Search results for "amine"

showing 10 items of 7299 documents

Arabidopsis Serine Decarboxylase 1 (SDC1) in Phospholipid and Amino Acid Metabolism

2018

Arabidopsis thaliana serine decarboxylase 1 (SDC1) catalyzes conversion of serine to ethanolamine, the first reaction step of phosphatidylcholine and phosphatidylethanolamine biosynthesis. However, an involvement of SDC1 in amino acid metabolism remains elusive despite that serine is the substrate of SDC1. Here, we showed that SDC1 localizes in mitochondria although phosphatidylcholine and phosphatidylethanolamine are known to be produced in the endoplasmic reticulum (ER). Moreover, we found that overexpression of SDC1 decreased levels of amino acid compounds derived from mitochondrial tricarboxylic acid cycle. These results suggest that mitochondria-localized SDC1 plays an important role i…

0301 basic medicinechemistry.chemical_classificationPhosphatidylethanolamineArabidopsis thalianaEndoplasmic reticulumPhospholipidPlant ScienceMetabolismlcsh:Plant cultureAmino acidSerineCitric acid cycle03 medical and health scienceschemistry.chemical_compound030104 developmental biologychemistryBiochemistryBiosynthesislcsh:SB1-1110phospholipid biosynthesisserine decarboxylaseglycerolipid metabolismphospholipidOriginal ResearchFrontiers in Plant Science
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Biomarkers and clinical characteristics of autoimmune chronic spontaneous urticaria: Results of the PURIST Study.

2019

Background: Autoimmune chronic spontaneous urticaria (aiCSU) is an important subtype of chronic spontaneous urticaria (CSU) in which functional IgG autoantibodies to IgE or its high-affinity receptor (FcεRI) induces mast cell degranulation and subsequent symptom development. However, it has not been tightly characterized. This study aimed to better define the clinical and immunological features and to explore potential biomarkers of aiCSU. Methods: This was a multinational, multicenter study of 182 CSU patients. The clinical features studied included: urticaria activity and impact (UAS7 and quality of life); autologous serum skin test (ASST); IgG anti-FcεRI and IgG anti-IgE; IgG-anti-thyrop…

0301 basic medicinechronic spontaneous urticariaMaleBasophilIgG autoantibodiesImmunoglobulin EAutoantigensHistamine Releasechemistry.chemical_compound0302 clinical medicineIron-Binding ProteinsImmunology and AllergyChronic UrticariaReceptorAutologous serum skin testbiologyDegranulationMiddle AgedChronic spontaneous urticariaAntibodies Anti-IdiotypicBasophilsmedicine.anatomical_structurePhenotypeAutologous serum skin testautologous serum skin testFemaleSymptom AssessmentHistamineAdultAdolescentImmunologyIodide PeroxidaseAutoimmune Diseases03 medical and health sciencesYoung Adultautoimmune CSUmedicineJournal ArticleHumansAgedAutoantibodiesbusiness.industryReceptors IgEAutoantibodyBasophil activation030104 developmental biology030228 respiratory systemchemistryImmunoglobulin GImmunologyBasophil activation assaysbiology.proteinAutoimmune CSUbusinessbasophil activation assaysBiomarkersAllergyREFERENCES
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Effects of Dopamine on the Immature Neurons of the Adult Rat Piriform Cortex

2020

The layer II of the adult piriform cortex (PCX) contains a numerous population of immature neurons. Interestingly, in both mice and rats, most, if not all, these cells have an embryonic origin. Moreover, recent studies from our laboratory have shown that they progressively mature into typical excitatory neurons of the PCX layer II. Therefore, the adult PCX is considered a “non-canonical” neurogenic niche. These immature neurons express the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a molecule critical for different neurodevelopmental processes. Dopamine (DA) is a relevant neurotransmitter in the adult CNS, which also plays important roles in neural development and …

0301 basic medicinedopamine D2 receptorPSA-NCAMPopulationBiologylcsh:RC321-57103 medical and health scienceschemistry.chemical_compoundpiriform cortex0302 clinical medicineDopaminePiriform cortexDopamine receptor D2medicineeducationNeurotransmitterlcsh:Neurosciences. Biological psychiatry. Neuropsychiatryeducation.field_of_studyGeneral NeuroscienceDopaminergicBrief Research ReportCell biology030104 developmental biologychemistrynervous systemplasticityNeural cell adhesion moleculedopamineNeural development030217 neurology & neurosurgeryNeurosciencemedicine.drug
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Melatonin reduces inflammatory response in human intestinal epithelial cells stimulated by interleukin‐1β

2019

Melatonin is the main secretory product of the pineal gland, and it is involved in the regulation of periodic events. A melatonin production independent of the photoperiod is typical of the gut. However, the local physiological role of melatonin at the intestinal tract is poorly characterized. In this study, we evaluated the anti-inflammatory activities of melatonin in an in vitro model of inflamed intestinal epithelium. To this purpose, we assessed different parameters usually associated with intestinal inflammation using IL-1 beta-stimulated Caco-2 cells. Differentiated monolayers of Caco-2 cells were preincubated with melatonin (1 nmol/L-50 mu mol/L) and then exposed to IL-1 beta. After …

0301 basic medicineendocrine systemmedicine.medical_specialtyantioxidantDNA damageInterleukin-1betainflammatory bowel diseasesdietary supplementsMelatonin03 medical and health sciencesPineal gland0302 clinical medicineEndocrinologyCell surface receptorSettore BIO/10 - BiochimicaInternal medicinemedicineHumansMelatoninInflammationN-acetyl-5-methoxy-tryptamineInterleukin-6Chemistryantioxidants; dietary supplements; DNA damage; DNA methylation; inflammatory bowel diseases; N-acetyl-5-methoxy-tryptamine; NF-κB activationInterleukin-8AntagonistCell DifferentiationEpithelial CellsDNA MethylationSettore CHIM/08 - Chimica FarmaceuticaIntestinal epitheliumIntestinesSettore BIO/18 - Geneticaantioxidants030104 developmental biologyEndocrinologymedicine.anatomical_structureNF-κB activationCyclooxygenase 2dietary supplementParacellular transportDNA damageCaco-2 CellsLuzindolehormones hormone substitutes and hormone antagonists030217 neurology & neurosurgerySignal Transductionmedicine.drugJournal of Pineal Research
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Structure-Activity Relationship Analysis of 3-Phenylcoumarin-Based Monoamine Oxidase B Inhibitors

2018

Monoamine oxidase B (MAO-B) catalyzes deamination of monoamines such as neurotransmitters dopamine and norepinephrine. Accordingly, small-molecule MAO-B inhibitors potentially alleviate the symptoms of dopamine-linked neuropathologies such as depression or Parkinson's disease. Coumarin with a functionalized 3-phenyl ring system is a promising scaffold for building potent MAO-B inhibitors. Here, a vast set of 3-phenylcoumarin derivatives was designed using virtual combinatorial chemistry or rationally de novo and synthesized using microwave chemistry. The derivatives inhibited the MAO-B at 100 nM−1 μM. The IC50 value of the most potent derivative 1 was 56 nM. A docking-based structure-activi…

0301 basic medicineentsyymitParkinson's diseaseParkinsonin tautita311101 natural scienceslääkesuunnittelumonoamine oxidase B (MAO-B)lcsh:Chemistry03 medical and health scienceschemistry.chemical_compoundstructure-activity relationship (SAR)Dopamine3-phenylcoumarinmedicineStructure–activity relationshipoksidoreduktaasitkumariinitta116ta317inhibiittoritOriginal Researchchemistry.chemical_classificationbiologyvirtual drug designta1182General ChemistryCoumarin3. Good health0104 chemical sciences010404 medicinal & biomolecular chemistryChemistry030104 developmental biologyMonoamine neurotransmitterEnzymeBiochemistrychemistrylcsh:QD1-999Docking (molecular)biology.proteinParkinson’s diseaseMonoamine oxidase BMonoamine oxidase Amedicine.drugFrontiers in Chemistry
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Endocannabinoid LTD in Accumbal D1 Neurons Mediates Reward-Seeking Behavior

2020

Summary The nucleus accumbens (NAc) plays a key role in drug-related behavior and natural reward learning. Synaptic plasticity in dopamine D1 and D2 receptor medium spiny neurons (MSNs) of the NAc and the endogenous cannabinoid (eCB) system have been implicated in reward seeking. However, the precise molecular and physiological basis of reward-seeking behavior remains unknown. We found that the specific deletion of metabotropic glutamate receptor 5 (mGluR5) in D1-expressing MSNs (D1miRmGluR5 mice) abolishes eCB-mediated long-term depression (LTD) and prevents the expression of drug (cocaine and ethanol), natural reward (saccharin), and brain-stimulation-seeking behavior. In vivo enhancement…

0301 basic medicineglutamate02 engineering and technologyMolecular neuroscienceBiologyNucleus accumbensMGLUR5 receptorsMedium spiny neuronArticleinduced reinstatementBehavioral Neuroscience03 medical and health sciencesDopamineDopamine receptor D2lipasemedicinelong-term depression[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]lcsh:ScienceLong-term depressionrelapseMultidisciplinaryMetabotropic glutamate receptor 5021001 nanoscience & nanotechnologyEndocannabinoid systemin-vivo exposure3. Good healthrats030104 developmental biologynervous systemethanol-seekingplasticitylcsh:Q[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Molecular Neuroscience0210 nano-technologyNeurosciencepsychological phenomena and processesNeurosciencemedicine.drugiScience
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Is there evidence for a rostral-caudal gradient in fronto-striatal loops and what role does dopamine play?

2018

Research has shown that the lateral prefrontal cortex (LPFC) may be hierarchically organized along a rostral-caudal functional gradient such that control processing becomes progressively more abstract from caudal to rostral frontal regions. Here, we briefly review the most recent functional MRI, neuropsychological, and electrophysiological evidence in support of a hierarchical LPFC organization. We extend these observations by discussing how such a rostral-caudal gradient may also exist in the striatum and how the dopaminergic system may play an important role in the hierarchical organization of fronto-striatal loops. There is evidence indicating that a rostral-caudal gradient of dopamine r…

0301 basic medicinehierarchical processingReviewStriatumBiologylcsh:RC321-57103 medical and health sciences0302 clinical medicineDopaminemedicineHierarchical organizationlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryGeneral NeuroscienceDopaminergicNeuropsychologylateral prefrontal cortexElectrophysiology030104 developmental biologynervous systemDopamine receptorfronto-striatal loopsreceptor distributiondopamineLateral prefrontal cortexNeuroscience030217 neurology & neurosurgeryNeurosciencemedicine.drug
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Acetaldehyde as the first hit of addictive behaviour

2016

Unhealthy alcohol use is common in the Western society, which puts risk of health consequences, causing multiple behavioural injuries. Increasing evidence focuses on acetaldehyde, the first metabolite of ethanol, as the mediator of the several behavioural actions of alcohol, including its rewarding and motivational effects. In particular, acetaldehyde induces dopamine release in the nucleus accumbens modulating primary alcohol rewarding effect, drug seeking, and relapse behaviour. Recent behavioural studies point at acetaldehyde as a drug of abuse since its oral self-administration is induced and maintained in an operant/conflict paradigm. These findings provide further evidence on the role…

0301 basic medicinemedicine.medical_specialtyAddictive behaviourmedia_common.quotation_subjectAlcohol abuseAlcoholAcetaldehydePlant SciencePharmacologyNucleus accumbensGeneral Biochemistry Genetics and Molecular BiologyEthanol-related effect03 medical and health scienceschemistry.chemical_compound0302 clinical medicineMediatorDopamineEthanol-related effectsAcetaldehyde; Addictive behaviour; Ethanol-related effects; Biochemistry Genetics and Molecular Biology (all); Plant Science; Biochemistry (medical)medicinePsychiatrylcsh:QH301-705.5media_commonBiochemistry Genetics and Molecular Biology (all)EthanolAddictionBiochemistry (medical)Acetaldehydemedicine.disease030104 developmental biologylcsh:Biology (General)chemistryPsychology030217 neurology & neurosurgerymedicine.drugJournal of Biological Research - Bollettino della Società Italiana di Biologia Sperimentale
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Pain-induced alterations in the dynorphinergic system within the mesocorticolimbic pathway: Implication for alcohol addiction.

2020

Latest studies have revealed that pain negatively impacts on reward processing and motivation leading to negative affective states and stress. These states not only reduce quality of life of patients by increasing the appearance of psychiatric comorbidities, but also have an important impact on vulnerability to drug abuse, including alcohol. In fact, clinical, epidemiological but also preclinical studies have revealed that the presence of pain is closely related to alcohol use disorders (AUDs). All this evidence suggests that pain is a factor that increases the risk of suffering AUD, predicting heavy drinking behavior and relapse drinking in those patients with a previous history of AUD. Th…

0301 basic medicinemedicine.medical_specialtyDopamineAlcohol use disorderMesolimbic pathwayκ-opioid receptor03 medical and health sciencesCellular and Molecular NeuroscienceReward system0302 clinical medicineQuality of lifeRewardmental disordersmedicineHumansPsychiatrybusiness.industryDopaminergicChronic painmedicine.diseaseSubstance abuseAlcoholism030104 developmental biologyQuality of LifeChronic Painbusiness030217 neurology & neurosurgeryJournal of neuroscience researchREFERENCES
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Neurotransmitters and Behavioral Alterations Induced by Nickel Exposure.

2020

Background:: Nickel ions (Ni2+) are a heavy metal with wide industrial uses. Environmental and occupational exposures to Ni are potential risk factors for brain dysfunction and behavioral and neurological symptoms in humans. Methods: We reviewed the current evidence about neurochemical and behavioral alterations associated with Ni exposure in laboratory animals and humans. Results: Ni2+ exposure can alter (both inhibition and stimulation) dopamine release and inhibit glutamate NMDA receptors. Few reports claim an effect of Ni2+ at the level of GBA and serotonin neurotransmission. At behavioral levels, exposure to Ni2+ in rodents alters motor activity, learning and memory as well as anxiety…

0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismPopulationStimulationEnvironmental Illness03 medical and health scienceschemistry.chemical_compound0302 clinical medicineNeurochemicalDopamineNickelInternal medicineImmunology and AllergyMedicineAnimalsHumanseducationNeurotransmittereducation.field_of_studyBehaviorNeurotransmitter Agentsbusiness.industryMental DisordersGlutamate receptorEnvironmental Exposure030104 developmental biologyEndocrinologychemistryNMDA receptorSerotoninbusiness030217 neurology & neurosurgerymedicine.drugEndocrine, metabolicimmune disorders drug targets
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