Search results for "acetylcholine."

showing 10 items of 620 documents

Release of [3H]acetylcholine from a modified rat phrenic nerve-hemidiaphragm preparation

1986

Two different preparations of the rat phrenic nerve-hemidiaphragm (whole nerve-muscle preparation, end-plate preparation) were used for studying synthesis and release of radioactive acetylcholine in the absence and presence of cholinesterase inhibitors. When the whole nerve-muscle preparation (110-180 mg) was incubated with [3H]choline, only small amounts of radioactive acetylcholine were synthesized within the tissue. Electrical nerve stimulation of the whole nerve-muscle preparation produced no increase in tritium outflow. Incubation of the end-plate preparation (16-29 mg) which was obtained after removal of most of the muscle mass led to the formation of large amounts of [3H]acetylcholin…

MaleDiaphragmNeuromuscular JunctionStimulationIn Vitro TechniquesNeuromuscular junctionCholinechemistry.chemical_compoundmedicineAnimalsCholinePhrenic nerveCholinesterasePharmacologyNeurotransmitter AgentsbiologyRats Inbred StrainsHemicholinium 3General MedicineAcetylcholineMuscle DenervationRatsPhrenic Nervemedicine.anatomical_structurechemistryAnesthesiaTetrodotoxinbiology.proteinBiophysicsLiberationAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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alpha-Bungarotoxin, kappa-bungarotoxin, alpha-cobratoxin and erabutoxin-b do not affect [3H]acetylcholine release from the rat isolated left hemidiap…

1995

Endplate preparations of the rat left hemidiaphragm were incubated with [3H]choline to label neuronal transmitter stores. Nerve evoked release of newly-synthesized [3H]acetylcholine was measured in the absence of cholinesterase inhibitors to investigate whether snake venom neurotoxins by blocking presynaptic nicotinic autoreceptors affect evoked transmitter release. Contractions of the indirectly stimulated hemidiaphragm were recorded to characterize the blocking effect of alpha-neurotoxins at the post-synaptic nicotinic receptors. Neither the long chain neurotoxins alpha-cobratoxin (1 microgram ml-1) and alpha-bungarotoxin (5 microgram ml-1) nor the short chain neurotoxin erabutoxin-b (0.1…

MaleDiaphragmNeurotoxinsPharmacologyReceptors NicotinicTritiumSynaptic TransmissionPostsynaptic potentialmedicineAnimalsCobra Neurotoxin ProteinsChromatography High Pressure LiquidCholinesterasePharmacologyErabutoxinsbiologyChemistryMuscle SmoothGeneral MedicineBungarotoxinmusculoskeletal systemBungarotoxinsAcetylcholineRatsPhrenic NerveNicotinic agonistSnake venomIsotope Labelingbiology.proteinAutoreceptorFemaleCobratoxinNeuroscienceAcetylcholinemedicine.drugMuscle ContractionSnake VenomsNaunyn-Schmiedeberg's archives of pharmacology
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Effects of scopolamine on dopamine neurons in the substantia nigra : role of the pedunculopontine tegmental nucleus

2009

Previous neurochemical and behavioral studies suggest that muscarinic receptor antagonism has an excitatory effect on the nigrostriatal dopamine (DA) system. Using in vivo extracellular single unit recording, this study examined whether blockade of the muscarinic receptor by scopolamine alters the firing properties of DA neurons in the substantia nigra (SN). Scopolamine was administered either systemically or locally to DA neurons using microiontophoresis. Surprisingly, scopolamine did not cause any significant change in either the firing rate or pattern of the spontaneously active DA neurons. However, systemic injection of scopolamine significantly increased the number of active DA neurons…

MaleDopamineParkinson's diseaseScopolamineAction PotentialsSubstantia nigraMuscarinic AntagonistsStriatumelectrophysiology microiontophoresisSettore BIO/09 - FisiologiaRats Sprague-DawleyCellular and Molecular NeuroscienceDopamineBasal gangliaMuscarinic acetylcholine receptorPedunculopontine Tegmental NucleusmedicineSubstantia nigraAnimalsPedunculopontine Tegmental NucleusNeuronsAnalysis of VarianceDose-Response Relationship DrugChemistryDrug Administration RoutesIontophoresisAcetylcholineRatsSubstantia Nigranervous systemAutoreceptorBasal gangliaNeuroscienceAcetylcholinemedicine.drug
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Muscarinic Properties of Compounds Related to Arecaidine Propargyl Ester

2011

A series of new analogues of the arecaidine propargyl ester (CAS 35516-99-5, APE, 1a) with alcohols consisting of 4 or 5 carbon atoms were investigated at muscarinic receptor subtypes. The muscarinic activity of the quaternary and tertiary salts of the APE-related compounds were assayed on the isolated guinea-pig ileum (M 3 receptor subtype) and guinea-pig left atria (M 2 receptor subtype) as well as on rabbit isolated vas deferens (M 1 receptor subtype). The structural variations made in the APE molecule, replacing the triple bond in the ester side chain with structures such as double bond, an allene moiety, a single bond, a cyclopropyl group or two triple bonds should alter the selectivit…

MaleDouble bondStereochemistryArecolineGuinea PigsCholinergic AgentsIn Vitro Techniqueschemistry.chemical_compoundVas DeferensIleumDrug DiscoveryMuscarinic acetylcholine receptorAnimalsMoietySingle bondReceptorReceptor Muscarinic M3chemistry.chemical_classificationReceptor Muscarinic M2Receptor Muscarinic M1HeartMuscle SmoothArecaidineTriple bondMyocardial ContractionReceptors MuscarinicchemistryPropargylFemaleRabbitsMuscle ContractionArzneimittelforschung
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Influence of experimental diabetes on regulatory mechanisms of vascular response of rabbit carotid artery to acetylcholine

2000

Summary The purpose of this study was to analyse the influence of experimental diabetes on vascular response of rabbit carotid artery to acetylcholine (Ach). We compared the Ach-induced relaxant response of isolated arterial segments obtained from both control and diabetic animals. To assess the influence of the endothelium, this cell layer was mechanically removed in some of the arterial segments (“rubbed arteries”) from each experimental group. Ach induced a concentration-related endothelium-mediated relaxation of carotid artery from control rabbits that was significantly higher with respect to that obtained in diabetic animals. Pre-treatment with NG-nitro-L-arginine (L-NA) induced a conc…

MaleGene isoformmedicine.medical_specialtyEndotheliumVasodilator AgentsIndomethacinProstacyclinGuanidinesNitroarginineGeneral Biochemistry Genetics and Molecular BiologyDiabetes Mellitus ExperimentalNitric oxidechemistry.chemical_compoundInternal medicineDiabetes mellitusAlloxanmedicineAnimalsEnzyme InhibitorsGeneral Pharmacology Toxicology and PharmaceuticsEndothelial dysfunctionbusiness.industryAnti-Inflammatory Agents Non-SteroidalGeneral Medicinemedicine.diseaseAcetylcholineCarotid ArteriesEndocrinologymedicine.anatomical_structurechemistryArachidonic acidEndothelium VascularRabbitsbusinessAcetylcholinemedicine.drugLife Sciences
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Role of epithelium in agonist-induced contractile responses of guinea-pig trachealis: influence of the surface through which drug enters the tissue.

1990

1. A method has been used in guinea-pig isolated tracheal rings to achieve selective drug entry from the adventitial or mucosal surface. A study has been made of the effects of epithelium removal on responses to spasmogens entering the tissue solely from the adventitial or the mucosal surface. 2. Cumulative concentration-response curves for KCl (1 to 100 mM), acetylcholine (0.1 microM to 10 mM) and histamine (1 microM to 1 mM) were constructed in intact and epithelium-denuded tracheal rings in circumstances where drug entry was unrestricted or restricted to the adventitial or mucosal surface. 3. Epithelium removal did not alter the responsiveness or sensitivity of tracheal rings to KCl eith…

MaleGuinea PigsBiologyPharmacologyIn Vitro TechniquesEpitheliumGuinea pigchemistry.chemical_compoundmedicineAnimalsRespiratory systemPharmacologyAirway ResistanceAnatomyEpitheliumAcetylcholineTracheamedicine.anatomical_structurechemistryTrachealis musclePotassiummedicine.symptomHistamineAcetylcholineMuscle contractionmedicine.drugRespiratory tractHistamineResearch ArticleBritish journal of pharmacology
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Isolation of Cholinergic Synaptic Vesicles from the Myenteric Plexus of Guinea-Pig Small Intestine

1980

The acetylcholine-rich myenteric plexus-longitudinal muscle preparation of the guinea-pig small intestine has been subjected to subcellular fractionation using modifications of both classical methods and that originally devised for bulk isolation of cholinergic synaptic vesicles from the electromotor nerve terminals of Torpedo marmorata by means of density gradient centrifugation in a zonal rotor. The latter method gave a vesicle fraction with the highest acetylcholine content so far recorded for a mammalian particulate fraction, 30.9 +/- S.E.M. 1.8 (5) nmol of acetylcholine . mg of protein-1. Electron-microscopical examination showed that it consisted of a homogeneous preparation of vesicl…

MaleGuinea PigsMyenteric PlexusBiologyCell FractionationBiochemistrySynaptic vesiclelaw.inventionCellular and Molecular NeurosciencelawIntestine SmallMyosinCentrifugation Density GradientmedicineAnimalsMyenteric plexusVesicleAcetylcholineMicroscopy ElectronBiochemistryBiophysicsCholinergicFemaleSynaptic VesiclesCell fractionationAcetylcholineTorpedomedicine.drugJournal of Neurochemistry
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Prejunctional M1 and postjunctional M3 muscarinic receptors in the circular muscle of the guinea-pig ileum.

1995

The effects of subtype-selective muscarinic receptor antagonists on electrically evoked release of acetylcholine and muscle contraction were compared in circular muscle preparations of the guinea-pig ileum. Incubation of the preparation with [3H]choline resulted in the formation of [3H]acetylcholine. Electrical stimulation caused the release of [3H]acetylcholine which was abolished by tetrodotoxin and omission of calcium from the medium. 5-Hydroxytryptamine (10 microM) and the nicotinic agonist 1,1-dimethyl-4-phenyl-piperazinium (300 microM) did not change acetylcholine release. The muscarinic antagonists pirenzepine (M1 selective), AF-DX 116 (M2 selective) and hexahydrosiladifenidol (M3 se…

MaleGuinea PigsNeuromuscular JunctionMuscarinic AntagonistsPharmacologyIn Vitro TechniquesCholinePiperidinesIleumMuscarinic acetylcholine receptorMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptor M4medicineAnimalsPharmacologyChemistryMuscarinic acetylcholine receptor M3ParasympatholyticsMuscarinic acetylcholine receptor M2Muscle SmoothGeneral MedicineMuscarinic acetylcholine receptor M1AnatomyPirenzepinePirenzepineReceptors MuscarinicAcetylcholineFemaleAcetylcholinemedicine.drugMuscle ContractionNaunyn-Schmiedeberg's archives of pharmacology
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Anti-inflammatory and antioxidant effects of muscarinic acetylcholine receptor (mAChR) activation in the rat hippocampus

2019

AbstractRecently we found that acute treatment with Oxotremorine (Oxo), a non-selective mAChRs agonist, up-regulates heat shock proteins and activates their transcription factor heat shock factor 1 in the rat hippocampus. Here we aimed to investigate: a) if acute treatment with Oxo may regulate pro-inflammatory or anti-inflammatory cytokines and oxidative stress in the rat hippocampus; b) if chronic restraint stress (CRS) induces inflammatory or oxidative alterations in the hippocampus and whether such alterations may be affected by chronic treatment with Oxo. In the acute experiment, rats were injected with single dose of Oxo (0.4 mg/kg) and sacrificed at 24 h, 48 h and 72 h. In the CRS ex…

MaleHydrocortisonemedicine.medical_treatmentInterleukin-1betaNeuroimmunologyAnti-Inflammatory Agentslcsh:MedicinePharmacologymedicine.disease_causeHippocampusSettore BIO/09 - FisiologiaAntioxidantsSuperoxide Dismutase-1Muscarinic acetylcholine receptorPhosphorylationlcsh:Sciencechemistry.chemical_classificationMultidisciplinarybiologyneurodegenerationAlzheimer's diseaseReceptors MuscarinicNeuroprotective AgentsCytokineSignal Transductionmedicine.drugRestraint PhysicalAgonistmedicine.drug_classScopolaminemuscarinic acetylcholine receptorMuscarinic AgonistsArticleOxotremorine anti-inflammatory cytokinesSuperoxide dismutaseHeat shock proteinOxotremorinemedicineAnimalsRats WistarInflammationReactive oxygen speciesInterleukin-6Superoxide DismutaseOxotremorinelcsh:RTranscription Factor RelARatsOxidative Stresschemistrybiology.proteinlcsh:QReactive Oxygen SpeciesProtein Processing Post-TranslationalOxidative stressScientific Reports
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Increased levels of Th17 cells are associated with non-neuronal acetylcholine in COPD patients.

2014

T-lymphocytes, including Th17-cells and T-cells expressing acetylcholine (ACh), are key components of systemic inflammation in chronic obstructive pulmonary disease (COPD). We investigated whether ACh promotes Th17 cells in COPD. ACh, IL-17A, IL-22, RORγt, FOXP3 expression and AChIL-17A, AChIL-22, AChRORγt coexpression was evaluated in peripheral blood mononuclear cells (PBMC) from COPD patients (n=16), healthy smokers (HS) (n=12) and healthy control subjects (HC) (n=13) (cultured for 48 h with PMA) by flow cytometry. Furthermore, we studied the effect of Tiotropium (Spiriva®) (100 nM) and Olodaterol (1nM) alone or in combination, and of hemicholinium-3 (50 μM) on AChIL-17A, AChIL-22, AChRO…

MaleImmunologyIntracellular SpaceScopolamine DerivativesPharmacologySystemic inflammationPeripheral blood mononuclear cellCholinergic AntagonistsFlow cytometrychemistry.chemical_compoundPulmonary Disease Chronic ObstructiveRAR-related orphan receptor gammaRisk FactorsmedicineImmunology and AllergyHumansTiotropium BromideAgedAged 80 and overCOPDmedicine.diagnostic_testbusiness.industryInterleukinsOlodaterolInterleukin-17FOXP3Forkhead Transcription FactorsHematologyMiddle AgedNuclear Receptor Subfamily 1 Group F Member 3medicine.diseaseAcetylcholineBenzoxazineschemistryLeukocytes MononuclearTh17 CellsFemalemedicine.symptombusinessAcetylcholinemedicine.drugImmunobiology
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