della fisica e della biologia, grazie all’ausilio di nuovi strumenti tecnologici che hanno ampliato l’orizzonte della ricerca. Le sperimentazioni su organismi geneticamente modificati (come la famosa clonazione della pecora Dolly) hanno spesso generato scalpore, turbando le coscienze. Tali interventi sono tollerati in quei settori come la medicina in cui possono portare benefici all’esistenza dell’uomo, mentre suscitano inquietudine e perplessità in campo artistico dove l’intervento sulle forme viventi, non trovando giustificazione sul principio di utilità, sembra sfociare nel futile edonismo. Tenendo presente il dibattito avviat…

biotech art aesthetics Edoardo Kac transgenic art history of ideasSettore M-FIL/04 - Esteticabiotech art estetica bioetica Edoardo Kac biotecnologie arte biotecnologica arte transgenica creatività storia delle idee estetiche
researchProduct

Creatività e arte transgenica

2012

Il brasiliano Edoardo Kac è stato tra i primi artisti biotecnologici ad applicare l’ingegneria genetica all’arte, dando avvio a quella che egli stesso nel 1998 ha definito Transgenic Art. Le sue creazioni di animali fluorescenti hanno turbato le coscienze e sono state spesso superficialmente reputate un’esibizione di mera spettacolarità al fine di emulare, non più solo a livello metaforico, il potere creativo di Dio. In realtà gli organismi fluorescenti che hanno conferito a Kac popolarità mediatica non s’iscrivono nella categoria della creatività volta a conferire nuovo prestigio all’artista-dio, secondo l’analogia imperante nel Rinascimento e nel Romanticismo, bensì in quell’estetica del …

biotech art estetica Edoardo Kac creatività storia delle idee arte transgenicaSettore M-FIL/04 - Estetica
researchProduct

Effects of the silencing of sorbitol dehydrogenase on sugar partitioning in vegetative sinks in apple

2011

carbohydrate – leaves – Malus – SDH – sorbitol – transgenics
researchProduct

Transgenic overexpression of corticotropin releasing hormone provides partial protection against neurodegeneration in an in vivo model of acute excit…

2008

Abstract Corticotropin releasing hormone (CRH) is the central modulator of the mammalian hypothalamic–pituitary–adrenal (HPA) axis. In addition, CRH affects other processes in the brain including learning, memory, and synaptic plasticity. Moreover, CRH has been shown to play a role in nerve cell survival under apoptotic conditions and to serve as an endogenous neuroprotectant in vitro . Employing mice overexpressing murine CRH in the CNS, we observed a differential response of CRH-overexpressing mice (CRH-COE hom -Nes) to acute excitotoxic stress induced by kainate compared with controls (CRH-COE con -Nes). Interestingly, CRH-overexpression reduced the duration of epileptic seizures and pre…

endocrine systemmedicine.medical_specialtyIndolesRNA UntranslatedCorticotropin-Releasing HormoneExcitotoxicityMice TransgenicNerve Tissue ProteinsBiologymedicine.disease_causeNeuroprotectionHippocampusNestinCorticotropin-releasing hormoneMiceIntermediate Filament ProteinsNeurotrophic factorsNeurofilament ProteinsSeizuresInternal medicineGlial Fibrillary Acidic Proteinpolycyclic compoundsmedicineExcitatory Amino Acid AgonistsReaction TimeAnimalsNeuroinflammationBrain-derived neurotrophic factorAnalysis of VarianceKainic AcidCell DeathGeneral NeuroscienceBrain-Derived Neurotrophic FactorNeurodegenerationProteinsLong-term potentiationmedicine.diseaseDisease Models AnimalEndocrinologynervous systemGene Expression RegulationNerve DegenerationNeurotoxicity SyndromesPlant Lectinshormones hormone substitutes and hormone antagonistsNeuroscience
researchProduct

Suppression and Replacement Gene Therapy for Autosomal Dominant Disease in a Murine Model of Dominant Retinitis Pigmentosa

2011

For dominantly inherited disorders development of gene therapies, targeting the primary genetic lesion has been impeded by mutational heterogeneity. An example is rhodopsin-linked autosomal dominant retinitis pigmentosa with over 150 mutations in the rhodopsin gene. Validation of a mutation-independent suppression and replacement gene therapy for this disorder has been undertaken. The therapy provides a means of correcting the genetic defect in a mutation-independent manner thereby circumventing the mutational diversity. Separate adeno-associated virus (AAV) vectors were used to deliver an RNA interference (RNAi)-based rhodopsin suppressor and a codon-modified rhodopsin replacement gene res…

genetic structuresGenetic enhancementMice TransgenicPolymerase Chain ReactionPhotoreceptor cellMiceRNA interferenceRetinitis pigmentosaDrug DiscoverymedicineGeneticsElectroretinographyAnimalsGeneMolecular BiologyPharmacologyGene therapy of the human retinabiologyAutosomal dominant traitGenetic Therapymedicine.diseaseMolecular biologyDisease Models Animalmedicine.anatomical_structureRhodopsinbiology.proteinMolecular MedicineOriginal Articlesense organsRetinitis PigmentosaMolecular Therapy
researchProduct

Characterization of Epileptic Spiking Associated With Brain Amyloidosis in APP/PS1 Mice

2019

Epileptic activity without visible convulsions is common in Alzheimer’s disease (AD) and may contribute adversely to the disease progress and symptoms. Transgenic mice with amyloid plaque pathology also display epileptic seizures, but those are too infrequent to assess the effect of anti-epileptic treatments. Besides spontaneous seizures, these mice also display frequent epileptic spiking in epidural EEG recordings, and these have provided a means to test potential drug treatment to AD-related epilepsy. However, the origin of EEG spikes in transgenic AD model mice has remained elusive, which makes it difficult to relate electrophysiology with underlying pathology at the cellular and molecul…

hippocampusamyloidoosiAlzheimer's diseaseAlzheimerin tautilcsh:RC346-429uni (lepotila)amyloid - beta- proteinaivokuoricortexNeurologymental disordersepilepsyNeurology (clinical)hippokampusEEGsleepepilepsialcsh:Neurology. Diseases of the nervous systemOriginal ResearchtransgenicFrontiers in Neurology
researchProduct