Search results for "anti-inflammation"

showing 4 items of 4 documents

Protective Effect of Pogostone on 2,4,6-Trinitrobenzenesulfonic Acid-Induced Experimental Colitis via Inhibition of T Helper Cell

2017

Inflammatory bowel disease (IBD) is a chronic immune-related disease mainly caused by the disequilibrium of T helper (Th) cell paradigm? Pogostone (PO) is one of the major chemical constituents of Pogostemon cablin (Blanco) Benth. The present study aims to investigate the potential benefit of PO against IBD in a 2,4,6-trinitrobenzenesulfonic acid (TNBS)-induced experimental colitis model. PO treatment by enema significantly brought down the disease activity index (DAI) of the TNBS-challenged rats, which was manifested by the ameliorated inflammatory features including ulceration, adhesion, and edema. Hematoxylin-eosin (HE) staining and immunohistochemistry analysis showed that PO effectivel…

0301 basic medicineexperimental colitisCellPharmacologyInflammatory bowel diseaseProinflammatory cytokine03 medical and health scienceschemistry.chemical_compound246-Trinitrobenzenesulfonic acidEdemamedicinePharmacology (medical)T helper cellOriginal ResearchPharmacologybiologyCell growthbusiness.industrylcsh:RM1-950pogostoneT helper cellmedicine.diseaseTNBSanti-inflammationdigestive system diseaseslcsh:Therapeutics. Pharmacology030104 developmental biologymedicine.anatomical_structurechemistryMyeloperoxidaseImmunologybiology.proteinmedicine.symptombusinessFrontiers in Pharmacology
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2′-O-Galloylhyperin Isolated From Pyrola incarnata Fisch. Attenuates LPS-Induced Inflammatory Response by Activation of SIRT1/Nrf2 and Inhibition of …

2018

2'-O-galloylhyperin, a major compound of Pyrola incarnata Fisch., possesses a variety of biological and pharmacological activities, including anti-oxidative and anti-inflammatory activities. Nevertheless, the underlying molecular mechanisms of 2'-O-GH in microbial infection and sepsis are not clear. In this study, we investigated the anti-inflammatory effects of 2'-O-GH. We found that 2'-O-GH significantly reduced the production of TNF-α, IL-6, and nitric oxide (NO), suppressed the expression levels of iNOS, blocked the translocation of NF-κB from the cytosol to nucleus, and decreased the MAPK activation in LPS-activated RAW 264.7 cells. 2'-O-GH also enhanced the nuclear translocation of Nr…

0301 basic medicinePharmacologylcsh:RM1-950Chromosomal translocationNF-κBPharmacologymedicine.diseaseIn vitroNF-κBanti-inflammationNrf2Nitric oxideSepsis03 medical and health sciencesCytosolchemistry.chemical_compound030104 developmental biologySIRT1lcsh:Therapeutics. PharmacologychemistryDownregulation and upregulationmedicinePharmacology (medical)Heme2′-O-galloylhyperinFrontiers in Pharmacology
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Phenolic Extract from Sonchus oleraceus L. Protects Diabetes-related Liver Injury in Rats through TLR4/NF-κB Signaling Pathway

2019

Sonchus oleraceus Linn. is of great interest in scientific platform because it exhibits significant biological activities. This study investigated the liver protective effects and mechanism of phenolic extract from S. oleraceus (SOL) on Streptozotocin (STZ)-induced diabetic rats. SOL significantly increased both superoxide dismutase activity and glutathione level, while causing a reduction of malondialdehyde level in the liver. Moreover, SOL ameliorates STZ-induced liver function and pathological damages. Diabetic rats fed with S. oleraceus daily for 6 weeks showed significantly decreased levels of tumor necrosis factor alpha and interleukin-1β in the liver. Sonchus oleraceus decreased the …

Liver injurybiologylcsh:TP368-456ChemistryAnti inflammationPharmacologybiology.organism_classificationmedicine.diseaseSonchus oleraceusin vivoSonchus oleraceuslcsh:Food processing and manufactureIn vivolcsh:RA1190-1270Diabetes mellitusAnti-inflammationTlr4 nf κbmedicineSignal transductionNF-κB signaling pathwayliver injurylcsh:Toxicology. PoisonseFood
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Antioxidation, Anti-Inflammation, and Regulation of SRD5A Gene Expression of Oryza sativa cv. Bue Bang 3 CMU Husk and Bran Extracts as Androgenetic A…

2022

Acknowledgments: The authors are grateful to the NRCT for supporting research facilities (grant no. NRCT5-RRI63004-P05), Chiang Mai University for the Fundamental Fund 2022, and the partially support grant. We would like to thank Lanna Rice Research Center, Chiang Mai University, and Saleekam Trading Co., Ltd., Thailand, for providing the rice bran and husk samples.

bioactive compoundsEcologyBotanyandrogenetic alopecia; anti-hair loss; anti-inflammation; bioactive compounds; <i>Oryza sativa</i>; 5α-reductase gene expression; rice bran; rice huskfood and beveragesOryza sativaPlant ScienceCuir cabellutanti-inflammationBioactive compounds5α-reductase gene expressionAnti-hair lossRice huskQK1-989Anti-inflammationRice brananti-hair lossandrogenetic alopecia<i>Oryza sativa</i>Androgenetic alopeciaAndrògensEcology Evolution Behavior and Systematics
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