Search results for "Wistar"

showing 10 items of 1094 documents

Nitric oxide is involved in anoxic preconditioning neuroprotection in rat hippocampal slices.

1999

Sublethal anoxia/ischemia protects against subsequent damaging insults in intact brain or hippocampal slices. To help further understand mechanisms underlying anoxic/ischemic preconditioning, we tested three hypotheses which were that: (a) anoxic preconditioning (APC) improves electrical recovery in rat hippocampal slices; (b) anoxic preconditioning requires nitric oxide (NO); and (c) anoxic preconditioning blocks mitochondrial dysfunction that occurs following re-oxygenation after anoxia. Control hippocampal slices underwent a single 'test' anoxic insult. Experimental slices were preconditioned by 3 short anoxic insults prior to the 'test' insult. Evoked potentials (EPs), and NADH redox st…

MaleCentral nervous systemIschemiaHippocampusPharmacologyMitochondrionHippocampal formationIn Vitro TechniquesNitric OxideNeuroprotectionHippocampusNitric oxidechemistry.chemical_compoundmedicineAnimalsRats WistarHypoxia BrainIschemic PreconditioningMolecular Biologybusiness.industryGeneral Neurosciencemedicine.diseaseNADRatsmedicine.anatomical_structureNeuroprotective AgentschemistrySynapsesIschemic preconditioningNeurology (clinical)businessNeuroscienceOxidation-ReductionDevelopmental BiologyBrain research
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Extracellular Vesicles from Hyperammonemic Rats Induce Neuroinflammation and Motor Incoordination in Control Rats.

2020

Minimal hepatic encephalopathy is associated with changes in the peripheral immune system which are transferred to the brain, leading to neuroinflammation and thus to cognitive and motor impairment. Mechanisms by which changes in the immune system induce cerebral alterations remain unclear. Extracellular vesicles (EVs) seem to play a role in this process in certain pathologies. The aim of this work was to assess whether EVs play a role in the induction of neuroinflammation in cerebellum and motor incoordination by chronic hyperammonemia. We characterized the differences in protein cargo of EVs from plasma of hyperammonemic and control rats by proteomics and Western blot. We assessed whether…

MaleCerebellumtnfαhepatic encephalopathyArticleExtracellular VesiclesImmune systemWestern blotmedicineAnimalsHumansHyperammonemiaRats WistarReceptorlcsh:QH301-705.5NeuroinflammationInflammationMicrogliamedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaHyperammonemiaGeneral Medicinetnfα receptor tnfr1medicine.diseaseRatsMotor Skills DisordersDisease Models Animalmedicine.anatomical_structurelcsh:Biology (General)glial activationTumor necrosis factor alphaNervous System DiseasesTNF alpha receptor TNFR1businessNeuroscienceTNF alpha
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Local Cerebral Blood Flow in a Rat Cortical Vein Occlusion Model

1996

The symptoms following sinus and vein occlusion observed in patients and experimental animals display a considerable variability that so far remains largely unexplained. In a rat cortical vein occlusion model using a photochemical thrombotic technique, we examined changes in the cerebral venous flow pattern by fluorescence angiography and regional cerebral blood flow (rCBF) and cerebral blood volume fraction (CBVF) by a modern laser Doppler “scanning” technique. Brain damage was assessed histologically. Fluorescence angiographic findings fell into two groups: group A, rats with an altered venous flow pattern after occlusion (n = 12), and group B, rats with interruption of blood flow and/or…

MaleCerebral veinsPathologymedicine.medical_specialtyIschemia030218 nuclear medicine & medical imaging03 medical and health sciencesCerebral circulation0302 clinical medicineOcclusionLaser-Doppler FlowmetryAnimalsMedicineFluorescein AngiographyRats WistarCerebral perfusion pressureCerebral CortexBlood Volumebusiness.industryIntracranial Embolism and Thrombosismedicine.diseaseCortical VeinVein occlusionRatsNeurologyCerebral blood flowCerebrovascular CirculationNeurology (clinical)Cardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgeryJournal of Cerebral Blood Flow & Metabolism
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Novel complement C1 inhibitor BSF468248 does not improve brain damage after cortical vein occlusion

2003

BSF468248 is a novel potent complement C1 inhibitor. To determine whether BSF468248 is effective against focal cerebral ischemia, we evaluated the change of cerebral blood flow (CBF) and infarction volume using a photochemically-induced cortical vein occlusion model in rats in blind studies. In 22 Wistar rats, two adjacent cortical veins were occluded by photochemical thrombosis and fiberoptic illumination under controlled anesthesia and ventilation. Just after the occlusion, BSF468248 or physiological saline was administrated. In the low-dose study, a treatment group (n = 7) was administered BSF468248 1 mg/kg bolus and 1 mg/kg continuously for 30 min. The same volume of saline was given to…

MaleCerebral veinsPhotochemistrymedicine.medical_treatmentComplement C1 Inactivator ProteinsRats Sprague-DawleyBolus (medicine)OcclusionmedicineAnimalsRats WistarInfusions IntravenousSalineCerebral Cortexbusiness.industryCerebral InfarctionBlood flowCortical Veinmedicine.diseaseCerebral VeinsThrombosisRatsDisease Models AnimalTreatment OutcomeCerebral blood flowRegional Blood FlowBrain InjuriesCerebrovascular CirculationAnesthesiaIntracranial ThrombosisbusinessOligopeptidesMethods and Findings in Experimental and Clinical Pharmacology
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Cluster analysis of mrna expression levels identifies multiple sequential patterns following focal cerebral ischemia

2012

AIM The purpose of this study is to detect gene expression patterns following focal cerebral ischemia. MATERIAL AND METHODS 25 male Wistar rats were divided into control (n = 8) and ischemic (n = 17) groups. In the ischemic group, slowly progressing focal ischemia was simulated by two-vein occlusion with spreading depression (SD) a cortical microinjection of KCl induced. Ischemic tissue was removed at 2, 8, 24, or 72 h postischemia. Using semiquantitative reverse transcription polymerase chain reaction, we investigated mRNA expression levels of 13 representative genes related to cerebral ischemia. Cluster analysis of the gene expression levels was done. RESULTS In the ischemic group, the ex…

MaleCerebral veinsmedicine.medical_specialtyTranscription GeneticPhotochemistryIschemiaNerve Tissue ProteinsReal-Time Polymerase Chain ReactionBioinformaticsPotassium ChlorideCyclin D1Internal medicineGene expressionElectric ImpedancemedicineAnimalsCluster AnalysisRNA MessengerRats WistarCerebral Cortexbusiness.industryCortical Spreading Depressionmedicine.diseaseCerebral VeinsRatsReverse transcription polymerase chain reactionmedicine.anatomical_structureEndocrinologyReal-time polymerase chain reactionIschemic Attack TransientCerebral cortexCortical spreading depressionSurgeryNeurology (clinical)Intracranial ThrombosisbusinessTurkish Neurosurgery
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Nanosuspension Formulations for Low-Soluble Drugs: Pharmacokinetic Evaluation Using Spironolactone as Model Compound

2005

Various particle sizes of spironolactone as a model low solubility drug were formulated to yield micro-and nanosuspensions of the type solid lipid nanoparticles and DissoCubes. Seven oral and one i.v. formulations were tested in an in vivo pharmacokinetic study in rats with the aim of characterizing the bioavailability of spironolactone on the basis of its metabolites canrenone and 7-alpha-thiomethylspirolactone. In addition, a dose escalation study was carried out using nonmicronized spironolactone suspension as well as a nanosuspension type DissoCubes. On the basis of AUC as well as Cmax ratios, three groups of formulations were distinguished. The biggest improvement was seen with a solid…

MaleChemistry PharmaceuticalCmaxAdministration OralBiological AvailabilityPharmaceutical ScienceSpironolactonePharmacologyDrug Delivery SystemsPharmacokineticsPulmonary surfactantOral administrationDrug DiscoverySolid lipid nanoparticlemedicineAnimalsCanrenoneRats WistarSolubilityDiureticsPharmacologyChemistryOrganic ChemistryRatsBioavailabilityArea Under Curvemedicine.drugDrug Development and Industrial Pharmacy
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IVIVC in oral absorption for fenofibrate immediate release tablets using a dissolution/permeation system.

2009

ABSTRACT: The usefulness of a dissolution/permeation (D/P) system to predict the in vivo performance of solid dosage forms containing the poorly soluble drug, fenofibrate, was studied. Biorelevant dissolution media simulating the fasted and fed state conditions of the human gastrointestinal tract were used in order to simulate the effect of food on the absorption of fenofibrate. Moreover, the results obtained from the D/P system were correlated with pharmacokinetic parameters obtained following in vivo studies in rats. The in vitro parameter (amount permeated in the D/P system) reflected well the in vivo performance in rats in terms of AUC and C max of fenofibric acid. This study thus demon…

MaleChemistry PharmaceuticalPharmaceutical ScienceAdministration OralAbsorption (skin)PharmacologyDosage formPermeabilityAbsorptionIVIVCPharmacokineticsFenofibrateIn vivoOral administrationmedicineAnimalsHumansRats WistarHypolipidemic AgentsFenofibrateChemistryPermeationRatsSolubilityCaco-2 Cellsmedicine.drugTabletsJournal of pharmaceutical sciences
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Assessment and modulation of acamprosate intestinal absorption: comparative studies using in situ, in vitro (CACO-2 cell monolayers) and in vivo mode…

2003

The purpose of this study was to explore the intestinal absorption mechanism of acamprosate and to attempt to improve the bioavailability (BA) of the drug through modulation of its intestinal absorption using two enhancers (polysorbate 80 and sodium caprate) based on in situ, in vitro and in vivo models and comparing the results obtained. Intestinal transport of the drug, in the absence and in presence of polysorbate 80 (0.06, 0.28 and 9.6 mM) or sodium caprate (13 and 16 mM) was measured by using an in situ rat gut technique and Caco-2 cell monolayers. Additionally, the effect of sodium caprate on drug oral bioavailability, measured as urinary recovery, was quantified by performing in vivo…

MaleChemistryTaurineAcamprosateCell MembranePharmaceutical ScienceAbsorption (skin)PharmacologyIn vitroIntestinal absorptionBioavailabilityRatsAcamprosatePharmacokineticsIntestinal AbsorptionIn vivoParacellular transportmedicineElectric ImpedanceAnimalsHumansCaco-2 CellsRats Wistarmedicine.drugEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
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Transcription of the MAT2A gene, coding for methionine adenosyltransferase, is up-regulated by E2F and Sp1 at a chromatin level during proliferation …

2006

Methionine adenosyltransferase (MAT) is an essential enzyme because it catalyzes the formation of S-adenosylmethionine, the main methyl donor. Two MAT-encoding genes (MAT1A, MAT2A) are found in mammals. The latter is expressed in proliferating liver, dedifferentiation and cancer, whereas MAT1A is expressed in adult quiescent hepatocytes. Here, we report studies on the molecular mechanisms controlling the induction of MAT2A in regenerating rat liver and in proliferating hepatocytes. The MAT2A is up-regulated at two discrete moments during liver regeneration, as confirmed by RNApol-ChIP analysis. The first one coincides with hepatocyte priming (i.e. G0-G1 transition), while the second one tak…

MaleChromatin ImmunoprecipitationTranscription GeneticSp1 Transcription FactorMolecular Sequence DataOligonucleotidesElectrophoretic Mobility Shift AssayBiologyBiochemistryS PhaseSequence Homology Nucleic AcidmedicineAnimalsE2F1Electrophoretic mobility shift assayRats WistarPromoter Regions GeneticE2FE2F4Cells CulturedCell ProliferationSp1 transcription factorBase SequenceG1 PhaseMethionine AdenosyltransferaseCell BiologyMolecular biologyChromatinLiver regenerationE2F Transcription FactorsLiver RegenerationRatsUp-Regulationmedicine.anatomical_structureLiverMethionine AdenosyltransferaseHepatocyteHepatocytesProtein BindingThe International Journal of Biochemistry & Cell Biology
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Effect of the extract of Ginkgo biloba (EGb 761) on the circulating and cellular profiles of polyunsaturated fatty acids: correlation with the anti-o…

2000

Abstract Ginkgo biloba extract (EGb 761) has beneficial effects on cognitive functions in aging patients, and on various pathologies, including cardiovascular diseases. Although the extract is known to have antioxidant properties and improve membrane fluidity, the cellular mechanisms underlying these effects have not been determined. Here, we examined the in vivo effects of EGb 761 on circulating and cellular lipids. EGb 761 treatment induced significant increases in the levels of circulating polyunsaturated fatty acids (PUFAs), and a decrease in the saturation index SI (saturated/polyunsaturated species). Plasma triglycerides and cholesterol were not affected, while phospholipids were slig…

MaleChromatography GasErythrocytesAntioxidantmedicine.medical_treatmentClinical BiochemistryPharmacologymedicine.disease_causeAntioxidantschemistry.chemical_compoundmedicineMembrane fluidityAnimalsGinkgoalesRats WistarPhospholipidsTriglycerideschemistry.chemical_classificationPlants MedicinalbiologyPlant ExtractsGinkgo bilobaCholesterolCell MembraneErythrocyte MembraneFatty AcidsGinkgo bilobaHydrogen PeroxideCell Biologybiology.organism_classificationEicosapentaenoic acidRatsOxidative StressCholesterolEicosapentaenoic AcidchemistryBiochemistryFatty Acids Unsaturatedlipids (amino acids peptides and proteins)Oxidative stressPolyunsaturated fatty acidProstaglandins, Leukotrienes and Essential Fatty Acids (PLEFA)
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