Search results for "Trophoblasts"

showing 5 items of 15 documents

Subcellular distribution of choline acetyltransferase by immunogold electron microscopy in non-neuronal cells: Placenta, airways and murine embryonic…

2012

Abstract Aims Acetylcholine is synthesized in more or less all mammalian cells. However, little is known about the subcellular location of acetylcholine synthesis. Therefore, in the present experiments the subcellular location of the synthesizing enzyme choline acetyltransferase (ChAT) was investigated by anti-ChAT immunogold electron microscopy in human placenta and airways as well as in a murine embryonic stem cell line (CGR8 cell line). Main methods Human tissue was obtained as so-called surplus tissue (after delivery/surgical removal because of lung tumor); the CGR8 stem cell line was cultured under standard conditions. For human tissue a monoclonal mouse anti-ChAT antibody (ab) was use…

PlacentaeducationBronchiRespiratory MucosaBiologyGeneral Biochemistry Genetics and Molecular BiologyCell LineCholine O-AcetyltransferaseCell membraneMicePregnancyCaveolaeMacrophages Alveolarmental disordersmedicineAnimalsHumansGeneral Pharmacology Toxicology and PharmaceuticsNuclear membraneCells CulturedEmbryonic Stem Cellshealth care economics and organizationsEpithelial CellsGeneral MedicineImmunogold labellingImmunohistochemistryCholine acetyltransferaseMolecular biologyCellular StructureshumanitiesTrophoblastsCell biologyMicroscopy ElectronCytosolCell nucleusmedicine.anatomical_structureCell cultureFemaleLife Sciences
researchProduct

Preeclampsia: a defect in decidualization is associated with deficiency of Annexin A2.

2020

Background Decidualization defects in the endometrium have been demonstrated at the time of delivery in women with severe preeclampsia and to linger for years, which suggests a maternal contribution to the pathogenesis of this condition. Global transcriptional profiling reveals alterations in gene expression, which includes down-regulation of Annexin A2 in severe preeclampsia patients with decidualization resistance. Objective We investigated the functional role of Annexin A2 deficiency during endometrial decidualization and its potential contribution to shallow trophoblast invasion during implantation and subsequent placentation using in vitro and in vivo modeling. Study Design Annexin A2 …

Stromal cellGene ExpressionEndometriumAndrology03 medical and health sciencesMice0302 clinical medicinePre-EclampsiaAnnexinCell MovementPregnancymedicineDeciduaAnimalsHumans030212 general & internal medicineEmbryo ImplantationRNA Small InterferingAnnexin A2Cells Cultured030219 obstetrics & reproductive medicinemedicine.diagnostic_testbusiness.industryObstetrics and GynecologyDecidualizationPlacentationTrophoblastActinsPlacentationProlactinTrophoblastsInsulin-Like Growth Factor Binding Protein 1Disease Models Animalmedicine.anatomical_structureFemaleStromal CellsbusinessAnnexin A2Endometrial biopsyAmerican journal of obstetrics and gynecology
researchProduct

Demonstration of P29, an oestrogen receptor-associated tumor marker, in human term placenta.

1991

A Mr29,000 serine phosphoprotein (P29) related to oestradiol receptor was studied in human term placenta with the use of a specific monoclonal antibody (D5). D5 was used with two different methods, immunohistochemistry and immunoradiometry. For immunohistochemistry, an indirect immunoperoxidase method was chosen to detect P29 in methacarn-fixed, wax-embedded sections. P29 was mostly confined to the syncytiotrophoblast surrounding placental villi, staining being positive in both cytoplasm and nuclei. The stroma of villi was negative. Content of P29 was uniformly high in crude placental cytosol, as measured by immunoradiometry assays. Specificity of D5 against P29 in placenta was tested by an…

medicine.drug_classPlacentaBiologyMonoclonal antibodySyncytiotrophoblastPregnancyPlacentamedicineBiomarkers TumorSerineHumansHeat-Shock ProteinsImmunoperoxidaseObstetrics and GynecologyAntibodies MonoclonalPhosphoproteinsMolecular biologyImmunohistochemistryStainingTrophoblastsmedicine.anatomical_structureReproductive MedicineReceptors EstrogenCytoplasmPhosphoproteinembryonic structuresImmunohistochemistryFemaleImmunoradiometric AssayDevelopmental BiologyPlacenta
researchProduct

Decidual endothelial cells express surface-bound C1q as a molecular bridge between endovascular trophoblast and decidual endothelium.

2008

This study was prompted by the observation that decidual endothelial cells (DECs), unlike endothelial cells (ECs) of blood vessels in normal skin, kidney glomeruli and brain, express surface-bound C1q in physiologic pregnancy. This finding was unexpected, because deposits of C1q are usually observed in pathologic conditions and are associated with complement activation. In the case of DECs, we failed to detect immunoglobulins and C4 co-localized with C1q on the cell surface. Surprisingly, DECs expressed mRNA for the three chains of C1q and secreted detectable level of this component in serum-free medium. The ability to synthesize C1q is acquired by DECs during pregnancy and is not shared by…

medicine.medical_specialtyC1q; Trophoblast; Endothelial cells; GlycosaminoglycansEndotheliumBlood VesselEndothelial cellsCellImmunologychemical and pharmacologic phenomenaBiologyurologic and male genital diseasesArticleEndothelial cellimmune system diseasesPregnancyInternal medicineparasitic diseasesmedicineCell AdhesionDeciduaHumansReceptorCell adhesionskin and connective tissue diseasesMolecular BiologyC1qGlycosaminoglycansC1q; Endothelial cells; Glycosaminoglycans; Trophoblast; Blood Vessels; Cell Adhesion; Complement C1q; Decidua; Endothelial Cells; Female; Humans; Membrane Glycoproteins; Pregnancy; Receptors Complement; Trophoblasts; Molecular Biology; ImmunologyEndothelial CellMembrane GlycoproteinsComplement C1qDeciduaTrophoblastTrophoblastComplement systemCell biologyTrophoblastsReceptors Complementmedicine.anatomical_structureEndocrinologyGlycosaminoglycanBlood VesselsFemaleMembrane GlycoproteinIntracellularHuman
researchProduct

An alternative role of C1q in cell migration and tissue remodeling: contribution to trophoblast invasion and placental development.

2010

Abstract Fetal trophoblast cells invading the decidua in the early phase of pregnancy establish complex interaction with the maternal extracellular matrix. We discovered that C1q was widely distributed in human decidual stroma in the absence of C4 and C3 and was actively synthesized by migrating extravillous trophoblasts. The cells expressed the messages for the three chains of C1q and secreted this complement component that interacted with the proteins of the decidual extracellular matrix. Solid phase-bound C1q promoted trophoblast adhesion and migration, and cell binding to C1q resulted in activation of ERK1/2 MAPKs. Ab inhibition experiments showed that the receptors for the globular hea…

medicine.medical_specialtyImmunologyCellIntegrinImmunoblottingchemical and pharmacologic phenomenaBiologyExtracellular matrixMicePre-Eclampsiaimmune system diseasesPregnancyInternal medicinemedicineCell AdhesionImmunology and AllergyAnimalsHumansImmunoprecipitationskin and connective tissue diseasesReceptorCell adhesionreproductive and urinary physiologyMicroscopy ConfocalC1q placental development.Reverse Transcriptase Polymerase Chain ReactionComplement C1qDeciduaTrophoblastPlacentationImmunohistochemistryPlacentationCell biologyTrophoblastsMice Inbred C57BLChemotaxis Leukocytemedicine.anatomical_structureEndocrinologyembryonic structuresbiology.proteinFemaleJournal of immunology (Baltimore, Md. : 1950)
researchProduct