Search results for "Virulence"

showing 10 items of 457 documents

Dosage-dependent roles of the Cwt1 transcription factor for cell wall architecture, morphogenesis, drug sensitivity and virulence in Candida albicans.

2009

The Cwt1 transcription factor is involved in cell wall architecture of the human fungal pathogen Candida albicans. We demonstrate here that deficiency of Cwt1 leads to decreased β1,6-glucan in the cell wall, while mannoproteins are increased in the cell wall of exponentially growing cells and are released into the medium of stationary phase cells. Hyphal morphogenesis of cwt1 mutants is reduced on the surfaces of some inducing media. Unexpectedly, the CWT1/cwt1 heterozygous strains shows some stronger in vitro phenotypes compared to the homozygous mutant. The heterozygous but not the homozygous strain is also strongly impaired for its virulence in a mouse model of systemic infection. We sug…

Antifungal AgentsMutantMorphogenesisGene DosageHyphaeVirulenceBioengineeringApplied Microbiology and BiotechnologyBiochemistryMicrobiologyCell wallFungal ProteinsMiceCell WallDrug Resistance FungalGene Expression Regulation FungalCandida albicansGeneticsMorphogenesisAnimalsHumansCandida albicansDNA FungalTranscription factorOligonucleotide Array Sequence AnalysisMembrane GlycoproteinsbiologyVirulenceHomozygoteCandidiasisbiology.organism_classificationPhenotypeCorpus albicansMutationBiotechnologyTranscription FactorsYeast (Chichester, England)
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Inhibition of Filamentation Can Be Used To Treat Disseminated Candidiasis

2006

ABSTRACT Candida albicans remains the leading causative agent of invasive fungal infection. Although the importance of filamentation in C. albicans pathogenesis has been extensively investigated, in vivo studies to date have been unable to dissect the role of this developmental process in the establishment of infection versus the development of active disease as characterized by damage to the host leading to mortality. To address this issue, we genetically engineered a C. albicans tet-NRG1 strain in which filamentation and virulence can be modulated both in vitro and in vivo simply by the presence or absence of doxycycline (DOX): this strain enabled us, in a prior study, to demonstrate that…

Antifungal AgentsSaccharomyces cerevisiae ProteinsHyphaeAntifungal drugVirulenceKidneyMicrobiologyMiceFilamentationIn vivoGene Expression Regulation FungalCandida albicansmedicineAnimalsExperimental TherapeuticsPharmacology (medical)Candida albicansPharmacologyDoxycyclineMice Inbred BALB CVirulencebiologyCandidiasisDisseminated Candidiasisbiology.organism_classificationCorpus albicansDNA-Binding ProteinsRepressor ProteinsInfectious DiseasesDoxycyclineFemaleGenetic Engineeringmedicine.drugAntimicrobial Agents and Chemotherapy
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Pga13 in Candida albicans is localized in the cell wall and influences cell surface properties, morphogenesis and virulence.

2011

The fungal cell wall is an essential organelle required for maintaining cell integrity and also plays an important role in the primary interactions between pathogenic fungi and their hosts. PGA13 encodes a GPI protein in the human pathogen Candida albicans, which is highly up-regulated during cell wall regeneration in protoplasts. The Pga13 protein contains a unique tandem repeat, which is present five times and is characterized by conserved spacing between the four cysteine residues. Furthermore, the mature protein contains 38% serine and threonine residues, and therefore probably is a highly glycosylated cell wall protein. Consistent with this, a chimeric Pga13-V5 protein could be localiz…

Antifungal AgentsSurface PropertiesCellMorphogenesisHyphaeCalcofluor-whiteKidneyMicrobiologyMicrobiologyCell wallFungal ProteinsMiceCell WallStress PhysiologicalOrganelleCandida albicansGeneticsmedicineCell AdhesionAnimalsHumansAmino Acid SequenceCell adhesionCandida albicansOligonucleotide Array Sequence AnalysisSequence DeletionFungal proteinMice Inbred BALB CbiologyVirulenceGene Expression ProfilingProtoplastsCandidiasisFlocculationbiology.organism_classificationCell biologymedicine.anatomical_structureFemaleSequence AlignmentFungal genetics and biology : FGB
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Antifungal and post-antifungal effects of chlorhexidine, fluconazole, chitosan and its combinations on Candida albicans

2010

Objective: The aim of this work was to assess the antifungal and post-antifungal effects of chlorhexidine, fluconazole, chitosan and its combinations on virulence factors of Candida albicans. Study Design: Ten isolated strains of Candida albicans obtained from 10 patients with oral candidiasis and a collection strain of C. albicans were treated with antifungal agents in different concentrations or combinations of them. Virulence factors analyzed were the cell surface hydrophobicity, the germinative tube development, the phospholipase activity and the post-antifungal effect of that exposure. Results: Virulence factors of the isolated strains obtained from patients together with the collectio…

Antifungal AgentsVirulencePhospholipaseBiologyMicrobiologyCandida albicansmedicineHumansOral mucosaCandida albicansGeneral DentistryFluconazoleChitosanStrain (chemistry)ChlorhexidineChlorhexidine:CIENCIAS MÉDICAS [UNESCO]biology.organism_classificationCorpus albicansmedicine.anatomical_structureOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASAnti-Infective Agents LocalSurgeryFluconazolemedicine.drug
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Diversity and Evolution of the Phenazine Biosynthesis Pathway

2010

ABSTRACT Phenazines are versatile secondary metabolites of bacterial origin that function in biological control of plant pathogens and contribute to the ecological fitness and pathogenicity of the producing strains. In this study, we employed a collection of 94 strains having various geographic, environmental, and clinical origins to study the distribution and evolution of phenazine genes in members of the genera Pseudomonas , Burkholderia , Pectobacterium , Brevibacterium , and Streptomyces . Our results confirmed the diversity of phenazine producers and revealed that most of them appear to be soil-dwelling and/or plant-associated species. Genome analyses and comparisons of phylogenies inf…

Antifungal Agentsgenome sequenceaeruginosa pao1virulence factorsphenazine-1-carboxylic acidVIRULENCE FACTORS GENE-CLUSTERApplied Microbiology and Biotechnologychemistry.chemical_compoundGene clusterEnvironmental MicrobiologyPhylogenySoil Microbiologyfluorescent pseudomonas2. Zero hungerGenetics0303 health sciencesEcologybiologyEPS-2PseudomonasPlants[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyMultigene FamilyHorizontal gene transferBiotechnologyDNA BacterialWashingtonPectobacteriumGene Transfer HorizontalGenotypeSequence analysisMolecular Sequence DataPhenazineerwinia-herbicola eh1087pseudomonas-chlororaphis pcl1391Evolution Molecular03 medical and health sciencesBacterial ProteinsPseudomonasBotanyEscherichia coli030304 developmental biologyBacteriaBase SequencePSEUDOMONAS-CHLORORAPHIS030306 microbiologybiological-controlGene Expression Regulation BacterialSequence Analysis DNA15. Life on landbiology.organism_classificationrpoBERWINIA-HERBICOLAPHENAZINEBiosynthetic Pathwaysgene-clusterLaboratorium voor PhytopathologieBurkholderiachemistryGenes BacterialLaboratory of PhytopathologyPhenazinesburkholderia-cepacia complexSequence AlignmentFood Science
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Role of the Non-Canonical RNAi Pathway in the Antifungal Resistance and Virulence of Mucorales

2021

Mucorales are the causal agents for the lethal disease known as mucormycosis. Mortality rates of mucormycosis can reach up to 90%, due to the mucoralean antifungal drug resistance and the lack of effective therapies. A concerning urgency among the medical and scientific community claims to find targets for the development of new treatments. Here, we reviewed different studies describing the role and machinery of a novel non-canonical RNAi pathway (NCRIP) only conserved in Mucorales. Its non-canonical features are the independence of Dicer and Argonaute proteins. Conversely, NCRIP relies on RNA-dependent RNA Polymerases (RdRP) and an atypical ribonuclease III (RNase III). NCRIP regulates the…

AntifungalTransposable element0301 basic medicineMucoralesAntifungal Agentstransposonmedicine.drug_classRNA Stability030106 microbiologyAntifungal drugVirulenceReviewQH426-470mucormycosis03 medical and health sciencesDrug Resistance FungalRNA interferenceFongsmedicineGeneticsbiochemistryRNA MessengerRibonuclease IIIepimutantGenetics (clinical)Genome stabilityGeneticsRdRPR3B2biologyMucormycosisnon-canonical RNAiRNA FungalArgonauteantifungal resistancemedicine.diseasebiology.organism_classificationvirulenceRNA silencing030104 developmental biologyNon canonicalbiology.proteinInfeccióMucoralesRNA Interferencegenome stabilitySignal TransductionDicerGenes
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Role of the virulence plasmid pR99 and the metalloprotease Vvp in resistance of Vibrio vulnificus serovar E to eel innate immunity

2007

Vibrio vulnificus biotype 2 serovar E (VSE) is a bacterial pathogen that produces a haemorrhagic septicaemia called vibriosis in eels. Its ability to grow in blood is conferred by a recently described virulence plasmid [Lee CT, Amaro C, Wu KM, Valiente E, Chang YF, Tsai SF, et al. A common virulence plasmid in biotype 2 Vibrio vulnificus and its dissemination aided by a conjugal plasmid. Journal of Bacteriology, submitted for publication.]. In this study, we analyzed the role of this plasmid together with the role played by the metalloprotease (Vvp) in the interaction between bacteria and eel innate immunity. To this end, we compared and statistically analyzed the differences in resistance …

Antimicrobial peptidesVirulenceMicrobial Sensitivity TestsVibrio vulnificusAquatic ScienceMicrobiologyPlasmidAnti-Infective AgentsBacterial ProteinsPhagocytosisBacteriologyAnimalsEnvironmental ChemistryImmunity MucosalVibrio vulnificusPathogenAntigens BacterialPhagocytesInnate immune systembiologyComplement Fixation TestsTransferrinMetalloendopeptidasesGeneral MedicineAnguillabiology.organism_classificationAntibodies BacterialVirologyImmunity InnateAntigens SurfaceMutationAlternative complement pathwayMuramidaseAntimicrobial Cationic PeptidesPlasmidsFish & Shellfish Immunology
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Pathogenic Aeromonas hydrophila Serogroup O:14 and O:81 Strains with an S Layer

2004

ABSTRACT Five autoagglutinating Aeromonas hydrophila isolates recovered from eels and humans were assigned to serogroups O:14 and O:81 of the Sakazaki and Shimada (National Institutes of Health) scheme. They had the following properties in common: positive precipitation after boiling, moderate surface hydrophobicity (salt-aggregation-test value around 1.2), pathogenicity for fish and mice (50% lethal dose, 10 4.61 to 10 7.11 ), lipopolysaccharides that contained O-polysaccharide chains of homogeneous chain length, and an external S layer peripheral to the cell wall observed by electron microscopy. A strong cross-reactivity was detected by immunoblotting between the homogeneous O-polysacchar…

Applied Microbiology and BiotechnologyMicrobiologyMiceBacterial ProteinsSpecies SpecificityVibrionaceaeEnvironmental MicrobiologyAnimalsHumansSerotypingEelsVirulenceEcologybiologyStrain (chemistry)EcologyStructural geneMembrane Proteinsbiology.organism_classificationAeromonas hydrophilaMolecular WeightMicroscopy ElectronAeromonas salmonicidaAeromonas hydrophilaAeromonasGenes BacterialS-layerBacteriaFood ScienceBiotechnologyApplied and Environmental Microbiology
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Invasion Ability and Disease Dynamics of Environmentally Growing Opportunistic Pathogens under Outside-Host Competition

2014

Most theories of the evolution of virulence concentrate on obligatory host-pathogen relationship. Yet, many pathogens replicate in the environment outside-host where they compete with non-pathogenic forms. Thus, replication and competition in the outside-host environment may have profound influence on the evolution of virulence and disease dynamics. These environmentally growing opportunistic pathogens are also a logical step towards obligatory pathogenicity. Efficient treatment methods against these diseases, such as columnaris disease in fishes, are lacking because of their opportunist nature. We present a novel epidemiological model in which replication and competition in the outside-hos…

Bacterial Diseases0106 biological sciencesPopulation ModelingDisease01 natural sciencesTheoretical EcologyMedicine and Health SciencesPathogenPOPULATIONmedia_common0303 health scienceseducation.field_of_studyPREDATIONMultidisciplinaryEcologyTransmission (medicine)EcologySimulation and ModelingQRCHANNEL CATFISHEVOLUTIONARY DYNAMICSCOMMUNITYInfectious DiseasesHost-Pathogen Interactions1181 Ecology evolutionary biologyMedicineAlgorithmsResearch ArticleTRANSMISSIONSciencemedia_common.quotation_subjecteducationPopulationVirulenceOpportunistic InfectionsBiologyResearch and Analysis Methods010603 evolutionary biologyCompetition (biology)03 medical and health sciences14. Life underwaterParasite EvolutioneducationEvolutionary dynamicsta413030304 developmental biologyEvolutionary BiologyMathematical ModelingSTABILITYMORTALITYEcology and Environmental SciencesBiology and Life SciencesComputational BiologyFLAVOBACTERIUM-COLUMNAREOutbreakModels TheoreticalEmerging Infectious DiseasesEvolutionary Ecologyta1181VIRULENCEParasitologyInfectious Disease ModelingPLoS ONE
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The widespread presence of a family of fish virulence plasmids in Vibrio vulnificus stresses its relevance as a zoonotic pathogen linked to fish farms

2021

Vibrio vulnificus is a pathogen of public health concern that causes either primary septicemia after ingestion of raw shellfish or secondary septicemia after wound exposure to seawater. In consequence, shellfish and seawater are considered its main reservoirs. However, there is one aspect of its biology that is systematically overlooked: its association with fish in its natural environment. This association led in 1975 to the emergence of a zoonotic clade within phylogenetic lineage 2 following successive outbreaks of vibriosis in farmed eels. Although this clade is now worldwide distributed, no new zoonotic clades were subsequently reported. In this work, we have performed phylogenetic, ge…

Bacterial ZoonosesEpidemiologyanimal diseasesFish farmingImmunologyVirulenceVibrio vulnificusAquacultureBiologyphylogenyMicrobiologyMicrobiologyFish DiseasesPlasmidVirologyDrug DiscoverymedicineAnimalsHumansPathogenZoonotic pathogenVibrio vulnificusShellfishintegumentary systemVirulenceZoonosisFishesGeneral Medicinevibriosiszoonosisbacterial infections and mycosesmedicine.diseasebiology.organism_classificationvirulence plasmidInfectious DiseasesVibrio InfectionsV. vulnificusParasitologyResearch ArticlePlasmids
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