Search results for "arginine"

showing 9 items of 389 documents

Vasoactive intestinal peptide stimulation of cyclic guanosine monophosphate formation: further evidence for a role of nitric oxide synthase and cytos…

1993

In the rat pineal gland vasoactive intestinal peptide (VIP) and beta-adrenergic agonists stimulate cyclic guanosine monophosphate (cGMP) formation and their action is amplified by alpha 1-adrenergic agonists. Since beta-adrenergic stimulation of cGMP is suggested to involve activation of nitric oxide (NO) synthase and NO-mediated activation of cytosolic guanylate cyclase (GC), we investigated the effects of the NO synthase inhibitor N-monomethyl-L-arginine (L-NMMA) and of the cytosolic GC inhibitor methylene blue (MB) on VIP receptor-stimulated cGMP formation. Both L-NMMA and MB depressed VIP-induced cGMP formation as well as alpha 1-adrenergic potentiation of VIP-stimulated cGMP formation …

medicine.medical_specialtyVasoactive intestinal peptideArgininePineal GlandPinealocyteNitric oxidechemistry.chemical_compoundPhenylephrineEndocrinologyCytosolInternal medicinemedicineAnimalsCyclic guanosine monophosphateCyclic GMPomega-N-MethylarginineATP synthasebiologyDrug SynergismRatsNitric oxide synthaseMethylene BlueEndocrinologychemistryGuanylate CyclaseSecond messenger systembiology.proteinOmega-N-MethylarginineAmino Acid OxidoreductasesNitric Oxide Synthasehormones hormone substitutes and hormone antagonistsVasoactive Intestinal PeptideEndocrinology
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Inhibition of growth of measles virus by mycoplasma in cell-cultures and the restoring effect of arginine

1970

Presence of mycoplasma in MK cell cultures caused an inhibition of measles virus, as revealed by a lower plating efficiency. When arginine is supplemented to the cell-system, mycoplasma contaminated cells yield plaque titers as high as those of uncontaminated cells. The essential role of arginine for plaque formation of measles virus is suggested also by experiments in which arginine added to the agar overlay restored the plating efficiency of measles virus in cultures maintained in a minimal nutrient medium.

medicine.medical_specialtyVirus CultivationPlating efficiencyArginineBiologyArginineKidneymedicine.disease_causeMeaslesCell LineMicrobiologyMeasles virusMycoplasmaMedical microbiologyCulture TechniquesVirologymedicineAnimalsHaplorhiniGeneral MedicineMycoplasmabiology.organism_classificationmedicine.diseaseVirologyCulture MediaTiterMeasles virusCell cultureArchiv f�r die gesamte Virusforschung
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Is oxidative stress a therapeutic target in cardiovascular disease?

2010

An abnormal production of reactive oxygen species (ROS) and the subsequent decrease in vascular bioavailability of nitric oxide (NO) have long been proposed to be the common pathogenetic mechanism of the endothelial dysfunction, resulting from diverse cardiovascular risk factors such as hypercholesterolaemia, diabetes mellitus, chronic smoking, metabolic syndrome, and hypertension. Superoxide produced by the nicotinamide dinucleotide phosphate (NADPH) oxidase, mitochondrial sources, or the xanthine oxidase may react with NO, thereby resulting in excessive formation of peroxynitrite, a reactive nitrogen species that has been demonstrated to accelerate the atherosclerotic process by causing d…

medicine.medical_specialtyXanthine OxidaseAntioxidantmedicine.medical_treatmentmedicine.disease_causeArginineAntioxidantschemistry.chemical_compoundRisk FactorsInternal medicinemedicineHumansProspective StudiesEndothelial dysfunctionXanthine oxidaseReactive nitrogen specieschemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologybusiness.industrySuperoxideNADPH OxidasesPolyphenolsVitaminsmedicine.diseasePrognosisMitochondriaOxidative StressEndocrinologychemistryCardiovascular Diseasesbiology.proteinEndothelium VascularNitric Oxide SynthaseCardiology and Cardiovascular MedicinebusinessReactive Oxygen SpeciesOxidative stressEuropean heart journal
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Selected peptides targeted to the NMDA receptor channel protect neurons from excitotoxic death

1998

Excitotoxic neuronal death, associated with neurodegeneration and stroke, is triggered primarily by massive Ca2+ influx arising from overactivation of glutamate receptor channels of the N-methyl-D-aspartate (NMDA) subtype. To search for channel blockers, synthetic combinatorial libraries were assayed for block of agonist-evoked currents by the human NR1-NR2A NMDA receptor subunits expressed in amphibian oocytes. A set of arginine-rich hexapeptides selectively blocked the NMDA receptor channel with IC50 approximately 100 nM, a potency similar to clinically tolerated blockers such as memantine, and only marginally blocked on non-NMDA glutamate receptors. These peptides prevent neuronal cell d…

medicine.medical_specialtyXenopusDrug Evaluation PreclinicalBiomedical EngineeringBioengineeringHippocampal formationBiologyPharmacologyArginineBinding CompetitiveHippocampusReceptors N-Methyl-D-AspartateApplied Microbiology and BiotechnologySubstrate SpecificityInternal medicinemedicineAnimalsHumansChannel blockerReceptorNeuronsCell DeathNeurodegenerationGlutamate receptorMemantinemedicine.diseaseRatsEndocrinologymedicine.anatomical_structurenervous systemDrug DesignOocytesMolecular MedicineNMDA receptorFemaleNeuronPeptidesBiotechnologymedicine.drugNature Biotechnology
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Myogenic effects enhance norepinephrine constriction: Inhibition by nitric oxide and felodipine

1998

Myogenic effects enhance norepinephrine constriction: Inhibition by nitric oxide and felodipine. Myogenic, pressure-induced vasoconstriction may amplify the effects of circulating vasoconstrictors. Through intravital microscopy in cremaster arterioles (31 to 115 μm diameter), the relative contribution of myogenic responses (MR) to norepinephrine (NE)-induced constriction and the inhibitor potency of nitric oxide (NO) or a Ca2+ entry blocker (CEB), felodipine (F), were examined. In 24 anesthetized hamsters, a vessel occluder was placed around the aorta to control cremaster vessel inflow pressure (IP). NE infusion increased blood pressure (by 50 ± 2mm Hg) and induced significant constriction …

medicine.medical_specialtyendotheliumVasodilator AgentsmicrocirculationMyogenic mechanismBayliss effectBlood PressureNitric OxideNitroarginineMuscle Smooth VascularConstrictionNitric oxideMicrocirculationNorepinephrine (medication)Norepinephrinechemistry.chemical_compoundCricetinaeInternal medicineintravital microscopymedicineAnimalsVasoconstrictor AgentsBayliss effectAorta AbdominalcremasterFelodipineCapillariesArteriolesEndocrinologychemistryFelodipineNephrologyAnesthesiacalcium entry blockerInjections Intravenouscardiovascular systemmedicine.symptomVasoconstrictionmedicine.drugKidney International
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Nitric oxide and sensory afferent neurones modulate the protective effects of low-dose endotoxin on rat gastric mucosal damage

1995

Pretreatment (1 h) with low doses (5-40 micrograms/kg i.p.) of Escherichia coli endotoxin dose dependently reduced the gastric mucosal damage induced by a 10 min challenge with 1 ml ethanol (50% and 100%) in conscious rats. Treatment with the nitric oxide synthesis inhibitor, NG-nitro-L-arginine methyl ester (L-NAME, 5 and 10 mg/kg i.p.), significantly inhibited the protective effects of endotoxin (40 micrograms/kg i.p.). The actions of L-NAME were reversed by the prior administration of L-arginine (100 mg/kg i.p.). The protective effects of endotoxin were not influenced by pretreatment with dexamethasone (5 mg/kg s.c. twice) or indomethacin (5 mg/kg s.c.). However, ablation of sensory affe…

medicine.medical_treatmentIndomethacinPharmacologyArginineDexamethasoneNitric oxideRats Sprague-Dawleychemistry.chemical_compoundEscherichia colimedicineAnimalsNeurons AfferentEnzyme InhibitorsAntidoteDexamethasonePharmacologyAnalysis of VarianceEthanolEthanolSensory neuronRatsEndotoxinsNG-Nitroarginine Methyl Estermedicine.anatomical_structureMechanism of actionchemistryGastric MucosaCapsaicinAnesthesiaToxicityFemaleCapsaicinNitric Oxide Synthasemedicine.symptomInjections Intraperitonealmedicine.drugEuropean Journal of Pharmacology
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Amphoteric, prevailingly cationic L-arginine polymers of poly(amidoamino acid) structure: Synthesis, acid/base properties and preliminary cytocompati…

2013

A linear amphoteric poly(amidoamino acid), L-ARGO7, is prepared by Michael-type polyaddition of L-arginine with N,N′-methylenebisacrylamide. Chain-extension of acrylamide end-capped L-ARGO7 oligomers with piperazine leads to high-molecular-weight copolymers in which L-arginine maintains its absolute configuration. Acid/base properties of L-ARGO7 polymers show isolectric points of ≈10 and positive net average charges per repeating unit at pH = 7.4 from 0.25 to 0.40. These arginine-rich synthetic polymers possibly share some of the unique biological properties of polyarginine cell-permeating peptides. In vitro tests with mouse embryo fibroblasts balb/3T3 clone A31 show that L-ARGO7 polymers a…

poly(amidoamine)Materials Chemistry2506 Metals and AlloyAcrylamidesCell Membrane PermeabilityPolymers and PlasticCell SurvivalL -arginine polymerMedicine (all)Static ElectricityBioengineeringHydrogen-Ion ConcentrationArgininebiological application of polymerBiomaterialPiperazinesMicebiocompatibilityNIH 3T3 CellsPolyaminesAnimalsIsoelectric Pointpoly(amidoamino acid)PeptidesHydrophobic and Hydrophilic InteractionsBiotechnology
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Homeostatic control of polyamine levels under long-term salt stress in Arabidopsis

2011

Salt stress has been frequently studied in its first osmotic phase. Very often, data regarding the second ionic phase is missing. It has also been suggested that Putrescine or/and Spermine could be responsible for salt resistance. In order to test this hypothesis under long-term salt stress, we obtained Arabidopsis thaliana transgenic plants harboring pRD29A::oatADC or pRD29A::GUS construction. Although Putrescine was the only polyamine significantly increased after salt acclimation in pRD29A::oatADC transgenic lines, this rendered in no advantage to this kind of stress. The higher Spermine levels found in WT and transgenic lines when compared to control conditions along with no increment o…

salt acclimationsperminepolyaminesputrescinearginine decarboxilasesalt overlay sensitive mutants
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The AQP2 mutation V71M causesnephrogenic diabetes insipidus in humans but does not impair the function of a bacterial homolog

2015

Graphical abstract

wt wild-typeGpA glycophorin AHM half-membrane-spanningurogenital systemQH301-705.5AquaporinNephrogenic diabetes insipidusAQP ER endoplasmic reticulumGlpF glycerol facilitatorActivityProtein oligomerizationResearch articleNDI nephrogenic diabetes insipidusAVP arginine vasopressinGlpF500 Natural sciences and mathematicsAQP aquaporin500 NaturwissenschaftenBiology (General)AVPR2 V2 receptorComputingMethodologies_COMPUTERGRAPHICSTM transmembraneFEBS Open Bio
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