Search results for "Umbilical Vein"

showing 10 items of 194 documents

Establishment of a quantitative RT-pCR for detection of vascular cell adhesion molecule-1 transcripts in endothelial cells after stimulation with adv…

1998

Advanced glycation endproducts (AGE) are supposed to increase endothelial expression of adhesion molecules like vascular cell adhesion molecule-1 (VCAM-1) by inducing an intracellular stress with subsequent activation of nuclear transcription factor NF-kappa-B. Quantitative analysis of VCAM-1-transcription has not been demonstrated concerning this topic. Thus, the aim of this study was to establish quantitative reverse transcription polymerase chain reaction (RT-PCR) assays using a spacer gene in order to measure the amounts of specific mRNA for VCAM-1 in human umbilical vein endothelial cells (HUVEC) which were stimulated with AGE-albumin (AGE-BSA). A recombinant RNA-standard was synthesiz…

Glycation End Products AdvancedCell adhesion moleculeReverse Transcriptase Polymerase Chain ReactionCellEndothelial CellsReproducibility of ResultsVascular Cell Adhesion Molecule-1Serum Albumin BovineGeneral MedicineCell cycleBiologyUmbilical veinCell biologyReverse transcription polymerase chain reactionReal-time polymerase chain reactionmedicine.anatomical_structureGeneticsmedicineHumansEndothelium VascularRNA MessengerCell adhesionIntracellularCells CulturedInternational journal of molecular medicine
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Cellular and tissue expression of DAPIT, a phylogenetically conserved peptide

2011

DAPIT (Diabetes Associated Protein in Insulin-sensitive Tissues) is a small, phylogenetically conserved, 58 amino acid peptide that was previously shown to be down-regulated at mRNA level in insulin-sensitive tissues of type 1 diabetes rats. In this study we characterize a custom made antibody against DAPIT and confirm the mitochondrial presence of DAPIT on cellular level. We also show that DAPIT is localized in lysosomes of HUVEC and HEK 293T cells. In addition, we describe the histological expression of DAPIT in several tissues of rat and man and show that it is highly expressed especially in cells with high aerobic metabolism and epithelial cells related to active transport of nutrients …

HistologyCellular respirationProtein subunitBiophysicsPeptideV-ATPaseBiologyMitochondrionAntibodiesMitochondrial ProteinsHuman Umbilical Vein Endothelial CellsV-ATPaseAnimalsHumansmitochondrionta315lcsh:QH301-705.5PhylogenyDAPIT mitochondrion V-ATPase type 1 diabeteschemistry.chemical_classificationRegulation of gene expressionOriginal Papertype 1 diabetes.HEK 293 cellsMembrane ProteinsCell BiologyProton PumpsCell biologyMitochondriaRatsHEK293 CellsMembrane proteinchemistryBiochemistryGene Expression Regulationlcsh:Biology (General)Organ SpecificityLysosomesDAPITEuropean Journal of Histochemistry
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Tropism of human cytomegalovirus for endothelial cells is determined by a post-entry step dependent on efficient translocation to the nucleus.

2000

Marked interstrain differences in the endothelial cell (EC) tropism of human cytomegalovirus (HCMV) isolates have been described. This study aimed to define the step during the replicative cycle of HCMV that determines this phenotype. The infection efficiency of various HCMV strains in EC versus fibroblasts was quantified by immunodetection of immediate early (IE), early and late viral antigens. Adsorption and penetration were analysed by radiolabelled virus binding assays and competitive HCMV-DNA-PCR. The translocation of penetrated viral DNA to the nucleus of infected cells was quantified by competitive HCMV-DNA-PCR in pure nuclear fractions. The intracytoplasmic translocation of capsids …

Human cytomegalovirusUmbilical VeinsvirusesBlotting WesternActive Transport Cell NucleusCytomegalovirusChromosomal translocationBiologyAntibodies ViralTransfectionVirus ReplicationVirusImmediate-Early ProteinsViral ProteinsViral Envelope ProteinsViral entryVirologyGene expressionmedicineHumansEndotheliumPromoter Regions GeneticAntigens ViralGenes Immediate-EarlyTropismCells CulturedCell NucleusMembrane GlycoproteinsAntibodies MonoclonalGenetic VariationFibroblastsmedicine.diseaseVirologyMolecular biologyCell nucleusMicroscopy Electronmedicine.anatomical_structureOrgan SpecificityDNA ViralTrans-ActivatorsAdsorptionImmunostainingThe Journal of general virology
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Comparative Studies on Vascular Endothelium in vitro

1994

Recent studies have presented evidence that the processes of hypoxaemia and reperfusion are involved in several pathogenetic mechanisms of atherosclerotic lesions. The ability of hypoxaemia to activate circulating white blood cells (WBCs) and enhance WBC-endothelial cell (EC) interactions is suspected to be a major factor in deleterious processes in the blood vessel wall. Various groups have suggested that cell adhesion molecules (CAMs), such as ICAM-1, VCAM-1 and E-selectin and their leukocyte ligands are involved in intercellular activities of the relevant cell types. We studied the effects of different oxygen tensions, simulating normoxic conditions, hypoxia and hyperoxia in vitro with t…

HyperoxiaCell adhesion moleculemedicine.medical_treatmentStimulationCell BiologyGeneral MedicineBiologyPharmacologyHypoxia (medical)Umbilical veinPathology and Forensic MedicineOxygen tensionCell biologyCytokinemedicine.anatomical_structuremedicinemedicine.symptomMolecular BiologyBlood vesselPathobiology
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Comparative Studies on Vascular Endothelium in vitro

1995

Endothelial cells (EC) are very responsive to proinflammatory cytokines, e.g. interleukin-1 (IL-1) and tumor necrosis factor alpha (TNF alpha), as well as to bacterial lipopolysaccharide. EC are stimulated by these substances to secrete chemotactic factors and to increase expression of cell adhesion molecules (CAM), leading to dramatically altered interactions with leukocytes, e.g. granulocytes and monocytes. In these interactions E-selectin, ICAM-1 and VCAM-1 are known to play an important role, as they are presented by the EC and interact with corresponding ligands on the white blood cell membranes. These adhesion molecules have been studied worldwide in a variety of in vitro experiments …

ICAM-1biologyCell adhesion moleculeIntercellular Adhesion Molecule-1Cell BiologyGeneral MedicineMolecular biologyUmbilical veinIn vitroPathology and Forensic MedicineProinflammatory cytokinechemistry.chemical_compoundchemistryE-selectinImmunologybiology.proteinVCAM-1Molecular BiologyPathobiology
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Role of Neutral Amino Acid Transport and Protein Breakdown for Substrate Supply of Nitric Oxide Synthase in Human Endothelial Cells

2003

Endothelial dysfunction is often associated with a relative substrate deficiency of the endothelial nitric oxide synthase (eNOS) in spite of apparently high intracellular arginine concentrations. For a better understanding of the underlying pathophysiological mechanisms, we aimed to characterize the intracellular arginine sources of eNOS. Our previous studies in human endothelial EA.hy926 cells suggested the existence of two arginine pools: pool I can be depleted by extracellular lysine, whereas pool II is not freely exchangeable with the extracellular space, but accessible to eNOS. In this study, we demonstrate that the eNOS accessible pool II is also present in human umbilical vein endoth…

Intracellular FluidUmbilical VeinsNitric Oxide Synthase Type IIIArginineEndotheliumPhysiologyGlutamineArginineTransfectionSubstrate Specificitychemistry.chemical_compoundEnosNeutral amino acid transportCitrullinemedicineAnimalsHumansAmino AcidsCells CulturedbiologyCarcinomaMembrane Transport ProteinsProteinsNitric Oxide Synthase Type IIIBiological Transportbiology.organism_classificationRatsEndothelial stem cellNitric oxide synthaseAmino Acid Transport Systems NeutralAmino Acids Neutralmedicine.anatomical_structureUrinary Bladder NeoplasmsBiochemistrychemistrybiology.proteinCitrullineEndothelium VascularNitric Oxide SynthaseCardiology and Cardiovascular MedicineCirculation Research
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In vitro and in vivo characterization of porcine acellular dermal matrix for gingival augmentation procedures

2013

Recently, porcine acellular dermal matrix (PADM) has been proposed as a possible alternative to autogenous grafts in periodontal plastic surgery. The aim of the present study was to investigate the in vitro responses of four different oral cell lines cultured on a novel PADM. Furthermore, tissue reaction to PADM was evaluated histologically after subcutaneous implantation in mice.Human gingival fibroblasts (HGF), human osteoblast-like cells, human umbilical vein endothelial cells and human oral keratinocytes (HOK) were cultured and transferred on to the PADM. A tissue culture polystyrene surface served as the control. The viability of all tested cell lines on PADM was measured by using the …

Keratinocytesmedicine.medical_specialtyTime FactorsCell SurvivalCell TransplantationSwineCell Culture TechniquesGingivaMice NudeTetrazolium SaltsAdenylate kinaseUmbilical veinCell LineAndrologyMiceSubcutaneous TissueIn vivoHuman Umbilical Vein Endothelial CellsmedicineAnimalsHumansCytotoxic T cellAcellular DermisColoring AgentsGingivoplastyOsteoblastsTissue EngineeringTissue ScaffoldsAugmentation procedureChemistryAdenylate KinaseSoft tissueFibroblastsIn vitroSurgeryThiazolesCell cultureGuided Tissue Regeneration PeriodontalPeriodonticsColorimetryFemaleIndicators and ReagentsJournal of Periodontal Research
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Kininogen binding protein p33/gC1qR is localized in the vesicular fraction of endothelial cells

1996

AbstractThe endothelial protein p33/gC1qR is thought to mediate the assembly of components of the kinin-forming and complement-activating pathways on the surface of cardiovascular cells. FACS analysis of intact human umbilical vein endothelial cells using specific antibodies to p33 revealed a minor fluorescence on the cell surface whereas permeabilized cells showed a bright fluorescence indicative of an intracellular localization of p33. Immunostaining of fixed cells confirmed the predominant intracellular localization of p33. Fractionation studies demonstrated that the vesicular but not the membrane fraction of EA.hy926 cells is rich in p33. We conclude that externalization of p33 must pre…

Kininogen bindingp33Kininogen binding proteinCellBiophysicsComplementFluorescent Antibody TechniqueBiologyBiochemistryUmbilical veinMitochondrial ProteinsStructural BiologyGeneticsmedicineHumansMolecular BiologyCells CulturedMembrane GlycoproteinsImmune SeraCell BiologyKininFlow CytometryKininFluorescenceReceptors ComplementCell biologyEndothelial stem cellSpecific antibodyHyaluronan Receptorsmedicine.anatomical_structuregC1qREndothelium VascularCarrier ProteinsImmunostainingFEBS Letters
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Human endothelial cell-based assay for endotoxin as sensitive as the conventional Limulus Amebocyte Lysate assay

2014

AbstractEndotoxin, also known as lipopolysaccharide (LPS) produced by bacteria can be present in any liquid or on any biomaterial even if the material is sterile. Endotoxin in mammals can cause fever, inflammation, cell and tissue damage and irreversible septic shock and death. In the body, endothelial cells making up the blood vasculature and endothelial cells in vitro rapidly react to minute amounts of endotoxin resulting in a rapid induction of the cell adhesion molecule E-selectin. In this study we have used immunofluorescent staining to evaluate the expression of E-selectin on human microvascular endothelial cells from the skin (HDMEC) and human umbilical vein endothelial cells (HUVEC)…

LipopolysaccharideCellBiophysicsLipopolysaccharideBioengineeringBiologyUmbilical veinEndothelialMicrobiologyBiomaterialschemistry.chemical_compoundEndotoxinLimit of DetectionHorseshoe CrabsmedicineAnimalsHumansCell adhesionCells CulturedCell adhesion moleculeIn vitroEndotoxinsEndothelial stem cellmedicine.anatomical_structurechemistryMechanics of MaterialsLimulus amebocyte lysateCeramics and CompositesLimulus amebocyte assayEndothelium VascularBiomaterials
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Conversion of biliverdin to bilirubin by biliverdin reductase contributes to endothelial cell protection by heme oxygenase-1—evidence for direct and …

2009

Heme oxygenase-1 (HO-1) is highly protective in various pathophysiological states such as cardiovascular and neurodegenerative diseases. HO-1-derived bilirubin is an efficient scavenger of reactive oxygen and nitrogen species (RONS). It remains to determine whether conversion of biliverdin to bilirubin is an essential step for HO-1-conferred protection of endothelial cells. RONS scavenging activities of biliverdin versus bilirubin were assessed by different RONS generating systems and detection techniques. We also silenced the biliverdin reductase (BVR) or HO-1 gene in cultured primary human endothelial cells (HUVECs) and measured the effect on RONS formation upon stimulation with lipopolys…

LipopolysaccharidesOxidoreductases Acting on CH-CH Group DonorsUmbilical VeinsXanthine OxidaseNeutrophilsBilirubinNitrosationModels BiologicalAntioxidantschemistry.chemical_compoundPeroxynitrous AcidLeukocytespolycyclic compoundsHumansGene SilencingMolecular BiologyHemeReactive nitrogen speciesRespiratory BurstBiliverdinAngiotensin IIBiliverdineBiliverdin reductaseEndothelial CellsBilirubinFree Radical ScavengersAngiotensin IIMitochondriaEndothelial stem cellHeme oxygenasechemistryBiochemistryCytoprotectionGene Knockdown TechniquesTyrosineReactive Oxygen SpeciesCardiology and Cardiovascular MedicineHeme Oxygenase-1Journal of Molecular and Cellular Cardiology
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