Search results for "Transporters"

showing 10 items of 203 documents

Delayed post-ischemic administration of CDP-choline increases EAAT2 association to lipid rafts and affords neuroprotection in experimental stroke

2007

Glutamate transport is the only mechanism for maintaining extracellular glutamate concentrations below excitotoxic levels. Among glutamate transporters, EAAT2 is responsible for up to 90% of all glutamate transport and has been reported to be associated to lipid rafts. In this context, we have recently shown that CDP-choline induces EAAT2 translocation to the membrane. Since CDP-choline preserves membrane stability by recovering levels of sphingomyelin, a glycosphingolipid present in lipid rafts, we have decided to investigate whether CDP-choline increases association of EAAT2 transporter to lipid rafts. Flotillin-1 was used as a marker of lipid rafts due to its known association to these m…

MaleCytidine Diphosphate CholineTime FactorsIschemiaGlutamic AcidContext (language use)PharmacologyBiologyCell FractionationNeuroprotectionlcsh:RC321-571chemistry.chemical_compoundMembrane MicrodomainsIschemiamedicineAnimalsCholineLipid raftlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryGlutamate transportersGlutamate receptorInfarction Middle Cerebral ArteryGlutamic acidmedicine.diseaseRats Inbred F344Ratscarbohydrates (lipids)Disease Models AnimalNeuroprotective AgentsExcitatory Amino Acid Transporter 2Gene Expression RegulationNeurologyBiochemistrychemistrylipids (amino acids peptides and proteins)GlutamateSphingomyelinNeurobiology of Disease
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Human cationic amino acid transporter hCAT-3 is preferentially expressed in peripheral tissues.

2001

At least five distinct carrier proteins form the family of mammalian cationic amino acid transporters (CATs). We have cloned a cDNA containing the complete coding region of human CAT-3. hCAT-3 is glycosylated and localized to the plasma membrane. Transport studies in Xenopus laevis oocytes revealed that hCAT-3 is selective for cationic L-amino acids and exhibits a maximal transport activity similar to other CAT proteins. The apparent substrate affinity and sensitivity to trans-stimulation of hCAT-3 resembles most closely hCAT-2B. This is in contrast to rat and murine CAT-3 proteins that have been reported to display a very low activity and to be inhibited by neutral and anionic L-amino acid…

MaleDNA ComplementaryGene ExpressionThymus GlandIn Vitro TechniquesBiochemistryCell LineMiceXenopus laevisComplementary DNACoding regionAnimalsHumansTissue DistributionAmino acid transporterAmino Acid SequenceCationic Amino Acid Transporterschemistry.chemical_classificationCATSBase SequenceChemistryCationic polymerizationBrainMembrane ProteinsAmino acidRatsBiochemistryCarrier proteinOocytesAmino Acid Transport Systems BasicFemaleCarrier ProteinsBiochemistry
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Tiagabine, a gamma-amino-butyric acid transporter inhibitor impairs spatial learning of rats in the Morris water-maze.

2002

Abstract γ-Amino-butyric acid (GABA) is cleaved from the synaptic cleft by uptake via specific transporters. Inhibition of such transporters increases the effectiveness of physiologically released GABA. Increased GABAergic neurotransmission has an impact on learning and memory. Therefore, effects of tiagabine, a GABA-transporter inhibitor, were investigated on spatial orientation in the Morris water-maze. Rats were given four training trials per day for 4 days and a probe trial without platform on the 5th day. Compared to saline treated rats, rats treated daily with 20 mg/kg tiagabine showed impaired learning during the acquisition trials. Retrieval was impaired in rats treated only at the …

MaleGABA Plasma Membrane Transport ProteinsSynaptic cleftTiagabinemedicine.medical_treatmentNipecotic AcidsMorris water navigation taskOrganic Anion TransportersPharmacologyBehavioral Neurosciencechemistry.chemical_compoundMemorymedicineGABA transporterAnimalsNeurotransmitterMaze LearningTiagabineSalineGABA AgonistsSwimmingbiologyMembrane ProteinsMembrane Transport ProteinsTransporterRats Inbred StrainsReceptors GABA-ARatschemistrybiology.proteinReuptake inhibitorCarrier ProteinsNeurosciencemedicine.drugBehavioural brain research
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Effects of polymorphisms in vitamin E-, vitamin C-, and glutathione peroxidase-related genes on serum biomarkers and associations with glaucoma.

2012

Purpose To study the association of selected polymorphism in genes related to vitamin E, vitamin C, and glutathione peroxidase with these biomarkers and primary open-angle glaucoma (POAG) risk. Methods A case-control study matched for age, sex, and bodyweight was undertaken. Two hundred fifty POAG cases and 250 controls were recruited from a Mediterranean population. Plasma concentrations of vitamin C, vitamin E, and glutathione peroxidase (GPx) activity were measured. We analyzed the polymorphisms rs1279683 in the Na+-dependent L-ascorbic acid transporter 2 (SLC23A2) gene, rs6994076 in the tocopherol alpha transfer protein (TTPA) gene, rs737723 in the tocopherol-associated protein (SEC14L2…

MaleGlutathione PeroxidasePolymorphism Geneticgenetic structuresLipoproteinsEpistasis GeneticAscorbic AcidMiddle AgedPhospholipid Hydroperoxide Glutathione PeroxidasePolymorphism Single Nucleotideeye diseasesRisk FactorsCase-Control StudiesTrans-ActivatorsHumansVitamin EFemaleCarrier ProteinsSodium-Coupled Vitamin C TransportersBiomarkersGenetic Association StudiesGlaucoma Open-AngleAgedResearch ArticleMolecular vision
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Goitrous Congenital Hypothyroidism and Hearing Impairment Associated with Mutations in the TPO and SLC26A4/PDS Genes

2006

Abstract Context: Pendred syndrome (PS) and thyroid peroxidase (TPO) deficiency are autosomal-recessive disorders that result in thyroid dyshormonogenesis. They share congenital hypothyroidism, goiter, and an iodide organification defect as common features. Whereas the hallmark of PS is sensorineural deafness, other forms of congenital hypothyroidism may also lead to hearing impairment. Therefore, a definite diagnosis may be difficult and require molecular genetic analyses. Case Report: The propositus presented at birth with primary hypothyroidism and goiter. He also had congenital bilateral moderate hearing loss, and PS was suspected. Methods: We sequenced the SLC26A4/PDS and TPO genes in …

MaleHeterozygoteendocrine systemmedicine.medical_specialtyGenotypeHearing lossEndocrinology Diabetes and MetabolismClinical BiochemistryMutation MissenseTransfectionIodide PeroxidaseBiochemistryEndocrinologyThyroid dyshormonogenesisThyroid peroxidaseInternal medicineCongenital Hypothyroidismotorhinolaryngologic diseasesHumansMedicineMissense mutationHearing LossPendred syndromebiologyGoiterbusiness.industryBiochemistry (medical)Infant NewbornPrimary hypothyroidismMembrane Transport ProteinsPendrinmedicine.diseasePedigreeCongenital hypothyroidismEndocrinologySulfate Transportersbiology.proteinmedicine.symptombusinessThe Journal of Clinical Endocrinology & Metabolism
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Alteration of hypothalamic glucose and lactate sensing in 48h hyperglycemic rats.

2013

International audience; Hypothalamic detection of nutrients is involved in the control of energy metabolism and is altered in metabolic disorders. Although hypothalamic detection of blood lactate lowers hepatic glucose production and food intake, it is unknown whether it also modulates insulin secretion. To address this, a lactate injection via the right carotid artery (cephalad) was performed in Wistar rats. This triggered a transient increase in insulin secretion. Rats made hyperglycemic for 48h exhibited prolonged insulin secretion in response to a glucose injection via the carotid artery, but lactate injection induced two types of responses: half of the HG rats showed no difference comp…

MaleMonocarboxylic Acid TransportersGene isoformmedicine.medical_specialtyTime Factors[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionHypothalamusMuscle ProteinsBiologyCarbohydrate metabolismInternal medicineInsulin SecretionBlood lactatemedicineExtracellularAnimalsInsulinLactic AcidRats WistarInsulin secretionSymportersGeneral NeuroscienceGlucose InjectionTransporterRatsGlucoseEndocrinologyHypothalamusHyperglycemia[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Association of Monocarboxylate Transporter-1 (MCT1) A1470T Polymorphism (rs1049434) with Forward Football Player Status.

2018

AbstractThe aim of this study was to investigate the association between the MCT1 (monocarboxylate transporter 1) A1470T polymorphism and positional roles in a large cohort of professional football players from five different countries. We compared genotype distributions of the MCT1 A1470T polymorphism between football players (n=694) and non-athlete controls (n=781) from Italy, Poland, Lithuania, Ukraine and Malta, and we analyzed the MCT1 genotype distributions with respect to the players’ positions in the field (e. g. forwards, midfielders, defenders and goalkeepers). Genomic DNA was extracted from either buccal epithelium or peripheral blood using a standard protocol. In the pooled coho…

MaleMonocarboxylic Acid TransportersGenotypePhysical Therapy Sports Therapy and RehabilitationFootball030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicineGene FrequencySoccerGenotypeHumansOrthopedics and Sports MedicineGenetic differences lactate monocarboxylate transporter-1 repeated sprint ability soccerAlleleAllele frequencyAllelesGenetic Association StudiesPolymorphism GeneticSymportersbiology030229 sport sciencesPeripheral bloodMonocarboxylate transporter 1CohortStandard protocolbiology.proteinDemography
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Elevated serum triiodothyronine and intellectual and motor disability with paroxysmal dyskinesia caused by a monocarboxylate transporter 8 gene mutat…

2008

Monocarboxylate transporter 8 (MCT8 or SLC16A2) is important for the neuronal uptake of triiodothyronine (T3) in its function as a specific and active transporter of thyroid hormones across the cell membrane, thus being essential for human brain development. We report on a German male with Allan-Herndon-Dudley syndrome presenting with severe intellectual and motor disability, paroxysmal dyskinesia combined with truncal muscular hypotonia, and peripheral muscular hypertonia at his current age of 9 years. Additionally, the patient has a lesion in the left putamen region revealed by magnetic resonance imaging and elevated serum T3 levels. The male appeared to have a hemizygous mutation (R271H)…

MaleMonocarboxylic Acid Transportersmedicine.medical_specialtyDevelopmental DisabilitiesDNA Mutational AnalysisEnzyme-Linked Immunosorbent AssayGene mutationArginineLesionDevelopmental NeuroscienceChoreaInternal medicineIntellectual DisabilitymedicineHumansHistidineChildMonocarboxylate transporterAllan–Herndon–Dudley syndromeTriiodothyroninebiologyMuscular hypotoniaSymportersParoxysmal dyskinesiamedicine.diseaseMagnetic Resonance ImagingEndocrinologyPediatrics Perinatology and Child HealthMutationbiology.proteinHypertoniaTriiodothyronineNeurology (clinical)medicine.symptomDevelopmental medicine and child neurology
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Dehydroepiandrosterone up-regulates the Adrenoleukodystrophy-related gene (ABCD2) independently of PPAR alpha in rodents

2007

International audience; X-linked adrenoleukodystrophy (X-ALD) is a neurodegenerative disease caused by mutations in the ABCD1 gene, which encodes a peroxisomal ABC transporter, ALDP, supposed to participate in the transport of very long chain fatty acids (VLCFA). The adrenoleukodystrophyrelated protein (ALDRP), which is encoded by the ABCD2 gene, is the closest homolog of ALDP and is considered as a potential therapeutic target since functional redundancy has been demonstrated between the two proteins. Pharmacological induction of Abcd2 by fibrates through the activation of PPARa has been demonstrated in rodent liver. DHEA, the most abundant steroid in human, is described as a PPARa activat…

MalePEROXISOMEProhormonePeroxisome proliferator-activated receptorATP-binding cassette transporterBiochemistryMice0302 clinical medicineABC TRANSPORTERSPPAR-ALPHAAdrenal GlandsTestisDHEACells Culturedchemistry.chemical_classification0303 health sciencesSex CharacteristicsbiologyBrainGeneral MedicineOrgan SizePeroxisome3. Good healthUp-RegulationLiverAdrenoleukodystrophyFemalemedicine.drugAndrostenediolmedicine.medical_specialtyADRENOLEUKODYSTROPHYATP Binding Cassette Transporter Subfamily D03 medical and health sciencesABCD3Internal medicinemedicineABCD2AnimalsPPAR alpha[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRats Wistar030304 developmental biologyActivator (genetics)Body Weightnutritional and metabolic diseasesMembrane ProteinsDehydroepiandrosteronemedicine.diseaseRatsMice Inbred C57BLEndocrinologychemistrybiology.proteinHepatocytesATP-Binding Cassette TransportersAcyl-CoA Oxidase030217 neurology & neurosurgery
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Regulation of organic anion transporters in a new rat model of acute and chronic cholangitis resembling human primary sclerosing cholangitis

2002

Primary sclerosing cholangitis (PSC) is a cholestatic liver disease of unknown etiology. Although the primary defect affects cholangiocytes, cholestatic injury of hepatocytes may promote further liver damage. Since down-regulation of hepatocellular organic anion transporters is implicated in the molecular pathogenesis of cholestasis, expression of these transporters was determined in a novel rat model, which closely resembles human PSC.Hepatic protein and mRNA expression of basolateral (Ntcp, Oatp1, 2 and 4) and canalicular (Mrp2, Bsep) organic anion transporters were analyzed 1, 4 and 12 weeks after induction of experimental PSC by 2,4,6-trinitrobenzenesulfonic acid (TNBS).Specific down-re…

MalePathologymedicine.medical_specialtyOrganic Cation Transport ProteinsOrganic anion transporter 1Cholangitis SclerosingGene ExpressionOrganic Anion Transporters Sodium-DependentInflammationOrganic Anion Transporters Sodium-Independentdigestive systemPrimary sclerosing cholangitisSolute Carrier Organic Anion Transporter Family Member 1B3CholestasismedicineAnimalsRNA MessengerChronic CholangitisLiver injurySymportersHepatologybiologybusiness.industryMultidrug resistance-associated protein 2Membrane Transport ProteinsTransportermedicine.diseasedigestive system diseasesRatsDisease Models AnimalRats Inbred LewAcute DiseaseChronic Diseasebiology.proteinATP-Binding Cassette TransportersFemalemedicine.symptomCarrier ProteinsbusinessJournal of Hepatology
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