Search results for "Transgenic"

showing 10 items of 552 documents

Interleukin 10 restores lipopolysaccharide-induced alterations in synaptic plasticity probed by repetitive magnetic stimulation

2020

Systemic inflammation is associated with alterations in complex brain functions such as learning and memory. However, diagnostic approaches to functionally assess and quantify inflammation-associated alterations in synaptic plasticity are not well-established. In previous work, we demonstrated that bacterial lipopolysaccharide (LPS)-induced systemic inflammation alters the ability of hippocampal neurons to express synaptic plasticity, i.e., the long-term potentiation (LTP) of excitatory neurotransmission. Here, we tested whether synaptic plasticity induced by repetitive magnetic stimulation (rMS), a non-invasive brain stimulation technique used in clinical practice, is affected by LPS-induc…

lcsh:Immunologic diseases. AllergyLipopolysaccharides0301 basic medicinenon-invasive brain stimulationInterleukin-1betaImmunologyTNFα-reporter mouseMice TransgenicStimulationNeurotransmissionHippocampusSynaptic TransmissionneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineGenes Reportertranscranial magnetic stimulationAnimalsImmunology and Allergyddc:610NeuroinflammationOriginal ResearchInflammationNeuronsNeuronal Plasticitysynaptic plasticityInterleukin-6Tumor Necrosis Factor-alphaChemistryLong-term potentiationInterleukin-10Mice Inbred C57BLOrganoids030104 developmental biologyBrain stimulationSynaptic plasticityExcitatory postsynaptic potentialTumor necrosis factor alphaMicrogliainterleukin 10lcsh:RC581-607Neuroscience030217 neurology & neurosurgery
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Transgenic Killer Commensal Bacteria as Mucosal Protectants

2001

As first line of defense against the majority of infections and primary site for their transmission, mucosal surfaces of the oral cavity and genitourinary, gastrointestinal, and respiratory tracts represent the most suitable sites to deliver protective agents for the prevention of infectious diseases. Mucosal protection is important not only for life threatening diseases but also for opportunistic infections which currently represent a serious burden in terms of morbidity, mortality, and cost of cures. Candida albicans is among the most prevalent causes of mucosal infections not only in immuno- compromised patients, such as HIV-infected subjects who are frequently affected by oral and esoph…

lcsh:Medicinemedicine.disease_causeEsophageal candidiasislcsh:TechnologyGeneral Biochemistry Genetics and Molecular BiologyMicrobiologymucosal immunotherapyCandida albicansmedicineAnimalsHumansTransgenesCandida albicanslcsh:ScienceKiller recombinant antibodiesAntibodies FungalGeneral Environmental ScienceMucous MembranebiologyGenitourinary systemTransmission (medicine)Streptococcuslcsh:Ttransgenic commensal bacterislcsh:RCandidiasisMucous membraneStreptococcusGeneral Medicinebiology.organism_classificationmedicine.diseasemedicine.anatomical_structureProtective AgentsToxicityImmunologyVaginalcsh:QFemaleDirections in ScienceThe Scientific World Journal
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Low cholesterol stimulates the nonamyloidogenic pathway by its effect on the α-secretase ADAM 10

2001

Biochemical, epidemiological, and genetic findings demonstrate a link between cholesterol levels, processing of the amyloid precursor protein (APP), and Alzheimer's disease. In the present report, we identify the α-secretase ADAM 10 ( a d isintegrin a nd m etalloprotease) as a major target of the cholesterol effects on APP metabolism. Treatment of various peripheral and neural cell lines with either the cholesterol-extracting agent methyl-β-cyclodextrin or the hydroxymethyl glutaryl-CoA reductase inhibitor lovastatin resulted in a drastic increase of secreted α-secretase cleaved soluble APP. This strong stimulatory effect was in the range obtained with phorbol esters and was further increa…

media_common.quotation_subjectMice TransgenicMicechemistry.chemical_compoundAlzheimer DiseasemedicineAmyloid precursor proteinAnimalsSecretionInternalizationmedia_commonAmyloid beta-PeptidesMultidisciplinarybiologyCholesterolBiological SciencesADAM ProteinsCell biologycarbohydrates (lipids)CholesterolGene Expression RegulationchemistryBiochemistryAlpha secretasebiology.proteinLovastatinAmyloid precursor protein secretaseProtein Bindingmedicine.drugProceedings of the National Academy of Sciences
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Evaluation of a novel monoclonal antibody against tumor-associated MUC1 for diagnosis and prognosis of breast cancer

2019

There is still a great unmet medical need concerning diagnosis and treatment of breast cancer which could be addressed by utilizing specific molecular targets. Tumor-associated MUC1 is expressed on over 90 % of all breast cancer entities and differs strongly from its physiological form on epithelial cells, therefore presenting a unique target for breast cancer diagnosis and antibody-mediated immune therapy. Utilizing an anti-tumor vaccine based on a synthetically prepared glycopeptide, we generated a monoclonal antibody (mAb) GGSK-1/30, selectively recognizing human tumor-associated MUC1. This antibody targets exclusively tumor-associated MUC1 in the absence of any binding to MUC1 on health…

medicine.drug_classEstrogen receptorMUC1Breast NeoplasmsMice TransgenicDeferoxamineMonoclonal antibody89Zr03 medical and health sciences0302 clinical medicineBreast cancerIn vivoCell Line TumorBiomarkers TumormedicineAnimalsHumansTissue Distributionskin and connective tissue diseasesMUC1Triple-negative breast cancermAbRadioisotopesMice Inbred BALB Cbiologybusiness.industryMucin-1breast cancer diagnosisAntibodies MonoclonalCancerGeneral MedicinePrognosismedicine.diseaseImmunohistochemistryMice Inbred C57BLPositron-Emission Tomographybiology.proteinCancer researchFemale030211 gastroenterology & hepatologyZirconiumAntibodybusinessResearch PaperInternational Journal of Medical Sciences
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Physical exercise neuroprotects ovariectomized 3xTg-AD mice through BDNF mechanisms.

2014

Postmenopausal women may be more vulnerable to cognitive loss and Alzheimer's disease (AD) than premenopausal women because of their deficiency in estrogens, in addition to their usually older age. Aerobic physical exercise has been proposed as a therapeutic approach for maintaining health and well-being in postmenopausal women, and for improving brain health and plasticity in populations at high risk for AD. To study the neuroprotective mechanisms of physical exercise in a postmenopausal animal model, we submitted previously ovariectomized, six-month old non-transgenic and 3xTg-AD mice to three months of voluntary exercise in a running wheel. At nine months of age, we observed lower grip s…

medicine.medical_specialtyBehavioral testsEndocrinology Diabetes and MetabolismOvariectomyP-CREBPhysical exerciseMice Transgenictau ProteinsCREBNeuroprotectionGrip strengthAmyloid beta-Protein PrecursorMiceEndocrinologyCognitionAlzheimer DiseaseInternal medicinePhysical Conditioning AnimalNeuroplasticitymedicinePresenilin-1DementiaAnimalsApathy3xTg-AD miceBiological PsychiatryNeuronsFrailtybiologyEndocrine and Autonomic SystemsBrain-Derived Neurotrophic FactorPhysical exerciseAlzheimer's diseaseCatalasemedicine.diseaseMice Inbred C57BLPsychiatry and Mental healthDisease Models AnimalBDNFEndocrinologyNeuroprotective AgentsCytoprotectionbiology.proteinOvariectomized ratFemalemedicine.symptomPsychologySignal TransductionPsychoneuroendocrinology
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Progressive Endoplasmic Reticulum Stress Contributes to Hepatocarcinogenesis in Fatty Acyl-CoA Oxidase 1–Deficient Mice

2011

Fatty acyl-coenzyme A oxidase 1 (ACOX1) knockout (ACOX1(-/-)) mice manifest hepatic metabolic derangements that lead to the development of steatohepatitis, hepatocellular regeneration, spontaneous peroxisome proliferation, and hepatocellular carcinomas. Deficiency of ACOX1 results in unmetabolized substrates of this enzyme that function as biological ligands for peroxisome proliferator-activated receptor-α (PPARα) in liver. Here we demonstrate that sustained activation of PPARα in ACOX1(-/-) mouse liver by these ACOX1 substrates results in endoplasmic reticulum (ER) stress. Overexpression of transcriptional regulator p8 and its ER stress-related effectors such as the pseudokinase tribbles h…

medicine.medical_specialtyGenotypePeroxisome proliferator-activated receptorPeroxisome ProliferationMice TransgenicBiologyEndoplasmic ReticulumModels BiologicalPathology and Forensic MedicineMiceInternal medicinemedicineAnimalsHumansAcyl-CoA oxidasePPAR alphaTransgenesDNA Primerschemistry.chemical_classificationLiver cellEndoplasmic reticulumLiver NeoplasmsRegular ArticlePeroxisomemedicine.diseaseNeoplasm ProteinsCell biologyDNA-Binding ProteinsMice Inbred C57BLEndocrinologyGene Expression RegulationLiverchemistryHepatocytesUnfolded protein responseAcyl-CoA OxidaseSteatohepatitisThe American Journal of Pathology
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Penicillin induced epileptiform activity and EEG spectrum analysis of BDNF heterozygous mice: an in vivo electrophysiological study.

2011

Brain-derived neurotrophic factor (BDNF) heterozygous mice (BDNF (+/-)) kindle slowly and have a higher seizure threshold. However, BDNF (+/-) mice exhibit reduced cortical inhibition and disrupted balance of excitation/inhibition synaptic transmission. We investigated penicillin-induced focal cortical epileptiform activity and electroencephalogram (EEG) spectral power of BDNF (+/-) mice, by using electrocorticogram (ECoG) recordings. BDNF (+/-) mice (n=10) and wild type littermates (n=9) were anesthetized with i.p. urethane (1.750g/kg). The recordings of ECoG were carried out by using a data acquisition system and 100IU penicillin was administered intracortically to induce epileptiform act…

medicine.medical_specialtyHeterozygoteMicroinjectionsMice TransgenicNeocortexPenicillinsElectroencephalographyNeurotransmissionMiceNeurotrophic factorsInternal medicinemedicineAnimalsCerebral CortexMice KnockoutEpilepsymedicine.diagnostic_testSeizure thresholdChemistryGeneral NeuroscienceBrain-Derived Neurotrophic FactorWild typeElectroencephalographyCortex (botany)Electrophysiological PhenomenaElectrophysiologyEndocrinologymedicine.anatomical_structurenervous systemCerebral cortexNeuroscienceBrain research bulletin
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Relationships between adipose tissues and brain: what do we learn from animal studies?

2010

International audience; Over the last decades, more and more data supporting the importance of the relationships between the brain and adipose tissues (white and brown) in regards of body weight regulation and energy homeostasis have been published. Indeed the brain via the autonomic nervous system participates to the regulation of different parameters such as the metabolic (lipolysis, lipogenesis and thermogeneis), and secretory (leptin and other adipokines) activities but also plasticity (proliferation differentiation and apoptosis) of adipose tissues. In turn the various fat pads will send information via sensory innervation of white adipose tissue as well as metabolic and hormonal signa…

medicine.medical_specialtyMESH: Mice Transgenic[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismCentral nervous systemAdipokineAdipose tissueMice TransgenicWhite adipose tissueBiologyMESH : Adipose TissueEnergy homeostasisMiceMESH: BrainEndocrinologyMESH : Lipid MetabolismInternal medicineMESH : MiceBrown adipose tissueInternal MedicinemedicineAnimalsHumansLipolysisMESH: AnimalsMESH: MiceMESH: Lipid MetabolismMESH: HumansLeptinMESH : HumansBrainGeneral MedicineLipid MetabolismMESH : Mice TransgenicMESH : Models Animalmedicine.anatomical_structureEndocrinologyAdipose TissueMESH : BrainMESH: Models AnimalModels AnimalMESH : AnimalsNeuroscience[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH: Adipose Tissue
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An O2-sensitive glomus cell-stem cell synapse induces carotid body growth in chronic hypoxia.

2013

Summary Neural stem cells (NSCs) exist in germinal centers of the adult brain and in the carotid body (CB), an oxygen-sensing organ that grows under chronic hypoxemia. How stem cell lineage differentiation into mature glomus cells is coupled with changes in physiological demand is poorly understood. Here, we show that hypoxia does not affect CB NSC proliferation directly. Rather, mature glomus cells expressing endothelin-1, the O 2 -sensing elements in the CB that secrete neurotransmitters in response to hypoxia, establish abundant synaptic-like contacts with stem cells, which express endothelin receptors, and instruct their growth. Inhibition of glomus cell transmitter release or their sel…

medicine.medical_specialtyMice TransgenicBiologyGeneral Biochemistry Genetics and Molecular BiologyProlyl HydroxylasesMiceGlomus cellNeural Stem CellsInternal medicinemedicineAnimalsRats WistarCell ProliferationCarotid BodyCell growthBiochemistry Genetics and Molecular Biology(all)Germinal centerCell DifferentiationHypoxia (medical)Respiratory CenterNeural stem cellRatsOxygenEndocrinologymedicine.anatomical_structureCarotid bodyStem cellmedicine.symptomEndothelin receptorCell
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Hepatocellular Hyperplasia, Plasmacytoma Formation, and Extramedullary Hematopoiesis in Interleukin (IL)-6/Soluble IL-6 Receptor Double-Transgenic Mi…

1998

Cytokines interact not only with membrane anchored receptors, but also with specific soluble receptors which circulate in the bloodstream. In general, soluble cytokine receptors such as soluble tumor necrosis factor receptor, soluble interleukin 1 receptor, and soluble interleukin 4 receptor compete with their membrane-bound counterparts for the ligands and therefore act as antagonists. In contrast, soluble receptors for cytokines of the interleukin-6 (IL-6) family complex with their ligands act agonistically. Interestingly, the complex of IL-6 and the soluble interleukin 6 receptor (sIL-6R) activates target cells that do not express the membrane-bound IL-6R and therefore cannot respond to …

medicine.medical_specialtyMice TransgenicInterleukin 1 receptor type IIInterleukin-1 receptorPathology and Forensic MedicineMiceNecrosisInterleukin-4 receptorInternal medicinemedicineAnimalsHumansReceptorInterleukin 6HyperplasiabiologyInterleukin-6Body WeightLiver NeoplasmsInterleukinAnimal ModelsOrgan SizeReceptors Interleukin-6EndocrinologyLiverHematopoiesis ExtramedullaryInterleukin-6 receptorCancer researchbiology.proteinInterleukin 1 receptor type ISpleenPlasmacytomaThe American Journal of Pathology
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