Search results for "Virology"

showing 10 items of 2354 documents

Chaperones Involved in Hepatitis B Virus Morphogenesis

1999

Little is known about host cell factors necessary for hepatitis B virus (HBV) assembly which involves envelopment of cytosolic nucleocapsids by the S, M and L transmembrane viral envelope proteins and subsequent budding into intraluminal cisternae. Central to virogenesis is the L protein that mediates hepatocyte receptor binding and envelopment of capsids. To serve these topologically conflicting roles, L protein exhibits an unusual dual membrane topology, disposing its N-terminal preS domain inside and outside of the virion lipid envelope. The mixed topology is achieved by posttranslational preS translocation of about half of the L protein molecules across a post-endoplasmic reticulum memb…

Hepatitis B virusProtein FoldingCalnexinHSC70 Heat-Shock ProteinsClinical BiochemistryBiochemistryViral Matrix ProteinsCytosolViral Envelope ProteinsViral envelopeCalnexinMorphogenesisAnimalsHumansHSP70 Heat-Shock ProteinsProtein PrecursorsMolecular BiologyHepatitis B Surface AntigensViral matrix proteinbiologyChemistryCalcium-Binding ProteinsHSC70 Heat-Shock ProteinsBiological TransportVirologyTransmembrane proteinCell biologyProtein BiosynthesisMembrane topologyChaperone (protein)COS Cellsbiology.proteinProtein foldingCarrier ProteinsMolecular ChaperonesBiological Chemistry
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Functional incorporation of green fluorescent protein into hepatitis B virus envelope particles

2004

AbstractThe envelope of hepatitis B virus (HBV), containing the L, M, and S proteins, is essential for virus entry and maturation. For direct visualization of HBV, we determined whether envelope assembly could accommodate the green fluorescent protein (GFP). While the C-terminal addition of GFP to S trans-dominant negatively inhibited empty envelope particle secretion, the N-terminal GFP fusion to S (GFP.S) was co-integrated into the envelope, giving rise to fluorescent particles. Microscopy and topogenesis analyses demonstrated that the proper intracellular distribution and folding of GFP.S, required for particle export were rescued by interprotein interactions with wild-type S. Thereby, a…

Hepatitis B virusRecombinant Fusion ProteinsGreen Fluorescent ProteinsRestriction MappingEnzyme-Linked Immunosorbent AssayBiologyTransfectionmedicine.disease_causeHBsAg particlesArticleViral envelopeGreen fluorescent proteinViral Envelope ProteinsViral envelopeViral entryVirologyChlorocebus aethiopsmedicineAnimalsHumansGreen fluorescent proteinSecretionPromoter Regions GeneticHepatitis B virusCOS cellsfungiTransfectionMolecular biologyCell biologyKineticsCOS CellsMetallothioneinVirus assembly and secretionProtein KinasesIntracellularVirology
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Hepatitis B virus assembly is sensitive to changes in the cytosolic S loop of the envelope proteins.

2000

Among the three related L, M, and S envelope proteins of the hepatitis B virus (HBV), the L and S polypeptides are required for virion production. Whereas the pivotal function of the pre-S region of L in nucleocapsid envelopment has been established, the contribution of its S domain and the S protein is less clear. In this study, we evaluated the role of the cytosolic S loop, common to L and S, in HBV assembly by performing mutagenesis experiments. To distinguish between the effect of the mutations on either envelope or virion formation, we investigated the ability of the mutants to assemble into secretable subviral empty envelopes and to replace the wild-type proteins in virion maturation,…

Hepatitis B virusRecombination GeneticMutationHepatitis B virusvirusesVirus AssemblyMutantMolecular Sequence DataMorphogenesisMutagenesis (molecular biology technique)Biologymedicine.disease_causeVirologyCell biologyLoop (topology)CytosolCytosolViral Envelope ProteinsSequence Analysis ProteinVirologymedicineMutagenesis Site-DirectedHumansAmino Acid SequenceFunction (biology)Virology
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Pre-core mutants of hepatitis B virus in patients receiving immunosuppressive treatment after orthotopic liver transplantation.

1996

Orthotopic liver transplantation (OLT) is a possible treatment for acute or chronic liver failure due to hepatitis B virus (HBV) infection, but reinfection of the graft can be a serious complication. The aim of this study was to monitor HBV markers, to analyse pre-core-/core-mutations as well as to identify the viral population causing reinfection after OLT, and to investigate the emergence or disappearance of these mutants in patients receiving immunosuppressive treatment. Fifty-four pre-and posttransplant serum samples of 17 patients were analysed. All patients underwent OLT for HBV-related liver disease and had HBV-DNA before and after OLT. Total DNA was extracted from all sera and a 240…

Hepatitis B virusTime Factorsmedicine.medical_treatmentPopulationLiver transplantationInterferon alpha-2medicine.disease_causeAntiviral AgentsLiver diseaseVirologyMedicineHumansHepatitis B e AntigensProtein PrecursorseducationHepatitis B viruseducation.field_of_studyHepatitis B Surface Antigensbiologybusiness.industryInterferon-alphaImmunosuppressionSequence Analysis DNAHepatitis Bbiology.organism_classificationmedicine.diseaseHepatitis BVirologyHepatitis B Core AntigensRecombinant ProteinsLiver TransplantationTransplantationsurgical procedures operativeInfectious DiseasesHepadnaviridaeDNA ViralbusinessImmunosuppressive AgentsFollow-Up StudiesJournal of medical virology
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Hepatitis B virus maturation is sensitive to functional inhibition of ESCRT-III, Vps4, and gamma 2-adaptin.

2007

ABSTRACT Hepatitis B virus (HBV) is an enveloped DNA virus that presumably buds at intracellular membranes of infected cells. HBV budding involves two endocytic host proteins, the ubiquitin-interacting adaptor γ2-adaptin and the Nedd4 ubiquitin ligase. Here, we demonstrate that HBV release also requires the cellular machinery that generates internal vesicles of multivesicular bodies (MVBs). In order to perturb the MVB machinery in HBV-replicating liver cells, we used ectopic expression of dominant-negative mutants of different MVB components, like the ESCRT-III complex-forming CHMP proteins and the Vps4 ATPases. Upon coexpression of mutated CHMP3, CHMP4B, or CHMP4C forms, as well as of ATPa…

Hepatitis B virusVacuolar Proton-Translocating ATPasesEndosomeImmunologyEndocytic cycleVesicular Transport Proteinsmacromolecular substancesEndosomesmedicine.disease_causeMicrobiologyESCRTVirusCell LineViral ProteinsVirologymedicineHumansAdaptor Protein Complex gamma SubunitsHepatitis B virusAdenosine TriphosphatasesMicroscopy ConfocalbiologyEndosomal Sorting Complexes Required for TransportVirus AssemblyDNA virusMolecular biologyUbiquitin ligaseCell biologyGenome Replication and Regulation of Viral Gene ExpressionMicroscopy FluorescenceInsect Sciencebiology.proteinHepatocytesATPases Associated with Diverse Cellular ActivitiesEctopic expressionJournal of virology
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Hepatitis B Virus Large Envelope Protein Interacts with γ2-Adaptin, a Clathrin Adaptor-Related Protein

2001

ABSTRACT For the outcome of a hepatitis B virus (HBV) infection, the viral L envelope protein with its pre-S domain performs pivotal functions by mediating attachment of HBV to liver cells, envelopment of viral capsids, release of (sub)viral particles, regulation of supercoiled DNA amplification, and transcriptional transactivation. To assess its multiple functions and host-protein assistance involved, we initiated a two-hybrid screen using the L-specific pre-S1 domain as bait. With this approach, we have identified γ2-adaptin, a putative member of the clathrin adaptor proteins responsible for protein sorting and trafficking, as a specific binding partner of L protein. Evidence for a physic…

Hepatitis B virusVesicle-associated membrane protein 8ImmunoprecipitationImmunologyGolgi ApparatusTransfectionmedicine.disease_causeMicrobiologyClathrinChromatography AffinityCytosolViral Envelope ProteinsMutant proteinYeastsVirologyProtein targetingmedicineAnimalsBinding siteAdaptor Protein Complex gamma SubunitsBinding SitesbiologyMembrane ProteinsPrecipitin TestsClathrinTransmembrane proteinVirus-Cell InteractionsCell biologyInsect ScienceCOS CellsMutationbiology.proteinClathrin adaptor proteinsProtein BindingJournal of Virology
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Selection of hepatitis B virus variants with aminoacid substitutions inside the core antigen during interferon-? therapy

2000

The hepatitis B virus (HBV) core antigen carries many epitopes relevant for B and T cell response that show aminoacid variation during viral infection. In a longitudinal analysis, sequential serum samples of 15 patients that suffered from chronic HBV infection were collected before, during, and after high-dose IFN-α treatment. The HBV preCore/Core (preC/C) sequence of the selected samples in each patient was determined and analysed for sequence variations compared to the pretreatment sample. The positions of HBV core aminoacid substitutions were assigned to immunodominant B, CD4+ and CD8+ cell epitopes. Seventy-five percent of all aminoacid substitutions were found within immunodominant T a…

Hepatitis B virusbiologyAlpha interferonmedicine.disease_causebiology.organism_classificationVirologyEpitopeVirusInfectious DiseasesAntigenOrthohepadnavirusHepadnaviridaeVirologymedicineAntigenic variationJournal of Medical Virology
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Possible role of human interleukin-6 and soluble interleukin-6 receptor in hepatitis B virus infection

2001

Human interleukin-6 has been shown to promote hepatitis B virus (HBV) infection. However, it is not clear whether this influence is the result of a direct interaction between interleukin-6 (IL-6) and the HBV envelope proteins or of a rather indirect mechanism. A direct interaction of IL-6 and the preS region of the large envelope protein (L-protein) of HBV has been reported. In this study we assessed the binding of IL-6 and of the IL-6 receptor subunits to the preS region of the L-protein of HBV. Binding of IL-6 and IL-6 receptor subunits sIL-6R and gp130 to preS was assessed by immunoprecipitation with recombinant preS proteins. In patient sera IL-6 and sIL-6R concentrations were analysed …

Hepatitis B virusmedicine.disease_causeHepatitis B virus PRE betalaw.inventionHepatitis B ChroniclawVirologyEscherichia colimedicineAnimalsHumansProtein PrecursorsInterleukin 6ReceptorCells CulturedHepatitis B virusHepatitis B Surface AntigensHepatologybiologyInterleukin-6Chemistryvirus diseasesViral LoadHepatitis BGlycoprotein 130medicine.diseasePrecipitin TestsReceptors Interleukin-6VirologyMolecular biologyRecombinant ProteinsInfectious DiseasesSolubilityCOS CellsRecombinant DNAbiology.proteinViral loadCell DivisionPlasmidsJournal of Viral Hepatitis
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An outbreak of HBV and HCV infection in a paediatric oncology ward: Epidemiological investigations and prevention of further spread

2003

Hospital-acquired hepatitis B (HBV) and C virus (HCV) infections continue to occur despite increased awareness of this problem among the medical community. One hundred six patients were infected in a haematology oncology ward for children, over the time period 1996 to 2000. Serum samples from 45 such patients and 3 from infected medical personnel were used for nucleic acid amplification. HBV core, as well as HCV core and hypervariable region 1 (HVR1) nucleotide sequences, were analysed by phylogenetic tree analysis, in order to characterise the epidemiological pattern of viral transmission on the ward. Samples from 32 patients were positive for HBV-DNA or HCV-RNA by PCR. Ten patients were p…

Hepatitis B virusmedicine.medical_specialtyAdolescentMolecular Sequence DataHepacivirusmedicine.disease_causePediatricsPolymerase Chain ReactionDisease OutbreaksFlaviviridaeOncology Service HospitalVirologyEpidemiologymedicineHumansInfection controlChildPhylogenyHepatitis B virusCross InfectionbiologyTransmission (medicine)business.industryIncidence (epidemiology)virus diseasesSequence Analysis DNAHepatitis BHepatitis Bmedicine.diseasebiology.organism_classificationHepatitis CVirologydigestive system diseasesInfectious DiseasesChild PreschoolDNA ViralRNA ViralViral diseasebusinessJournal of Medical Virology
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From viral pathobiology to the treatment of hepatitis B virus infection EASL Monothematic Conference (Istanbul, Turkey, October 6-8, 2005).

2006

Hepatitis B virus infection, which was for over a decade put aside by the rising star of hepatitis C, has seen over the last years a resurgence of interest. This stemmed from a better knowledge of the virus itself, from the availability of new drugs and combinations, and from the realization that far from being eradicated by mass immunization programs HBV is still on a worldwide basis the major cause of cirrhosis and hepatocellular carcinoma, alone or in combinations with other viruses, alcohol andmetabolic cofactors. This prompted the EASL Scientific Committee to organize a Monothematic Conference on HBV. Experts from Europe, USA, Canada, Australia, the Far East and other parts of the worl…

Hepatitis B virusmedicine.medical_specialtyHepatitis B virusHepatologybusiness.industryIstanbul turkeyhepatitis B viruHepatitis Chepatocellular carcinomamedicine.disease_causemedicine.diseaseHepatitis BVirologyHepatitis B AntigensMass immunizationChronic hepatitischronic hepatitiFamily medicinemedicineHumansViremiabusinesscirrhosiJournal of hepatology
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