Search results for "apolipoprotein B"

showing 10 items of 170 documents

Effectiveness of cascade filtration plasmapheresis in two patients affected by familial hypercholesterolemia

1995

Hypercholesterolemia has been recognised as a primary risk factor for coronary heart disease. Reduction of plasma levels of total and LDL cholesterol has been shown to decrease coronary atherosclerosis. Plasmapheresis represents an useful non-pharmacological tool to treat severe hypercholesterolemias. We have evaluated the effectiveness of a system of plasmapheresis using a cascade filtration method in two young male subjects (aged 16 and 26 years) with homozygous familial hypercholesterolemia. Both showed severe coronary atherosclerosis as determined by angiography. Procedures were performed at intervals of 7 days in each case. We observed a mean reduction of plasma levels of total cholest…

AdultMalemedicine.medical_specialtyAdolescentApolipoprotein Bmedicine.medical_treatmentFamilial hypercholesterolemiaFibrinogenGastroenterologyHyperlipoproteinemia Type IIchemistry.chemical_compoundInternal medicinemedicineHumansHyperlipoproteinemia Type IIRisk factorCoronary atherosclerosisbiologybusiness.industryCholesterolCholesterol LDLPlasmapheresisHematologyGeneral Medicinemedicine.diseaseEndocrinologychemistrybiology.proteinlipids (amino acids peptides and proteins)PlasmapheresisbusinessFiltrationmedicine.drugJournal of Clinical Apheresis
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Association of TaqIB polymorphism in the cholesteryl ester transfer protein gene with plasma lipid levels in a healthy Spanish population.

2000

Genetic variants at the cholesteryl ester transfer protein (CETP) locus have been associated with CETP activity and mass, as well as plasma high density lipoprotein cholesterol (HDL-C) and apolipoprotein A-I levels. We have examined allele frequencies and lipid associations for the common CETP TaqIB polymorphism in a sample of 514 healthy subjects (231 men, mean age 37.4 years, and 283 women, mean age 35.7 years) residing in Valencia (Spain). The frequency of the less common TaqIB2 allele (0.351; 95% CI: 0.322-0. 380) was significantly lower than those reported for Northern European populations. Consistent with previous studies, we found a significant association of the TaqIB polymorphism w…

AdultMalemedicine.medical_specialtyApolipoprotein BAdolescentAlcohol DrinkingGenotypeCoronary DiseaseBody Mass Indexchemistry.chemical_compoundHigh-density lipoproteinGene FrequencyInternal medicineGenotypeCholesterylester transfer proteinmedicineHumansTaq PolymeraseAlleleAllele frequencyAllelesAgedGlycoproteinsGeneticsbiologyCholesterolCholesterol HDLSmokingGenetic VariationMiddle AgedLipidsCholesterol Ester Transfer ProteinsEndocrinologyApolipoproteinsCross-Sectional StudieschemistrySpainMultivariate Analysisbiology.proteinlipids (amino acids peptides and proteins)FemaleCardiology and Cardiovascular MedicineCarrier ProteinsBody mass indexAtherosclerosis
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Small and dense LDL in Familial Combined Hyperlipidemia and N291S polymorphism of the lipoprotein lipase gene

2009

18 pagesm 2 figures. -- PMID: 19335919 [PubMed]

AdultMalemedicine.medical_specialtyApolipoprotein BAdolescentEndocrinology Diabetes and MetabolismPopulationClinical BiochemistryHyperlipidemia Familial CombinedMutation MissenseBlood lipidschemistry.chemical_compoundYoung AdultHigh-density lipoproteinEndocrinologyInternal medicinePrevalenceMedicineShort PaperHumanseducationlcsh:RC620-627AgedBiochemistry medicalLipoprotein lipaseeducation.field_of_studyPolymorphism Geneticmedicine.diagnostic_testbiologyCholesterolbusiness.industryBiochemistry (medical)Cholesterol LDLMiddle AgedAtherosclerosisLipidslcsh:Nutritional diseases. Deficiency diseasesLipoprotein LipaseEndocrinologychemistrySpainLow-density lipoproteinCase-Control Studiesbiology.proteinFemalelipids (amino acids peptides and proteins)businessLipid profile
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Low intestinal cholesterol absorption is associated with a reduced efficacy of phytosterol esters as hypolipemic agents in patients with metabolic sy…

2010

Summary Background & aims Phytosterols (PS) lower LDLc, but their effect on metabolic syndrome (MetS) remains unknown. We evaluated whether low-fat milk enriched with PS improves cardiovascular risk factors in these patients. Methods A randomised parallel trial employing 24 moderate-hypercholesterolaemic MetS patients and consisting of two 3-month intervention phases. After a 3-month healthy diet, patients were divided into two intervention groups: diet (n = 10) and diet + PS (n = 14) (2 g/day). A control group of 24 moderate-hypercholesterolaemic patients without MetS (matched in age and BMI) underwent the same procedure. Results Neither dietary intervention nor enrichment of PS induced an…

AdultMalemedicine.medical_specialtyApolipoprotein BHomocysteineLipoproteinsHypercholesterolemiaPopulationCritical Care and Intensive Care MedicineSeverity of Illness IndexCholesterol Dietarychemistry.chemical_compoundRisk FactorsInternal medicinemedicineHumanseducationHypolipidemic AgentsMetabolic Syndromeeducation.field_of_studyNutrition and Dieteticsmedicine.diagnostic_testbiologybusiness.industryAnticholesteremic AgentsPhytosterolsMiddle Agedmedicine.diseaseSitosterolsCholesterolEndocrinologyIntestinal AbsorptionchemistryCardiovascular DiseasesSpainDietary SupplementsIntestinal cholesterol absorptionbiology.proteinFemaleMetabolic syndromebusinessLipid profileBody mass indexLipoproteinClinical Nutrition
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Lipid-free apolipoprotein (apo) A-I is converted into alpha-migrating high density lipoproteins by lipoprotein-depleted plasma of normolipidemic dono…

1998

Plasma of patients with Tangier disease (TD) is devoid of alpha-LpA-I (apolipoprotein A-I-containing lipoprotein), which in normolipidemic plasma constitutes the majority of high density lipoprotein (HDL). The residual amounts of apolipoprotein A-I (apo A-I) in TD plasma have electrophoretic prebeta1-LpA-I mobility. We have previously demonstrated that TD plasma does not convert prebeta1-LpA-I into alpha-LpA-I. In this study we found that plasmas of normolipidemic controls, apo A-I-deficient patients and patients with fish-eye disease, but not plasmas of six TD patients, convert biotinylated lipid-free apo A-I into alpha-LpA-I. Supplementation of plasma with free oleic acid or fatty acid fr…

AdultMalemedicine.medical_specialtyApolipoprotein BLipoproteinsBlood Donorschemistry.chemical_compoundTangier diseaseHigh-density lipoproteinReference ValuesPhospholipid transfer proteinInternal medicinemedicineHumansPhospholipidsTangier DiseaseAgedApolipoprotein A-IbiologyChemistryCholesterolVesicleAlbuminMiddle Agedmedicine.diseaseEndocrinologybiology.proteinFemalelipids (amino acids peptides and proteins)Lipoproteins HDLCardiology and Cardiovascular MedicineLipoproteinAtherosclerosis
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High Plasma Phospholipid Transfer Protein Levels as a Risk Factor for Coronary Artery Disease

2003

Objective— Plasma phospholipid transfer protein (PLTP) mediates both net transfer and exchange of phospholipids between different lipoproteins. Animal studies have shown that it is closely related to the development of atherosclerosis. PLTP-deficient mice have demonstrated increased antioxidation potential as well as a decrease in apolipoprotein B secretion and atherosclerotic lesions. In humans, high PLTP is associated with type II diabetes and obesity. Methods and Results— To assess the relationship between PLTP activity and coronary artery disease (CAD), a novel, high-throughput method to measure plasma PLTP activity was used, relating it to CAD in 1102 cases and 444 controls. This demo…

AdultMalemedicine.medical_specialtyApolipoprotein BLipoproteinsMyocardial InfarctionCoronary Artery DiseaseAngina PectorisAnginaCoronary artery diseaseReference ValuesRisk FactorsPhospholipid transfer proteinInternal medicinemedicineHumansAngina UnstablePhospholipid Transfer ProteinsRisk factorPhospholipidsAgedbiologybusiness.industryCase-control studyMembrane ProteinsBiological activityMiddle Agedmedicine.diseaseLogistic ModelsEndocrinologyCase-Control Studiesbiology.proteinFemaleAnimal studiesCarrier ProteinsCardiology and Cardiovascular MedicinebusinessArteriosclerosis, Thrombosis, and Vascular Biology
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Inability of HDL from abdominally obese subjects to counteract the inhibitory effect of oxidized LDL on vasorelaxation.

2007

Abdominal obesity is associated with a decreased plasma concentration of HDL cholesterol and with qualitative modifications of HDL, such as triglyceride enrichment. Our aim was to determine, in isolated aorta rings, whether HDL from obese subjects can counteract the inhibitory effect of oxidized low density lipoprotein (OxLDL) on endothelium-dependent vasodilation as efficiently as HDL from normolipidemic, lean subjects. Plasma triglycerides were 74% higher (P < 0.005) in obese subjects compared with controls, and apolipoprotein A-I (apoA-I) and HDL cholesterol concentrations were 12% and 17% lower (P < 0.05), respectively. HDL from control subjects significantly reduced the inhibitory effe…

AdultMalemedicine.medical_specialtyApolipoprotein BVasodilator Agentsapolipoprotein A-IVasodilationQD415-436In Vitro TechniquesBiochemistrychemistry.chemical_compoundEndocrinologyHigh-density lipoproteinInternal medicinemedicineAnimalsHumansObesityInhibitory effectAbdominal obesityAortaTriglyceridesbiologyTriglycerideCholesterolCholesterol HDLnutritional and metabolic diseasesCell BiologyMiddle Agedmedicine.diseaseObesityAcetylcholineLipoproteins LDLVasodilationEndocrinologychemistryhigh density lipoproteinbiology.proteinoxidized low density lipoproteinlipids (amino acids peptides and proteins)FemaleCholesterol EstersRabbitsmedicine.symptomJournal of lipid research
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No change in apolipoprotein AI metabolism when subcutaneous insulin infusion is replaced by intraperitoneal insulin infusion in type 1 diabetic patie…

2006

In type 1 diabetic patients, the replacement of subcutaneous insulin infusion by intraperitoneal insulin infusion restores the normal physiological gradient between the portal vein and the peripheral circulation, which is likely to modify HDL metabolism. This stable isotope kinetic study was designed to compare HDL apolipoprotein (apo) AI metabolism in seven type 1 diabetic patients first treated by continuous subcutaneous insulin infusion by an external pump and then 3 months after the beginning of intraperitoneal insulin infusion by an implantable pump. Glycaemic control was comparable under subcutaneous and intraperitoneal insulin infusion (HbA1c=7.34+/-0.94% versus 7.24+/-1.00%, NS). HD…

AdultMalemedicine.medical_specialtyApolipoprotein Bmedicine.medical_treatmentCarbon Compounds InorganicSulfidesAdministration Cutaneouschemistry.chemical_compoundInsulin Infusion SystemsDiabetes mellitusInternal medicinemedicineHumansInsulinInfusions ParenteralPancreatic hormoneType 1 diabetesTriglyceridebiologyApolipoprotein A-Ibusiness.industryCholesterolInsulinMetabolismMiddle Agedmedicine.diseaseKineticsEndocrinologyDiabetes Mellitus Type 1chemistrybiology.proteinFemaleCardiology and Cardiovascular MedicinebusinessLipoproteins HDLAtherosclerosis
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Association of C677T polymorphism in MTHFR gene, high homocysteine and low HDL cholesterol plasma values in heterozygous familial hypercholesterolemi…

2010

Aim: to investigate the association of C677T polymorphism in the methylene tetrahydrofolate reductase (MTHFR) gene, homocysteine plasma values (Hcy), and plasma HDL cholesterol in heterozy-gous familial hypercholesterolemia (hFH).Methods: One hundred and twenty-five hFH subjects were studied. Plasma lipid, lipoprotein, vitamin B12, folic acid and Hcy values were determined. C677T polymorphism in the MTHFR gene was detected by SSCP-PCR. Genetic diagnosis of FH was determined by a three-step protocol using SSCP-PCR, Southern blot, long PCR and automatic sequencing.Results: We found significant differences in plasma HDL-C (CC 1.39±0.34, CT 1.33±0.39 and TT 1.14±0.26 mmol/L, p=0.028) between th…

AdultMalemedicine.medical_specialtyHeterozygoteApolipoprotein BHomocysteineHypercholesterolemiaFamilial hypercholesterolemiaPolymerase Chain Reactionchemistry.chemical_compoundInternal medicineGenotypeInternal MedicinemedicineHumansVitamin B12HomocysteineMethylenetetrahydrofolate Reductase (NADPH2)Polymorphism Single-Stranded ConformationalApolipoproteins BGeneticsPolymorphism GeneticbiologyCholesterolbusiness.industryBiochemistry (medical)Cholesterol HDLMiddle Agedmedicine.diseaseEndocrinologychemistryReceptors LDLMethylenetetrahydrofolate reductaseMutationbiology.proteinFemaleCardiology and Cardiovascular MedicinebusinessLipoproteinJournal of atherosclerosis and thrombosis
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Familial HDL deficiency due to ABCA1 gene mutations with or without other genetic lipoprotein disorders

2004

Mutations in ABCA1 have been shown to be the cause of Tangier disease (TD) and some forms of familial hypoalphalipoproteinemia (HA), two genetic disorders characterized by low plasma HDL levels. Here we report six subjects with low HDL, carrying seven ABCA1 mutations, six of which are previously unreported. Two mutations (R557X and H160FsX173) were predicted to generate short truncated proteins; two mutations (E284K and Y482C) were located in the first extracellular loop and two (R1901S and Q2196H) in the C-terminal cytoplasmic domain of ABCA1. Two subjects found to be compound heterozygotes for ABCA1 mutations did not have overt clinical manifestations of TD. Three subjects, all with prema…

AdultMalemedicine.medical_specialtyHeterozygoteSettore MED/09 - Medicina InternaApolipoprotein BAdolescentPremature coronary artery diseaseTangier diseaseCoronary DiseaseBiologyGene mutationmedicine.disease_causeCompound heterozygosityTangier diseaseInternal medicineGenotypeABCA1 genemedicineHumansChildHypoalphalipoproteinemiaSelection BiasAgedApolipoproteins BGeneticsMutationFamilial defective Apo B (FDB)Apolipoprotein A-ICholesterol HDLnutritional and metabolic diseasesMiddle Agedmedicine.diseaseLipoprotein lipaseTangier disease; Familial HDL deficiency; ABCA1 gene; Familial defective Apo B (FDB); Lipoprotein lipase; Premature coronary artery diseaseEndocrinologyChild PreschoolMutationbiology.proteinlipids (amino acids peptides and proteins)Allelic heterogeneityATP-Binding Cassette TransportersFemaleCardiology and Cardiovascular MedicineFamilial HDL deficiencyATP Binding Cassette Transporter 1
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