Search results for "Up-Regulation"

showing 10 items of 455 documents

Up-regulation of 1-deoxy-D-xylulose-5-phosphate synthase enhances production of essential oils in transgenic spike lavender.

2006

Abstract Spike lavender (Lavandula latifolia) is an aromatic shrub cultivated worldwide for the production of essential oils. The major constituents of these oils are monoterpenes, which are obtained from isopentenyl diphosphate and dimethylallyl diphosphate precursors through the plastidial methylerythritol phosphate (MEP) pathway and/or the cytosolic mevalonate pathway. 1-Deoxy-d-xylulose-5-P synthase (DXS) catalyzes the first step of the MEP pathway. A cDNA coding for the Arabidopsis (Arabidopsis thaliana) DXS was constitutively expressed in spike lavender. Gas chromatography/mass spectrometry analyses revealed that transgenic plants accumulated significantly more essential oils compared…

ChlorophyllPhysiologyTransgeneMolecular Sequence DataLavandula latifoliaPlant ScienceGenetically modified cropslaw.inventionlawGene Expression Regulation PlantTransferasesArabidopsisBotanyGeneticsOils VolatileArabidopsis thalianaEssential oilbiologyATP synthasefood and beveragesbiology.organism_classificationPlants Genetically ModifiedCarotenoidsUp-RegulationPlant LeavesLavandulaBiochemistrybiology.proteinMevalonate pathwayResearch ArticlePlant physiology
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Up-regulation of an N-terminal truncated 3-hydroxy-3-methylglutaryl CoA reductase enhances production of essential oils and sterols in transgenic Lav…

2007

Spike lavender (Lavandula latifolia) essential oil is widely used in the perfume, cosmetic, flavouring and pharmaceutical industries. Thus, modifications of yield and composition of this essential oil by genetic engineering should have important scientific and commercial applications. We generated transgenic spike lavender plants expressing the Arabidopsis thaliana HMG1 cDNA, encoding the catalytic domain of 3-hydroxy-3-methylglutaryl CoA reductase (HMGR1S), a key enzyme of the mevalonic acid (MVA) pathway. Transgenic T0 plants accumulated significantly more essential oil constituents as compared to controls (up to 2.1- and 1.8-fold in leaves and flowers, respectively). Enhanced expression …

ChlorophyllTransgeneArabidopsisStigmasterolLavandula latifoliaMevalonic AcidPlant ScienceMevalonic acidReductaselaw.inventionchemistry.chemical_compoundlawBotanyOils VolatileArabidopsis thalianaCarotenoidEssential oilchemistry.chemical_classificationStigmasterolbiologyPhytosterolsfood and beveragesPigments BiologicalPlants Genetically Modifiedbiology.organism_classificationCarotenoidsSitosterolsUp-RegulationPlant LeavesLavandulachemistryBiochemistryMonoterpenesHydroxymethylglutaryl CoA ReductasesSesquiterpenesAgronomy and Crop ScienceBiotechnologyPlant Biotechnology Journal
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Down-regulation of transcription factors AP-1, Sp-1, and NF-kappa B precedes myocyte differentiation.

1996

Terminal differentiation of myocytes involves withdrawal from the cell cycle, induction of myogenin expression, and finally formation of myotubes. To study the factors that regulate the initial phase of muscle differentiation, we analyzed the binding activities of transcription factors AP-1, Sp-1, and NF-kappa B in L6, C2C12, and rhabdomyosarcoma BA-Han-1C cells. Temporal changes in transcription factor binding activities were compared to the activation of myogenin promoter-driven CAT reporter gene and the expression level of myogenin, a master gene of myogenic differentiation. We observed a prominent decrease in the nuclear binding activities of AP-1, Sp-1, and NF-kappa B already 12 to 24 …

Cholera ToxinSp1 Transcription FactorCellular differentiationBiophysicsDown-RegulationBiologyMuscle DevelopmentBiochemistryRetinoblastoma ProteinCell FusionMiceOkadaic AcidTumor Cells CulturedMyocyteAnimalsMuscle SkeletalMolecular BiologyTranscription factorMyogeninCell fusionMyogenesisNF-kappa BCell DifferentiationCell BiologyCell cyclemusculoskeletal systemMolecular biologyRatsUp-RegulationTranscription Factor AP-1MyogeninC2C12Protein BindingBiochemical and biophysical research communications
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Collagen-induced differential expression of an RNA polymerase subunit by breast cancer cells

2005

It was previously reported that the stroma of ductal infiltrating carcinoma (DIC) of the human breast contains considerable amount of an embryo-foetal collagen type, OF/LB (onco-foetal/laminin-binding), and that adhesion of 8701-BC DIC cells onto OF/LB collagen substrates selectively promotes cell growth, motility, production of extracellular lytic enzymes and invasion "in vitro" if compared with other collagen species. To detect possible transcriptional differences for regulatory proteins following OF/LB collagen-cell interactions, we submitted RNA preparations from 8701-BC cells grown on collagen type I, IV and OF/LB to "differential display"-PCR in the presence of degenerate C(2)H(2) zin…

Collagen Type IVProtein subunitBreast NeoplasmsBiologyPolymerase Chain ReactionBiochemistryCollagen Type Ichemistry.chemical_compoundBreast cancerRNA polymeraseRNA Ribosomal 18STumor Cells CulturedExtracellularHumansSettore BIO/06 - Anatomia Comparata E CitologiaGeneCell growthRNACell DifferentiationGeneral MedicineMolecular biologyUp-RegulationEnzyme ActivationGene Expression Regulation NeoplasticProtein SubunitschemistryCell cultureRNA polymeraseFemaleLamininRNA Polymerase IICollagenCell cultureGlyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating)Tyrosine kinase
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Low doses of paclitaxel potently induce apoptosis in human retinoblastoma Y79 cells by up-regulating E2F1.

2008

Paclitaxel (PTX) is an anticancer drug currently in phase II clinical trials. This study shows for the first time that low doses of PTX (5 nM) potently induce apoptosis in human retinoblastoma Y79 cells. The effect of PTX is accompanied by a potent induction of E2F1 which appears to play a critical role in the effects induced by PTX. PTX induced a dose- and time-dependent effect, with G2/M arrest, cyclines A, E and B1 accumulation and a marked modification in the status of Cdc2-cyclin B1 complex, the major player of the G2/M checkpoint. Apoptosis followed G2/M arrest. An early and prolonged increase in p53 expression with its stabilization by phosphorylation and acetylation and its nuclear …

Cyclin-Dependent Kinase Inhibitor p21G2 Phaseendocrine systemCancer ResearchProgrammed cell deathPaclitaxelApoptosisBiologyretinoblastoma apoptosis paclitaxelp14arfSettore BIO/10 - BiochimicaCell Line TumorE2F1HumansFragmentation (cell biology)PhosphorylationMembrane Potential MitochondrialRetinoblastomaCell cycleAntineoplastic Agents PhytogenicUp-RegulationGene Expression Regulation NeoplasticOncologyApoptosisCancer researchPhosphorylationApoptosomeTumor Suppressor Protein p53Cell DivisionE2F1 Transcription FactorInternational journal of oncology
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Loss of Neuroglobin Expression Alters Cdkn1a/Cdk6-Expression Resulting in Increased Proliferation of Neural Stem Cells

2018

Abstract: In the quest to unravel its functional significance, neuroglobin (Ngb), a brain-specific neuroprotective protein, has recently been proposed as an actor in neurodevelopment. As neural stem cells (NSCs) are fundamental during brain development, the present study aimed at investigating the role of Ngb in the growth and proliferation of NSCs by comparing an Ngb-floxed (Ngb(fl)-)NSC line, equivalent to the wild-type cellular situation, with an in-house created Ngb knockout (Ngb(KO)-)NSC line. Ngb(KO)-NSCs were characterized by an increased growth and proliferation capacity in vitro, supported by RNA sequencing and western blot results reporting the downregulation of Cdkn1a and the upr…

Cyclin-Dependent Kinase Inhibitor p21Male0301 basic medicineCell signalingDown-RegulationNeuroglobinNerve Tissue ProteinsBiologyNeuroprotectionTranscriptomeMice03 medical and health sciences0302 clinical medicineNeural Stem CellsDownregulation and upregulationAnimalsBiologyCells CulturedCell ProliferationCell CycleCyclin-Dependent Kinase 6Cell BiologyHematologyCell cycleNeural stem cellUp-RegulationCell biologyMice Inbred C57BL030104 developmental biologynervous systemNeuroglobinbiology.proteinFemaleHuman medicineCyclin-dependent kinase 6Tumor Suppressor Protein p53Proto-Oncogene Proteins c-akt030217 neurology & neurosurgerySignal TransductionDevelopmental BiologyStem Cells and Development
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The DNA-binding subunit p140 of replication factor C is upregulated in cycling cells and associates with G 1 phase cell cycle regulatory proteins

1999

The DNA-binding subunit of replication factor C (RFCp140) plays an important role in both DNA replication and DNA repair. The mechanisms regulating activation of RFCp140 thereby controlling replication and cellular proliferation are largely unknown. We analyzed protein expression of RFCp140 during cell cycle progression and investigated the association of RFCp140 with cell cycle regulatory proteins in cell lines of various tissue origin and in primary hematopoietic cells. Western and Northern blot analyses of RFCp140 from synchronized cells showed downregulation of RFCp140 when cells enter a G0-like quiescent state and upregulation of RFCp140 in cycling cells. Translocation from the cytopla…

CytoplasmSaccharomyces cerevisiae ProteinsT-LymphocytesCyclin ACell Cycle ProteinsEukaryotic DNA replicationCell LineMinor Histocompatibility AntigensDNA replication factor CDT1MiceReplication factor CControl of chromosome duplicationDrug DiscoveryAnimalsHumansReplication Protein CGenetics (clinical)Cell NucleusHomeodomain ProteinsbiologyG1 PhaseS-phase-promoting factor3T3 CellsCell cycleMolecular biologyUp-RegulationCell biologyDNA-Binding ProteinsRepressor ProteinsProto-Oncogene Proteins c-bcl-2biology.proteinMolecular MedicineOrigin recognition complexJournal of Molecular Medicine
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Upregulation of Major Histocompatibility Complex Class I on Liver Cells by Hepatitis C Virus Core Protein via p53 and TAP1 Impairs Natural Killer Cel…

2003

ABSTRACTThe mechanisms of immune evasion and the role of the early immune response in chronic infection caused by hepatitis C virus (HCV) are still unclear. Here, we present evidence for a cascade of molecular events that the virus initiates to subvert the innate immune attack. The HCV core protein induced p53-dependent gene expression of TAP1 (transporter associated with antigen processing 1) and consecutive major histocompatibility complex (MHC) class I upregulation. Moreover, in p53-deficient liver cell lines, only reconstitution with wild-type p53, but not mutated p53 lacking DNA binding capacity, showed this effect. As a consequence of increased MHC class I expression, a significantly …

Cytotoxicity ImmunologicImmunologyAntigen presentationHepacivirusMajor histocompatibility complexMicrobiologyCell LineNatural killer cellAntigenVirologyMHC class ImedicineHumansATP Binding Cassette Transporter Subfamily B Member 2Cells CulturedLymphokine-activated killer cellbiologyViral Core ProteinsHistocompatibility Antigens Class IHepatitis C ChronicNatural killer T cellVirologyUp-RegulationKiller Cells Naturalmedicine.anatomical_structureInsect ScienceImmunologyHepatocytesbiology.proteinPathogenesis and ImmunityATP-Binding Cassette TransportersTumor Suppressor Protein p53CD8Journal of Virology
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DNA polymeraseθ up-regulation is associated with poor survival in breast cancer, perturbs DNA replication, and promotes genetic instability

2010

“Replicative stress” is one of the main factors underlying neoplasia from its early stages. Genes involved in DNA synthesis may therefore represent an underexplored source of potential prognostic markers for cancer. To this aim, we generated gene expression profiles from two independent cohorts (France,n= 206; United Kingdom,n= 117) of patients with previously untreated primary breast cancers. We report here that among the 13 human nuclear DNA polymerase genes, DNA Polymerase θ (POLQ) is the only one significantly up-regulated in breast cancer compared with normal breast tissues. Importantly,POLQup-regulation significantly correlates with poor clinical outcome (4.3-fold increased risk of de…

DNA ReplicationGenome instabilityDNA damageDNA polymerase[SDV]Life Sciences [q-bio]DNA Polymerase ThetaBreast NeoplasmsDNA-Directed DNA PolymeraseKaplan-Meier Estimatemedicine.disease_causeBioinformaticsGenomic InstabilityCell LineCohort Studies03 medical and health sciences0302 clinical medicineBreast cancerCell Line TumorChromosome instabilityCyclin EmedicineHumansComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesMultidisciplinarybiologyReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingCancerMiddle AgedBiological SciencesPrognosismedicine.diseaseUnited KingdomUp-RegulationGene Expression Regulation Neoplastic030220 oncology & carcinogenesisCancer researchbiology.proteinFemaleRNA InterferenceFranceCarcinogenesisDNA DamageProceedings of the National Academy of Sciences
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'Up-regulation of histone acetylation induced by social defeat mediates the conditioned rewarding effects of cocaine

2016

Social defeat (SD) induces a long-lasting increase in the rewarding effects of psychostimulants measured using the self-administration and conditioned place procedures (CPP). However, little is known about the epigenetic changes induced by social stress and about their role in the increased response to the rewarding effects of psychostimulants. Considering that histone acetylation regulates transcriptional activity and contributes to drug-induced behavioral changes, we addressed the hypothesis that SD induces transcriptional changes by histone modifications associated with the acquisition of place conditioning. After a fourth defeat, H3(K9) acetylation was decreased in the hippocampus, whil…

Dominance-SubordinationMaleCurcuminHippocampusSpatial BehaviorPharmacologyHippocampusChromatin remodelingEpigenesis GeneticSocial defeatHistone H4Histones03 medical and health sciencesMice0302 clinical medicineRewardCocaineConditioning PsychologicalValproic acidAnimalsEpigeneticsBiological PsychiatryHistone AcetyltransferasesPharmacologySocial stressCerebral CortexbiologyValproic AcidAcetylation030227 psychiatryUp-RegulationHistone Deacetylase InhibitorsDisease Models AnimalHistoneHistone acetylationAcetylationbiology.proteinCentral Nervous System StimulantsPsychologySocial defeat stress030217 neurology & neurosurgeryStress Psychological
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