Search results for "WTH"

showing 10 items of 5145 documents

Developmental and tumoral vascularization is regulated by G protein-coupled receptor kinase 2

2012

Tumor vessel dysfunction is a pivotal event in cancer progression. Using an in vivo neovascularization model, we identified G protein–coupled receptor kinase 2 (GRK2) as a key angiogenesis regulator. An impaired angiogenic response involving immature vessels was observed in mice hemizygous for Grk2 or in animals with endothelium-specific Grk2 silencing. ECs isolated from these animals displayed intrinsic alterations in migration, TGF-β signaling, and formation of tubular networks. Remarkably, an altered pattern of vessel growth and maturation was detected in postnatal retinas from endothelium-specific Grk2 knockout animals. Mouse embryos with systemic or endothelium-selective Grk2 ablation …

medicine.medical_specialtyG-Protein-Coupled Receptor Kinase 2Angiogenic SwitchAngiogenesisMedicinaActivin Receptors Type IIMelanoma ExperimentalReceptor Transforming Growth Factor-beta Type INeovascularization PhysiologicProtein Serine-Threonine KinasesBiologyMural cellGrk2Transforming Growth Factor beta1NeovascularizationMiceDownregulation and upregulationCell MovementPregnancyInternal medicinemedicineAnimalsHumansCell ProliferationHemizygoteMice KnockoutG protein-coupled receptor kinaseTumorNeovascularization PathologicEndothelial CellsRetinal VesselsG proteinGeneral MedicineCell biologyEndocrinologymedicine.anatomical_structurecardiovascular systemFemalePericyteSignal transductionmedicine.symptomActivin Receptors Type IReceptors Transforming Growth Factor betaSignal TransductionResearch Article
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Neuroprotective Actions of Estradiol and Novel Estrogen Analogs in Ischemia: Translational Implications

2010

This review highlights our investigations into the neuroprotective efficacy of estradiol and other estrogenic agents in a clinically relevant animal model of transient global ischemia, which causes selective, delayed death of hippocampal CA1 neurons and associated cognitive deficits. We find that estradiol rescues a significant number of CA1 pyramidal neurons that would otherwise die in response to global ischemia, and this is true when hormone is provided as a long-term pretreatment at physiological doses or as an acute treatment at the time of reperfusion. In addition to enhancing neuronal survival, both forms of estradiol treatment induce measurable cognitive benefit in young animals. Mo…

medicine.medical_specialtyGPR30hippocampusIschemiaEstrogen receptorHippocampusNeuroprotectionArticleEstradiol CongenersIschemiaInternal medicineestradiolmedicineAnimalsHumansEndocrine and Autonomic Systemsbusiness.industryCREBEstrogen analogapoptosismedicine.diseasestrokeglobal ischemiainsulin-like growth factor-1EndocrinologyNeuroprotective AgentsneuroprotectionEstradiol CongenersbusinessGPERhormones hormone substitutes and hormone antagonistsHormoneestrogen receptor
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Diacylglycerol kinase α mediatses 17-β-estradiol-induced proliferation, motility, and anchorage-independent growth of Hec-1A endometrial cancer cell …

2011

Increased levels of endogenous and/or exogenous estrogens are one of the well known risk factors of endometrial cancer. Diacylglycerol kinases (DGKs) are a family of enzymes which phosphorylate diacylglycerol (DAG) to produce phosphatidic acid (PA), thus turning off and on DAG-mediated and PA-mediated signaling pathways, respectively. DGK α activity is stimulated by growth factors and oncogenes and is required for chemotactic, proliferative, and angiogenic signaling in vitro. Herein, using either specific siRNAs or the pharmacological inhibitor R59949, we demonstrate that DGK α activity is required for 17-β-estradiol (E2)-induced proliferation, motility, and anchorage-independent growth of …

medicine.medical_specialtyGPR30medicine.drug_classCell SurvivalDiacylglycerol kinaseMotilityEstrogen receptorEnzyme AssayEndometrial carcinomaBiologyQuinazolinoneReceptors G-Protein-CoupledPiperidinePiperidinesCell MovementInternal medicineCell Line TumormedicineCell AdhesionHumansEndometrial NeoplasmEnzyme AssaysQuinazolinonesDiacylglycerol kinaseCell ProliferationEstradiolCell growthKinaseCell BiologyDiacylglycerol kinase; Endometrial carcinoma; Estrogen; GPR30; Cell BiologyEstrogenEndometrial NeoplasmsCell biologyEnzyme ActivationLipoprotein LipaseEndocrinologyReceptors EstrogenEstrogenGene Knockdown TechniquesGene Knockdown TechniqueFemaleRNA InterferenceSignal transductionGPERHuman
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The COMT val158met Polymorphism Is Associated with Early Pubertal Development, Height and Cortical Bone Mass in Girls

2005

Estrogens are involved in accretion of bone mass during puberty. Catechol-O-Methyltransferase (COMT) is involved in the degradation of estrogens. In this cross-sectional study we investigated associations between the COMT val158met polymorphism, which results in a 60-75% difference in enzyme activity between the val (high activity = H) and the met (low activity = L) variant, and skeletal phenotypes in 246 healthy pre/early pubertal girls. Girls with COMT(LL) were 5.4 cm taller than COMT(HH) girls. Dual x-ray absorptiometry showed higher values of bone mineral content (BMC), and larger areas of total body, femur and spine in COMT(LL). Cortical BMC, measured by peripheral quantitative compute…

medicine.medical_specialtyGenotypeBone densitymedicine.medical_treatmentCatechol O-Methyltransferasebehavioral disciplines and activitiesBone and BonesInsulin-like growth factorAbsorptiometry PhotonMethionineBone DensityInternal medicinemental disordersGenotypemedicineHumansFemurTibiaChildBone mineralPolymorphism GeneticCatechol-O-methyl transferaseEstradiolbusiness.industryPubertyfungiEstrogensValineBody HeightPhenotypemedicine.anatomical_structureEndocrinologynervous systemPediatrics Perinatology and Child HealthBody CompositionRegression AnalysisFemaleCortical boneTomography X-Ray ComputedbusinessPediatric Research
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Comparison of Postural Features and Muscle Strength between Children with Idiopathic Short Stature and Healthy Peers in Relation to Physical Exercise

2020

Previous research has reported that children with idiopathic short stature (ISS) showed functional and cognitive impairments. The purpose of this study was to compare muscle strength and body posture between children with ISS treated with growth hormone (GH) and healthy peers (healthy children, HC), and to analyze whether these parameters were affected by physical exercise. Eighteen children for the ISS group (mean age: 10.96 ± 1.68 years) and 26 children for the HC group (mean age: 10.19 ± 1.06 years) were recruited for the study. All participants performed the following assessments: handgrip and Sargent test for the muscle strength evaluation; baropodometric and stabilometric test for the…

medicine.medical_specialtyGeography Planning and Developmentlcsh:TJ807-830lcsh:Renewable energy sourcesphysical activity030209 endocrinology & metabolismPhysical exercisebody postureManagement Monitoring Policy and LawGrowth hormone03 medical and health sciences0302 clinical medicineQuality of lifeGH treatmentidiopathic short statureStatistical significancemedicine030212 general & internal medicinelcsh:Environmental scienceslcsh:GE1-350Settore M-EDF/02 - Metodi E Didattiche Delle Attivita' SportiveRenewable Energy Sustainability and the EnvironmentBody posturebusiness.industrylcsh:Environmental effects of industries and plantsMean agemedicine.diseaseSargent testIdiopathic short staturelcsh:TD194-195idiopathic short stature; physical activity; muscle strength; body posture; handgrip test; Sargent test; GH therapy; GH treatmentMuscle strengthPhysical therapymuscle strengthhandgrip testbusinesshuman activitiesGH therapySettore M-EDF/01 - Metodi E Didattiche Delle Attivita' Motorie
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Trajectories of motor coordination in 4–7-years-old children: A latent growth curve analysis

2014

Introduction For being able to take part in physical activities typical to the given age, children need proficiency in several motor tasks. Gross motor coordination (MC) is a basis for executing motor tasks and for learning new movement patterns. This study aimed to identify different MC trajectories in children aged 4–7-years-old. Methods Motor coordination of children ( n  = 94, mean age 6.1 ± 1.3 years at baseline) was measured at 0, 6 and 12 months by KTK-test battery [1] . To identify different MC trajectories, developmental trend of the mean score of KTK was analyzed by using a latent growth curve and growth mixture analysis. Results In general, MC developed significantly over the yea…

medicine.medical_specialtyGross motor skillmedicineOrthopedics and Sports MedicineMean ageAudiologyPsychologyGrowth curve (statistics)Motor coordinationScience & Sports
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Normal-weight obesity and cardiometabolic risk: A 7-year longitudinal study in girls from prepuberty to early adulthood

2017

Objective To study whether normal-weight obesity in childhood is associated with increased cardiometabolic risk in early adulthood. Methods This study assessed data for 236 girls followed from prepuberty to early adulthood. Growth chart data were obtained from birth to 18 years. Body composition was assessed by dual-energy x-ray absorptiometry and cardiometabolic risk by calculating continuous clustered risk score (at ages 11, 14, and 18). The association of body weight status with cardiometabolic risk from childhood to early adulthood was examined. Results Subjects with normal-weight obesity were virtually indistinguishable from their normal-weight lean peers in terms of relative body weig…

medicine.medical_specialtyGrowth chartLongitudinal studyPediatricsNutrition and DieteticsFramingham Risk Scorebusiness.industryEndocrinology Diabetes and MetabolismMedicine (miscellaneous)030209 endocrinology & metabolismmedicine.diseaseObesityBody fat percentage03 medical and health sciencesNormal weight obesity0302 clinical medicineEndocrinologyEndocrinologyPrepubertyInternal medicinemedicine030212 general & internal medicinebusinessBody mass indexObesity
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New Melanocortin 1 Receptor Binding Motif Based on the C-Terminal Sequence of ?-Melanocyte-Stimulating Hormone

2006

The C-terminal tripeptide of the alpha-melanocyte stimulating hormone (alpha-MSH11-13) possesses strong antiinflammatory activity without known cellular target. In order to better understand the structural requirements for function of such motif, we designed, synthesized and tested out Trp- and Tyr-containing analogues of the alpha-MSH11-13. Seven alpha-MSH11-13 analogues were synthesized and characterized for their binding to the melanocortin receptors recombinantly expressed in insect (Sf9) cells, infected with baculovirus carrying corresponding MC receptor DNA. We also tested these analogues on B16-F1 mouse melanoma cells endogenously expressing the MC1 receptor for binding and for abili…

medicine.medical_specialtyGrowth-hormone-releasing hormone receptorProtein ConformationAmino Acid MotifsMelanoma ExperimentalBiologyToxicologyBinding CompetitiveMiceThyrotropin-releasing hormone receptorInternal medicineChlorocebus aethiopsmedicineEnzyme-linked receptorAnimalsHumansACTH receptorMelanocyte-Stimulating HormonesReceptorPharmacologyGeneral MedicineMelanocortin 3 receptorCell biologyEndocrinologyCOS CellsEstrogen-related receptor gammaMelanocortinReceptor Melanocortin Type 1Basic <html_ent glyph="@amp;" ascii="&"/> Clinical Pharmacology <html_ent glyph="@amp;" ascii="&"/> Toxicology
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Home Parenteral Nutrition

2012

Parenteral nutrition (PN) involves the administration of fluids and nutrients using routes other than the gut, although usually involves central venous catheters (CVC). Home PN (HPN) refers to instances where this nutritional support is administered in the patient's home. Although this form of treatment is expensive and complex, it facilitates patients’ social rehabilitation, returning them to their home environment, reducing healthcare costs, and improving quality of life (QoL). Pediatric patients present specific technical characteristics, as not only should a proper nutritional status be maintained, but also optimal growth and development.

medicine.medical_specialtyHome environmentbusiness.industrymedicine.medical_treatmentNutritional statusClinical nutritionParenteral nutritionQuality of lifeHealth caremedicineOptimal growthbusinessIntensive care medicineCentral venous catheter
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The estrogen receptor α:insulin receptor substrate 1 complex in breast cancer: structure–function relationships

2007

Background: Insulin receptor substrate 1 (IRS-1) is a signaling molecule that exerts a key role in mediating cross talk between estrogen receptor a (ERa) and insulin-like growth factor 1 (IGF-1) in breast cancer cells. Previously, we demonstrated that a fraction of IRS-1 binds ERa, translocates to the nucleus, and modulates ERa-dependent transcription at estrogen response elements (ERE). Here, we studied structure-function relationships of the ER-a:IRS-1 complex under IGF-1 and/or estradiol (E 2 ) stimulation. Materials and methods: ERa and IRS-1 deletion mutants were used to analyze structural and functional ERα/IRS-1 interactions. IRS-1 binding to ERE and IRS-1 role in ERa-dependent ERE t…

medicine.medical_specialtyInsulin Receptor Substrate ProteinsActive Transport Cell NucleusEstrogen receptorRepressorBreast NeoplasmsBiologyStructure-Activity Relationshipestrogen receptor alpha (ERa) Insulin receptor substrate 1 (IRS-1) breast cancerCell Line TumorInternal medicineCoactivatormedicineHumansInsulin-Like Growth Factor IReceptors InterferonEstradiolEstrogen Receptor alphaHematologyDNA-binding domainPhosphoproteinsPeptide FragmentsReceptor InsulinProtein Structure TertiaryCell biologyIRS1Repressor ProteinsPleckstrin homology domainEndocrinologyOncologyInsulin Receptor Substrate ProteinsFemaleChromatin immunoprecipitationProtein BindingAnnals of Oncology
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