Search results for "acids"

showing 10 items of 3520 documents

Induction of the fatty acid transport protein 1 and acyl-CoA synthase genes by dimer-selective rexinoids suggests that the peroxisome proliferator-ac…

2000

The intracellular fatty acid content of insulin-sensitive target tissues determines in part their insulin sensitivity. Uptake of fatty acids into cells is a controlled process determined in part by a regulated import/export system that is controlled at least by two key groups of proteins, i.e. the fatty acid transport protein (FATP) and acyl-CoA synthetase (ACS), which facilitate, respectively, the transport of fatty acids across the cell membrane and catalyze their esterification to prevent their efflux. Previously it was shown that the expression of the FATP-1 and ACS genes was controlled by insulin and by peroxisome proliferator-activated receptor (PPAR) agonists in liver or in adipose t…

MalePeroxisome proliferator-activated receptor gammaTime FactorsReceptors Retinoic AcidRetinoic acidReceptors Cytoplasmic and NuclearPeroxisome proliferator-activated receptorTretinoinRetinoid X receptorBiologyFatty Acid-Binding ProteinsBiochemistryMicechemistry.chemical_compoundCoenzyme A LigasesTumor Cells CulturedAnimalsHumansTissue DistributionMolecular BiologyNucleic Acid Synthesis InhibitorsCell Nucleuschemistry.chemical_classificationDose-Response Relationship DrugFatty AcidsMembrane ProteinsFatty acidMembrane Transport ProteinsSerum Albumin Bovine3T3 CellsCell BiologyFatty Acid Transport ProteinsRatsRats ZuckerRetinoic acid receptorRetinoid X ReceptorschemistryBiochemistryDactinomycinFree fatty acid receptorRNAPeroxisome proliferator-activated receptor alphaCaco-2 CellsCarrier ProteinsTranscription Factors
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The effects of three absorption-modifying critical excipients on the in vivo intestinal absorption of six model compounds in rats and dogs.

2018

Pharmaceutical excipients that may affect gastrointestinal (GI) drug absorption are called critical pharmaceutical excipients, or absorption-modifying excipients (AMEs) if they act by altering the integrity of the intestinal epithelial cell membrane. Some of these excipients increase intestinal permeability, and subsequently the absorption and bioavailability of the drug. This could have implications for both the assessment of bioequivalence and the efficacy of the absorption-enhancing drug delivery system. The absorption-enhancing effects of AMEs with different mechanisms (chitosan, sodium caprate, sodium dodecyl sulfate (SDS)) have previously been evaluated in the rat single-pass intestin…

MalePharmaceutical ScienceExcipientBiological Availability02 engineering and technologyBioequivalencePharmacology030226 pharmacology & pharmacyIntestinal absorptionPermeabilityExcipients03 medical and health sciences0302 clinical medicineDogsIn vivomedicineAnimalsPharmaceutical sciencesIntestinal MucosaChitosanIntestinal permeabilityChemistrySodium Dodecyl Sulfate021001 nanoscience & nanotechnologymedicine.diseaseBioavailabilityRatsIntestinesIntestinal AbsorptionPharmaceutical PreparationsDrug delivery0210 nano-technologyDecanoic Acidsmedicine.drugInternational journal of pharmaceutics
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Phenytoin-induced glutathione depletion in rat peripheral nerve

1995

Abstract Administration of high doses (150–250 mg/kg body weight) of phenytoin (DPH) promote a 40% decrease in glutathione (GSH) content of rat sciatic nerve. This DPH-induced GSH depletion is accompanied with an electrophysiological impairment of peripheral neuromuscular function. H7 (20 mg/kg body weight IP, 30 min prior to DPH), a protein kinase C inhibitor, was able to prevent the DPH-induced GSH depletion only at the lower DPH dose used. This same inhibitor completely prevented the electrophysiological impairment at the lower DPH dose, and only partially at the higher DPH dose used. These results confirm the hypothesis of a DPH-dependent activation of PKC (that might be triggered by, o…

MalePhenytoinAntioxidantmedicine.medical_treatmentAction PotentialsIn Vitro TechniquesPharmacologymedicine.disease_causeBiochemistryPiperazineschemistry.chemical_compound1-(5-Isoquinolinesulfonyl)-2-MethylpiperazinePhysiology (medical)polycyclic compoundsmedicineAnimalsEnzyme InhibitorsRats WistarMuscle SkeletalEvoked PotentialsProtein Kinase CProtein kinase CMotor NeuronsAnalysis of Variancetechnology industry and agricultureNeurotoxicityGlutathioneIsoquinolinesmedicine.diseaseGlutathioneSciatic NerveRatsKineticschemistryBiochemistryPhenytoinAnticonvulsantslipids (amino acids peptides and proteins)Sciatic nerveOxidative stressIntracellularmedicine.drugFree Radical Biology and Medicine
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Non-alcoholic fatty liver disease and the development of nephrolithiasis: A cohort study.

2017

Background Non-alcoholic fatty liver disease (NAFLD), a hepatic manifestation or precursor of metabolic syndrome, may increase nephrolithiasis, a renal manifestation of insulin resistance, but the prospective association between NAFLD and incident nephrolithiasis has not been evaluated. We examined the association of NAFLD with the development of nephrolithiasis in a large cohort of Korean men and women. Methods We performed a cohort study of 208,578 Korean adults who underwent a health checkup examination between January 2002 and December 2014 and were followed-up annually or biennially through December 2014. NAFLD was defined as the presence of fatty liver in the absence of excessive alco…

MalePhysiology030232 urology & nephrologylcsh:MedicineBlood PressureGastroenterologyVascular MedicineBody Mass IndexCohort Studies0302 clinical medicineEndocrinologyNon-alcoholic Fatty Liver DiseaseMedicine and Health Scienceslcsh:ScienceMultidisciplinaryAlcohol ConsumptionIncidence (epidemiology)Liver DiseasesHazard ratioFatty liverMiddle AgedChemistryPhysiological ParametersResearch DesignHypertensionPhysical Sciences030211 gastroenterology & hepatologyFemaleCohort studyResearch ArticleAdultmedicine.medical_specialtyEndocrine DisordersGastroenterology and HepatologyNephrolithiasisResearch and Analysis Methods03 medical and health sciencesInsulin resistanceInternal medicineDiabetes mellitusmedicineDiabetes MellitusHumansNutritionEndocrine Physiologybusiness.industrylcsh:RBody WeightChemical Compoundsnutritional and metabolic diseasesBiology and Life Sciencesmedicine.diseasedigestive system diseasesDietUric AcidFatty LiverMetabolic Disorderslcsh:QMetabolic syndromeInsulin ResistancebusinessBody mass indexAcidsPloS one
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Role of HDL function and LDL atherogenicity on cardiovascular risk: a comprehensive examination

2019

[Background] High-density lipoprotein (HDL) functionality and low-density lipoprotein (LDL) atherogenic traits can describe the role of both particles on cardiovascular diseases more accurately than HDL- or LDL-cholesterol levels. However, it is unclear how these lipoprotein properties are particularly affected by different cardiovascular risk factors.

MalePhysiologyhumanosLipoproteïnesBlood PressureCardiovascular Medicine030204 cardiovascular system & hematologyOxidacióBiochemistryVascular Medicinelipoproteínas616.1 - Patologia del sistema circulatori dels vasos sanguinis. Trastorns cardiovasculars:Ciencias de la Salud::Cardiología [Materias Investigacion]Endocrinology0302 clinical medicineRisk FactorsSex factorsSistema cardiovascular--MalaltiesMedicine and Health SciencesMedicine030212 general & internal medicinemediana edadancianoMultidisciplinaryQChemical ReactionsAge FactorsRMiddle AgedadultoLipidsBody FluidsLipoproteins LDLLipoproteïnes de densitat baixaChemistryBloodCholesterolCardiovascular DiseasesPhysical SciencesHypertensiondiabetes mellitusMedicineFemalelipids (amino acids peptides and proteins)AnatomyLipoproteins HDLColesterolResearch ArticleAdultEndocrine DisordersScienceLipoproteinsdislipidemiasenfermedades cardiovascularesBlood Plasma03 medical and health sciencesSex FactorsOxidationHumansfactores de riesgoAgedDyslipidemiasbusiness.industryCholesterol HDLBiology and Life SciencesProteinsCholesterol LDLAtherosclerosisSistema cardiovascular -- Malalties -- Factors de riscCross-Sectional StudiesLipoproteïnes de densitat altaDyslipidemiaDiabetes Mellitus Type 2Metabolic DisordersaterosclerosisbusinessHumanitiesestudios transversales
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Circulating, cell-free DNA as a marker for exercise load in intermittent sports

2017

Background Attempts to establish a biomarker reflecting individual player load in intermittent sports such as football have failed so far. Increases in circulating DNA (cfDNA) have been demonstrated in various endurance sports settings. While it has been proposed that cfDNA could be a suitable marker for player load in intermittent sports, the effects on cfDNA of repeated sprinting as an essential feature in intermittent sports are unknown. For the first time, we assessed both alterations of cfDNA due to repeated maximal sprints and due to a professional football game. Methods Nine participants were subjected to a standardised sprint training session with cross-over design of five maximal s…

MalePhysiologylcsh:MedicineBiochemistryBlood Plasma796 Athletic and outdoor sports and gamesRunningMedicine and Health SciencesHumansPublic and Occupational HealthSports and Exercise Medicinelcsh:ScienceExerciseBehavior796 SportBiological Locomotionlcsh:RBiology and Life SciencesPhysical ActivitySports ScienceBody FluidsCapillariesBloodPhysical FitnessCardiovascular AnatomyRecreationBlood VesselsFemalelcsh:QAnatomyGamesCell-Free Nucleic Acidshuman activitiesBiomarkersSportsResearch Article
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Metabolomics of the aqueous humor in the rat glaucoma model induced by a series of intracamerular sodium hyaluronate injection

2015

Glaucoma models are helpful to study disease characteristics and to design new therapeutic options. Metabolomic profiling approach have been used to elucidating the molecular characteristics of the aqueous humor. Juvenile male Wistar rats experimental (n = 15) and controls (n = 6) were used for these studies. Experimental rats received weekly intracamerular injection of 25 µl of sodium hyaluronate in the left eye and sterile saline solution in the right eye, consecutively for ten weeks. Rats were subjected to anterior/posterior eye segment examinations, intraocular pressure (IOP), and flash electroretinograms (ERG). The aqueous humor was collected at endpoints and analyzed by Nuclear Magnet…

MalePosterior Eye Segmentmedicine.medical_specialtyIntraocular pressureMagnetic Resonance Spectroscopygenetic structuresSodium hyaluronateCarbohydratesGlaucomaAqueous humorAqueous humor Glaucoma model Metabolomics Nuclear magnetic resonance Rats Sodium hyaluronateInjectionsAqueous HumorPathogenesisCellular and Molecular Neurosciencechemistry.chemical_compoundMetabolomicsAnterior Eye SegmentOphthalmologyElectroretinographymedicineAnimalsMetabolomicsAmino AcidsHyaluronic AcidRats WistarIntraocular PressureGlaucomamedicine.diseaseLipidseye diseasesSensory SystemsRatsSurgeryDisease Models AnimalOphthalmologychemistrysense organsErgFollow-Up StudiesExperimental Eye Research
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2-Hydroxyoleate, a nontoxic membrane binding anticancer drug, induces glioma cell differentiation and autophagy

2012

Despite recent advances in the development of new cancer therapies, the treatment options for glioma remain limited, and the survival rate of patients has changed little over the past three decades. Here, we show that 2-hydroxyoleic acid (2OHOA) induces differentiation and autophagy of human glioma cells. Compared to the current reference drug for this condition, temozolomide (TMZ), 2OHOA combated glioma more efficiently and, unlike TMZ, tumor relapse was not observed following 2OHOA treatment. The novel mechanism of action of 2OHOA is associated with important changes in membrane-lipid composition, primarily a recovery of sphingomyelin (SM) levels, which is markedly low in glioma cells bef…

MaleProgrammed cell deathTime FactorsCell SurvivalMAP Kinase Signaling SystemCellular differentiationMice NudeAntineoplastic AgentsOleic AcidsBiologyglioma biomarkerfatty acidsMembrane LipidsMicePhosphatidylinositol 3-Kinases2-Hydroxyoleic AcidGliomaCell Line TumormedicineAutophagyTemozolomideAnimalsHumansPI3K/AKT/mTOR pathwayCell ProliferationMultidisciplinaryTemozolomideMicroscopy ConfocalDose-Response Relationship DrugCell growthCell MembraneRetinoblastoma proteinCell DifferentiationGliomaBiological Sciencesmedicine.diseaseXenograft Model Antitumor AssaysCell biologyDacarbazineProtein TransportCancer researchbiology.proteinras Proteinssphingomyelin synthaseProto-Oncogene Proteins c-aktcancer drug targetmedicine.drug
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Heterogeneity of osteogenesis imperfecta. Biochemical and morphological findings in a case of type III according to Sillence.

1986

A male infant with pale-blue sclerae, who died at the age of 6 weeks through the aspiration of food, presented multiple fractures and deformation of the long tubular bones. The clinical and radiological findings and the course indicated osteogenesis imperfecta, type III, according to Sillence's classification. The family history was unremarkable. Light and electron microscopic studies of iliac crest bone obtained postmortem, showed an abrupt interruption of endochondral ossification, with an active periosteal ossification. In the region of the fractures, a mixed desmochondral callus was seen. The endoplasmic reticulum of the osteoblasts was markedly dilated, the mitochondria were swollen. T…

MaleProlineEndoplasmic ReticulumHydroxylationIliac crestHydroxylysineBone and BonesOsteogenesis Imperfecta Type IIIchemistry.chemical_compoundMedicineHumansAmino AcidsEndochondral ossificationSkinOsteoblastsbusiness.industryOsteoidCartilageInfantAnatomyOsteogenesis Imperfectamedicine.diseaseChromatography Ion ExchangeHydroxylysinemedicine.anatomical_structureCartilagechemistryOsteogenesis imperfectaPediatrics Perinatology and Child HealthCollagenbusinessMitochondrial SwellingReticulumEuropean journal of pediatrics
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Inhibition of Proteasomal Glucocorticoid Receptor Degradation Restores Dexamethasone-Mediated Stabilization of the Blood–Brain Barrier After Traumati…

2013

To establish the molecular background for glucocorticoid insensitivity, that is, failure to reduce edema formation and to protect blood-brain barrier integrity after acute traumatic brain injury.Controlled animal study.University research laboratory.Male C57Bl/6N mice.Mechanical brain lesion by controlled cortical impact.Our study demonstrates that 1) proteasomal glucocorticoid receptor degradation is established in brain endothelial cells after traumatic brain injury as a form of posttranslational glucocorticoid receptor modification; 2) inhibition of the proteasomal degradation pathway with bortezomib (0.2 mg/kg) in combination with the glucocorticoid dexamethasone (10 mg/kg) by subcutane…

MaleProteasome Endopeptidase ComplexTraumatic brain injuryBlotting WesternBrain EdemaPharmacologyReal-Time Polymerase Chain ReactionCritical Care and Intensive Care MedicineBlood–brain barrierSensitivity and SpecificityDexamethasoneStatistics NonparametricBortezomibMiceRandom AllocationReceptors GlucocorticoidGlucocorticoid receptorReference ValuesmedicineAnimalsRNA MessengerReceptorDexamethasonebusiness.industryBortezomibmedicine.diseaseBoronic AcidsImmunohistochemistryMice Inbred C57BLBlotDisease Models Animalmedicine.anatomical_structureBlood-Brain BarrierBrain InjuriesPyrazinesMultivariate AnalysisBlood Gas AnalysisbusinessGlucocorticoidmedicine.drugCritical Care Medicine
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