Search results for "Uremia"

showing 10 items of 29 documents

Impaired cellular immune responses in chronic renal failure: Evidence for a T cell defect

1986

Impaired cellular immune responses in chronic renal failure: Evidence for a T cell defect. Cellular immune responses in vitro were studied in 24 patients on chronic hemodialysis and 16 healthy volunteers with normal kidney function. Patients on maintenance hemodialysis had lymphopenia with diminished numbers of both T4 + and T8 + T-lymphocytes. The T4/T8 ratios were within the normal range. Peripheral blood lymphocytes (PBL) showed a diminished proliferative response upon stimulation with concanavalin A, phytohemagglutinin and poke weed mitogen. When cell surface antigens were used for stimulation (mixed lymphocyte culture) uremic lymphocytes also showed a lower proliferation rate. Although…

AdultMaleIsoantigensT-LymphocytesLymphocyteT cellLymphocyte CooperationRenal functionStimulationchemical and pharmacologic phenomenaLymphocyte ActivationLeukocyte CountImmune systemmedicineHumansLymphocytesImmunodeficiencyAgedUremiaB-LymphocytesImmunity Cellularbiologybusiness.industryAntibodies MonoclonalMiddle Agedmedicine.diseaseIn vitromedicine.anatomical_structureNephrologyConcanavalin AAntibody FormationImmunologybiology.proteinInterleukin-2Kidney Failure ChronicFemaleMitogensbusinessKidney International
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Effects of Some Guanidino Compounds on Human Cerebral Arteries

1999

Background and Purpose —Accumulation of endogenous guanidino-substituted analogues of l -arginine in chronic renal failure might contribute to some of the vascular and neurological disorders of this pathology. We tested the hypothesis that in human cerebral arteries, some guanidino compounds may increase vascular tone, through nitric oxide (NO) synthase inhibition, and impair endothelium-dependent relaxation. Methods —Rings of human middle cerebral artery were obtained during autopsy of 26 patients who had died 3 to 12 hours before. The rings were suspended in organ baths for isometric recording of tension. We then studied the responses to N G -monomethyl- l -arginine (L-NMMA), N G , N G -…

AdultMaleMiddle Cerebral Arterymedicine.medical_specialtyEndotheliumVasodilator AgentsCerebral arteriesEndogenyMethylguanidineArginineGuanidinesNitric oxidechemistry.chemical_compoundmedicine.arteryInternal medicinemedicineHumansVasoconstrictor AgentsEnzyme InhibitorsAgedEC50Advanced and Specialized Nursingomega-N-Methylargininebusiness.industryMiddle Agedmedicine.diseaseUremiaSurgeryEndocrinologymedicine.anatomical_structurechemistryMiddle cerebral arteryFemaleNeurology (clinical)Nitric Oxide SynthaseCardiology and Cardiovascular MedicinebusinessAcetylcholinemedicine.drugStroke
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LOW-DOSE INTERLEUKIN-2 INDUCES SYSTEMIC IMMUNE RESPONSES AGAINST HBsAg IN IMMUNODEFICIENT NON-RESPONDERS TO HEPATITIS B VACCINATION

1989

Abstract A metabolic monocyte defect appears to correlate with non-responsiveness to hepatitis B vaccine in many patients on haemodialysis. This defect prevents production of interleukin-2 during T-cell activation after antigen contact. Receptors for interleukin-2 are, however, expressed in greater numbers than in healthy subjects or uraemic responders to hepatitis B vaccination. In this study, ten uraemic patients, previous non-responders to vaccination against hepatitis B, were revaccinated with the same vaccine combined with one intramuscular injection (2·5 × 10 5 U) of natural human interleukin-2. Systemic production of antibodies against hepatitis B surface antigen was initiated in tho…

AdultMaleViral Hepatitis VaccinesInterleukin 2HBsAgHepatitis B vaccinePilot ProjectsImmunoenzyme TechniquesImmune systemAntigenmedicineHumansHepatitis B VaccinesAgedUremiaHepatitis B Surface Antigensbiologybusiness.industryImmunologic Deficiency SyndromesReceptors Interleukin-2General MedicineMiddle AgedHepatitis Bmedicine.diseaseVirologyVaccinationAntibody FormationImmunologybiology.proteinInterleukin-2FemaleAntibodybusinessmedicine.drugThe Lancet
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Analysis of F-wave in metabolic neuropathies: a comparative study in uremic and diabetic patients.

1987

Motor nerve conduction study along the entire length of the ulnar and tibialis posterior nerves was carried out in 30 diabetics compared with 30 uremic patients and 30 control subjects. The conduction in the proximal and the distal nerve segments was evaluated by the determination of the M and F latencies, MNCV (between the stimulus sites), FWCV (between the spinal cord and the stimulus sites), and F-ratio (conduction time ratio of proximal to distal segment). In both groups of patients the lower limbs appear much more involved than the upper, where the ulnar nerve is more commonly affected in uremic than in diabetic patients. In diabetic neuropathy the motor conduction abnormalities are di…

AdultMalemedicine.medical_specialtyDiabetic neuropathyNeural ConductionMotor nerveAction PotentialsNerve conduction velocityF waveDiabetic NeuropathiesElectroneuronographymedicineReaction TimeHumansUlnar nerveTibial nerveUlnar NerveAgedUremiaNeural ConductionMotor Neuronsbusiness.industryPeripheral Nervous System DiseasesGeneral MedicineAnatomyMiddle Agedmedicine.diseaseSurgeryNeurologyFemaleNeurology (clinical)Tibial NervebusinessActa neurologica Scandinavica
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T cell activation defect in hemodialysis patients: Evidence for a role of the B7/CD28 pathway

1993

T cell activation defect in hemodialysis patients: Evidence for a role of the B7/CD28 pathway. The immunosuppressive effect of chronic renal failure is correlated with an impaired proliferation of peripheral blood leukocytes in vitro . This is mainly due to an impaired function of the accessory cells rather than the T cells. Here we tried to define a missing accessory signal for T cell activation in hemodialysis patients. We substituted cell surface bound molecules by adding tumor cell lines to the in vitro assays that express different patterns of accessory molecules. Cell lines that express the costimulatory B7 molecule reconstituted the activation of patients' cells whereas B7 negative c…

Antigens Differentiation T-LymphocyteT-LymphocytesT cellCellLymphocyte ActivationTransfectionMonocytesMiceImmune systemCD28 AntigensAntigens CDRenal DialysisTumor Cells CulturedmedicineAnimalsHumansPhytohemagglutininsAntigen-presenting cellAgedUremiabusiness.industryCD283T3 CellsT lymphocyteTransfectionMiddle AgedBurkitt LymphomaPhenotypemedicine.anatomical_structureNephrologyCell cultureAntigens SurfaceImmunologyB7-1 AntigenCancer researchInterleukin-2businessKidney International
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Serum- und Erythrocyten-Magnesium bei renaler Insuffizienz

1963

1. Bei akuten und chronischen Niereninsuffizienzen finden sich normale und erhohte Serum-Magnesium-Werte. Eine Beziehung zwischen Serum-Magnesium und Serum-Rest-N besteht nicht.

Blood Chemical Analysismedicine.medical_specialtyMagnesiumbusiness.industrychemistry.chemical_elementGlomerulonephritisGeneral Medicinemedicine.diseaseMolecular medicineGastroenterologyUremiachemistryInternal medicineDrug DiscoverymedicineMolecular MedicinebusinessNephrotic syndromeNephritisGenetics (clinical)Kimmelstiel-Wilson syndromeKlinische Wochenschrift
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Management of secondary hyperparathyroidism in uremic patients : the role of the new vitamin D analogs

2007

Secondary hyperparathyroidism - a common comorbid condition in patients with chronic renal insufficiency - is considered a consequence of critical determinants such as hypocalcemia, phosphate retention and reduced levels of calcitriol production. In this complex mechanism, the skeletal apparatus and the nonskeletal targets such as vascular and heart valves are often involved, thus explaining the increased risk of cardiovascular morbidity and mortality of uremic patients. In this review we will focus on the major role played by Calcitriol deficiency as a trigger of secondary hyperparathyroidism and the crucial need for obiquitous vitamin D receptor activation in order to have an optimal PTH …

Bone Density Conservation AgentsCalcitriolHypocalcemiaErgocalciferolsHumansReceptors CalcitriolHyperparathyroidism SecondaryRenal Insufficiency ChronicVitamin DVitamin D DeficiencyPhosphatesUremia
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Primary angiosarcoma of the alveolar mucosa in a haemodialysis patient: case report and discussion

1994

A case of a haemodialysis patient with a primitive angiosarcoma of the alveolar mucosa is reported. The vascular origin of the tumor was confirmed by the immunohistochemical data which showed strong positivity for Factor VIII-related antigen and for vimentin, whereas stains for desmin and cytokeratins were negative.

Cancer ResearchPathologymedicine.medical_specialtyHemangiosarcomaVimentinMandiblemacromolecular substancesPathology and Forensic MedicineImmunoenzyme TechniquesAntigenRenal Dialysisvon Willebrand FactorHumansVimentinMedicineAngiosarcomaDental alveolusAlveolar mucosaUremiaGingival Neoplasmsbiologybusiness.industryMouth MucosaMiddle AgedPrimary AngiosarcomaOtorhinolaryngologybiology.proteinPeriodonticsImmunohistochemistryFemaleDesminOral SurgerybusinessJournal of Oral Pathology and Medicine
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Does uremic enterocolitis exist?

1981

In rats a severe but compensated chronic renal insufficiency was induced by stepwise 9/10 nephrectomy. Despite this severe chronic renal insufficiency we observed no relevant pathological changes in the intestinal mucosa. In particular, we found no evidence of mucosal erosions, ulceration or pseudomembranous colitis, findings which are traditionally thought to be characteristic of the uremic state. This was also true of those animals dying prematurely from uremia. Thus serious doubts arise about the existence of “uremic enterocolitis”, doubts which also proved justified after a critical review of the literature on human pathology.

EnterocolitisPathologymedicine.medical_specialtybusiness.industrymedicine.medical_treatmentPseudomembranous colitismedicine.diseaseIntestinal absorptionNephrectomyUremiaIntestinal mucosamedicinemedicine.symptombusinessHuman PathologyPathologicalVirchows Archiv B Cell Pathology Including Molecular Pathology
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Bedeutung der Autolyse f�r die Ur�miediagnostik durch Kreatininbestimmung am Leichenherzen

1968

Die Kreatininkonzentrationen im Herzmuskel wurden untersucht in Abhangigkeit von der Lagerungstemperatur und dem zeitlichen Intervall zwischen Todeseintritt und chemischer Bestimmung. Fur die Bestimmung der Todesursache und der Todeszeit ergaben sich folgende Konsequenzen: 1. Eine Beurteilung der intravitalen Nierenfunktion ist bis zu 3 Tagen post mortem moglich. 2. Werte unter 5 und uber 20 mg/100 g schliesen ein Leichenalter von mehr als 5 Tagen aus. 3. Verlaufsbeobachtungen der Kreatininkonzentration am obduzierten Herzen erlauben Ruckschlusse auf eine renale Todesursache und die Todeszeit. 4. Mit geringen Erniedrigungen der Kreatininwerte 1–4 Tage post mortem bei niedrigen Ausentemperat…

Gynecologymedicine.medical_specialtyAutolysis (biology)Creatininechemistry.chemical_compoundchemistrybusiness.industrymedicinebusinessmedicine.diseaseUremiaPathology and Forensic MedicineDeutsche Zeitschrift f�r die Gesamte Gerichtliche Medizin
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