Search results for "VASODILATION"

showing 10 items of 228 documents

Relaxant effect of sildenafil in the rabbit basilar artery

2005

We hypothesized that sildenafil, inhibitor of phosphodiesterase-5 (PDE-5), interacts with the nitric oxide (NO)-cGMP pathway in the cerebral arteries and shows vasoactive effects. To prove it in the isolated rabbit basilar artery, we compared the effects of sildenafil with other PDE-5 inhibitors, assessed the endothelial dependence of the vasoactive responses, and used modulators of the cGMP and cAMP signaling processes. Sildenafil (10 nM-0.1 mM) induced concentration-dependent relaxations of endothelin-1 (10 nM)-precontracted basilar artery, which were partially inhibited both in endothelium-denuded arteries and in arteries precontracted by depolarization with KCl (50 mM). Endothelin-1 (1 …

Malemedicine.medical_specialtyPhosphodiesterase InhibitorsPhysiologySildenafilVasodilator AgentsCerebral arteriesVasodilationIn Vitro TechniquesPiperazinesSildenafil Citratechemistry.chemical_compound3'5'-Cyclic-GMP PhosphodiesterasesQuinoxalinesmedicine.arteryInternal medicinemedicineBasilar arteryAnimalsSulfonesCyclic Nucleotide Phosphodiesterases Type 5PharmacologyOxadiazolesDose-Response Relationship DrugPhosphoric Diester HydrolasesPDE5 drug designVasodilationNG-Nitroarginine Methyl EsterEndocrinologychemistryGuanylate CyclasePurinesBasilar Arterycardiovascular systemMolecular MedicineRabbitsSodium nitroprussideNitric Oxide SynthaseSoluble guanylyl cyclaseZaprinastSignal Transductionmedicine.drugVascular Pharmacology
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Diabetes impairs the atrial natriuretic peptide relaxant action mediated by potassium channels and prostacyclin in the rabbit renal artery.

2012

Diabetes is associated with increased prevalence of hypertension, cardiovascular and renal disease. Atrial natriuretic peptide (ANP) plays an important role in cardiovascular pathophysiology and is claimed to have cardioprotective and renoprotective effect in diabetic patients. The working hypothesis was that alloxan-induced diabetes might modify the vascular effects of ANP in isolated rabbit renal arteries and the mechanisms involved in such actions. Plasma ANP levels were higher in diabetic rabbits than in control rabbits. ANP (10(-12)-10(-7)M) induced a relaxation of precontracted renal arteries, which was lower in diabetic than in control rabbits. In arteries from both groups of animals…

Malemedicine.medical_specialtyPotassium ChannelsEndotheliumProstacyclinIn Vitro TechniquesDiabetes Mellitus ExperimentalGlibenclamideThromboxane A2chemistry.chemical_compoundThromboxane A2Renal ArteryAtrial natriuretic peptidemedicine.arteryDiabetes mellitusInternal medicinemedicineAnimalsRenal arteryPharmacologybusiness.industryTetraethylammoniummedicine.diseaseEpoprostenolPotassium channelVasodilationEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemEndothelium VascularRabbitsbusinesshormones hormone substitutes and hormone antagonistsAtrial Natriuretic Factorcirculatory and respiratory physiologymedicine.drugPharmacological research
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Effects of a nitrate-free interval on tolerance, vasoconstrictor sensitivity and vascular superoxide production

2000

Abstract OBJECTIVES In the present study, we tested whether a nitrate-free interval is able to prevent increases in vascular superoxide (O2•−) and the development of hypersensitivity to vasoconstrictors and whether this may result in restoration of vascular nitroglycerin (NTG) sensitivity. BACKGROUND Intermittent NTG-patch treatment (12 h patch on/patch off) has been shown to increase ischemic periods in patients with stable coronary arteries, suggesting a rebound-like situation during the patch-off period. Recently, we demonstrated that long-term treatment with NTG induces tolerance, which was in part related to increases in vascular O2•− and increased vasoconstrictor sensitivity. METHODS …

Malemedicine.medical_specialtyTime Factorsgenetic structuresVasodilator AgentsBlotting WesternVasodilationIn Vitro TechniquesSuperoxide dismutaseNitroglycerinchemistry.chemical_compoundDrug toleranceInternal medicinemedicineAnimalsEndothelial dysfunctionPhenylephrinebiologySuperoxide DismutaseSuperoxidebusiness.industryDrug Tolerancemedicine.diseaseAngiotensin IIAcetylcholineeye diseasesOxidative StressEndocrinologychemistryVasoconstrictionbiology.proteinFemaleEndothelium VascularRabbitsmedicine.symptombusinessCardiology and Cardiovascular MedicineVasoconstrictionmedicine.drugJournal of the American College of Cardiology
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Comparison between local and regional anesthesia in arteriovenous fistula creation.

2011

Purpose Assessment of the effectiveness of Brachial Plexus Block (BPB) via axillary approach compared to regional anesthesia for arteriovenous fistula surgery in patients affected by end-stage renal disease. Methods We compared forty patients randomly divided into two groups. Group A underwent BPB procedure with 15 mL ropivacaine 1% and 10 mL of saline (0.9% NaCl) via axillary approach. Group B received local anesthesia with lidocaine 2%. The forearm blood vessels were assessed by Doppler ultrasonography before and after the intervention. Results BPB performed on Group A was associated with a considerable venous dilation and a significant decrease (48.7%, P<.05) in pulsatility index (PI)…

Malemedicine.medical_specialtyTime Factorslocal and regional anesthesiaFistulaArteriovenous fistulaSettore MED/41 - AnestesiologiaVeinsArteriovenous Shunt SurgicalForearmRenal DialysisOcclusionmedicineHumansLocal anesthesiaBrachial PlexusRopivacaineAnesthetics Localarteriovenous fistulaBrachial plexus blockarteriovenous fistula; local and regional anesthesiaAgedRopivacainebusiness.industryLidocaineNerve BlockUltrasonography DopplerMiddle Agedmedicine.diseaseThrombosisAmidesSurgeryVasodilationSettore MED/18 - Chirurgia GeneraleForearmmedicine.anatomical_structureTreatment OutcomeItalyNephrologyAnesthesiaRadial ArteryKidney Failure ChronicSurgeryFemalebusinessmedicine.drugAnesthesia LocalThe journal of vascular access
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Pharmacological profile of phytoestrogens in cerebral vessels: in vitro study with rabbit basilar artery.

2003

As a previous step to consider their use in the pharmacology for stroke, we investigated the effects of four phytoestrogens (i.e. genistein, daidzein, zearalanone and biochanin A) on cerebral vessels. Cerebral vascular responses were analyzed by conventional recording of isometric tension in rabbit basilar artery segments kept in organ bath under standard conditions. The four phytoestrogens elicited concentration-dependent relaxant responses of different potency in basilar artery segments previously contracted with either 5x10(-2) M KCl or 10(-4) M UTP. Neither endothelium removal, 10(-4) M N(omega)-nitro-L-arginine methyl ester (L-NAME, nitric oxide (NO) synthase inhibitor), 10(-5) M1 H-[1…

Malemedicine.medical_specialtyVascular smooth muscleEndotheliumCerebral arteriesStimulationPhytoestrogensBiologyIn Vitro TechniquesNitric oxidechemistry.chemical_compoundInternal medicinemedicine.arterymedicineBasilar arteryAnimalsPharmacologyDose-Response Relationship DrugIsoflavonesVasodilationEndocrinologymedicine.anatomical_structurechemistryVasoconstrictionBasilar ArteryPhytoestrogensPlant PreparationsRabbitsSoluble guanylyl cyclaseEuropean journal of pharmacology
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Increased responsiveness of human pulmonary arteries in patients with positive bronchodilator response.

1996

Abstract 1. The effects of noradrenaline, endothelin-1, acetylcholine and sodium nitroprusside were studied in isolated pulmonary arteries obtained from 14 patients undergoing lobectomy for lung carcinoma. Seven patients had shown increased response to a bronchodilator test prior to operation. In the remaining patients (control) the bronchodilator test was negative. 2. Artery rings from patients with a positive bronchodilator response showed greater contraction to noradrenaline (pD2 = 6.44 +/- 0.1; Emax = 93 +/- 9% of response to 100 mM KCl) and endothelin-1 (pD2 = 8.92 +/- 0.1; Emax = 130 +/- 16%) than the rings from control patients (pD2 = 6.04 +/- 0.08; Emax = 56 +/- 8% for noradrenaline…

Malemedicine.medical_specialtyVascular smooth musclemedicine.drug_classVasodilator AgentsVasodilationIn Vitro TechniquesPulmonary ArteryInternal medicineBronchodilatormedicineHumansVasoconstrictor AgentsEndotheliumEnzyme InhibitorsAgedPharmacologybusiness.industryBronchodilator Agentsmedicine.anatomical_structureNG-Nitroarginine Methyl EsterBronchodilator AgentsDilatorAnesthesiaCardiologySodium nitroprussidemedicine.symptomBronchial HyperreactivityNitric Oxide SynthasebusinessVasoconstrictionArterymedicine.drugResearch ArticleBritish journal of pharmacology
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Nitric oxide-mediated beta 2-adrenoceptor relaxation is impaired in mesenteric veins from portal-hypertensive rats.

1996

Abstract BACKGROUND & AIMS: beta-Adrenergic relaxation seems to be mediated by nitric oxide. The aim of this study was to evaluate changes induced by portal hypertension in beta 2-adrenergic vasorelaxation. METHODS: Isolated rat mesenteric veins were relaxed by salbutamol, and nerve- mediated vasocontractions were induced by electrical field stimulation. Responses were evaluated in the presence of NG-nitro-L-arginine methyl ester (L-NAME) or tetrodotoxin. Immunocytochemical techniques were used for localization of neuronal NO synthase. RESULTS: Salbutamol-induced relaxations were decreased in rings from portal-hypertensive animals. L- NAME reduced these relaxations, but its effects were mor…

Malemedicine.medical_specialtyVasodilator AgentsStimulationVasodilationTetrodotoxinIn Vitro TechniquesS-Nitroso-N-AcetylpenicillamineNitric OxideNitric oxideRats Sprague-Dawleychemistry.chemical_compoundMesenteric VeinsInternal medicinePapaverineHypertension PortalReceptors Adrenergic betamedicineAnimalsAlbuterolHepatologybiologyPenicillamineGastroenterologyAdrenergic beta-AgonistsImmunohistochemistryElectric StimulationRatsNitric oxide synthaseVasodilationEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterchemistryTetrodotoxinbiology.proteinS-Nitroso-N-acetylpenicillamineNitric Oxide SynthaseFree nerve endingBlood vesselGastroenterology
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No Improvement of High-Density Lipoprotein (HDL) Vasorelaxant Effect Despite Increase in HDL Cholesterol Concentration in Type 2 Diabetic Patients Tr…

2014

Abstract Context: High-density lipoproteins (HDLs) from type 2 diabetic patients are unable to counteract the inhibitory effect of oxidized low-density lipoproteins (ox-LDLs) on vasorelaxation. We hypothesized that glitazones, which improve glycemic control and dyslipidemia, could correct this abnormality. Objectives and Design: We compared the ability of HDL from controls (n = 12) and from type 2 diabetic patients before and after 6 months of treatment with either rosiglitazone (n = 11) or pioglitazone (n = 8) to counteract the inhibitory effect of ox-LDL on vasodilatation of rabbit aorta rings. Results: Rosiglitazone induced a decrease in hemoglobin A1c (7.7% ± 1.1% vs 9.8% ± 1.0%, P = .0…

Malemedicine.medical_specialtymedicine.drug_classEndocrinology Diabetes and MetabolismClinical BiochemistryContext (language use)BiochemistryRosiglitazonechemistry.chemical_compoundEndocrinologyHigh-density lipoproteinInternal medicineDiabetes mellitusmedicineAnimalsHumansHypoglycemic AgentsThiazolidinedioneAortaAgedDyslipidemiasPioglitazoneCholesterolbusiness.industryCholesterol HDLBiochemistry (medical)Middle Agedmedicine.diseaseLipoproteins LDLVasodilationEndocrinologyDiabetes Mellitus Type 2chemistryFemaleThiazolidinedionesEndothelium VascularRabbitsLipoproteins HDLRosiglitazonebusinessPioglitazoneDyslipidemiamedicine.drugThe Journal of Clinical Endocrinology & Metabolism
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Sevoflurane Impairs Cerebral Blood Flow Autoregulation in Rats: Reversal by Nonselective Nitric Oxide Synthase Inhibition

2005

UNLABELLED In this study, we investigated the effects of 1.0 and 2.0 minimum alveolar anesthetic concentration (MAC) sevoflurane on cerebral blood flow (CBF) autoregulation before and after nonselective inhibition of nitric oxide (NO) synthase in rats. Rats were randomly assigned as follows: Group 1 (n = 8): 1.0 MAC sevoflurane; Groups 2 and 3 (n = 8 per group): 2.0 MAC sevoflurane. Assessment of autoregulation within a mean arterial blood pressure range of 140-60 mm Hg was performed by graded hemorrhage before and after administration of l-arginine methyl ester (l-NAME, 30 mg/kg IV, Groups 1 and 2) or during hypocapnia (Group 3). In 10 additional animals, brain tissue NO(2)(-) concentratio…

Methyl EthersBlood PressureVasodilationPharmacologyNitric OxideSevofluraneNitric oxideRats Sprague-DawleySevofluranechemistry.chemical_compoundHypocapniaAnimalsHomeostasisHyperventilationMedicineAutoregulationEnzyme InhibitorsCerebral HemorrhageBrain ChemistryBlood VolumeDose-Response Relationship Drugbiologybusiness.industrymedicine.diseaseRatsNitric oxide synthaseNG-Nitroarginine Methyl EsterAnesthesiology and Pain MedicineCerebral blood flowchemistryCerebrovascular CirculationAnesthesiaAnesthetics InhalationAnestheticbiology.proteinNitric Oxide Synthasebusinessmedicine.drugAnesthesia & Analgesia
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Manganese superoxide dismutase and aldehyde dehydrogenase deficiency increase mitochondrial oxidative stress and aggravate age-dependent vascular dys…

2008

AimsImbalance between pro- and antioxidant species (e.g. during aging) plays a crucial role for vascular function and is associated with oxidative gene regulation and modification. Vascular aging is associated with progressive deterioration of vascular homeostasis leading to reduced relaxation, hypertrophy, and a higher risk of thrombotic events. These effects can be explained by a reduction in free bioavailable nitric oxide that is inactivated by an age-dependent increase in superoxide formation. In the present study, mitochondria as a source of reactive oxygen species (ROS) and the contribution of manganese superoxide dismutase (MnSOD, SOD-2) and aldehyde dehydrogenase (ALDH-2) were inves…

Mitochondrial ROSMaleAgingPhysiologyVasodilator AgentsMitochondrionVascular dysfunctionmedicine.disease_causeMitochondria HeartMuscle Smooth Vascularchemistry.chemical_compoundMiceEndothelial dysfunctionAortachemistry.chemical_classificationMice KnockoutbiologySuperoxideAldehyde Dehydrogenase MitochondrialAge FactorsVasodilationBiochemistryCardiology and Cardiovascular MedicineMitochondrial aldehyde dehydrogenasemedicine.medical_specialty8-oxodGOxidative phosphorylationDNA MitochondrialSuperoxide dismutaseManganese superoxide dismutaseddc:570Physiology (medical)Internal medicinemedicineAnimalsReactive oxygen speciesDose-Response Relationship DrugSuperoxide DismutaseMitochondrial oxidative stressOriginal ArticlesAldehyde Dehydrogenasemedicine.diseaseMice Inbred C57BLOxidative StressEndocrinologychemistrybiology.proteinEndothelium VascularReactive Oxygen SpeciesOxidative stressDNA DamageCardiovascular research
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