Search results for "Zidovudine"

showing 10 items of 22 documents

Synergistic effect of recombinant CD4-immunoglobulin in combination with azidothymidine, dideoxyinosine and 0.5 beta-monoclonal antibody on human imm…

1994

Data are presented which indicate that combinations of rCD4 immunoglobulin with azidothymidine, dideoxyinosine or 0.5 beta mouse monoclonal antibodies directed against the V3 region of HIV-1, were more effective in treatment of acute HIV infection in vitro than each compound alone. It is suggested that combination therapy with these compounds is more beneficial in treatment of HIV-infected patients than monotherapy, especially with respect to a reduction of the known side effects and the formation of resistant HIV strains after treatment with nucleoside analogues.

Combination therapymedicine.drug_classmedicine.medical_treatmentHIV InfectionsBiologyMonoclonal antibodyApplied Microbiology and BiotechnologyVirusZidovudinemedicineHumansLymphocytesCells Culturedvirus diseasesAntibodies MonoclonalDrug SynergismImmunotherapyVirologyIn vitroAnti-Bacterial AgentsDidanosinebiology.proteinDrug Therapy CombinationAntibodyNucleosideZidovudineCD4 Immunoadhesinsmedicine.drugLetters in applied microbiology
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Coupling of the antiviral agent zidovudine to polyaspartamide and in vitro drug release studies.

1998

A macromolecular prodrug of the known antiretroviral agent zidovudine and alpha, beta-poly(N-2-hydroxyethyl)-DL-aspartamide (PHEA) was synthesized. A succinic spacer was present between the polymer and the drug, and 1,1'-carbonyldiimidazole was used as the coupling agent. In vitro drug release studies at pH 1.1, 5.5 and 7.4 indicated that limited amounts of intact drug were released from the conjugate. At pH 1.1 and 7.4 succinylzidovudine was released, and this was hydrolysed to give free zidovudine. In the presence of alpha-chymotrypsin, zidovudine was released preferentially in comparison with the succinyl derivative. The amounts of released zidovudine and succinylzidovudine were greater …

DrugActive ingredientDrug CarriersChemistryAnti-HIV Agentsmedia_common.quotation_subjectHydrolysisPharmaceutical ScienceProdrugPharmacologyHydrogen-Ion ConcentrationIn Vitro TechniquesIn vitroZidovudinemedicineLiberationChymotrypsinHumansProdrugsDrug carrierPeptidesZidovudinemedia_commonmedicine.drugConjugateJournal of controlled release : official journal of the Controlled Release Society
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Biowaiver monographs for immediate-release solid oral dosage forms: Zidovudine (azidothymidine).

2012

Literature data on the properties of zidovudine relevant to waiver of in vivo bioequivalence (BE) testing requirements for the approval of immediate-release (IR) solid oral dosage forms containing zidovudine alone or in combination with other active pharmaceutical ingredients (APIs) are reviewed. Solubility, dissolution, and permeability data for zidovudine, along with its dosing schedule, therapeutic index and pharmacokinetic properties, and reports related to BE/bioavailability were all taken into consideration. Data for solubility and permeability suggest that zidovudine belongs to Class I according to the Biopharmaceutics Classification System. Also, zidovudine is not a narrow therapeut…

DrugAnti-HIV Agentsmedia_common.quotation_subjectPharmaceutical ScienceExcipientAdministration OralHIV InfectionsPharmacologyBioequivalenceDosage formPermeabilityCell LineExcipientsZidovudineDogsPharmacokineticsBIOEQUIVALÊNCIAMedicineAnimalsHumansmedia_commonActive ingredientbusiness.industryBiopharmaceutics Classification SystemSolubilityTherapeutic EquivalencyCaco-2 CellsbusinessZidovudinemedicine.drugJournal of pharmaceutical sciences
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Protection of Azidothymidine-Induced Cardiopathology in Mice by Mildronate, a Mitochondria-Targeted Drug

2006

Azidothymidine, a nucleoside-analogue reverse transcriptase inhibitor (NRTI), is a commonly used antiretroviral drug in AIDS treatment, however its use is limited by severe toxic side effects due to its influence on mitochondria that result in myopathy, particularly affecting the cardiac muscle. We suggest that effective protection of azidothymidine- induced cardiopathology can be expected from drugs that are capable of targeting mitochondria. Therefore the present study in mice was carried out with mildronate, a cardioprotective drug of the aza-butyrobetaine class, which previously has been shown to act as a highly potent protector of mitochondrial processes. In our study, saline (control)…

DrugHeart Diseasesmedia_common.quotation_subjectInflammationMitochondrionPharmacologyToxicologymedicine.disease_causeMiceZidovudinemedicineAnimalsmedia_commonPharmacologyMice Inbred ICRbiologyReverse-transcriptase inhibitorCardiovascular AgentsGeneral MedicineVirologyMitochondriaDisease Models AnimalEnzyme inhibitorbiology.proteinmedicine.symptomZidovudineNucleosideOxidative stressMethylhydrazinesmedicine.drugBasic <html_ent glyph="@amp;" ascii="&"/> Clinical Pharmacology <html_ent glyph="@amp;" ascii="&"/> Toxicology
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Future Perspectives in NNRTI-Based Therapy: Bases for Understanding Their Toxicity

2011

Continuous administration of the drugs included under the term Highly Active Antiretroviral Therapy (HAART) has turned AIDS into a chronic disease, at least in developed countries (Panos et al., 2008). The initial development of these drugs was particularly rapid and focused on clinical efficacy before all other considerations. However, as the disease has come under control, there has been growing emphasis on the long-term adverse effects associated with this therapy. The first drug for the treatment of HIV infection, zidovudine (AZT), was approved in 1987. The number of other antiretroviral drugs already approved for use or under development continues to grow, and the primary aim of resear…

EfavirenzNevirapinebusiness.industryvirus diseasesIntegrase inhibitorCCR5 receptor antagonistPharmacologyReverse transcriptaseNucleoside Reverse Transcriptase Inhibitorchemistry.chemical_compoundZidovudinechemistrymedicinebusinessViral loadmedicine.drug
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Abacavir and didanosine induce the interaction between human leukocytes and endothelial cells through Mac-1 upregulation

2010

Objective: Abacavir and didanosine are nucleoside reverse transcriptase inhibitors (NRTI) widely used in therapy for HIV-infection but which have been linked to cardiovascular complications. The objective of this study was to analyze the effects of clinically relevant doses of abacavir and didanosine on human leukocyte―endothelium interactions and to compare them with those of other NRTIs. Design and methods: The interactions between human leukocytes ― specifically peripheral blood polymorphonuclear (PMN) or mononuclear (PBMC) cells ― and human umbilical vein endothelial cells were evaluated in a flow chamber system that reproduces conditions in vivo. The expression of adhesion molecules wa…

EndotheliumImmunologyMacrophage-1 AntigenCell CommunicationPharmacologyBiologyPeripheral blood mononuclear cellZidovudineimmune system diseasesAbacavirLeukocytesmedicineHumansImmunology and AllergyDidanosineAnalysis of VarianceCell adhesion moleculeEndothelial Cellsvirus diseasesLamivudineDideoxynucleosidesUp-RegulationEndothelial stem cellDidanosineInfectious Diseasesmedicine.anatomical_structureCardiovascular DiseasesImmunologyReverse Transcriptase InhibitorsEndothelium VascularCell Adhesion Moleculesmedicine.drugAIDS
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Neuroprotective properties of mildronate, a mitochondria-targeted small molecule.

2010

Mildronate, a representative of the aza-butyrobetaine class of drugs with proven cardioprotective efficacy, was recently found to prevent dysfunction of complex I in rat liver mitochondria. The present study demonstrates that mildronate also acts as a neuroprotective agent. In a mouse model of azidothymidine (anti-HIV drug) neurotoxicity, mildronate reduced the azidothymidine-induced alterations in mouse brain tissue: it normalized the increase in caspase-3, cellular apoptosis susceptibility protein (CAS) and iNOS expression assessed by quantitative and semi-quantitative analysis. Mildronate also normalized the changes in cytochrome c oxidase (COX) expression, reduced the expression of glia…

MaleCell signalingAnti-HIV AgentsNitric Oxide Synthase Type IIMice Inbred StrainsMitochondrionPharmacologyNeuroprotectionElectron Transport Complex IVMiceCellular Apoptosis Susceptibility ProteinGlial Fibrillary Acidic ProteinmedicineAnimalsLymphocytesNeuroinflammationGlial fibrillary acidic proteinbiologyCaspase 3General NeuroscienceNeurodegenerationNeurotoxicityBrainmedicine.diseaseDisease Models AnimalNeuroprotective AgentsBiochemistrybiology.proteinNeurotoxicity SyndromesZidovudineCellular apoptosis susceptibility proteinMethylhydrazinesNeuroscience letters
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Distinct influence of atypical 1,4-dihydropyridine compounds in azidothymidine-induced neuro- and cardiotoxicity in mice ex vivo.

2008

This study demonstrates the effective protection by compounds of atypical 1,4-dihydropyridine (DHP) series cerebrocrast, glutapyrone and tauropyrone against neuro- and cardiotoxicity caused by the model compound azidothymidine, a well-known mitochondria-compromising anti-HIV drug. In previous in vitro experiments, we have demonstrated distinct effects of these DHP compounds to influence mitochondrial functioning. In the present in vivo experiments, DHP compounds were administered intraperitoneally in mice daily for 2 weeks, per se and in combinations with azidothymidine at doses: azidothymidine 50 mg/kg; cerebrocrast 0.1 mg/kg; glutapyrone 1 mg/kg; and tauropyrone 1 mg/kg. At the end of the…

MaleDihydropyridinesHeart DiseasesRatónAnti-HIV AgentsTaurineApoptosisBiologyPharmacologyToxicologyMiceGlutamatesIn vivomedicineAnimalsPharmacologyCerebral CortexInflammationCardiotoxicityMice Inbred ICRCaspase 3DihydropyridineTranscription Factor RelAGeneral MedicineBiochemistryGene Expression RegulationEnzyme inhibitorApoptosisToxicitybiology.proteinNeurotoxicity SyndromesZidovudineEx vivomedicine.drugBasicclinical pharmacologytoxicology
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Mitochondria as the target for mildronate's protective effects in azidothymidine (AZT)-induced toxicity of isolated rat liver mitochondria

2008

Previously mildronate, an aza-butyrobetaine derivative, was shown to be a cytoprotective drug, through its mechanism of action of inhibition of carnitine palmitoyltransferase-1, thus protecting mitochondria from long-chain fatty acid accumulation and subsequent damage. Recently in an azidothymidine (AZT)-induced cardiotoxicity model in vivo (in mice), we have found mildronate's ability of protecting heart tissue from nuclear factor kappaB abnormal expression. Preliminary data also demonstrate cerebro- and hepatoprotecting properties of mildronate in AZT-toxicity models. We suggest that mildronate may target its action predominantly to mitochondria. The present study in isolated rat liver mi…

MaleMitochondrial DiseasesBioenergeticsAntimetabolitesCell RespirationClinical BiochemistryMitochondria LiverIn Vitro TechniquesMitochondrionPharmacologyBiologymedicine.disease_causeBiochemistryPermeabilityRespiratory electron transport chainDrug Delivery SystemsmedicineAnimalsCarnitineRats WistarCardiotoxicityCell BiologyGeneral MedicineRatsDisease Models AnimalMechanism of actionBiochemistryToxicitymedicine.symptomEnergy MetabolismZidovudineOxidative stressMethylhydrazinesmedicine.drug
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AZT induces oxidative damage to cardiac mitochondria: Protective effect of vitamins C and E

2004

Abstract AZT (zidovudine) is a potent inhibitor of HIV replication and a major antiretroviral drug used for AIDS treatment. A major limitation in the use of AZT is the occurrence of severe side effects. The aim of this work was to test whether AZT causes oxidative damage to heart mitochondria and whether this can be prevented by supranutritional doses of antioxidant vitamins. An experimental animal model was used in which mice were treated with AZT for 35 days (10 mg/kg/day) in drinking water. Animals treated with antioxidant vitamins were fed the same diet as controls but supplemented with vitamins C (ascorbic acid, 10 g/ kg diet) and E (α-dl-tocopherol, 0.6 g/kg diet) for 65 days before s…

MaleMitochondrial Diseasesmedicine.medical_treatmentAscorbic AcidOxidative phosphorylationMitochondrionPharmacologyBiologymedicine.disease_causeDNA MitochondrialMitochondria HeartGeneral Biochemistry Genetics and Molecular BiologyLipid peroxidationMiceZidovudinechemistry.chemical_compoundmedicineAnimalsVitamin Eheterocyclic compoundsGeneral Pharmacology Toxicology and PharmaceuticsVitamin CVitamin EDeoxyguanosineGeneral MedicineAscorbic acidGlutathioneBiochemistrychemistry8-Hydroxy-2'-DeoxyguanosineLipid PeroxidationZidovudineOxidative stressmedicine.drugLife Sciences
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