Search results for "acids"

showing 10 items of 3520 documents

The BIOSAFEPAPER project for in vitro toxicity assessments: preparation, detailed chemical characterisation and testing of extracts from paper and bo…

2008

International audience; Nineteen food contact papers and boards and one non-food contact board were extracted following test protocols developed within European Union funded project BIOSAFEPAPER. The extraction media were either hot or cold water, 95% ethanol or Tenax, according to the end use of the sample. The extractable dry matter content of the samples varied from 1200 to 11,800 mg/kg (0.8-35.5 mg/dm2). According to GC-MS the main substances extracted into water were pulp-derived natural products such as fatty acids, resin acids, natural wood sterols and alkanols. Substances extracted into ethanol particularly, were diisopropylnaphthalenes, alkanes and phthalic acid esters. The non-foo…

PaperPolymersCytotoxicityTenaxFood Contamination[SDV.TOX.TCA]Life Sciences [q-bio]/Toxicology/Toxicology and food chainToxicologymedicine.disease_causeRisk Assessment01 natural sciencesGas Chromatography-Mass Spectrometrychemistry.chemical_compound0404 agricultural biotechnologyBacillus cereusToxicity TestsmedicineAnimalsHumansBioassaymedia_common.cataloged_instanceChemical analysisEuropean unionmedia_commonChromatographyEthanolMutagenicity TestsFatty Acids010401 analytical chemistryFood PackagingWaterEnvironmental Exposure04 agricultural and veterinary sciencesGeneral MedicineBSTFA040401 food science0104 chemical sciencesSterolsPhthalic acidchemistryGlobal bioassayBIOSAFEPAPERToxicityBiological AssaySafetyGas chromatography–mass spectrometryGenotoxicityFood Science
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The nucleus negatively controls the synthesis of mitochondrial proteins in the sea urchin egg.

1983

Enucleation of Paracentrotus lividus eggs, followed by parthenogenetic activation induces a sharp increase in the synthesis of mitochondrial proteins as shown by electrofluorography after in vivo labeling with radioactive amino acids. These results further substantiate the hypothesis that the cell nucleus negatively controls mitochondrial replication in the sea urchin egg.

ParthenogenesisBiologyParacentrotus lividusbiology.animalmedicineProtein biosynthesisAnimalsAmino AcidsSea urchinPolyacrylamide gel electrophoresisOvumchemistry.chemical_classificationCell NucleusProteinsCell BiologyParthenogenesisAnatomybiology.organism_classificationAmino acidCell biologyMitochondriaCell nucleusmedicine.anatomical_structurechemistryProtein BiosynthesisSea Urchinsembryonic structuresElectrophoresis Polyacrylamide GelFemaleNucleusCell biology international reports
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Desipramine induces disorder in cholesterol-rich membranes:implications for viral trafficking

2009

In this study, the effect of desipramine (DMI) on phospholipid bilayers and parvoviral entry was elucidated. In atomistic molecular dynamics simulations, DMI was found to introduce disorder in cholesterol-rich phospholipid bilayers. This was manifested by a decrease in the deuterium order parameter S(CD) as well as an increase in the membrane area. Disordering of the membrane suggested DMI to destabilize cholesterol-rich membrane domains (rafts) in cellular conditions. To relate the raft disrupting ability of DMI with novel biological relevance, we studied the intracellular effect of DMI using canine parvovirus (CPV), a virus known to interact with endosomal membranes and sphingomyelin, as …

Parvovirus CanineEndosomeBiophysicsPhospholipidBiologyAntidepressive Agents Tricyclicchemistry.chemical_compoundDogsStructural BiologyDesipraminemedicineAnimalsComputer SimulationMolecular BiologyCells CulturedMolecular StructureVesicleCell MembraneDesipramineCell BiologyRaftDisease Models AnimalMembraneCholesterolchemistryBiochemistryBiophysicslipids (amino acids peptides and proteins)Sphingomyelinhuman activitiesIntracellularmedicine.drug
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Familial hypercholesterolaemia: A global call to arms

2015

Familial Hypercholesterolaemia (FH) is the commonest autosomal co-dominantly inherited condition affecting man. It is caused by mutation in one of three genes, encoding the low-density lipoprotein (LDL) receptor, or the gene for apolipoprotein B (which is the major protein component of the LDL particle), or in the gene coding for PCSK9 (which is involved in the degradation of the LDL-receptor during its cellular recycling). These mutations result in impaired LDL metabolism, leading to life-long elevations in LDL-cholesterol (LDL-C) and development of premature atherosclerotic cardiovascular disease (ASCVD) [1], [2] and [3]. If left untreated, the relative risk of premature coronary artery d…

PathologyApolipoprotein BDisease030204 cardiovascular system & hematologymedicine.disease_causeGlobal HealthDISEASEDoenças Cardio e Cérebro-vasculares0302 clinical medicineHyperlipoproteinemia Type IISocieties MedicalRISK0303 health sciencesMutationbiology3. Good healthPREVALENCEEuropelipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicineFamilial hypercholesterolaemiaLife Sciences & Biomedicinemedicine.medical_specialtyHeterozygote1102 Cardiovascular Medicine And HaematologyHyperlipoproteinemia Type II03 medical and health sciencesInternal medicinemedicineHumans030304 developmental biologyScience & Technologybusiness.industryGUIDANCEPCSK9Heterozygote advantage1103 Clinical SciencesEndocrinologyPeripheral Vascular DiseaseCardiovascular System & HematologyReceptors LDLRECEPTORES DE LIPOPROTEÍNASRelative riskMutationbiology.proteinCardiovascular System & CardiologyFamilial HypercholesterolaemiabusinessCLINICIANLipoprotein
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Lung myofibroblasts are characterized by down-regulated cyclooxygenase-2 and its main metabolite, prostaglandin E2.

2013

Background: Prostaglandin E2 (PGE(2)), the main metabolite of cyclooxygenase (COX), is a well-known anti-fibrotic agent. Moreover, myofibroblasts expressing alpha-smooth muscle actin (alpha-SMA), fibroblast expansion and epithelial-mesenchymal transition (EMT) are critical to the pathogenesis of idiopathic pulmonary fibrosis (IPF). Our aim was to investigate the expression of COX-2 and PGE(2) in human lung myofibroblasts and establish whether fibroblast-myofibroblast transition (FMT) and EMT are associated with COX-2 and PGE(2) down-regulation. Methods: Fibroblasts obtained from IPF patients (n = 6) and patients undergoing spontaneous pneumothorax (control, n = 6) and alveolar epithelial ce…

PathologyPulmonologyMetaboliteImmunofluorescencelcsh:MedicineBiochemistrychemistry.chemical_compoundIdiopathic pulmonary fibrosisMolecular Cell BiologyPulmonary fibrosisProstaglandin E2Myofibroblastslcsh:ScienceLungCells CulturedFisiologia cel·lularMultidisciplinarybiologyFibrosi pulmonarrespiratory systemExtracellular Matrixmedicine.anatomical_structureCytokinesMedicinelipids (amino acids peptides and proteins)Immunohistochemical AnalysisMyofibroblastResearch ArticleSignal Transductionmedicine.drugmedicine.medical_specialtyEpithelial-Mesenchymal TransitionImmunologyInterstitial Lung DiseasesDinoprostonePulmonary fibrosisTransforming Growth Factor beta1ImmunofluorescènciaGrowth FactorsCell Line TumormedicineHumansEpithelial–mesenchymal transitionFibroblastBiologyCell Proliferationlcsh:RProteinsEpithelial Cellsmedicine.diseaseActinsIdiopathic Pulmonary Fibrosisrespiratory tract diseasesGene Expression RegulationchemistryCyclooxygenase 2Immune SystemCase-Control StudiesImmunologic Techniquesbiology.proteinCancer researchClinical Immunologylcsh:QCyclooxygenaseBiomarkersPLoS ONE
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The role of hyaluronic acid and amino acid against the aging of the human skin: A clinical and histological study.

2020

Background In esthetic medicine, different techniques have been used against the aging of the human skin especially in the facial area. Hyaluronic acid is used for improving the quantity of water and extracellular matrix molecule. The aim of this study is a clinical and histological evaluation of the effect of low-molecular-weight hyaluronic acid fragments mixed with amino acid (HAAM) on the rejuvenation the face skin treated with intradermal microinjections. Methods Twenty women with mean age 45 range from 35 to 64 were studied, thereof 8 in menopause and 12 of childbearing age. The patients were treated with the HAAM products by mesotherapy technique; before and after 3 months of the ther…

Pathologymedicine.medical_specialtyAgingHuman skinDermatologyCosmetic Techniqueshyaluronic acid fragmentsExtracellular matrix030207 dermatology & venereal diseases03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDermisHyaluronic acidMedicineHumansRejuvenationAmino AcidsHyaluronic AcidFibroblastchemistry.chemical_classificationbusiness.industryMiddle AgedAmino acidSkin AgingMesotherapymedicine.anatomical_structurechemistry030220 oncology & carcinogenesisReticular connective tissueFemalebusinessamino acidbiomodulationJournal of cosmetic dermatologyREFERENCES
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Swelling, Acidosis, and Irreversible Damage of Glial Cells from Exposure to Arachidonic Acid in vitro

1994

Swelling and damage of C6 glioma cells and of primary cultured astrocytes were analyzed in vitro during incubation with arachidonic acid (AA; 20:4). The cells were suspended in a physiological medium supplemented with AA at concentrations of 0.001–1.0 m M. Cell swelling was quantified by flow cytometry with hydrodynamic focusing. Flow cytometry was also utilized for assessment of cell viability by exclusion of the fluorescent dye propidium iodide and for measurement of the intracellular pH (pHi) by 2′,7′-bis-(2-carboxyethyl)−5(and −6)carboxyfluorescein. Administration of AA caused an immediate dose-dependent swelling of C6 glioma cells, even at a concentration of 0.01 m M. At this level cel…

Pathologymedicine.medical_specialtyCell SurvivalLinoleic acidIntracellular pHBiologychemistry.chemical_compoundTumor Cells CulturedmedicineLactic AcidViability assayPropidium iodideCell damageArachidonic AcidFatty AcidsSodiumHydrogen-Ion Concentrationmedicine.diseaseMolecular biologyNeurologychemistryCell cultureAstrocytesLactatesSteroidsArachidonic acidNeurology (clinical)Swellingmedicine.symptomAcidosisCardiology and Cardiovascular MedicineNeurogliaJournal of Cerebral Blood Flow & Metabolism
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Evaluation of polysialic acid in the diagnosis of Wilms' tumor. A comparative study on urinary tract tumors and non-neuroendocrine tumors.

1988

The polysialic acid moiety of the neural cell adhesion molecule has been shown to represent an onco-developmental antigen which can be detected in both embryonic human kidney and Wilms’ tumor but not in normal adult human kidney. In the present comparative study, Wilms’ tumors, clear cell (bone-metastasizing) sarcomas of kidney, cystic nephromas, renal cell carcinomas, transitional cell carcinomas and papillomas of the renal pelvis, ureter and urinary bladder (as well normal transitional epithelium from these regions), Ewing sarcomas, hepatoblastomas, rhabdomyosarcomas, and carcinomas of the stomach, colon, exocrine pancreas, lung, and esophagus, were investigated immunohistochemically for …

Pathologymedicine.medical_specialtyKidneyUrologic NeoplasmsPolysialic acidImmunoblottingAntibodies MonoclonalWilms' tumorGeneral MedicineBiologyNeuroendocrine tumorsTransitional epitheliummedicine.diseaseImmunohistochemistryWilms TumorKidney NeoplasmsMicemedicine.anatomical_structuremedicineBiomarkers TumorSialic AcidsAnimalsNeural cell adhesion moleculeRenal pelvisClear cellVirchows Archiv. B, Cell pathology including molecular pathology
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The Participation of the Complement System in Atherosclerotic Vascular Disease

1991

Atherosclerosis is a vascular disease of large and medium-sized arteries wherein the tunica intima becomes thickened due to lipid accumulation, mostly cholesterol and its esters, smooth muscle cell proliferation, and increased deposition of connective tissue matrix. A major risk factor in the development of this disease is hypercholesterolemia arising from elevated levels of low density lipoproteins (LDL). The earliest recognizable lesion, which may be a precursor to the fibrofatty plaque, is the fatty streak. It is predominantly composed of monocyte-derived macrophage foam cells, i.e. cells ladened with intracellular lipid droplets. Hence, a fundamental aspect of atherogenesis is the insud…

Pathologymedicine.medical_specialtyVascular smooth muscleChemistryMonocyteFatty streakConnective tissueTunica intimaLesionmedicine.anatomical_structureLipid dropletmedicineMacrophagelipids (amino acids peptides and proteins)medicine.symptom
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Niemann-Pick Type C-2 Disease: Identification by Analysis of Plasma Cholestane-3β,5α,6β-Triol and Further Insight into the Clinical Phenotype.

2014

Niemann-Pick type C disease is a rare disorder caused by impaired intracellular lipid transport due to mutations in either the NPC1 or the NPC2 gene. Ninety-five % of NPC patients show mutations in the NPC1 gene. A much smaller number of patients suffer from NPC2 disease and present respiratory failure as one of the most frequent symptoms. Several plasma oxysterols are highly elevated in NPC1 and can be used as a biomarker in the diagnosis of NPC1.Plasma cholestane-3β,5α,6β-triol was evaluated as biomarker for NPC2 by GC/MS and LC-MS/MS analysis. The diagnosis was confirmed by Sanger sequencing and filipin staining.We report three NPC2 patients with typical respiratory problems and a detail…

Pathologymedicine.medical_specialtycongenital hereditary and neonatal diseases and abnormalitiesbusiness.industrynutritional and metabolic diseasesDiseaseIntracellular lipid transportmedicine.diseaseArticlenervous system diseasesRespiratory failurehemic and lymphatic diseasesImmunologyMedicineCholestane 3β 5α 6β triolBiomarker (medicine)lipids (amino acids peptides and proteins)NPC1businessPulmonary alveolar proteinosisGeneJIMD reports
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