Search results for "VOD"

showing 10 items of 84 documents

Deep Brain Stimulation and L-DOPA Therapy: Concepts of Action and Clinical Applications in Parkinson's Disease.

2018

L-DOPA is still the most effective pharmacological therapy for the treatment of motor symptoms in Parkinson's disease (PD) almost four decades after it was first used. Deep brain stimulation (DBS) is a safe and highly effective treatment option in patients with PD. Even though a clear understanding of the mechanisms of both treatment methods is yet to be obtained, the combination of both treatments is the most effective standard evidenced-based therapy to date. Recent studies have demonstrated that DBS is a therapy option even in the early course of the disease, when first complications arise despite a rigorous adjustment of the pharmacological treatment. The unique feature of this therapeu…

0301 basic medicineLevodopaParkinson's diseaseDeep brain stimulationglobus pallidus internus (GPi)medicine.medical_treatmentParkinson's diseaseCentral nervous systemStimulationDiseaseReviewlcsh:RC346-42903 medical and health sciencesTherapeutic approach0302 clinical medicinemedicinelevodopadeep brain stimulation (DBS)lcsh:Neurology. Diseases of the nervous systembusiness.industryDopaminergicmedicine.diseasenervous system diseases030104 developmental biologymedicine.anatomical_structureNeurologyNeurology (clinical)businessNeurosciencesubthalamic nucleus (STN)030217 neurology & neurosurgerymedicine.drugFrontiers in neurology
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Medicinal Plants and Natural Products as Potential Sources for Antiparkinson Drugs

2016

Parkinsonʼs disease is a progressive neurodegenerative dysfunction characterized by the loss of pigmented dopaminergic neurons of the nigrostriatal system with a consequent dopamine decrease. The reduction of dopamine levels produces neuronal damage, depigmentation of the substantia nigra, and the presence of intracellular inclusions in dopaminergic neurons. Treatments for Parkinsonʼs disease aim for improving these motor symptoms by increasing the dopaminergic signal in the striatum with levodopa in combination with enzyme inhibitors or anticholinergic drugs. Nevertheless, natural products can act as neuroprotective agents by reducing the progression of the disease and the inflammatory pro…

0301 basic medicineLevodopaPharmaceutical ScienceSubstantia nigraPharmacologyBiologyNeuroprotectionAnalytical ChemistryAntiparkinson Agents03 medical and health sciences0302 clinical medicineDopamineDrug DiscoverymedicineAnimalsHumansPharmacologyBiological ProductsPlants MedicinalPlant ExtractsOrganic ChemistryDopaminergicAntiparkinsonian AgentAntiparkinson drug030104 developmental biologynervous systemComplementary and alternative medicineAntiparkinson AgentsMolecular Medicine030217 neurology & neurosurgeryPhytotherapymedicine.drugPlanta Medica
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Pre-dopa Deep Brain Stimulation: Is Early Deep Brain Stimulation Able to Modify the Natural Course of Parkinson’s Disease?

2020

Deep brain stimulation (DBS) is an established therapy for the management of Parkinson's disease (PD). However, DBS is indicated as the disease progresses and motor complications derived from pharmacological therapy arise. Here, we evaluate the potential of DBS prior to levodopa (L-Dopa) in improving quality of life (QoL), challenging the state of the art for DBS therapy. We present data on clinical manifestation, decision finding during early indication to DBS, and trajectories after DBS. We further discuss current paradigms for DBS and hypothesize on possible mechanisms. Six patients, between 50 and 67 years old, presenting at least 5 years of PD symptoms, and without L-Dopa therapy initi…

0301 basic medicineLevodopamedicine.medical_specialtyDeep brain stimulationParkinson's diseasemedicine.medical_treatmentDiseaselcsh:RC321-57103 medical and health sciences0302 clinical medicinePhysical medicine and rehabilitationQuality of lifemedicineAdverse effectlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal Researchsubthalamic nucleusbusiness.industryGeneral Neuroscienceearly deep brain stimulationmedicine.diseasenervous system diseasesdeep brain stimulationClinical trialSubthalamic nucleusearly intervention030104 developmental biologysurgical procedures operativenervous systemParkinson’s diseasebusinesstherapeutics030217 neurology & neurosurgerymedicine.drugNeuroscienceFrontiers in Neuroscience
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Attenuated beta rebound to proprioceptive afferent feedback in Parkinson's disease.

2018

AbstractMotor symptoms are defining traits in the diagnosis of Parkinson’s disease (PD). A crucial component in motor function and control of movements is the integration of efferent signals from the motor network to the peripheral motor system, and afferent proprioceptive sensory feedback. Previous studies have indicated abnormal movement-related cortical oscillatory activity in PD, but the role of the proprioceptive afference on abnormal oscillatory activity in PD has not been elucidated. In the present study, we examine the role of proprioception by studying the cortical processing of proprioceptive stimulation in PD patients, ON/OFF levodopa medication, as compared to that of healthy co…

0301 basic medicineMaleParkinson's diseaselcsh:MedicineStimulationAntiparkinson AgentsLevodopa0302 clinical medicineFeedback SensoryMedicine:Science::Medicine [DRNTU]lcsh:Science0303 health sciencesMultidisciplinarymedicine.diagnostic_testMotor CortexMagnetoencephalographyParkinson DiseaseMiddle Aged3. Good healthProprioceptive functioncortexmedicine.anatomical_structureFemaleMotor cortexmedicine.drugAdultLevodopaParkinsonin tautiSensory systemArticle03 medical and health sciencesmotor cortexMotor systemHumans030304 developmental biologyAgedProprioceptionbusiness.industrylcsh:RMagnetoencephalographyIndex fingermedicine.diseaseProprioceptionaivokuori030104 developmental biologylcsh:QbusinessBeta RhythmNeuroscience030217 neurology & neurosurgeryScientific reports
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Effect of high-frequency subthalamic neurostimulation on gait and freezing of gait in Parkinson's disease: a systematic review and meta-analysis.

2016

The aim of this meta-analysis was to summarize the short- and long-term effects of bilateral deep brain stimulation of the subthalamic nucleus (STN-DBS) on gait and freezing of gait (FOG) in Parkinson's disease and to detect predictors of post-stimulation outcome. A comprehensive review of the literature was conducted up to October 2015 using Medline Ovid databases for studies analyzing the effect of bilateral STN-DBS on FOG and/or gait. Sixteen studies with available data for the gait item (no. 29) of the Unified Parkinson's Disease Rating Scale (UPDRS) and six studies with the FOG item (no. 14) were included. Data were summarized for the following follow-up periods: 6-15, 24-48 and48 mont…

0301 basic medicinemedicine.medical_specialtyLevodopaDeep brain stimulationParkinson's diseasemedicine.medical_treatmentDeep Brain Stimulation03 medical and health sciences0302 clinical medicineGait (human)Physical medicine and rehabilitationRating scaleSubthalamic NucleusmedicineHumansNeurostimulationGaitGait Disorders Neurologicbusiness.industryParkinson Diseasemedicine.diseasenervous system diseasesSubthalamic nucleus030104 developmental biologyTreatment OutcomeNeurologyMeta-analysisPhysical therapyNeurology (clinical)business030217 neurology & neurosurgerymedicine.drugEuropean journal of neurology
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Vulvodynia¿It Is Time to Accept a New Understanding from a Neurobiological Perspective

2021

Vulvodynia is one the most common causes of pain during sexual intercourse in premenopausal women. The burden of vulvodynia in a woman’s life can be devastating due to its consequences in the couple’s sexuality and intimacy, in activities of daily living, and psychological well-being. In recent decades, there has been considerable progress in the understanding of vulvar pain. The most significant change has been the differentiation of vulvar pain secondary to pathology or disease from vulvodynia. However, although it is currently proposed that vulvodynia should be considered as a primary chronic pain condition and, therefore, without an obvious identifiable cause, it is still believed that …

Activities of daily livingcognitive-behavioral therapyFisioteràpiaVulvodyniaHealth Toxicology and Mutagenesismedicine.medical_treatmentSexual BehaviorHuman sexualityDiseaseReview03 medical and health sciences0302 clinical medicineActivities of Daily LivingmedicineHumans030219 obstetrics & reproductive medicinebusiness.industryRPublic Health Environmental and Occupational HealthChronic painCoituscentral sensitization painmedicine.diseaseCognitive behavioral therapySexual intercourseSexual PartnersMedicineVulvodyniaFemalecomplex painbusinessPsychosocial030217 neurology & neurosurgeryClinical psychology
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Effects of levodopa oral bolus on the kinematics of the pointing movements in Parkinson's disease patients.

2005

We studied the time-course of a levodopa oral bolus effects on the kinematics of patients affected by a mild akinetic-rigid form of idiopathic Parkinson's disease (PD). Eleven PD patients were evaluated: a) in OFF-state, that is before their first medication or after its withdrawal, b) in ON-state, that is at 1/2, 1, 2, 3, 4, 5, 6, 24, 30 and 48 hours after the administration of 250 mg of levodopa plus 25mg of carbidopa. The main kinematics (i. e.movement time, peak of velocity, peak of acceleration and peak of deceleration) of pointing movements to six target-stimuli placed on the horizontal plane of a table were recorded. Clinical conditions were assessed according to the Motor Examinatio…

AdultMaleLevodopamedicine.medical_specialtyTime FactorsNeurologyParkinson's diseaseAdministration Oralparkinson's diesease clinical neurophysiology kinematicsKinematicsClinical neurophysiologyAntiparkinson AgentsLevodopaCentral nervous system diseaseBolus (medicine)Internal medicinemedicineHumansAgedbusiness.industryParkinson DiseaseMiddle Agedmedicine.diseaseBiomechanical PhenomenaSurgeryNeurologyCarbidopaCardiologyFemaleSettore MED/26 - NeurologiaNeurology (clinical)businessPsychomotor Performancemedicine.drug
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Increased Neural Activity in Mesostriatal Regions after Prefrontal Transcranial Direct Current Stimulation and L-DOPA Administration

2019

Dopamine dysfunction is associated with a wide range of neuropsychiatric disorders commonly treated pharmacologically or invasively. Recent studies provide evidence for a nonpharmacological and noninvasive alternative that allows similar manipulation of the dopaminergic system: transcranial direct current stimulation (tDCS). In rodents, tDCS has been shown to increase neural activity in subcortical parts of the dopaminergic system, and recent studies in humans provide evidence that tDCS over prefrontal regions induces striatal dopamine release and affects reward-related behavior. Based on these findings, we used fMRI in healthy human participants and measured the fractional amplitude of low…

AdultMaleLevodopamedicine.medical_treatmentDopaminePrefrontal CortexTranscranial Direct Current StimulationLevodopa03 medical and health sciencesNeural activitySpatial similarityYoung Adult0302 clinical medicineDopamineMedicineAnimalsHumansSingle-Blind MethodResearch Articles030304 developmental biologyNeurons0303 health sciencesBrain MappingResting state fMRITranscranial direct-current stimulationbusiness.industryReceptors Dopamine D2General NeuroscienceReceptors Dopamine D1DopaminergicMagnetic Resonance ImagingCorpus StriatumDopamine receptorRats Inbred LewFemalebusinessNeuroscience030217 neurology & neurosurgerymedicine.drug
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Single dose of l-dopa makes extinction memories context-independent and prevents the return of fear

2013

Traumatic events can engender persistent excessive fear responses to trauma reminders that may return even after successful treatment. Extinction, the laboratory analog of behavior therapy, does not erase conditioned fear memories but generates competing, fear-inhibitory "extinction memories" that, however, are tied to the context in which extinction occurred. Accordingly, a dominance of fear over extinction memory expression--and, thus, return of fear--is often observed if extinguished fear stimuli are encountered outside the extinction (therapy) context. We show that postextinction administration of the dopamine precursor L-dopa makes extinction memories context-independent, thus strongly…

AdultMaleVentromedial prefrontal cortexPrefrontal CortexContext (language use)AmygdalaDevelopmental psychologyExtinction PsychologicalLevodopaMiceMemorymedicineAnimalsHumansFear conditioningPrefrontal cortexFear processing in the brainMultidisciplinarysocial sciencesExtinction (psychology)FearMiddle AgedAmygdalahumanitiesMice Inbred C57BLmedicine.anatomical_structurePNAS PlusAnxietymedicine.symptomPsychologyNeuroscience
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Study of pituitary secretion in relation to retinopathy in patients with juvenile diabetes mellitus.

1981

Fifteen juvenile diabetic patients with normal eye fundus, 6 with non proliferative retinopathy, 5 with proliferative retinopathy and 5 healthy control subjects were studied in order to investigate pituitary function in relation to diabetic retinopathy. ACTH values at 08(00) and 18(00), hPRL and TSh secretion in response to 200 microgram TRH i.v., and GH secretion in response to 500 mg oral L-dopa were evaluated. In all diabetic subjects, 08(00) ACTH levels were lower than in controls. Basal hPRL, TSH and GH values of the diabetics did not differ from those of the controls. No significant differences were found in hPRL levels in response to TRH, whereas significantly lower TSH responses wer…

AdultMaleendocrine systemPituitary glandmedicine.medical_specialtyAdolescentEndocrinology Diabetes and MetabolismThyrotropin-releasing hormoneThyrotropinAdrenocorticotropic hormoneLevodopaEndocrinologyAdrenocorticotropic HormoneInternal medicineDiabetes mellitusInternal MedicineMedicineHumansThyrotropin-Releasing HormoneDiabetic Retinopathybusiness.industryGeneral MedicineDiabetic retinopathymedicine.diseaseGrowth hormone secretionProlactinProlactinKineticsmedicine.anatomical_structureEndocrinologyDiabetes Mellitus Type 1Growth HormonePituitary GlandFemalebusinesshormones hormone substitutes and hormone antagonistsRetinopathyActa diabetologica latina
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