Search results for "Unity"

showing 10 items of 3852 documents

Immune regulatory neural stem/precursor cells protect from central nervous system autoimmunity by restraining dendritic cell function.

2009

Background: The systemic injection of neural stem/precursor cells (NPCs) provides remarkable amelioration of the clinicopathological features of experimental autoimmune encephalomyelitis (EAE). This is dependent on the capacity of transplanted NPCs to engage concurrent mechanisms of action within specific microenvironments in vivo. Among a wide range of therapeutic actions alternative to cell replacement, neuroprotective and immune modulatory capacities of transplanted NPCs have been described. However, lacking is a detailed understanding of the mechanisms by which NPCs exert their therapeutic plasticity. This study was designed to identify the first candidate that exemplifies and sustains …

Central Nervous SystemEncephalomyelitis Autoimmune ExperimentalCell Transplantationmedicine.medical_treatmentScienceAutoimmunityNeurological Disorders/Multiple Sclerosis and Related DisordersBiologyMiceImmune systemPrecursor cellmedicineotorhinolaryngologic diseasesAnimalsLymph nodeInflammationNeuronsMultidisciplinaryStem CellsExperimental autoimmune encephalomyelitisMesenchymal stem cellQRStem-cell therapyDendritic cellDendritic Cellsmedicine.diseaseCell biologyDevelopmental Biology/Stem CellsMicroscopy Electronstomatognathic diseasesmedicine.anatomical_structureImmune SystemImmunologyBone Morphogenetic ProteinsMedicineFemaleLymph NodesStem cellNeuroscience/Neurobiology of Disease and RegenerationResearch ArticlePLoS ONE
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OTUB1 inhibits CNS autoimmunity by preventing IFN-γ-induced hyperactivation of astrocytes.

2019

Astrocytes are critical regulators of neuroinflammation in multiple sclerosis (MS) and its animal model experimental autoimmune encephalomyelitis (EAE). Growing evidence indicates that ubiquitination of signaling molecules is an important cell‐intrinsic mechanism governing astrocyte function during MS and EAE. Here, we identified an upregulation of the deubiquitinase OTU domain, ubiquitin aldehyde binding 1 (OTUB1) in astrocytes during MS and EAE. Mice with astrocyte‐specific OTUB1 ablation developed more severe EAE due to increased leukocyte accumulation, proinflammatory gene transcription, and demyelination in the spinal cord as compared to control mice. OTUB1‐deficient astrocytes were hy…

Central Nervous SystemEncephalomyelitis Autoimmune ExperimentalNeuroimmunomodulationmedicine.medical_treatmentexperimental autoimmune encephalomyelitisAutoimmunityBiologymultiple sclerosisubiquitinationGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineastrocytemedicineAnimalsMolecular BiologyCells CulturedNeuroinflammation030304 developmental biologyMice Knockout0303 health sciencesGeneral Immunology and MicrobiologySuppressor of cytokine signaling 1General NeuroscienceExperimental autoimmune encephalomyelitisArticlesmedicine.disease3. Good healthCell biologyMice Inbred C57BLCysteine EndopeptidasesCytokinemedicine.anatomical_structureAnimals NewbornAstrocytesSTAT proteinOTUB1FemaleNeurogenic InflammationJanus kinase030217 neurology & neurosurgeryAstrocyte
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Innate and adaptive immune responses in the CNS.

2015

Almost every disorder of the CNS is said to have an inflammatory component, but the precise nature of inflammation in the CNS is often imprecisely defined, and the role of CNS-resident cells is uncertain compared with that of cells that invade the tissue from the systemic immune compartment. To understand inflammation in the CNS, the term must be better defined, and the response of tissue to disturbances in homoeostasis (eg, neurodegenerative processes) should be distinguished from disorders in which aberrant immune responses lead to CNS dysfunction and tissue destruction (eg, autoimmunity). Whether the inflammatory tissue response to injury is reparative or degenerative seems to be depende…

Central Nervous SystemInnate immunologyAutoimmunityInflammationContext (language use)610 Medicine & healthAdaptive ImmunityBiologymedicine.disease_cause10263 Institute of Experimental ImmunologyAutoimmunity03 medical and health sciences0302 clinical medicineImmune systemCentral Nervous System DiseasesResponse to injuryImmunitymedicineAnimalsHumans030304 developmental biology0303 health sciencesImmunity Innate2728 Neurology (clinical)Immunology570 Life sciences; biologyNeurology (clinical)medicine.symptomNeuroscience030217 neurology & neurosurgeryHomeostasisThe Lancet. Neurology
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Host microbiota constantly control maturation and function of microglia in the CNS.

2015

As the tissue macrophages of the CNS, microglia are critically involved in diseases of the CNS. However, it remains unknown what controls their maturation and activation under homeostatic conditions. We observed substantial contributions of the host microbiota to microglia homeostasis, as germ-free (GF) mice displayed global defects in microglia with altered cell proportions and an immature phenotype, leading to impaired innate immune responses. Temporal eradication of host microbiota severely changed microglia properties. Limited microbiota complexity also resulted in defective microglia. In contrast, recolonization with a complex microbiota partially restored microglia features. We determ…

Central Nervous SystemMaleCellGut–brain axis610 Medicine & healthBiologydigestive systemReceptors G-Protein-CoupledMiceImmunitymedicineAnimalsHomeostasis10239 Institute of Laboratory Animal ScienceReceptorInnate immune systemMicrogliaGeneral NeuroscienceMicrobiota2800 General NeuroscienceFatty Acids VolatilePhenotypeImmunity InnateMice Inbred C57BLmedicine.anatomical_structurenervous systemImmunology570 Life sciences; biology590 Animals (Zoology)FemaleMicrogliaNeuroscienceHomeostasisNature neuroscience
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Comprehensive analysis of expression, subcellular localization, and cognate pairing of SNARE proteins in oligodendrocytes

2009

Oligodendrocytes form the central nervous system myelin sheath by spiral wrapping of their plasma membrane around axons, necessitating a high rate of exocytic membrane addition to the growing myelin membrane. Membrane fusion is mediated by soluble N-ethylmaleimide-sensitive factor attachment protein receptor proteins (SNAREs), which act by specific pairing of vesicle (R)- and target (Q)-SNAREs. To characterize oligodendroglial SNAREs and their trafficking pathways, we performed a detailed expression analysis of SNAREs in differentiating cultured oligodendrocytes and myelin and determined their subcellular localization. Expression of the plasma membrane Q-SNAREs syntaxin 3, syntaxin 4, SNAP2…

Central Nervous SystemMaleVesicle-Associated Membrane Protein 3SynaptobrevinGolgi ApparatusBiologyMembrane FusionR-SNARE ProteinsMiceCellular and Molecular NeuroscienceSNAP23AnimalsSyntaxinQc-SNARE ProteinsTransport VesiclesCells CulturedMyelin SheathR-SNARE ProteinsQa-SNARE ProteinsVesicleCell MembraneLipid bilayer fusionQb-SNARE ProteinsSyntaxin 3Cell CompartmentationTransport proteinCell biologyOligodendrogliaProtein Transportnervous systemFemalebiological phenomena cell phenomena and immunitySNARE ProteinsDimerizationJournal of Neuroscience Research
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IL-17 and related cytokines involved in the pathology and immunotherapy of multiple sclerosis: Current and future developments.

2014

Multiple sclerosis (MS), an autoimmune neurological disorder, is driven by self-reactive T helper (Th) cells. Research on the role of Th17 lymphocytes in MS pathogenesis has made significant progress in identifying various immunological as well as environmental factors that induce the differentiation and expansion of these cells, different subsets of Th17 cells with varying degrees of pathogenicity, and the role of the secreted effector cytokines. While approved therapies for MS offer significant benefit to patients, there remain unmet needs. Ongoing clinical trials aim to translate the advanced knowledge of Th17 cytokines to improved therapies. This review discusses the current status and …

Central Nervous SystemPathologymedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisEndocrinology Diabetes and Metabolismmedicine.medical_treatmentImmunologyAutoimmunityNeurological disorderGeneral Biochemistry Genetics and Molecular BiologyUnmet needsPathogenesisMicemedicineImmunology and AllergyAnimalsHumansEffectorbusiness.industryMultiple sclerosisInterleukin-17Cell DifferentiationImmunotherapyInterferon-betamedicine.diseaseClinical trialImmunologyTh17 CellsInterleukin 17ImmunotherapyInflammation MediatorsbusinessCytokinegrowth factor reviews
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The Community Structure of the Global Corporate Network

2013

We investigate the community structure of the global ownership network of transnational corporations. We find a pronounced organization in communities that cannot be explained by randomness. Despite the global character of this network, communities reflect first of all the geographical location of firms, while the industrial sector plays only a marginal role. We also analyze the network in which the nodes are the communities and the links are obtained by aggregating the links among firms belonging to pairs of communities. We analyze the network centrality of the top 50 communities and we provide the first quantitative assessment of the financial sector role in connecting the global economy.

Character (mathematics)Secondary sector of the economyCommunity structureQuantitative assessmentCorporate networkBusinessEconomic geographyLocationCentralityFinancial sectorSSRN Electronic Journal
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Caritas e bene comune

2014

Tra il XIII e il XIV secolo in Italia, e soprattutto a Firenze, prende forma una nuova ideologia del «bene comune», molto legata al rafforzamento dello «stato» comunale durante la fase caratterizzata dal dominio del «popolo» . Alla base di questo rinnovamento sta l’esplicita politicizzazione della virtù cristiana della carità. Il legame politico rientrava tradizionalmente nelle categorie dell’amicizia e dell’amore, non della carità, virtù «universale» che legava l’uomo a Dio e gli uomini tra di loro nella comunità della chiesa: il sistema tomista fornisce un rappresentazione codificata di questa logica. Viceversa, alcuni autori, da Tolomeo da Lucca a Remigio de’Girolami a Coluccio Salutati,…

Charity common good commons political community Remigio de’ GirolamiCarità bene comune beni comuni comunità politica Remigio de’GirolamiSettore M-STO/01 - Storia Medievale
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In vitro evaluation of probiotic strains for lactic acid production

2019

Background The growing interest on usage of probiotic lactobacilli in maintaining oral health has posed number of questions on its probable side effects. One such consideration could be an increased acid production in dental plaque, in turn leading to dental caries. Thus, the aim of this study was to comparatively evaluate the lactic acid producing ability of L. acidophilus and L. plantarum with and without dental plaque. Material and Methods The study consisted of five groups: 3 control groups (Supragingival plaque, L. acidophilus and L. plantarum) and 2 test groups (Supragingival plaque with L. acidophilus and Supragingival plaque with L. plantarum). 26 samples for each group were collect…

ChemistryResearchfood and beveragesFructoseOral health:CIENCIAS MÉDICAS [UNESCO]Dental plaquemedicine.diseaseCommunity and Preventive DentistryAcid productionIn vitroLactic acidlaw.inventionchemistry.chemical_compoundProbioticfluids and secretionslawUNESCO::CIENCIAS MÉDICASmedicinebacteriaCentrifugationFood scienceGeneral DentistryJournal of Clinical and Experimental Dentistry
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IL-23-mediated mononuclear phagocyte crosstalk protects mice from Citrobacter rodentium-induced colon immunopathology.

2014

Gut homeostasis and mucosal immune defense rely on the differential contributions of dendritic cells (DC) and macrophages. Here we show that colonic CX3CR1+ mononuclear phagocytes are critical inducers of the innate response to Citrobacter rodentium infection. Specifically, the absence of IL-23 expression in macrophages or CD11b+ DC results in the impairment of IL-22 production and in acute lethality. Highlighting immunopathology as a death cause, infected animals are rescued by the neutralization of IL-12 or IFNγ. Moreover, mice are also protected when the CD103+ CD11b− DC compartment is rendered deficient for IL-12 production. We show that IL-12 production by colonic CD103+ CD11b− DC is r…

ChemokineColonCX3C Chemokine Receptor 1General Physics and Astronomychemical and pharmacologic phenomenaMice TransgenicInterleukin-23General Biochemistry Genetics and Molecular BiologyMonocytesArticleMicrobiologyInterferon-gammaMiceIntestinal mucosaAntigens CDImmunopathologyCitrobacter rodentiummedicineAnimalsHomeostasisInterferon gammaIntestinal MucosaImmunity MucosalMultidisciplinaryCD11b AntigenbiologyInterleukinsMacrophagesEnterobacteriaceae InfectionsGeneral ChemistryMononuclear phagocyte systemDendritic CellsInterleukin-12Survival AnalysisImmunity InnateIntegrin alpha MGene Expression RegulationImmunologyInterleukin 12biology.proteinCitrobacter rodentiumTh17 CellsReceptors ChemokineIntegrin alpha Chainsmedicine.drugSignal TransductionNature communications
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