Search results for "Trophy"

showing 10 items of 10031 documents

Estimation of Location and Activation Time of Purkinje Myocardial Junctions from Sparse and Noisy Endocardial Electrical Samples

2017

The activation of the myocardial muscle is triggered by Purkinje-myocardial junctions (PMJs), which are the terminal sites of the specialised cardiac conduction system (CCS). Obtaining the location of the PMJs and other sources of endocardial ectopic activity would be desirable for building computer models of cardiac electrophysiology and planning ablation interventions. We present a method to estimate the location and activation times of endocardial electrical sources in a 3D model of the ventricles. The algorithm requires a set of discrete electrical samples on the endocardium, which can include errors in location and activation time. The estimated sources are properly placed with a locat…

03 medical and health sciences0302 clinical medicineObservational errorTerminal (electronics)Computer scienceCardiac electrophysiologyTime error3d model030204 cardiovascular system & hematologyElectrical conduction system of the heartEndocardium030218 nuclear medicine & medical imagingBiomedical engineeringComputing in Cardiology Conference (CinC)
researchProduct

Dual-sided Mapping During Global Stretch Using a Custom Miniaturized Endocardial Balloon with a Multipurpose Multichannel Acquisition System for Prec…

2017

Acute regional myocardial stretch (STR) is known to induce myocardial electrophysiological modifications via mechano-electric feedback (MEF), including abnormal repolarization, premature excitation and increased complexity of activation leading to the initiation, maintenance and acceleration of arrhythmic events. Due to technical limitations little is known about intramural and endo-epicardial (ENDO-EPI) phenomena during these events. Understanding ENDO-EPI modifications produced by STR through MEF may require a dual-sided approach to simultaneously correlate macroscopic surface modifications and their independent or interconnected components. For that purpose, we optically mapped (OM) the …

0301 basic medicine030103 biophysics03 medical and health sciencesElectrophysiologyComputer scienceRepolarizationBalloonBiomedical engineeringComputing in Cardiology Conference (CinC)
researchProduct

Novel Autotrophic Organisms Contribute Significantly to the Internal Carbon Cycling Potential of a Boreal Lake

2018

ABSTRACT Oxygen-stratified lakes are typical for the boreal zone and also a major source of greenhouse gas emissions in the region. Due to shallow light penetration, restricting the growth of phototrophic organisms, and large allochthonous organic carbon inputs from the catchment area, the lake metabolism is expected to be dominated by heterotrophic organisms. In this study, we test this assumption and show that the potential for autotrophic carbon fixation and internal carbon cycling is high throughout the water column. Further, we show that during the summer stratification carbon fixation can exceed respiration in a boreal lake even below the euphotic zone. Metagenome-assembled genomes an…

0301 basic medicine030106 microbiologyHeterotrophDNA RibosomaljärvetMicrobiologyCarbon cycle03 medical and health sciencesWater columnTotal inorganic carbonRNA Ribosomal 16SVirologyCluster AnalysisPhotic zoneAnaerobiosis14. Life underwaterAutotrophFinlandPhylogeny030304 developmental biologyTotal organic carbon0303 health sciencesAutotrophic ProcessesmetagenomicsPhototroph030306 microbiologygreenhouse gas emissionsEcologyhiilen kiertoCarbon fixationSequence Analysis DNAiron oxidizers15. Life on landBiotaCarbonQR1-502Food webLakesMikrobiologi13. Climate actionmikro-organismitredox gradientEnvironmental sciencechemoautotrophyResearch ArticlemBio
researchProduct

Systemic blockade of ACVR2B ligands prevents chemotherapy-induced muscle wasting by restoring muscle protein synthesis without affecting oxidative ca…

2016

AbstractDoxorubicin is a widely used and effective chemotherapy drug. However, cardiac and skeletal muscle toxicity of doxorubicin limits its use. Inhibiting myostatin/activin signalling can prevent muscle atrophy, but its effects in chemotherapy-induced muscle wasting are unknown. In the present study we investigated the effects of doxorubicin administration alone or combined with activin receptor ligand pathway blockade by soluble activin receptor IIB (sACVR2B-Fc). Doxorubicin administration decreased body mass, muscle size and bone mineral density/content in mice. However, these effects were prevented by sACVR2B-Fc administration. Unlike in many other wasting situations, doxorubicin indu…

0301 basic medicineACUTE DOXORUBICIN CARDIOTOXICITYEXPRESSIONmedicine.medical_specialtyMDX MICEhuumeetlihaksetMyostatinProtein degradationEXERCISE PROTECTSMYOSTATINArticledrugs03 medical and health sciencesInternal medicinemedicineDoxorubicinCANCER CACHEXIApreclinical researchWastingaineenvaihduntaMultidisciplinaryCARDIOMYOPATHYbiologyRECEPTORbusiness.industrychemotheraphyta1182Skeletal muscleta3141Activin receptorta3122Muscle atrophy3. Good health030104 developmental biologyEndocrinologymedicine.anatomical_structurebiology.proteinSKELETAL-MUSCLEHEARTmuscles3111 Biomedicinemedicine.symptombusinessmetabolismACVR2Bmedicine.drug
researchProduct

Cognitive network hyperactivation and motor cortex decline correlate with ALS prognosis.

2021

We aimed to quantitatively characterize progressive brain network disruption in Amyotrophic Lateral Sclerosis (ALS) during cognition using the mismatch negativity (MMN), an electrophysiological index of attention switching. We measured the MMN using 128-channel EEG longitudinally (2-5 timepoints) in 60 ALS patients and cross-sectionally in 62 healthy controls. Using dipole fitting and linearly constrained minimum variance beamforming we investigated cortical source activity changes over time. In ALS, the inferior frontal gyri (IFG) show significantly lower baseline activity compared to controls. The right IFG and both superior temporal gyri (STG) become progressively hyperactive longitudina…

0301 basic medicineAdultMaleAgingmedicine.medical_specialtyMismatch negativityPrefrontal CortexElectroencephalographyAudiologybehavioral disciplines and activities03 medical and health sciences0302 clinical medicineCognitionmedicineHumansAttentionCognitive DysfunctionAmyotrophic lateral sclerosisAgedAged 80 and overHyperactivationmedicine.diagnostic_testbusiness.industryGeneral NeuroscienceAmyotrophic Lateral SclerosisMotor CortexCognitionElectroencephalographyMiddle Agedmedicine.diseasePrognosisTemporal LobeElectrophysiology030104 developmental biologymedicine.anatomical_structureCross-Sectional StudiesDisinhibitionFemaleNeurology (clinical)Geriatrics and Gerontologymedicine.symptombusiness030217 neurology & neurosurgeryDevelopmental BiologyMotor cortexNeurobiology of aging
researchProduct

The largest caucasian kindred with dentatorubral-pallidoluysian atrophy: A founder mutation in italy

2019

BACKGROUND: Dentatorubral-pallidoluysian atrophy is a hereditary neurodegenerative disease prevalently reported in Japan but rare in Caucasians. The objective of this study was to reconstruct the pedigree of Italian dentatorubral-pallidoluysian atrophy familial cases describing their clinical features. METHODS: We investigated 6 apparently unrelated dentatorubral-pallidoluysian atrophy families comprising a total of 51 affected individuals: 13 patients were clinically examined, and for 38 patients clinical data were collected from clinical sources. The dentatorubral-pallidoluysian atrophy diagnosis was genetically confirmed in 18 patients. Genealogical data from historical archives were ana…

0301 basic medicineAdultMalePediatricsmedicine.medical_specialtyAdolescentDiseaseNeuropsychological TestsWhite People03 medical and health sciencesEpilepsyYoung Adult0302 clinical medicineAtrophyTrinucleotide Repeatsdentatorubral-pallidoluysian atrophymedicineHumansFamilyATN1 geneChildFounder mutationAgedDentatorubral-pallidoluysian atrophyEpilepsybusiness.industrygenealogical methodMiddle Agedmedicine.diseaseMyoclonic Epilepsies ProgressivePedigree030104 developmental biologyfounder effectNeurologyCerebellar cognitive affective syndromeItalycerebellar cognitive-affective syndromeMutationFemaleNeurology (clinical)business030217 neurology & neurosurgeryFounder effect
researchProduct

Cortical network fingerprints predict deep brain stimulation outcome in dystonia.

2018

AbstractBackgroundDeep brain stimulation (DBS) is an effective evidence-based therapy for dystonia. However, no unequivocal predictors of therapy responses exist. We investigate whether patients optimally responding to DBS present distinct brain network organization and structural patterns.MethodsBased on a German multicentre cohort of eighty-two dystonia patients with segmental and generalized dystonia, who received DBS implantation in the globus pallidus internus patients were classified based on the clinical response 36 months after DBS, as superior-outcome group or moderate-outcome group, as above or below 70% motor improvement, respectively. Fifty-one patients met MRI-quality and treat…

0301 basic medicineAdultMaleTreatment responsemedicine.medical_specialtyDeep brain stimulationMovement disordersmedicine.medical_treatmentDeep Brain Stimulation610 MedizinStimulationGrey matterGlobus PallidusSeverity of Illness IndexCohort Studies03 medical and health sciences0302 clinical medicineText miningPhysical medicine and rehabilitationAtrophy610 Medical sciencesmedicineHumansddc:610Dystoniabusiness.industryStructural integrityMiddle Agedmedicine.diseasenervous system diseasesDystoniamedicine.anatomical_structure030104 developmental biologyNeurologyCortical networkDystonic DisordersCohortFemaleNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryMovement disorders : official journal of the Movement Disorder SocietyReferences
researchProduct

Long-term physical activity modulates brain processing of somatosensory stimuli: Evidence from young male twins.

2016

Leisure-time physical activity is a key contributor to physical and mental health. Yet the role of physical activity in modulating cortical function is poorly known. We investigated whether precognitive sensory brain functions are associated with the level of physical activity. Physical activity history (3-yr-LTMET), physiological measures and somatosensory mismatch response (sMMR) in EEG were recorded in 32 young healthy twins. In all participants, 3-yr-LTMET correlated negatively with body fat%, r = −0.77 and positively with VO2max, r = 0.82. The fat% and VO2max differed between 15 physically active and 17 inactive participants. Trend toward larger sMMR was seen in inactive compared to ac…

0301 basic medicineAdultMalehuman electrophysiologyFITNESSMISMATCH NEGATIVITY MMN515 PsychologyPhysical activityMonozygotic twinprecognitive brain functionPhysical exerciseSensory systemCHILDRENGatingElectroencephalographyMotor ActivitySomatosensory system03 medical and health sciences0302 clinical medicinephysical exercisemedicineEVOKED-POTENTIALSHumansHealthy Lifestyleta315Evoked PotentialsExerciseYoung malemedicine.diagnostic_testLATENCYGeneral NeuroscienceMEMORYta3141ADULTSSomatosensory Cortex16. Peace & justice3142 Public health care science environmental and occupational healthta3124030104 developmental biologyNeuropsychology and Physiological PsychologyDISCRIMINATIONFemalePsychologyNeurosciencesomatosensory mismatch response030217 neurology & neurosurgeryRESPONSESBiological psychology
researchProduct

Clinical implications of serum neurofilament in newly diagnosed MS patients: a longitudinal multicentre cohort study

2020

Abstract Background We aim to evaluate serum neurofilament light chain (sNfL), indicating neuroaxonal damage, as a biomarker at diagnosis in a large cohort of early multiple sclerosis (MS) patients. Methods In a multicentre prospective longitudinal observational cohort, patients with newly diagnosed relapsing-remitting MS (RRMS) or clinically isolated syndrome (CIS) were recruited between August 2010 and November 2015 in 22 centers. Clinical parameters, MRI, and sNfL levels (measured by single molecule array) were assessed at baseline and up to four-year follow-up. Findings Of 814 patients, 54.7% (445) were diagnosed with RRMS and 45.3% (369) with CIS when applying 2010 McDonald criteria (R…

0301 basic medicineAdultMalemedicine.medical_specialtyResearch paperClinical Decision-MakingIntermediate Filamentslcsh:Medicine610 Medicine & healthNewly diagnosedGeneral Biochemistry Genetics and Molecular BiologyMultiple sclerosis03 medical and health sciences0302 clinical medicineAtrophyMultiple Sclerosis Relapsing-RemittingNeurofilament ProteinsInternal medicineGermanymedicineHumansLongitudinal StudiesProspective Studiesddc:610610 Medicine & healthNeurofilament light chainlcsh:R5-920Clinically isolated syndromebusiness.industryMultiple sclerosislcsh:RMcDonald criteriaGeneral MedicineBiomarkermedicine.diseasesNfL030104 developmental biology030220 oncology & carcinogenesisCohortDisease ProgressionCommentaryBiomarker (medicine)Femalelcsh:Medicine (General)businessPredictionFunction and Dysfunction of the Nervous SystemBiomarkersCohort study
researchProduct

Molecular and clinical studies in five index cases with novel mutations in the GLA gene

2016

Fabry disease is a metabolic and lysosomal storage disorder caused by the functional defect of the α-galactosidase A enzyme; this defect is due to mutations in the GLA gene, that is composed of seven exons and is located on the long arm of the X-chromosome (Xq21–22). The enzymatic deficit is responsible for the accumulation of glycosphingolipids in lysosomes of different cellular types, mainly in those ones of vascular endothelium. It consequently causes a cellular and microvascular dysfunction. In this paper, we described five novel mutations in the GLA gene, related to absent enzymatic activity and typical manifestations of Fabry disease. We identified three mutations (c.846_847delTC, p.E…

0301 basic medicineAdultMalep.R227Pnovel moutationAdolescentc.639 + 5G > TMutation MissenseBiologyLeft ventricular hypertrophy03 medical and health sciencesExonYoung Adult0302 clinical medicineSettore BIO/13 - Biologia ApplicataGeneticsmedicinefabry; novel moutationMissense mutationAlpha-galactosidase AHumansPoint MutationCornea verticillataGenetic Predisposition to DiseaseChildfabryGLA genec.846_847delTCGeneticsAlpha-galactosidasePoint mutationFabry disease; Alpha-galactosidase A; c.846_847delTC; p.E341X; p.C382X; p.R227P; c.639 + 5G > Tp.E341XGeneral MedicineMiddle Agedmedicine.diseaseMolecular biologyFabry diseaseStop codon030104 developmental biologyp.C382Xalpha-Galactosidasebiology.proteinFabry DiseaseFemalemedicine.symptom030217 neurology & neurosurgery
researchProduct