Search results for "VASODILATION"

showing 10 items of 228 documents

Sirolimus-Induced Vascular Dysfunction

2008

Objectives This study sought to analyze mechanisms that mediate vascular dysfunction induced by sirolimus. Background Despite excellent antirestenotic capacity, sirolimus-eluting stents have been found to trigger coronary endothelial dysfunction and impaired re-endothelialization. Methods To mimic the continuous sirolimus exposure of a stented vessel, Wistar rats underwent drug infusion with an osmotic pump for 7 days. Results Sirolimus treatment caused a marked degree of endothelial dysfunction as well as a desensitization of the vasculature to the endothelium-independent vasodilator nitroglycerin. Also, sirolimus stimulated intense transmural superoxide formation as detected by dihydroeth…

medicine.medical_specialtyEndotheliumVasodilationNitric oxidechemistry.chemical_compoundInternal medicinemedicinecardiovascular diseasesEndothelial dysfunctionNADPH oxidaseNicotinamidebiologybusiness.industrySuperoxideequipment and suppliesmedicine.diseasesurgical procedures operativemedicine.anatomical_structureEndocrinologychemistrySirolimuscardiovascular systembiology.proteinCardiology and Cardiovascular Medicinebusinessmedicine.drugJournal of the American College of Cardiology
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Deleterious effect of glycation on the ability of HDL to counteract the inhibitory effect of oxidized LDL on endothelium-dependent vasorelaxation

2013

Background Contrary to high-density lipoprotein (HDL) from normolipidaemic and normoglycaemic subjects, HDL from diabetic patients loses its ability to reverse the inhibition of vasorelaxation induced by oxidized low-density lipoprotein (LDL). The aim of this study was to analyze the role of glycation, a major abnormality observed in diabetes, on the impairment of the vasorelaxant effect of HDL. Methods HDL from healthy subjects was glycated in vitro by incubation in glucose 200 mmol/L for 3 days. Vasoreactivity was evaluated by the relaxation response to acetylcholine of rabbit aorta rings pre-contracted with noradrenaline, before and after 2 h incubation with or without different lipoprot…

medicine.medical_specialtyEndotheliumbusiness.industryEndocrinology Diabetes and MetabolismVasodilationmedicine.diseasechemistry.chemical_compoundEndocrinologymedicine.anatomical_structureEndocrinologyFructosaminechemistryGlycationInternal medicineDiabetes mellitusInternal Medicinemedicinelipids (amino acids peptides and proteins)businessIncubationAcetylcholinemedicine.drugLipoproteinDiabetes/Metabolism Research and Reviews
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The impact of alpha1-adrenoceptors up-regulation accompanied by the impairment of beta-adrenergic vasodilatation in hypertension

2008

9 pages, 7 figures, 3 tables.-- PMID: 19060223 [PubMed]

medicine.medical_specialtyG-Protein-Coupled Receptor Kinase 2Adrenergic receptorSystolemedia_common.quotation_subjectAdrenergicVasodilationModels BiologicalRats Inbred WKYDownregulation and upregulationHeart RateRats Inbred SHRReceptors Adrenergic alpha-1Internal medicineReceptors Adrenergic betamedicineAnimalsHumansRNA MessengerReceptorInternalizationAortamedia_commonPharmacologybiologyChemistryKinaseBeta adrenergic receptor kinaseRatsUp-RegulationVasodilationEndocrinologyHypertensionbiology.proteinMolecular Medicine
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Basal release of nitric oxide in the mesenteric artery in portal hypertension and cirrhosis: Role of dimethylarginine dimethylaminohydrolase

2013

Background and Aim Increased basal release of nitric oxide (NO) in the splanchnic circulation contributes to elevated plasma levels of NO observed in decompensated cirrhosis. We evaluated in rat mesenteric arteries whether the differences in basal release of NO, revealed by asymmetric dimethylarginine (ADMA)- and NG-nitro-L-arginine methyl ester (L-NAME)-induced contractions, were associated with changes in messenger RNA (mRNA) expression of endothelial NO synthase (eNOS) and dimethylarginine dimethylaminohydrolases (DDAHs). Methods Rat small mesenteric arteries from 14 Sham-control, from 14 with partial portal vein ligation (PPVL), and from 14 with bile duct excision (BDE)-induced cirrhosi…

medicine.medical_specialtyHepatologybiologybusiness.industryGastroenterologyVasodilationmedicine.diseasebiology.organism_classificationApaminNitric oxidechemistry.chemical_compoundEndocrinologymedicine.anatomical_structurechemistryEnosInternal medicinemedicinePortal hypertensionbusinessAsymmetric dimethylarginineMesenteric arteriesArteryJournal of Gastroenterology and Hepatology
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Genistein and endothelial function in postmenopausal women with metabolic syndrome

2013

BackgroundPrevious data have suggested that genistein could exert beneficial effects on endothelial function and on predictors of cardiovascular risk in healthy postmenopausal women. In a randomized clinical trial, we studied the effects of genistein on endothelial function in postmenopausal women with metabolic syndrome (MS). MethodsTwenty postmenopausal women with MS, according to modified NCEP-ATP III criteria were randomly assigned to receive placebo or genistein (54mg/day) for 6months, along with a Mediterranean-style diet. Postmenopausal women without MS (n=15), served as controls. The primary goal was the assessment of endothelial function by flow-mediated vasodilation (FMD) of brach…

medicine.medical_specialtyHomocysteineClinical BiochemistryGenisteinmenopausePhytoestrogensPilot ProjectsVasodilationPlaceboBiochemistrymetabolic syndromeClinical studygenisteinchemistry.chemical_compoundendothelial functionmedicine.arteryInternal medicinemedicineHumansAnkle Brachial IndexBrachial arteryAdiponectinbusiness.industryClinical study; Endothelial function; Genistein; Menopause; Metabolic syndromeGeneral MedicineMiddle Agedmedicine.diseasePostmenopauseVasodilationMenopauseTreatment OutcomeEndocrinologychemistryFemaleEndothelium VascularMetabolic syndromebusiness
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Treatment of pulmonary hypertension in patients undergoing cardiac surgery with cardiopulmonary bypass: a randomized, prospective, double-blind study

2006

Pulmonary hypertension can already be present in patients undergoing cardiac surgery or can be exacerbated by cardiopulmonary bypass. Postoperative treatment is still a challenge for physicians. The aim of this study was to evaluate the effects of inhaled prostacyclin (iPGI2) and nitric oxide (iNO) compared with those of intravenous vasodilators.This prospective, randomized, double-blind study included 58 patients affected by severe mitral valve stenosis and pulmonary hypertension with high pulmonary vascular resistance (250 dynes x s x cm(-5)) and a mean pulmonary artery pressure25 mmHg. All patients were monitored by central venous, radial arterial and Swan-Ganz catheters. Data were recor…

medicine.medical_specialtyHypertension PulmonaryHemodynamicsProstacyclinVasodilationNitric Oxidelaw.inventionDouble-Blind MethodRandomized controlled triallawInternal medicineAdministration InhalationCardiopulmonary bypassmedicineHumansMitral Valve StenosisProspective StudiesProspective cohort studyAntihypertensive AgentsAgedCardiopulmonary Bypassbusiness.industryfungiHemodynamicsfood and beveragesGeneral MedicineLength of StayMiddle Agedmedicine.diseaseEpoprostenolPulmonary hypertensionBronchodilator AgentsCardiac surgeryAnesthesiaCardiologyCardiology and Cardiovascular Medicinebusinessmedicine.drugJournal of Cardiovascular Medicine
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Effects of Long-Term Nitroglycerin Treatment on Endothelial Nitric Oxide Synthase (NOS III) Gene Expression, NOS III–Mediated Superoxide Production, …

2000

Abstract —Long-term nitroglycerin (NTG) treatment has been shown to be associated with cross-tolerance to endothelium-dependent vasodilators. It may involve increased production of reactive oxygen species (such as superoxide, O 2 ·− ) that rapidly inactivate the nitric oxide (NO) released from the endothelial cells. It remains to be elucidated, however, whether long-term treatment with NTG alters the activity and expression of the endothelial NO synthase (NOS III) and whether this enzyme can contribute to O 2 ·− formation. We studied the influence of long-term NTG treatment on the expression of NOS III as assessed by RNase protection assay and Western blot. Tolerance was measured ex vivo i…

medicine.medical_specialtyIndolesNitric Oxide Synthase Type IIIPhysiologyCarbazolesBiological AvailabilityVasodilationArginineNitric OxideGene Expression Regulation EnzymologicTimeNitric oxideNitroglycerinchemistry.chemical_compoundAlkaloidsSuperoxidesInternal medicinemedicineAnimalsRNA MessengerLucigeninCloning MolecularEnzyme InhibitorsRats WistarCalcimycinProtein Kinase CProtein kinase CBenzophenanthridineschemistry.chemical_classificationReactive oxygen speciesSuperoxideAcetylcholinePhenanthridinesRatsVasodilationEndocrinologychemistryBiochemistryEndothelium VascularNitric Oxide SynthaseCardiology and Cardiovascular MedicineEx vivoAcetylcholinemedicine.drugCirculation Research
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LEUKOTRIENE RECEPTORS ON HUMAN PULMONARY VASCULAR ENDOTHELIUM

1995

1. Cysteinyl-leukotrienes cause contractions and/or relaxations of human isolated pulmonary vascular preparations. Although, the localization and nature of the receptors through which these effects are mediated have not been fully characterized, some effects are indirect and not mediated via the well-described LT1 receptor. 2. In human pulmonary veins (HPV) with an intact endothelium, leukotriene D4 (LTD4) induced contraction above basal tone. This response was observed at lower concentrations of LTD4 in the presence of nitric oxide synthase inhibitor N omega-nitro-L-arginine (L-NOARG). Contractions (in the absence and presence of L-NOARG) were partially blocked by the LT1 antagonists (MK 5…

medicine.medical_specialtyLeukotriene D4EndotheliumVasodilationPulmonary ArteryArginineNitroarginineMuscle Smooth VascularNitric oxideLeukotriene D4Nitroargininechemistry.chemical_compoundNorepinephrineInternal medicinemedicineHumansEnzyme InhibitorsReceptorPharmacologyReceptors LeukotrieneLeukotrieneAnalysis of Variancebiologyrespiratory systemNitric oxide synthaseVasodilationmedicine.anatomical_structureEndocrinologychemistryPulmonary VeinsVasoconstrictionbiology.proteinlipids (amino acids peptides and proteins)Endothelium VascularResearch Article
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Endotheliale Dysfunktion: Pathophysiologie, Diagnostik und prognostische Bedeutung

2008

The endothelium plays a crucial role in the regulation of vascular tone. Recent studies have indicated that endothelial dysfunction develops in the presence of cardiovascular risk factors such as hypertension, diabetes mellitus, hypercholesterolemia and in chronic smokers, as well as in patients with a family history of cardiovascular disease. It has now been established that endothelial dysfunction represents the first indicator of vascular damage. Endothelial function can be assessed in coronary and peripheral conductance and resistance vessels by means of invasive and noninvasive (ultrasound-guided) methods such as intracoronary infusion of acetylcholine, the endothelium-dependent vasodi…

medicine.medical_specialtyNADPH oxidaseEndotheliumbiologybusiness.industrySuperoxideVasodilationGeneral Medicinemedicine.diseasemedicine.disease_causeNitric oxide synthasechemistry.chemical_compoundEndocrinologymedicine.anatomical_structurechemistryInternal medicinebiology.proteinMedicineEndothelial dysfunctionbusinessXanthine oxidaseOxidative stressDMW - Deutsche Medizinische Wochenschrift
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NADPH Oxidase Accounts for Enhanced Superoxide Production and Impaired Endothelium-Dependent Smooth Muscle Relaxation in BKβ1 −/− Mice

2006

Objective— Nitric oxide (NO)-induced vasorelaxation involves activation of large conductance Ca 2+ -activated K + channels (BK). A regulatory BKβ1 subunit confers Ca 2+ , voltage, and NO/cGMP sensitivity to the BK channel. We investigated whether endothelial function and NO/cGMP signaling is affected by a deletion of the β1-subunit. Methods and Results— Vascular superoxide in BKβ1 −/− was measured using the fluorescent dye hydroethidine and lucigenin-enhanced chemiluminescence. Vascular NO formation was analyzed using electron paramagnetic resonance (EPR), expression of NADPH oxidase subunits, the endothelial NO synthase (eNOS), the soluble guanylyl cyclase (sGC), as well as the activity a…

medicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumAorta ThoracicNitric OxideMuscle Smooth VascularNitric oxideMicechemistry.chemical_compoundSuperoxidesInternal medicineCyclic GMP-Dependent Protein KinasesmedicineAnimalsHumansProtein IsoformsNADH NADPH OxidoreductasesLarge-Conductance Calcium-Activated Potassium ChannelsMice KnockoutNADPH oxidasebiologySuperoxideMicrofilament ProteinsNADPH OxidasesPhosphoproteinsMolecular biologyVasodilationEndocrinologymedicine.anatomical_structurechemistryGuanylate CyclaseNAD(P)H oxidaseNOX1ApocyninNADPH Oxidase 1biology.proteinEndothelium VascularCardiology and Cardiovascular MedicineSoluble guanylyl cyclaseCell Adhesion MoleculesSignal TransductionArteriosclerosis, Thrombosis, and Vascular Biology
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