Search results for "Vasoconstriction"

showing 10 items of 134 documents

Catecholamine release in human skin--a microdialysis study.

2003

Dermal microdialysis might be a promising tool to investigate properties of sympathetic neurons in the skin as investigation of peripheral noradrenergic neurons in humans usually relies on highly variable vasoconstrictor reflexes or on indirect measurements like skin temperature recordings. To evaluate this technique, 21 experiments were performed in 15 healthy subjects with four intracutaneous microdialysis fibers (diameter, 200 microm; cutoff, 5 kDa) at hands or feet. After 60 min, saline perfusion tyramine at concentrations of 0.195 to 200 microg/ml was applied for 15 min followed by a 15-min saline perfusion again. Catecholamine concentrations were detected through high-performance liqu…

AdultMaleMicrodialysisSympathetic nervous systemmedicine.medical_specialtyDopamineMicrodialysisPresynaptic TerminalsTyramineHuman skinSweatingNorepinephrinechemistry.chemical_compoundNorepinephrineCatecholaminesSympathetic Fibers PostganglionicDevelopmental NeuroscienceInternal medicinemedicineHumansSkinDose-Response Relationship DrugChemistrySecretory VesiclesTyramineAxonsUp-RegulationEpinephrinemedicine.anatomical_structureEndocrinologyNeurologyVasoconstrictionCatecholamineFemalePerfusionmedicine.drugExperimental neurology
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Contractile effects of arginine analogues on human internal thoracic and radial arteries

2000

Abstract Objectives: Plasma levels of endogenous guanidino-substituted analogues of L -arginine are increased in various pathologic conditions. In the present study we determined the effects of some of these compounds on basal and stimulated release of nitric oxide in human internal thoracic and radial arteries. Methods: Rings of human internal thoracic and radial arteries were obtained from 16 multiorgan donors. The rings were suspended in organ baths for isometric recording of tension. Results: N G -monomethyl L -arginine (10 –6 to 10 –3 mol/L) and N G ,N G -dimethyl L -arginine (10 –6 to 10 –3 mol/L) caused concentration- and endothelium-dependent contractions. Maximal force of contracti…

AdultMalePulmonary and Respiratory Medicinemedicine.medical_specialtyContraction (grammar)AdolescentArginineInternal thoracic arteryIn Vitro TechniquesArginineNitric OxideGuanidinesNitric oxidechemistry.chemical_compoundThoracic ArteriesInternal medicinemedicine.arteryMolemedicineMethylguanidineHumansVasoconstrictor AgentsAnalysis of Variancebusiness.industryAnatomyMiddle AgedEndocrinologychemistryRadial ArteryRegression AnalysisFemaleSurgerymedicine.symptomCardiology and Cardiovascular MedicinebusinessVasoconstrictionAcetylcholinemedicine.drugThe Journal of Thoracic and Cardiovascular Surgery
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Comparison of the anticonstrictor action of dihydropyridines (nimodipine and nicardipine) and Mg2+ in isolated human cerebral arteries.

1992

The isometric tension recorded from ring segments of branches of human middle cerebral artery was the parameter used to study the inhibition of spasmogen-induced contractions as model for cerebral vasospasm. Concentration-response curves to 5-hydroxytryptamine (10(-9)-3 x 10(-5) M) and prostaglandin F2 alpha (10(-7)-3 x 10(-5) M) were inhibited in Ca(2+)-free medium and in Ca(2+)-free medium to which EGTA (1 mM) had been added, respectively. Nimodipine (10(-7), 10(-5) M), nicardipine (10(-7), 10(-5) M) and Mg2+ (magnesium sulfate 10(-4), 10(-2) M) inhibited the 5-HT-elicited contractions, and this inhibition was similar for the highest concentrations tested. In contrast, nimodipine and nica…

AdultMaleSerotoninNicardipineCerebral arteriesProstaglandinPharmacologyIn Vitro TechniquesDinoprostchemistry.chemical_compoundNicardipineCerebral vasospasmmedicine.arterymedicineHumansMagnesiumNimodipineAgedPharmacologyAged 80 and overChemistryCerebral ArteriesMiddle AgedEGTAIschemic Attack TransientVasoconstrictionAnesthesiaMiddle cerebral arteryCirculatory systemCalciumFemaleNimodipinemedicine.drugEuropean journal of pharmacology
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Contractile responses of human thyroid arteries to vasopressin

2013

Abstract Aims In the present study we investigated the intervention of nitric oxide and prostacyclin in the responses to vasopressin of isolated thyroid arteries obtained from multi-organ donors. Main methods Paired artery rings from glandular branches of the superior thyroid artery, one normal and the other deendothelised, were mounted in organ baths for isometric recording of tension. Concentration–response curves to vasopressin were determined in the absence and in the presence of either the vasopressin V 1 receptor antagonist d(CH 2 ) 5 Tyr(Me)AVP (10 − 8  M), the nitric oxide synthase inhibitor N G -monomethyl- l -arginine ( L -NMMA, 10 − 4  M), or the inhibitor of prostaglandins indom…

AdultMaleVasopressinmedicine.medical_specialtyEndotheliumVasopressinsIndomethacinThyroid GlandProstacyclinIn Vitro TechniquesGeneral Biochemistry Genetics and Molecular BiologyNitric oxideSuperior thyroid arterychemistry.chemical_compoundmedicine.arteryInternal medicinemedicineHumansDrug InteractionsGeneral Pharmacology Toxicology and PharmaceuticsAgedVasopressin receptoromega-N-MethylarginineDose-Response Relationship DrugbiologyChemistryThyroidArteriesGeneral MedicineMiddle AgedArginine VasopressinNitric oxide synthaseEndocrinologymedicine.anatomical_structureVasoconstrictionbiology.proteinFemalehormones hormone substitutes and hormone antagonistsmedicine.drugLife Sciences
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Endothelium-independent contractions of human cerebral arteries in response to vasopressin.

1990

We studied the effects of vasopressin in isolated segments from branches (500-700 micrograms in external diameter) of human middle cerebral arteries obtained during autopsy of 15 patients who had died 3-8 hours before. Paired segments, one normal and the other de-endothelized by gentle rubbing, were mounted for isometric recording of tension in organ baths. In 11 normal segments, vasopressin produced concentration-dependent contractions with an EC50 of 7.0 X 10(-10) M. Removal of the endothelium from 12 segments did not significantly affect vasopressin-induced contractions. Vasopressin produced further contractions in arterial segments with (n = 4) or without (n = 5) endothelium precontract…

AdultMaleVasopressinmedicine.medical_specialtyEndotheliummedicine.drug_classVasopressinsCerebral arteriesNeuropeptideIn Vitro TechniquesPotassium Chloridemedicine.arteryInternal medicinemedicineHumansAgedAdvanced and Specialized NursingDose-Response Relationship Drugbusiness.industryOsmolar ConcentrationCerebral ArteriesMiddle AgedReceptor antagonistAcetylcholineArginine Vasopressinmedicine.anatomical_structureEndocrinologyVasoconstrictionMiddle cerebral arteryFemaleNeurology (clinical)Endothelium Vascularmedicine.symptomCardiology and Cardiovascular MedicinebusinessAcetylcholineVasoconstrictionmedicine.drugStroke
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Effects of vasopressin on human renal arteries

1996

The effects of vasopressin were studied in isolated rings from branches (2-3 mm in external diameter) of human renal arteries obtained from 18 patients undergoing nephrectomy for non-obstructive neoplasia. In arterial rings under resting tension, vasopressin produced concentration-dependent and endothelium-independent contractions with an EC 50 of 9.1 x 10 -10 mol L -1 . The vasopressin V 1 receptor antagonist d(CH 2 ) 5 Tyr(Me)AVP (10 -6 mol L -1 ) displaced the control curve to vasopressin 564-fold to the right in a parallel manner. In precontracted arterial rings and previously treated with the V 1 antagonist (10 -6 mol L -1 ) vasopressin caused endothelium-independent relaxation. The re…

AdultMaleVasopressinmedicine.medical_specialtyVasopressinsmedicine.drug_classMuscle RelaxationIndomethacinClinical BiochemistryNeuropeptideBiologyBiochemistryNorepinephrineRenal ArteryInternal medicinemedicineHumansVasoconstrictor AgentsAgedVasopressin receptorKidneyDose-Response Relationship DrugAnti-Inflammatory Agents Non-SteroidalAntagonistGeneral MedicineMiddle AgedReceptor antagonistmedicine.anatomical_structureEndocrinologyCirculatory systemFemaleEndothelium Vascularmedicine.symptomhormones hormone substitutes and hormone antagonistsVasoconstrictionMuscle ContractionEuropean Journal of Clinical Investigation
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Effects of sildenafil on human penile blood vessels.

2000

Abstract Objectives. To investigate the effects of sildenafil on human penile blood vessels and evaluate the interaction of sildenafil with neurogenic-mediated responses. Sildenafil is currently used in the treatment of erectile dysfunction. Methods. Penile dorsal arteries and deep dorsal veins were obtained from 14 multiorgan donors. Vascular rings were suspended in organ bath chambers, and the isometric tension was recorded. We then studied the effects of sildenafil on precontracted vessels and the neurogenic (noradrenergic and nitrergic) responses. Results. Sildenafil (10 −9 to 3 × 10 −6 M) caused concentration-dependent relaxation and amplified the relaxation induced by sodium nitroprus…

AdultMalemedicine.medical_specialtyAdolescentSildenafilPhosphodiesterase InhibitorsUrologyMuscle RelaxationPiperazinesSildenafil CitrateNitric oxideVeinschemistry.chemical_compoundInternal medicineMedicineHumansSulfonesGuanethidinebusiness.industrySmooth muscle contractionArteriesMiddle AgedPDE5 drug designrespiratory tract diseasesVasodilationEndocrinologymedicine.anatomical_structurechemistryPurinesVasoconstrictioncardiovascular systemSodium nitroprussidebusinessZaprinastBlood vesselmedicine.drugMuscle ContractionPenisUrology
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Endothelium-dependent component in the contractile responses of human omental arteries to adrenergic stimulation

1993

Abstract The present study was designed to investigate the influence of endothelium-derived nitric oxide on the contractile responses of isolated human omental arteries to electrical field stimulation and noradrenaline. We measured isometric tension in artery rings obtained from portions of human omentum during the course of abdominal operations (32 patients). Electrical field stimulation induced frequency-dependent contractions which were abolished by tetrodotoxin (10−6 M) and prazosin (10−6 M), thus indicating that this effect was due to noradrenaline released from adrenergic nerves acting on α1-adrenoceptors. The increases in tension induced by electrical field stimulation were of greate…

AdultMalemedicine.medical_specialtyArginineEndotheliumIndomethacinTetrodotoxinIn Vitro TechniquesArginineNitric OxideMuscle Smooth VascularNitric oxideNorepinephrinechemistry.chemical_compoundInternal medicinemedicinePrazosinHumansAgedPharmacologybusiness.industryStereoisomerismArteriesMiddle AgedElectric StimulationNG-Nitroarginine Methyl EsterEndocrinologymedicine.anatomical_structurechemistryVasoconstrictionCirculatory systemTetrodotoxinFemaleEndothelium VascularbusinessOmentumMuscle Contractionmedicine.drugBlood vesselArteryEuropean Journal of Pharmacology
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Role of endothelium and calcium channels in endothelin-induced contraction of human cerebral arteries

1990

Endothelin constricted human isolated cerebral arteries in a concentration-dependent manner. The maximal tension developed, as well as EC50 values were similar in arteries with and without endothelium. Removal of extracellular calcium or addition of the calcium antagonist nicardipine (10(-6)M), attenuated but did not abolish responses to endothelin. These experiments show that the endothelin-induced contraction in human cerebral arteries is not linked to the presence of intact endothelial cells. The data also show that the contractile effects of endothelin cannot be explained solely by an action on voltage-dependent calcium channels.

AdultMalemedicine.medical_specialtyCerebral arterieschemistry.chemical_elementIn Vitro TechniquesCalciumMuscle Smooth VascularNicardipineInternal medicinemedicineHumansAgedPharmacologyVoltage-dependent calcium channelbusiness.industryEndothelinsT-type calcium channelAnatomyCerebral ArteriesMiddle AgedEndocrinologychemistryCirculatory systemcardiovascular systemFemaleCalcium ChannelsEndothelium Vascularmedicine.symptomPeptidesEndothelin receptorbusinessVasoconstrictionMuscle ContractionResearch ArticleMuscle contraction
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Endothelium-dependent responses in human isolated thyroid arteries from donors

2004

The functional properties of the endothelium of human thyroid arteries remain unexplored. We investigated the intervention of nitric oxide (NO), prostacyclin (PGI(2)) and endothelium-derived hyperpolarizing factor (EDHF) in the responses to acetylcholine and noradrenaline in isolated thyroid arteries obtained from multi-organ donors. Artery rings were suspended in organ baths for isometric recording of tension. The contribution of NO, PGI(2) and EDHF to endothelium-dependent relaxation was determined by the inhibitory effects of N(G)-monomethyl-L-arginine (L-NMMA), indomethacin, and K(+) channel inhibitors respectively. Acetylcholine induced concentration-dependent relaxation; this effect w…

AdultMalemedicine.medical_specialtyCharybdotoxinCharybdotoxinEndotheliumEndocrinology Diabetes and MetabolismIndomethacinThyroid GlandProstacyclinNitric OxideApaminOuabainNitric oxideBiological FactorsNorepinephrinechemistry.chemical_compoundEndocrinologyCulture TechniquesInternal medicinePotassium Channel BlockersmedicineHumansomega-N-MethylarginineDose-Response Relationship DrugArteriesMiddle AgedEpoprostenolAcetylcholineEndocrinologymedicine.anatomical_structureApaminchemistryVasoconstrictioncardiovascular systemFemaleEndothelium VascularNitric Oxide SynthaseSodium-Potassium-Exchanging ATPasePeptidesAcetylcholinemedicine.drugArteryJournal of Endocrinology
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