Search results for "Vasoconstrictor Agent"

showing 10 items of 62 documents

Intensive care unit strain should not rush physicians into making inappropriate decisions, but merely reduce the time to the right decisions being ma…

2016

The effect of capacity strain in an ICU on the timing of end-of-life decision-making is unknown. We sought to determine how changes in strain impact timing of new do-not-resuscitate (DNR) orders and of death.Retrospective cohort study of 9891 patients dying in the hospital following an ICU stay ≥72 h in Project IMPACT, 2001-2008. We examined the effect of ICU capacity strain (measured by standardized census, proportion of new admissions, and average patient acuity) on time to initiation of DNR orders and time to death for all ICU decedents using fixed-effects linear regression.Increases in strain were associated with shorter time to DNR for patients with limitations in therapy (predicted ti…

MaleTime FactorsDatabases Factualcommunication strategySeverity of Illness Indexlaw.invention0302 clinical medicinelawicuVasoconstrictor Agents030212 general & internal medicineHospital MortalityComputingMilieux_MISCELLANEOUSmedia_commonResuscitation OrdersAged 80 and overTerminal CaresupportGeneral MedicineMiddle AgedIntensive care unit3. Good healthIntensive Care UnitsEditorialqualityFemaleMedical emergencyof-life practicesAutonomyAdultmedicine.medical_specialtymedia_common.quotation_subjectCritical IllnessDecision Making03 medical and health sciencesQuality of life (healthcare)[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologymedicineendHumansQuality (business)surrogateIntensive care medicineAgedRetrospective Studiescapacity strainbusiness.industry030208 emergency & critical care medicinefamily membersLength of Staymedicine.diseaseRespiration ArtificialcultureLinear Modelsbusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Sex-dependent changes in the pulmonary vasoconstriction potential of newborn rats following short-term oxygen exposure

2012

Chronic exposure to supplemental oxygen (O(2)) induces lung damage and mortality in a sex-dependent manner. The effect of short-term hyperoxia on the newborn pulmonary vasculature is unknown but is, however, of clinical significance in the neonatal resuscitation context. We hypothesize that short-term hyperoxia has a sex-dependent effect on the pulmonary vasculature.Following 1-h 100% O(2) exposure, the pulmonary arteries and lung tissues of newborn rats were evaluated.Superoxide dismutase 3 (SOD3) expression in female pups' lungs was increased as compared with that in the lungs of male pups. As compared with air-treated pups, the response of male pups to thromboxane was increased by O(2), …

MaleTime FactorsHypertension PulmonaryHyperoxiaPulmonary ArteryRats Sprague-DawleySex FactorsSuperoxidesPeroxynitrous AcidHypoxic pulmonary vasoconstrictionAnimalsVasoconstrictor AgentsMedicineFamilial Primary Pulmonary Hypertensionskin and connective tissue diseasesOXYGEN EXPOSURELungrho-Associated KinasesDose-Response Relationship DrugSuperoxide Dismutasebusiness.industryFree Radical ScavengersHydrogen PeroxideRatsUp-RegulationEnzyme ActivationDisease Models AnimalOxidative StressAnimals NewbornVasoconstrictionAnesthesiaPediatrics Perinatology and Child HealthFemalesense organsbusinessPediatric Research
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A critical role for TNFα in the selective attachment of mononuclear leukocytes to angiotensin-II-stimulated arterioles

2007

Abstract Angiotensin II (Ang-II) exerts inflammatory activity and is involved in different cardiovascular disorders. This study has evaluated the involvement of tumor necrosis factor alpha (TNFα) in the leukocyte accumulation elicited by Ang-II. Ang-II (1 nM intraperitoneally in rats) induced TNFα release at 1 hour followed by neutrophil and mononuclear cell recruitment. The administration of an antirat TNFα antiserum had no effect on Ang-IIinduced neutrophil accumulation but inhibited the infiltration of mononuclear cells and reduced CC chemokine content in the peritoneal exudate. Pretreatment with either an anti-TNFα or an anti-IL-4 antiserum decreased Ang-II-induced arteriolar mononuclea…

MaleUmbilical Veinsmedicine.medical_specialtyEndotheliummedicine.medical_treatmentImmunologyBiologyBiochemistryPeripheral blood mononuclear cellMicrocirculationRats Sprague-DawleyInternal medicineCell AdhesionmedicineAnimalsHumansVasoconstrictor AgentsRNA MessengerVenuleReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaAngiotensin IICell BiologyHematologyAngiotensin IIRatsArteriolesMononuclear cell infiltrationmedicine.anatomical_structureCytokineEndocrinologyLeukocytes MononuclearTumor necrosis factor alphaEndothelium VascularInterleukin-4ChemokinesInjections IntraperitonealBlood
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Role of central oxytocin in the inhibition by endotoxin of distension-stimulated gastric acid secretion

2000

The gastric acid hyposecretory state associated with endotoxemia is mediated by a nervous reflex involving the central nervous system. The aim of the present study was to analyse the central effects of different peptides on distension-stimulated gastric acid secretion and the endogenous role of such peptides on the hyposecretory effects of endotoxin. The effect of an intracisternal (i.c.) administration of oxytocin, vasopressin, corticotropin releasing factor (CRF), bombesin, somatostatin and the opioid receptor agonist BW443C or an intravenous (i.v.) injection of a small dose of endotoxin on distension-stimulated gastric acid secretion was studied in the continuously perfused stomach of an…

MaleVasopressinendotoxinCorticotropin-Releasing HormonevasopressinNarcotic AntagonistsGastric DilatationOxytocinchemistry.chemical_compoundVasoconstrictor AgentsReceptorChemistryStomachBombesincorticotropin-releasing factorGeneral MedicineSomatostatinmedicine.anatomical_structurebombesinReceptors Oxytocingastric acid secretionBombesinFemaleSomatostatingastric distensionOligopeptidesAntidiuretic Hormone Receptor Antagonistshormones hormone substitutes and hormone antagonistsmedicine.drugmedicine.medical_specialtyVasopressinsReceptors Corticotropin-Releasing HormoneGastric AcidAdrenergic AgentsInternal medicineoxytocinmedicineAnimalsRats WistarInjections IntraventricularPharmacologyDose-Response Relationship Drugcentral nervous systemOxytocin receptorEndotoxemiaHormonesRatsEndotoxinsReceptors BombesinEndocrinologyOxytocinGastric MucosaGastric acid
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Comparative effects of the novel vasotocin analogue F-180 vs. vasopressin and terlipressin on systemic and splanchnic isolated vessels from portal hy…

2001

F-180 has been proposed as a new vasopressin analogue for the treatment of portal hypertension. This study investigates the contractile profile of F-180 compared to vasopressin and its analogue terlipressin on isolated systemic and splanchnic vessels from sham-operated and partial portal vein ligated (PPVL) rats. F-180 (10(-9)-10(-6) M), vasopressin (10(-11)-10(-8) M) and terlipressin (10(-9)-10(-4) M) induced contraction of the mesenteric vein, aorta, iliac, tail and mesenteric arteries. The order of potency in these vessels was vasopressin (pD2 approximately 9)F-180 (pD2 approximately 8)terlipressin (pD2 approximately 6). Significant (P0.01) differences between sham-operated and PPVL rats…

MaleVasopressinmedicine.medical_specialtymedicine.drug_classVasopressinsPharmacology toxicologyPortal veinLypressinVasotocinMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundMesenteric VeinsInternal medicineHypertension PortalmedicineAnimalsVasoconstrictor AgentsPharmacologybusiness.industryGeneral Medicinemedicine.diseaseMesenteric ArteriesRatsEndocrinologychemistryVasoconstrictioncardiovascular systemPortal hypertensionVasopressin AnalogueSplanchnicbusinessTerlipressinTerlipressinmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Increased contraction to noradrenaline by vasopressin in human renal arteries

2002

Arginine vasopressin (AVP) not only acts directly on blood vessels through vasopressin V1 receptor stimulation but also may modulate adrenergic-mediated responses in animal experiments. The aim of the present study was to assess whether subpressor concentrations of AVP could contribute to an abnormal adrenergic contractile response of human renal arteries.Renal artery rings were obtained from 27 patients undergoing nephrectomy. The rings were suspended in organ bath chambers for isometric recording of tension.AVP (10(-10) mol/l) and the vasopressin V1 receptor agonist [Phe2, Orn8]-vasotocin (10(-10) mol/l) produced a leftward shift of the concentration-response curve to noradrenaline (half-…

Maleendocrine systemVasopressinmedicine.medical_specialtyContraction (grammar)NifedipineArgininePhysiologyNeuropeptideStimulationPotassium ChlorideNorepinephrineRenal ArteryInternal medicineInternal MedicinemedicineHumansVasoconstrictor AgentsEnzyme InhibitorsAgedVasopressin receptoromega-N-MethylarginineDose-Response Relationship Drugurogenital systembusiness.industryMiddle AgedCalcium Channel BlockersArginine VasopressinEndocrinologymedicine.anatomical_structureVasoconstrictionCirculatory systemFemaleCardiology and Cardiovascular MedicinebusinessAntidiuretic Hormone Receptor Antagonistshormones hormone substitutes and hormone antagonistsBlood vesselJournal of Hypertension
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Vasopressor Cumulative Dose Requirement and Risk of Early Death During Septic Shock: An Analysis From The EPISS Cohort

2018

Septic shock is the primary cause of death in intensive care units, with about 20% of patients dying in the first 3 days. To design future trials focused on early mortality, we require knowledge of early indicators that can detect patients at high risk of early death from refractory septic shock.The aim of this study was to assess whether the cumulative dose of vasopressors (CDV), calculated as the cumulative dose of epinephrine + norepinephrine, is a predictor of early death (within 72 hours) attributable to refractory septic shock (EDASS). This substudy of the EPISS trial was based on 370 patients admitted to a French ICU for septic shock between 2009 and 2011. The area under the receivin…

Malemedicine.medical_specialty030204 cardiovascular system & hematologyCritical Care and Intensive Care MedicineDisease-Free Survival03 medical and health sciences0302 clinical medicineRisk FactorsInternal medicineIntensive caremedicineHumansVasoconstrictor AgentsProspective StudiesIntensive care medicineProspective cohort studySurvival rateAgedCause of deathAged 80 and overCumulative doseSeptic shockbusiness.industry030208 emergency & critical care medicineMiddle Agedmedicine.diseaseShock SepticSurvival RateIntensive Care UnitsROC CurveShock (circulatory)CohortEmergency MedicineFemalemedicine.symptombusinessShock
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Intubation Practices and Adverse Peri-intubation Events in Critically Ill Patients From 29 Countries

2021

Importance: Tracheal intubation is one of the most commonly performed and high-risk interventions in critically ill patients. Limited information is available on adverse peri-intubation events. Objective: To evaluate the incidence and nature of adverse peri-intubation events and to assess current practice of intubation in critically ill patients. Design, Setting, and Participants: The International Observational Study to Understand the Impact and Best Practices of Airway Management in Critically Ill Patients (INTUBE) study was an international, multicenter, prospective cohort study involving consecutive critically ill patients undergoing tracheal intubation in the intensive care units (ICUs…

Malemedicine.medical_specialtyCritical Illnessmedicine.medical_treatmentAged; Critical Illness; Female; Heart Arrest; Humans; Hypotension; Hypoxia; Intensive Care Units; Intubation Intratracheal; Logistic Models; Male; Medical Errors; Middle Aged; Prospective Studies; Respiration Artificial; Respiratory Insufficiency; Vasoconstrictor Agents01 natural sciencesNOtracheal intubation ; adverse peri-intubation events03 medical and health sciences0302 clinical medicineInterquartile rangeIntensive careSettore MED/41 - ANESTESIOLOGIAIntubation IntratrachealHumansVasoconstrictor AgentsMedicineIntubationIntubation Critical CareProspective Studies030212 general & internal medicine0101 mathematics610 Medicine & healthHypoxiaProspective cohort studyAgedMedical Errorsbusiness.industryRespiration010102 general mathematicsTracheal intubationGeneral MedicineMiddle AgedRespiration ArtificialHeart ArrestIntratrachealIntensive Care UnitsIntubation procedureLogistic ModelsRespiratory failureArtificialEmergency medicineFemaleAirway managementHypotensionIntubationRespiratory Insufficiencybusiness
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U-46619-induced potentiation of noradrenergic constriction in the human saphenous vein: antagonism by thromboxane receptor blockade.

2001

Objective: We investigated the potentiating effect of U-46619, a synthetic analogue of thromboxane A2 (TXA2), on the adrenergic responses in human saphenous vein. Methods: Saphenous vein rings were obtained from 35 patients undergoing coronary artery bypass surgery. The rings were suspended in organ bath chambers for isometric recording of tension. Results: U-46619 (10−10–3×10−7 mol/l) produced concentration-dependent and endothelium-independent contractile responses. U-46619 (10−10 mol/l) potentiated the contractions elicited by electrical stimulation and potassium chloride, and produced leftward shifts of the concentration–response curve for noradrenaline. The TXA2 receptor antagonist SQ-…

Malemedicine.medical_specialtyDihydropyridinesNifedipinePhysiologymedicine.drug_classThromboxaneReceptors ThromboxaneAdrenergicIn Vitro TechniquesPotassium ChlorideThromboxane receptorThromboxane A2chemistry.chemical_compoundNorepinephrineThromboxane A2Physiology (medical)Internal medicinemedicineHumansVasoconstrictor AgentsSaphenous VeinAgedVoltage-dependent calcium channelDose-Response Relationship DrugChemistryCalcium channelDihydropyridineDrug SynergismMiddle AgedReceptor antagonistCalcium Channel BlockersElectric StimulationStimulation ChemicalEndocrinology15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidFemaleEndothelium VascularCardiology and Cardiovascular Medicinemedicine.drugCardiovascular research
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Direct evidence of leukocyte adhesion in arterioles by angiotensin II

2004

AbstractAlthough leukocytes adhere in arteries in various vascular diseases, to date no endogenous proinflammatory molecule has been identified to initiate leukocyte adhesion in the arterial vasculature. This study was undertaken to assess angiotensin II (Ang II)-induced leukocyte adhesion in arterioles in vivo. Rats received intraperitoneal injections of Ang II; 4 hours later, leukocyte recruitment in mesenteric microcirculation was examined using intravital microscopy. Ang II (1 nM) produced significant arteriolar leukocyte adhesion of mononuclear cells. Using function-blocking monoclonal antibodies (mAbs) against different rat cell adhesion molecules (CAMs), we discovered that this effec…

Malemedicine.medical_specialtyEndotheliumIntegrin alpha4ImmunologyIntercellular Adhesion Molecule-1Vascular Cell Adhesion Molecule-1BiologyBiochemistryRats Sprague-DawleyVenulesInternal medicineCell AdhesionLeukocytesmedicineAnimalsVasoconstrictor AgentsLeukocyte RollingCell adhesionCell adhesion moleculeAngiotensin IINF-kappa BCell BiologyHematologyIntercellular Adhesion Molecule-1Angiotensin IIRatsArteriolesmedicine.anatomical_structureEndocrinologyCD18 AntigensImmunologyEndothelium VascularIntravital microscopySelectinArteryBlood
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