Search results for "Wistar"

showing 10 items of 1094 documents

Protein synthesisin vivoin rats fed on lipid-rich liquid diets

1994

Changes in tissue composition and protein synthesis ratio were studied in the major tissues of the body in young rats fed on lipid-rich, isonitrogenous purified liquid diets, a convenient method for inducing voluntary overfeeding under controlled nutritional conditions. Overfed rats showed faster growth induced by the energy excess. Analysis of tissue composition (protein, DNA and RNA contents) revealed that growth was due mainly to tissue hyperplasia in which protein and DNA contents increased in parallel. Fractional protein synthesis ratio measuredin vivoby the flooding-dose method of phenylalanine showed a marked increase in all tissues. This change could be attributed to an increase in …

MaleMedicine (miscellaneous)PhenylalanineGrowthBiologychemistry.chemical_compoundIn vivoProtein biosynthesismedicineAnimalsRats WistarHyperplasiaNutrition and DieteticsProteinsRNADNARibosomal RNAHyperplasiamedicine.diseaseDietary FatsRatschemistryBiochemistryProtein BiosynthesisRNAAnimal Nutritional Physiological PhenomenaEnergy sourceDNABritish Journal of Nutrition
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Free [NADH]/[NAD+] regulates sirtuin expression

2011

Sirtuins are deacetylases involved in metabolic regulation and longevity. Our aim was to test the hypothesis that they are subjected to redox regulation by the [NADH]/[NAD(+)] ratio. We used NIH3T3 fibroblasts in culture, Drosophila fed with or without ethanol and exercising rats. In all three models an increase in [NADH]/[NAD(+)] came up with an increased expression of sirtuin mRNA and protein. PGC-1α (a substrate of sirtuins) protein level was significantly increased in fibroblasts incubated with lactate and pyruvate but this effect was lost in fibroblasts obtained from sirtuin-deficient mice. We conclude that the expression of sirtuins is subject to tight redox regulation by the [NADH]/[…

MaleMetaboliteBiophysicsBiochemistryMicechemistry.chemical_compoundPhysical Conditioning AnimalPyruvic AcidAnimalsSirtuinsLactic AcidRNA MessengerRats WistarEthanol metabolismMolecular BiologyCells CulturedGlyceraldehyde 3-phosphate dehydrogenaseRegulation of gene expressionMessenger RNAEthanolbiologyFibroblastsNADPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaRatsCell biologyDrosophila melanogasterGlycerol-3-phosphate dehydrogenaseGene Expression RegulationchemistryBiochemistrySirtuinNIH 3T3 CellsTrans-Activatorsbiology.proteinNAD+ kinaseOxidation-ReductionTranscription FactorsArchives of Biochemistry and Biophysics
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Putative role of nitric oxide synthase isoforms in the changes of nitric oxide concentration in rat brain cortex and cerebellum following sevoflurane…

2005

We have previously observed an increase in nitric oxide (NO) content in rat brain cortex following halothane, sevoflurane or isoflurane anaesthesia. This study was undertaken in order to determine whether isoform-specific nitric oxide synthase (NOS) inhibitors and inducers could modify these increases in NO contents. Rats were subjected to isoflurane and sevoflurane anaesthesia with concomitant administration of neuronal nitric oxide synthase (nNOS) inhibitor 7-Nitro-indazole (7-NI), inducible nitric oxide synthase (iNOS) inhibitor 2-amino-5,6-dihydro-6-methyl-4H-1,3-thiazine (AMT) or lipopolysaccharide. NO concentration in different organs was measured by electron paramagnetic resonance (E…

MaleMethyl Ethersmedicine.medical_specialtyCentral nervous systemNitric Oxide Synthase Type IINerve Tissue ProteinsNitric Oxide Synthase Type INitric OxideSevofluraneNitric oxidechemistry.chemical_compoundSevofluraneInternal medicineCortex (anatomy)CerebellummedicineAnimalsRats WistarPharmacologyCerebral CortexbiologyIsofluraneRecombinant ProteinsRatsNitric oxide synthaseIsoenzymesmedicine.anatomical_structureEndocrinologyIsofluranechemistryBiochemistryCerebral cortexAnesthetics Inhalationbiology.proteinHalothaneNitric Oxide Synthasemedicine.drugEuropean journal of pharmacology
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Controlled transdermal iontophoresis for poly-pharmacotherapy: Simultaneous delivery of granisetron, metoclopramide and dexamethasone sodium phosphat…

2015

Iontophoresis has been used to deliver small molecules, peptides and proteins into and across the skin. In principle, it provides a controlled, non-invasive method for poly-pharmacotherapy since it is possible to formulate and to deliver multiple therapeutic agents simultaneously from the anodal and cathodal compartments. The objective of this proof-of-principle study was to investigate the simultaneous anodal iontophoretic delivery of granisetron (GST) and metoclopramide (MCL) and cathodal iontophoresis of dexamethasone sodium phosphate (DEX-P). In addition to validating the hypothesis, these are medications that are routinely used in combination to treat chemotherapy-induced emesis. Two p…

MaleMetoclopramideSwinePharmaceutical Science02 engineering and technologyPharmacologyGranisetronAdministration Cutaneous030226 pharmacology & pharmacyDexamethasoneGranisetron03 medical and health sciences0302 clinical medicineDexamethasone Sodium PhosphatePharmacokineticsIn vivomedicineAnimalsRats WistarDexamethasoneActive metaboliteTransdermalSkinIontophoresisChemistryHydrolysisIontophoresis021001 nanoscience & nanotechnologyRatsPolypharmacy0210 nano-technologymedicine.drugEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
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Genomic response of the rat brain to global ischemia and reperfusion

2008

To identify genes that are involved in ischemia response of the brain, we have evaluated changes of gene expression in rat cerebrum after 15 min complete global ischemia, followed by reperfusion for 1 h, 6 h or 24 h. The expression profiles of approximately 30,000 transcripts from three subjects in each group (including sham-operated controls) were monitored employing oligonucleotide microarrays. About 20,000 transcripts were detectable in rat brains. The levels of 576 transcripts (approximately 2.9%) were significantly altered in response to experimental ischemia. 419 transcripts were up- and 157 downregulated; 39 transcripts changed after 1 h reperfusion, 174 after 6 h and 462 after 24 h.…

MaleMicroarrayIschemiaBiologyBrain IschemiaGene expressionmedicineAnimalsCluster AnalysisRats WistarMolecular BiologyOligonucleotide Array Sequence AnalysisRegulation of gene expressionReverse Transcriptase Polymerase Chain ReactionMicroarray analysis techniquesGene Expression ProfilingGeneral NeuroscienceBrainmedicine.diseaseMolecular biologyRatsGene expression profilingReverse transcription polymerase chain reactionReal-time polymerase chain reactionGene Expression RegulationReperfusionRNANeurology (clinical)Developmental BiologyBrain Research
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Induction of brain CYP2E1 changes the effects of ethanol on dopamine release in nucleus accumbens shell.

2009

CYP2E1 is an important enzyme involved in the brain metabolism of ethanol that can be induced by chronic consumption of alcohol. Recent works have highlighted the importance of this system in the context of the behavioural effects of ethanol. Unfortunately, the underlying neurochemical events for these behavioural changes, has not been yet explored. In this work, we have started this exploration by analyzing the possible changes in the neurochemical response of the mesolimbic system to ethanol after pharmacological induction of brain CYP2E1. We have used the dopamine extracellular levels in nucleus accumbens (NAc) core and shell, measured by means of microdialysis in vivo, as an index of th…

MaleMicrodialysisDopamineContext (language use)Nucleus accumbensPharmacologyToxicologyNucleus Accumbenschemistry.chemical_compoundNeurochemicalDopaminemedicineAnimalsPharmacology (medical)Rats WistarNeurotransmitterInfusions IntravenousPharmacologyEthanolEthanolBrainCytochrome P-450 CYP2E1RatsPsychiatry and Mental healthchemistryEnzyme InductionCatecholamineNeurosciencemedicine.drugDrug and alcohol dependence
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Local salsolinol modulates dopamine extracellular levels from rat nucleus accumbens: shell/core differences.

2008

Salsolinol (SAL), a condensation product of dopamine and acetaldehyde that appears in the rat and human brain after ethanol ingestion, has been largely implicated in the aetiology of alcoholism. Although the behavioural consequences of systemic or intracerebral SAL administrations have been described, the neurochemical effects of pharmacologically relevant doses of SAL and other tetrahydroisoquinolines (THIQs) in the brain areas involved in alcohol addiction are practically unknown. To gain an insight into this topic, male Wistar rats were stereotaxically implanted with one concentric microdialysis probe in either the shell or the core of the nucleus accumbens (NAc). Treatments involved loc…

MaleMicrodialysisDopamineMicrodialysisDown-RegulationAcetaldehydePharmacologyNucleus accumbensSynaptic TransmissionNucleus AccumbensCellular and Molecular Neurosciencechemistry.chemical_compoundNeurochemicalAlcohol-Induced Disorders Nervous SystemRewardDopamineparasitic diseasesBasal gangliamedicineAnimalsEthanol metabolismRats WistarNeurotransmitterChromatography High Pressure LiquidDose-Response Relationship DrugEthanolChemistryExtracellular FluidCell BiologyIsoquinolinesRatsUp-RegulationAlcoholismCatecholamineNeurosciencemedicine.drugNeurochemistry international
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Acetylcholine release in the hippocampus of the urethane anaesthetised rat positively correlates with both peak theta frequency and relative power in…

2000

The need to achieve a clearer understanding of relations between hippocampal theta characteristics and cholinergic septohippocampal neuron activity, prompted us to re-examine, in the urethane-anaesthetised rat, the statistical relationships between the electrophysiological and neurochemical variables using a procedure which is believed to enhance significantly the degree of confidence with which parameters of theta recorded with classic macroelectrodes can be related to concomitant acetylcholine output measured by high-performance liquid chromatography with electrochemical detection. Firstly, the theta rhythm and the acetylcholine content were derived from the same hippocampus. Secondly, th…

MaleMicrodialysisHippocampusElectroencephalographyHippocampal formationHippocampusUrethanechemistry.chemical_compoundmedicineElectrochemistryAnimalsRats WistarTheta RhythmNeurotransmitterMolecular BiologyChromatography High Pressure Liquidmedicine.diagnostic_testGeneral NeuroscienceElectroencephalographyAcetylcholineRatsElectrophysiologymedicine.anatomical_structurechemistryAnesthesia IntravenousCholinergicNeurology (clinical)NeuronPsychologyNeuroscienceAcetylcholineDevelopmental Biologymedicine.drugBrain research
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Stimulatory and inhibitory effects of ethanol on hippocampal acetylcholine release

1998

Using the microdialysis technique and sensitive HPLC procedures for the determination of acetylcholine (ACh) and ethanol, we investigated the release of ACh in rat hippocampus after acute ethanol administration. Systemic administration of ethanol (0.8 and 2.4 g/kg i.p.) led to peak ethanol concentrations of 21 and 42 mM in the hippocampus, respectively. The high dose caused a long-lasting inhibition of basal ACh release by up to 33%. Local infusion of scopolamine (1 microM) enhanced hippocampal ACh release up to eightfold in the presence of neostigmine (10 microM), and this stimulated release was also inhibited after systemic ethanol administration (by up to 45%). The low dose of ethanol (0…

MaleMicrodialysisMicrodialysisScopolamineHippocampusStimulationMuscarinic AntagonistsHippocampal formationPharmacologyHippocampuschemistry.chemical_compoundmedicineAnimalsRats WistarChromatography High Pressure LiquidPharmacologyEthanolEthanolCentral Nervous System DepressantsGeneral MedicineAcetylcholineRatsKineticschemistrySystemic administrationCholinergicExtracellular SpaceAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Bilobalide, a constituent of Ginkgo biloba , inhibits NMDA-induced phospholipase A 2 activation and phospholipid breakdown in rat hippocampus

2000

In rat hippocampal slices superfused with magnesium-free buffer, glutamate (1 mM) caused the release of large amounts of choline due to phospholipid breakdown. This phenomenon was mimicked by N-methyl-D-aspartate (NMDA) in a calcium-sensitive manner and was blocked by NMDA receptor antagonists such as MK-801 and 7-chlorokynurenate. The NMDA-induced release of choline was not caused by activation of phospholipase D but was mediated by phospholipase A2 (PLA2) activation as the release of choline was accompanied by the formation of lyso-phosphatidylcholine (lyso-PC) and glycerophospho-choline (GPCh) and was blocked by 5-[2-(2-carboxyethyl)-4-dodecanoyl-3,5-dimethylpyrrol-1-yl]pentano ic acid, …

MaleMicrodialysisN-MethylaspartateMicrodialysisGlycineCyclopentanesPharmacologyHippocampal formationHippocampusReceptors N-Methyl-D-AspartatePhospholipases ACholinechemistry.chemical_compoundPhospholipase A2BilobalideSeizuresAnimalsCholineRats WistarFuransCells CulturedPhospholipidsPharmacologyPlants MedicinalDose-Response Relationship DrugbiologyPhospholipase DGlutamate receptorGinkgo bilobaLysophosphatidylcholinesGeneral MedicineGlycerylphosphorylcholineRatsEnzyme ActivationPhospholipases A2Ginkgolideschemistrybiology.proteinNMDA receptorDiterpenesNaunyn-Schmiedeberg's Archives of Pharmacology
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