Search results for "URSODEOXYCHOLIC ACID"

showing 10 items of 22 documents

Reply to Dr Michaudet al.

2015

There is no abstract

Cholagogues and Cholereticsbusiness.industryEndoplasmic reticulumAmyotrophic Lateral SclerosisTaurochenodeoxycholic acidTauroursodeoxycholic acidMitochondrionPharmacologyEndoplasmic Reticulummedicine.diseaseTaurochenodeoxycholic Acidchemistry.chemical_compoundNeurologychemistryImmunologyUnfolded Protein ResponsemedicineUnfolded protein responseAnimalsHumansTUDCA ALSSettore MED/26 - NeurologiaNeurology (clinical)Amyotrophic lateral sclerosisbusinessEuropean Journal of Neurology
researchProduct

Tauroursodeoxycholic acid for the treatment of chronic hepatitis: a multicentre dose-response study

1998

Ursodeoxycholic acid (UDCA) improves the biochemical expression of chronic liver disease. Tauroursodeoxycholic acid (TUDCA) was recently shown to have more favourable metabolic properties. We designed a multicenter, randomized, controlled study, aimed at assessing the efficacy of TUDCA for the treatment of chronic hepatitis. One hundred and fifty-five patients with chronic hepatitis were randomly assigned to receive TUDCA at the daily doses of 250, 500 and 1000 mg, or no treatment for 6 months. Aminotransferase and gamma-glutamyl transpeptidase (GGT) serum levels decreased with each dose of TUDCA compared with controls (P<0.001). The 1000 mg dose was followed by more marked improvement comp…

Hepatitismedicine.medical_specialtyChemotherapyHepatologybusiness.industrymedicine.medical_treatmentTauroursodeoxycholic acidChronic liver diseasemedicine.diseaseGastroenterologyUrsodeoxycholic acidchemistry.chemical_compoundLiver diseaseDose–response relationshipInfectious DiseasesEndocrinologychemistryInternal medicineMedicineLiver functionbusinessmedicine.drugHepatology Research
researchProduct

Ursodeoxycholic acid protects against secondary biliary cirrhosis in rats by preventing mitochondrial oxidative stress

2004

Ursodeoxycholic acid (UDCA) improves clinical and biochemical indices in primary biliary cirrhosis and prolongs survival free of liver transplantation. Recently, it was suggested that the cytoprotective mechanisms of UDCA may be mediated by protection against oxidative stress, which is involved in the development of cirrhosis induced by chronic cholestasis. The aims of the current study were 1) to identify the mechanisms involved in glutathione depletion, oxidative stress, and mitochondrial impairment during biliary cirrhosis induced by chronic cholestasis in rats; and 2) to determine the mechanisms associated with the protective effects of UDCA against secondary biliary cirrhosis. The find…

Malemedicine.medical_specialtyCirrhosisCardiolipinsGlutamate-Cysteine LigaseBiliary cirrhosisPopulationBiologymedicine.disease_causeMembrane Potentialschemistry.chemical_compoundPrimary biliary cirrhosisInternal medicinemedicineCardiolipinAnimalsRats Wistareducationeducation.field_of_studyCholestasisHepatologyLiver Cirrhosis BiliaryUrsodeoxycholic AcidCystathionine gamma-LyaseGlutathionemedicine.diseaseGlutathioneUrsodeoxycholic acidMitochondriaPeroxidesRatsOxidative StressEndocrinologyLiverchemistryChronic DiseaseHepatocytesOxidation-ReductionOxidative stressmedicine.drugHepatology
researchProduct

Obeticholic Acid and Fibrates in Primary Biliary Cholangitis: Comparative Effects in a Multicentric Observational Study

2021

INTRODUCTION: Obeticholic acid (OCA) and fibrates therapy results in biochemical improvement in placebo-controlled trials in patients with primary biliary cholangitis and insufficient response to ursodeoxycholic acid. There is scarce information outside of clinical trials. Therefore, we have assessed the effectiveness and adverse events of these treatments. METHODS: Data from patients included in the ColHai registry treated with OCA, fibrates, or both were recorded during a year, as well as adverse events and treatment discontinuation. RESULTS: Eighty-six patients were treated with OCA, 250 with fibrates (81% bezafibrate; 19% fenofibrate), and 15 with OCA plus fibrates. OCA group had baseli…

Malemedicine.medical_specialtyPROGNOSISChenodeoxycholic AcidPLACEBO-CONTROLLED TRIALGastroenterology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineFenofibrateInternal medicinemedicineHumansBIOCHEMICAL RESPONSEAdverse effectCIRRHOSISRetrospective StudiesFenofibrateBezafibrateHepatologybusiness.industryLiver Cirrhosis BiliaryGastroenterologyObeticholic acidMiddle AgedBEZAFIBRATEUrsodeoxycholic acideye diseasesDiscontinuationURSODEOXYCHOLIC ACIDClinical trialTreatment Outcomechemistry030220 oncology & carcinogenesisAlkaline phosphatase030211 gastroenterology & hepatologyFemaleBezafibratebusinessmedicine.drug
researchProduct

Effect of different doses of ursodeoxycholic acid in chronic liver disease

1989

Recent clinical studies have indicated that ursodeoxycholic acid (ursodiol), administered at dosages ranging between 10 and 15 mg/kg/day, improves liver function indices in both cholestatic and inflammatory chronic liver diseases. These dosages would be considered high for the use of ursodiol in gallstone dissolution therapy. To investigate the dose-response relationship to ursodiol administration, we planned a few studies in patients with primary biliary cirrhosis (PBC), primary sclerosing cholangitis (PSC), and chronic hepatitis (CH). Patients with PBC were subdivided into two groups on the basis of their serum bilirubin values, with 2 mg/dl as the dividing line. Ursodiol was given at dos…

Malemedicine.medical_specialtyTime FactorsPhysiologyBilirubinCholangitis SclerosingChronic liver diseaseGastroenterologyPrimary sclerosing cholangitisRandom Allocationchemistry.chemical_compoundPrimary biliary cirrhosisLiver Function TestsInternal medicinemedicineBileHumansHepatitis ChronicHepatitisDose-Response Relationship Drugmedicine.diagnostic_testLiver Cirrhosis Biliarybusiness.industryUrsodeoxycholic AcidGastroenterologyBilirubinMiddle AgedLipid Metabolismmedicine.diseaseUrsodeoxycholic acidchemistryFemaleLiver functionLiver function testsbusinessDeoxycholic Acidmedicine.drugDigestive Diseases and Sciences
researchProduct

Primary biliary cirrhosis and hereditary hemorrhagic telangiectasia: When two rare diseases coexist

2013

Primary biliary cirrhosis is a slowly progressive cholestatic autoimmune liver disease that mainly affects middle- aged women with an estimated prevalence ranging from 6.7 to 402 cases per million. Hereditary hemorrhagic telangiectasia, or Rendu-Osler-Weber disease, is an autosomal dominant disorder characterized by angiodysplastic lesions (telangiectases and arteriovenous malformations) that can affect many organs, including liver, with a prevalence of 1-2 cases per 10000. We describe the coexistence, for the first time to our knowledge, of these two rare diseases in a 50-year old Caucasian woman. In this setting, the relevance of an accurate medical history, the role of liver histology an…

Pathologymedicine.medical_specialtySettore MED/12 - GastroenterologiaPrimary biliary cirrhosiHepatologybusiness.industryFocal nodular hyperplasiaCase ReportDiseaseTelangiectasesImmunostainingSettore MED/08 - Anatomia Patologicamedicine.diseaseUrsodeoxycholic acidPrimary biliary cirrhosisHereditary hemorragic telangiectasiaUrsodeoxycholic acidFocal nodular hyperplasiamedicineMedical historymedicine.symptomAutoimmune liver diseasebusinessTelangiectasiamedicine.drug
researchProduct

Effect of tauroursodeoxycholic acid on bile acid-induced apoptosis in primary human hepatocytes

2000

Background/aims The accumulation of endogenous bile acids contributes to hepatocellular damage during cholestatic liver disease. To evaluate the potential role of apoptotic cell death due to increased concentrations of bile acids, primary human hepatocytes were treated with hydrophobic and hydrophilic bile acids. Because the Fas receptor–ligand system may mediate apoptosis in human liver cells, the effect of toxic bile acids on hepatocellular Fas receptor expression was evaluated. Materials and methods Primary human hepatocytes were incubated with 50 and 100 μM glycochenodeoxycholic acid (GCDCA) and co-incubated with equimolar concentrations of tauroursodeoxycholic acid (TUDCA). To evaluate…

medicine.medical_specialtyBile acidmedicine.drug_classClinical BiochemistryApoptotic DNA fragmentationTaurochenodeoxycholic acidTauroursodeoxycholic acidGeneral MedicineBiologyFas receptorBiochemistryMolecular biologyUrsodeoxycholic acidchemistry.chemical_compoundEndocrinologychemistryApoptosisInternal medicinemedicineGlycochenodeoxycholic acidmedicine.drugEuropean Journal of Clinical Investigation
researchProduct

Primary Biliary Cholangitis: advances in management and treatment of the disease

2017

Primary Biliary Cholangitis, previously known as Primary Biliary Cirrhosis, is a rare disease, which mainly affects women in their fifth to seventh decades of life. It is a chronic autoimmune disease characterized by a progressive damage of interlobular bile ducts leading to ductopenia, chronic cholestasis and bile acids retention. Even if the disease usually presents a long asymptomatic phase and a slow progression, in many patients it may progress faster toward cirrhosis and its complications. The 10Â year mortality is greater than in diseases such as human immunodeficiency virus/Hepatitis C Virus coinfection and breast cancer. Ursodeoxycholic acid is the only treatment available today, b…

medicine.medical_specialtyCholagogues and CholereticsCirrhosisPrimary Biliary CholangitisCholangitisDiseaseChenodeoxycholic AcidGastroenterologyEnd Stage Liver Disease03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePrimary biliary cirrhosisDuctopeniaAlkaline phosphatase; Budesonide; Fibrates; Obeticholic acid; Ursodeoxycholic acid; Hepatology; GastroenterologyMED/12 - GASTROENTEROLOGIAInternal medicineAlkaline phosphatasemedicineHumansFibrateSettore SECS-P/01 - Economia PoliticaBudesonideCholestasisHepatologyAlkaline phosphatase; Budesonide; Fibrates; Obeticholic acid; Ursodeoxycholic acidbusiness.industryGastroenterologyObeticholic acidHepatologymedicine.diseaseUrsodeoxycholic acidchemistryUrsodeoxycholic acid030220 oncology & carcinogenesisObeticholic acidDisease Progression030211 gastroenterology & hepatologyDrug Therapy CombinationbusinessFibratesbiologicalRare diseasemedicine.drug
researchProduct

Multicentre randomized placebo-controlled trial of ursodeoxycholic acid with or without colchicine in symptomatic primary biliary cirrhosis

2000

Aim: To establish the efficacy of combination therapy with ursodeoxycholic acid (UDCA) and colchicine in patients with symptomatic primary biliary cirrhosis (PBC), defined by the presence of liver cirrhosis, pruritus or bilirubin exceeding 2 mg/mL. Methods: A total of 90 patients were randomly assigned to ursodeoxycholic acid 500 mg/daily plus placebo (UDCA group, n=44), or ursodeoxycholic acid at the same dosage plus colchicine, 1 mg/daily (UDCA/C group, n=46). The two groups were comparable for age, sex, stage of disease, severity of pruritus, bilirubin, and Mayo score. All patients underwent clinical, ultrasonographic, and biochemical examinations at entry and then every 6 months up to 3…

medicine.medical_specialtyCirrhosisHepatologymedicine.diagnostic_testbusiness.industryBiliary cirrhosisGastroenterologyPlacebo-controlled studymedicine.diseasePlaceboGastroenterologyUrsodeoxycholic acidPrimary biliary cirrhosisCholestasisLiver biopsyInternal medicinemedicinePharmacology (medical)businessmedicine.drugAlimentary Pharmacology &amp; Therapeutics
researchProduct

Primary Biliary Cholangitis management: controversies, perspectives, and daily practice implications from an expert panel

2020

Primary biliary cholangitis (PBC) is a rare progressive immune-mediated liver disease that, if not adequately treated, may culminate in end-stage disease and need for transplantation. According to current guidelines, PBC is diagnosed in the presence of antimitochondrial antibodies (AMA) or specific antinuclear antibodies, and of a cholestatic biochemical profile, while biopsy is recommended only in selected cases. All patients receive ursodeoxycholic acid (UDCA) in first line; the only registered second-line therapy is obeticholic acid (OCA) for UDCA-inadequate responders. Despite the recent advances in understanding PBC pathogenesis and developing new treatments, many grey areas remain. Si…

medicine.medical_specialtyPopulationReceptors Cytoplasmic and NuclearDiseaserisk stratificationfibrates; liver biopsy; management; obeticholic acid; primary biliary cholangitis; risk stratificationprimary biliary cholangitiBile Acids and Salts03 medical and health scienceschemistry.chemical_compoundLiver disease0302 clinical medicineobeticholic acidBiopsymedicineHumansIntensive care medicineeducationliver biopsyeducation.field_of_studyCholestasisfibrateHepatologymedicine.diagnostic_testbusiness.industryprimary biliary cholangitisLiver Cirrhosis BiliaryUrsodeoxycholic AcidObeticholic acidmedicine.diseaseUrsodeoxycholic acidBile Acids and SaltTransplantationchemistryCholestasi030220 oncology & carcinogenesisLiver biopsy030211 gastroenterology & hepatologyfibratesbusinessmanagementmedicine.drugHuman
researchProduct