Search results for "Tumor necrosis factor-alpha"

showing 10 items of 504 documents

Clinical staging and serum cytokines in bipolar patients during euthymia

2017

Aims: Changes in serum cytokines and altered neutrophin concentration have been associated with bipolar disorder (BD). Our aim here was to analyze peripheral blood biomarkers according to the clinical stages of BD. Method: Euthymic BD-I patients were grouped according to their level of functioning in early-stage (n = 25) and late-stage (n = 23), and compared to healthy siblings (n = 23) and genetically unrelated healthy controls (n = 21). Neurotrophin (neurotrophin-3 and BDNF) concentration and biomarkers of inflammation, including cytokines (IL-6, IL-10 and TNF-alpha), leukocytes count and acute phase proteins, were measured. Results: IL-10 concentration was significantly increased in earl…

AdultMaleBipolar DisorderBipolar disorderInflammationNeurotrophinsYoung Adult03 medical and health sciences0302 clinical medicineNeurotrophin 3medicineLeukocytesHumansNerve Growth FactorsBipolar disorderBiological PsychiatryPharmacologyInflammationbiologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryBrain-Derived Neurotrophic FactorSiblingsAcute-phase proteinMiddle Agedmedicine.diseaseBlood Cell CountInterleukin-10030227 psychiatryPeripheralAffectSerum cytokineClinical stagingCase-Control StudiesImmunologybiology.proteinBiomarker (medicine)CytokinesFemaleTumor necrosis factor alphamedicine.symptombusinessBiomarkers030217 neurology & neurosurgeryAcute-Phase ProteinsNeurotrophin
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TNF-α in CRPS and 'normal' trauma--significant differences between tissue and serum.

2011

Posttraumatic TNF-alpha signaling may be one of the factors responsible for pain and hyperalgesia in complex regional pain syndromes (CRPS). In order to further specify the role of TNF-alpha we investigated tissue (skin) and serum concentrations in three different patient groups: patients with osteoarthritis and planned surgery, with acute traumatic upper limb bone fracture waiting for surgery, and with CRPS I. Thirty patients (10 in each group) were recruited. Mean CRPS duration was 36.1 ± 8.1 weeks (range 8- 90 weeks). Skin punch biopsies were taken at the beginning of the surgery in osteoarthritis and fracture patients and from the affected side in CRPS patients. Blood samples were taken…

AdultMaleBone pathologyEnzyme-Linked Immunosorbent AssayPilot ProjectsOsteoarthritisFractures BoneOsteoarthritismedicineHumansAgedSkinAged 80 and overmedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaBone fractureMiddle Agedmedicine.diseaseUp-RegulationAnesthesiology and Pain Medicinemedicine.anatomical_structureComplex regional pain syndromeNeurologyAnesthesiaSkin biopsyHyperalgesiaAcute DiseaseUpper limbWounds and InjuriesTumor necrosis factor alphaFemaleNeurology (clinical)medicine.symptombusinessComplex Regional Pain SyndromesPainReferences
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Elevated serum E-selectin in patients with liver metastases of colorectal cancer

1996

E-selectin, an endothelial cell adhesion molecule, mediates the initial step of leucocyte adhesion to activated vascular endothelium. The soluble isoform of E-selectin promotes angiogenesis in rat cornea. In the present study, we investigated whether leucocyte adhesion and angiogenesis are also involved in tumour progression and metastasis of colorectal cancer. Therefore, we determined the level of circulating soluble E-selectin in serum samples of 38 patients with colorectal cancer; 20 patients with non-metastatic and 18 patients with metastatic disease. Median levels of soluble E-selectin were found to be significantly higher in metastatic tumour disease (88.7 ng/ml, range 25-203 ng/ml) t…

AdultMaleCancer ResearchPathologymedicine.medical_specialtyAngiogenesisColorectal cancerFibrinogenMetastasisE-selectinmedicineCarcinomaHumansAgedbiologyTumor Necrosis Factor-alphaCell adhesion moleculebusiness.industryLiver NeoplasmsMiddle Agedmedicine.diseaseNeoplasm ProteinsC-Reactive ProteinSolubilityOncologyTumor progressionCancer researchbiology.proteinFemaleColorectal NeoplasmsE-Selectinbusinessmedicine.drugEuropean Journal of Cancer
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IL-10 and TNF-α polymorphisms and the recovery from HCV infection

2003

Hepatitis C virus (HCV) infection becomes chronic in about 85% of infected individuals, whereas only 15% of infected people clear spontaneously the virus. It is conceivable that the host immunogenetic background influences the course of infection in term of recovery. Thus, in this study we have evaluated the effect of functionally relevant polymorphisms at tumor necrosis factor-alpha (TNF-alpha, i.e., 2 biallelic polymorphisms at nt -863 and nt-308 of the promoter) and interleukin-10 (IL-10) loci (i.e., 1 biallelic polymorphism at nt -1082 of the promoter), on the clearance of HCV infection. To this purpose, we compared 18 Sicilian patients who had spontaneously recovered from previous HCV …

AdultMaleGenotypeHepatitis C virusmedicine.medical_treatmentImmunologyInfection groupBiologymedicine.disease_causePolymorphism Single NucleotideViruspolymorphismImmunoenzyme TechniquesGenotypemedicinecytokineImmunology and AllergyHumansIn patientTumor Necrosis Factor-alphaGeneral MedicineHepatitis C ChronicVirologyHepatitis CInterleukin-10Interleukin 10CytokineTNF-αImmunologyHCVIL-10Tumor necrosis factor alphaFemale
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The -308G/A polymorphism of TNF-alpha influences immunological parameters in old subjects affected by infectious diseases.

2005

Abnormal increments of pro-inflammatory cytokines (IL-6 and TNF-alpha) characterize the outbreak of infectious diseases, which are the major cause of death in the elderly. A counterbalance to the inflammation is exerted by IL-10 with an inhibitory role on TNF-alpha production. As is well known, some cytokine gene polymorphisms influence the cytokine production, playing a role as susceptibility or resistance factors against immune-mediated and infectious disease. Genetic variations in the -308A/G locus for TNF-alpha seems to affect the clinical outcome of some infectious diseases. In fact, the -308A allele is associated with severe septic shock and death. On this basis, we have screened heal…

AdultMaleGenotypemedicine.medical_treatmentImmunologyCommunicable DiseasesGene FrequencyGenotypeGeneticsmedicineBronchopneumoniaHumansAlleleInterleukin 6Molecular BiologyAllele frequencyGenetics (clinical)AgedPolymorphism GeneticbiologyInterleukin-6Tumor Necrosis Factor-alphaOutbreakGeneral MedicineMiddle AgedBronchitis ChronicKiller Cells NaturalZincCytokineInfectious disease (medical specialty)Immunologybiology.proteinTumor necrosis factor alphaFemaleMetallothioneinInternational journal of immunogenetics
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No detection of occult HBV-DNA in patients with various rheumatic diseases treated with anti-TNF agents: a two-year prospective study.

2013

OBJECTIVES: The widespread use of tumour necrosis factor (TNF)-targeted therapies in patients with rheumatic, digestive and dermatologic diseases has been associated with reports of reactivation of HBV replication and ensuing hepatitis flares both in asymptomatic HBsAg carriers and in subjects with occult HBV infection. The aim of our work was to investigate in a two-year prospective study the potential for HBV reactivation in patients with inflammatory joint diseases undergoing anti-TNF treatment from a southern Mediterranean area. METHODS: Fifty-seven consecutive outpatients attending the Academic Unit of Rheumatology at the University of Palermo (12 with rheumatoid arthritis, 17 with pso…

AdultMaleHepatitis B virusTime Factorsoccult HBV-DNAAntiviral AgentsPolymerase Chain ReactionRisk AssessmentRisk FactorsRheumatic Diseasesoccult HBV-DNA rheumatic diseases anti-TNFHumansProspective Studiesrheumatic diseaseAcademic Medical CentersHepatitis B Surface AntigensTumor Necrosis Factor-alphavirus diseasesanti-TNFMiddle AgedHepatitis Bdigestive system diseasesTreatment OutcomeItalyLamivudineAntirheumatic AgentsDNA ViralFemaleVirus ActivationBiomarkers
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Stress-induced pro- and anti-inflammatory cytokine concentrations in panic disorder patients

2018

Abstract Background An attenuated responsivity of the hypothalamus–hypophysis-adrenal (HPA) axis upon challenge and an increased risk for cardiac events are relatively consistent findings in panic disorder (PD) patients. Due to cytokine-HPA interactions, an altered HPA-axis responsivity may be accompanied by altered cytokine concentrations. Immunological reactions under stress might be considered the missing link for explaining an increased cardiac risk. This study analyzed stress-induced cytokine levels in PD patients. Methods A total of n = 32 PD patients and n = 32 healthy control individuals performed the Trier Social Test (TSST). Blood sample collection accompanied the TSST for the col…

AdultMaleHypothalamo-Hypophyseal Systemmedicine.medical_specialtyHydrocortisonemedicine.drug_classEndocrinology Diabetes and Metabolismmedicine.medical_treatmentPituitary-Adrenal SystemDiseaseAnti-inflammatory03 medical and health sciences0302 clinical medicineEndocrinologyInternal medicinemedicineHumansSalivaBiological PsychiatryPsychiatric Status Rating ScalesInterleukin-6Tumor Necrosis Factor-alphaEndocrine and Autonomic Systemsbusiness.industryPanic disorderPanicRepeated measures designMiddle Agedmedicine.diseaseInterleukin-10030227 psychiatryPsychiatry and Mental healthDistressEndocrinologyCytokineCase-Control StudiesCytokinesPanic DisorderFemaleSample collectionmedicine.symptombusinessStress Psychological030217 neurology & neurosurgeryPsychoneuroendocrinology
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Differential association of polymorphisms in the TNFalpha region with psoriatic arthritis but not psoriasis.

2002

To investigate the potential association of tumour necrosis factor alpha (TNFalpha) microsatellite and promoter alleles with psoriatic arthritis (PsA).DNA from 89 white patients with PsA, 65 patients with psoriasis, and 99 healthy white controls was investigated for two TNFalpha promoter (-238 and -308) and three microsatellite polymorphisms (TNFa, c, and d). Patients had previously been studied by serology for HLA class I antigens and by sequence-specific polymerase chain reaction for DRB1* alleles. In addition, TNFalpha production of Ficoll separated peripheral blood mononuclear cells (PBMC) into culture supernatants after stimulation with lipopolysaccharide, alphaCD3 antibodies, phytohae…

AdultMaleImmunologyArthritisEnzyme-Linked Immunosorbent AssayHuman leukocyte antigenurologic and male genital diseasesPeripheral blood mononuclear cellPolymerase Chain ReactionGeneral Biochemistry Genetics and Molecular BiologyStatistics NonparametricPsoriatic arthritisRheumatologyPsoriasismedicineOdds RatioImmunology and AllergyHumansPsoriasisPromoter Regions GeneticAllelesCells CulturedPhytohaemagglutininAgedAged 80 and overChi-Square DistributionPolymorphism Geneticbiologybusiness.industryTumor Necrosis Factor-alphaHaplotypeArthritis PsoriaticMiddle Agedmedicine.diseaseExtended ReportCase-Control StudiesImmunologybiology.proteinLeukocytes MononuclearFemaleAntibodybusinessMicrosatellite RepeatsAnnals of the rheumatic diseases
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Enhanced inflammatory and T-helper-1 type responses but suppressed lymphocyte proliferation in patients with seasonal affective disorder and treated …

2015

Abstract Background Animals show seasonal changes in the endocrine and immune system in response to winter stressors. Even though increased inflammation has been implicated in the pathophysiology of depression, whether immune disorder is a key mediator in seasonal affective depression (SAD) is unknown. Here, we hypothesized that short photoperiods in winter may induce inflammatory response, which contributes to SAD, and that light treatments should normalize immune function and improve depressive symptoms. Methods Twenty patients with a diagnosis of SAD, and a score on the HAM-29 of 20 or higher were recruited for this study. Twenty-one healthy subjects with no personal and family history o…

AdultMaleLight therapymedicine.medical_specialtymedicine.medical_treatmentLymphocyteLymphocyte proliferationProinflammatory cytokineInterferon-gammaInternal medicinemedicineAnimalsHumansLymphocytesInflammationAnalysis of VarianceTumor Necrosis Factor-alphaSeasonal Affective DisorderPhototherapymedicine.diseasePsychiatry and Mental healthClinical Psychologymedicine.anatomical_structureCytokineEndocrinologyImmunologyCytokinesMajor depressive disorderFemaleImmune disorderPsychologyHypothalamic–pituitary–adrenal axisInterleukin-1Journal of Affective Disorders
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Relevance of gamma interferon, tumor necrosis factor alpha, and interleukin-10 gene polymorphisms to susceptibility to Mediterranean spotted fever.

2009

Several studies have demonstrated that cellular immunity plays a critical role in the protective immune response against Rickettsia conorii. Immune CD4+ and CD8+ T cells are both involved in the control of rickettsial infection (38). Perivascular infiltrated CD4+ and CD8+ T lymphocytes, macrophages, and natural killer cells produce chemokines and cytokines that activate endothelial rickettsicidal activities. Infected human cells, including endothelial cells, hepatocytes, and macrophages, activated by gamma interferon (IFN-γ), tumor necrosis factor alpha (TNF-α), and interleukin-1β (IL-1β), kill intracellular rickettsiae by one or a combination of three mechanisms, involving nitric oxide syn…

AdultMaleMicrobiology (medical)Meningitidesgene polymorphismgene polymorphisms; mediterranean spotted fevermedicine.medical_treatmentClinical BiochemistryImmunologyBiologyBoutonneuse FeverPolymorphism Single NucleotideInterferon-gammaImmune systemInterferonmedicineHumansImmunology and AllergyCytotoxic T cellSicilyTumor Necrosis Factor-alphaMiddle AgedInterleukin-10mediterranean spotted feverInterleukin 10CytokineImmunologyFemaleTumor necrosis factor alphaDisease SusceptibilityMicrobial ImmunologyCD8medicine.drug
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