Search results for "autism"

showing 10 items of 457 documents

Fragile-X carrier females: evidence for a distinct psychopathological phenotype?

1996

The present study examined 35 mothers (29 premutation carriers) of children with fragile-X syndrome in measures of intelligence and psychiatric disorders by comparing them with two control groups: a) 30 mothers of children in the general population and b) 17 mothers of non-fra-X retarded children with autism. Premutation carriers had a higher frequency of affective disorders than mothers from the general population. Preliminary data indicate that normally intelligent premutation carriers of the fra-X genetic abnormality have a similar frequency of affective disorders (DSM-III-R criteria [APA, 1987]) than mothers of autistic children. Neither carriers of the premutation nor carriers of the f…

AdultHeterozygotePopulationIntelligenceMothersSchizoaffective disorderTrinucleotide RepeatsReference ValuesIntellectual DisabilitymedicineHumansSchizophreniform disorderAutistic DisordereducationChildGenetics (clinical)education.field_of_studybusiness.industryWechsler ScalesMiddle Agedmedicine.diseaseFragile X syndromeSchizophreniaFragile X SyndromeMutationSchizophreniaAutismAge of onsetbusinessClinical psychologyPsychopathologyAmerican journal of medical genetics
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Higher vitamin B12 levels in neurodevelopmental disorders than in healthy controls and schizophrenia

2020

Recent studies suggest that both high and low levels of vitamin B12 (vitB12) may have negative health impacts. We measured VitB12 in patients with the Neurodevelopmental disorders (ND) (n = 222), comprised of Autism Spectrum Disorders, specific Developmental disorders, and Intellectual Disability (aged 2-53 years), schizophrenia (n = 401), and healthy controls (HC) (n = 483). Age-and gender-adjusted vitB12 z-scores were calculated by comparisons with a reference population (n = 76 148). We found higher vitB12 in ND (median 420 pmol/L, mean z-score: 0.30) than in HC (316 pmol/L, z-score: 0.06, P < .01) and schizophrenia (306 pmol/L, z-score: -0.02, P < .001), which was significant after adju…

AdultMale0301 basic medicinemedicine.medical_specialtyAdolescentRenal functionBiochemistryCobalaminHemoglobinsYoung Adult03 medical and health scienceschemistry.chemical_compoundFolic Acid0302 clinical medicineInternal medicineIntellectual disabilityLeukocytesGeneticsHumansMedicineIn patientVitamin B12ChildMolecular Biologybusiness.industryVitamin B 12 DeficiencyMiddle Agedmedicine.diseaseVitamin B 12030104 developmental biologyEndocrinologychemistryNeurodevelopmental DisordersSchizophreniaCase-Control StudiesChild PreschoolDietary SupplementsSchizophreniaAutismFemaleHemoglobinbusiness030217 neurology & neurosurgeryBiotechnologyThe FASEB Journal
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Chromosome 15q BP3 to BP5 deletion is a likely locus for speech delay and language impairment: Report on a four‐member family and an unrelated boy

2020

Abstract Background Deletions in chromosome 15q13 have been reported both in healthy people and individuals with a wide range of behavioral and neuropsychiatric disturbances. Six main breakpoint (BP) subregions (BP1‐BP6) are mapped to the 15q13 region and three further embedded BP regions (BP3‐BP5). The deletion at BP4‐BP5 is the rearrangement most frequently observed compared to other known deletions in BP3‐BP5 and BP3‐BP4 regions. Deletions of each of these three regions have previously been implicated in a variable range of clinical phenotypes, including minor dysmorphism, developmental delay/intellectual disability, epilepsy, autism spectrum disorders, behavioral disturbances, and speec…

AdultMale0301 basic medicinespeech delayAdolescentlcsh:QH426-470BP3-BP5 deletionspeech delay.Chromosome DisordersLocus (genetics)030105 genetics & heredity03 medical and health sciencesEpilepsySettore MED/38 - Pediatria Generale E SpecialisticaSeizuresIntellectual DisabilityIntellectual disabilitychromosome 15 q13GeneticsmedicineHumansLanguage Development DisordersChildMolecular BiologyGenetics (clinical)GeneticsChromosomes Human Pair 15business.industryBreakpointlanguage impairmentOriginal Articlesmedicine.diseasePhenotypePedigreeBP3‐BP5 deletiondevelopmental delayLanguage developmentlcsh:GeneticsPhenotype030104 developmental biologyBP3-BP5 deletion; chromosome 15 q13; developmental delay; language impairment; speech delaySpeech delayAutismFemaleOriginal ArticleChromosome Deletionmedicine.symptombusinessMolecular Genetics & Genomic Medicine
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Genetic and environmental contributions to the inverse association between specific autistic traits and experience seeking in adults

2015

Autistic traits are characterized by social and communication problems, restricted, repetitive and stereotyped patterns of behavior, interests and activities. The relation between autistic traits and personality characteristics is largely unknown. This study focused on the relation between five specific autistic traits measured with the abridged version of the Autism Spectrum Quotient ("social problems," "preference for routine," "attentional switching difficulties," "imagination impairments," "fascination for numbers and patterns") and Experience Seeking (ES) in a general population sample of adults, and subsequently investigated the genetic and environmental etiology between these traits.…

AdultMaleAutism-spectrum quotientmedicine.medical_specialtyInverse Associationmedia_common.quotation_subjectTwinsEnvironmentDevelopmental psychology03 medical and health sciencesCellular and Molecular NeuroscienceRisk-Taking0302 clinical medicinemedicineHumansSensation seekingPersonalityAttention0501 psychology and cognitive sciencesAutistic DisorderAssociation (psychology)Genetics (clinical)Netherlandsmedia_commonGeneticsSiblings05 social sciencesmedicine.diseasePreferenceSubstance abusePsychiatry and Mental healthAutistic traitsEtiologyMedical geneticsAutismFemalePsychology030217 neurology & neurosurgeryPersonality050104 developmental & child psychologyAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
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The 2q37-deletion syndrome: an update of the clinical spectrum including overweight, brachydactyly and behavioural features in 14 new patients

2012

International audience; The 2q37 locus is one of the most commonly deleted subtelomeric regions. Such a deletion has been identified in >100 patients by telomeric fluorescence in situ hybridization (FISH) analysis and, less frequently, by array-based comparative genomic hybridization (array-CGH). A recognizable ‘2q37-deletion syndrome’ or Albright’s hereditary osteodystrophy-like syndrome has been previously described. To better map the deletion and further refine this deletional syndrome, we formed a collaboration with the Association of French Language Cytogeneticists to collect 14 new intellectually deficient patients with a distal or interstitial 2q37 deletion characterized by FISH and …

AdultMaleCandidate geneAdolescentDNA Copy Number Variations[SDV]Life Sciences [q-bio]Chromosome DisordersLocus (genetics)BiologyFibrous Dysplasia PolyostoticBioinformaticsArticleYoung Adult03 medical and health sciences0302 clinical medicineIntellectual DisabilityGeneticsmedicineHumansChildGenetic Association StudiesGenetics (clinical)030304 developmental biologyKIF1AGeneticsBehaviorComparative Genomic Hybridization0303 health sciences[ SDV ] Life Sciences [q-bio]medicine.diagnostic_testBrachydactylyBrachydactylyChromosome MappingOverweightSubtelomeremedicine.disease[SDV] Life Sciences [q-bio]Child PreschoolChromosomes Human Pair 2AutismFemaleChromosome Deletion030217 neurology & neurosurgeryComparative genomic hybridizationFluorescence in situ hybridizationEuropean Journal of Human Genetics
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Screening of subtelomeric rearrangements in autistic disorder: identification of a partial trisomy of 13q34 in a patient bearing a 13q;21p translocat…

2006

Within the framework of a FISH screening protocol to detect cryptic subtelomeric rearrangements in autistic disorder (AD), a patient bearing three copies of the subtelomeric portion of the q arm of chromosome 13 has been identified. Beside AD, the patient also has severe mental retardation and displays several dysmorphic features. Further FISH analyses revealed that the trisomy was caused by the translocation of a 13q subtelomeric fragment to the acrocentric tip of one chromosome 21 [46,XY.ish der(21) t(13;21) (q34;p13)(D13S1825+)]. Gene dosage experiments carried out with three multiallelic polymorphisms of the subtelomeric region of chromosome 13q showed that the putative length of the tr…

AdultMaleDerivative chromosomeAdolescentGene DosageautismChromosomal translocationTrisomyBiologyGene dosagePolymerase Chain ReactionTranslocation GeneticCellular and Molecular NeurosciencemedicineHumansAutistic DisorderChildGenetics (clinical)In Situ Hybridization FluorescenceChromosome 13GeneticsChromosomes Human Pair 13ChromosomeTelomereSubtelomeremedicine.diseasePsychiatry and Mental healthfrontal bossingFemaleTrisomyChromosome 21American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics
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Cognitive and social cognitive functioning in spinocerebellar ataxia : a preliminary characterization

2006

INTRODUCTION : The spinocerebellar ataxias (SCAs), are rare neurodegenerative disorders caused by distinct genetic mutations. Clinically, the SCAs are characterised by progressive ataxia and a variety of other features, including cognitive dysfunction. The latter is consistent with a growing body of evidence supporting a cognitive as well as motor role for the cerebellum. Recent suggestions of cerebellar involvement in social cognition have not been extensively explored in these conditions. The availability of definitive molecular diagnosis allows genetically defined subgroups of SCA patients, with distinct patterns of cerebellar and extracerebellar involvement, to be tested comparatively u…

AdultMaleEmotionsNeuropsychological TestsSocial Environmentcognitive functioningDisability EvaluationCognitionSocial cognitionCerebellumTheory of mindmedicineHumansSpinocerebellar AtaxiasCognitive skillSocial BehaviorAgedIntelligence TestsVerbal BehaviorCognitive disorderNeuropsychologyRecognition PsychologyCognitionMachado-Joseph DiseaseMiddle Agedmedicine.diseaseNeurologyMental RecallSpinocerebellar ataxiaAutismFemaleAtaxiaNeurology (clinical)PsychologyNeurosciencePsychomotor Performance
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Ten new cases further delineate the syndromic intellectual disability phenotype caused by mutations in DYRK1A.

2015

The dual-specificity tyrosine-phosphorylation-regulated kinase 1A (DYRK1A) gene, located on chromosome 21q22.13 within the Down syndrome critical region, has been implicated in syndromic intellectual disability associated with Down syndrome and autism. DYRK1A has a critical role in brain growth and development primarily by regulating cell proliferation, neurogenesis, neuronal plasticity and survival. Several patients have been reported with chromosome 21 aberrations such as partial monosomy, involving multiple genes including DYRK1A. In addition, seven other individuals have been described with chromosomal rearrangements, intragenic deletions or truncating mutations that disrupt specificall…

AdultMaleMicrocephalyMonosomyDown syndromeAdolescentChromosomes Human Pair 21BiologyProtein Serine-Threonine KinasesArticleIntellectual DisabilityIntellectual disabilityGeneticsmedicineHumansAutistic DisorderChildGenetics (clinical)Chromosomal DeletionGeneticsProtein-Tyrosine Kinasesmedicine.diseasePhenotypeChild PreschoolSpeech delayMutationMicrocephalyAutismFemalemedicine.symptomChromosome DeletionDown SyndromeChromosome 21European journal of human genetics : EJHG
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Autism is a prenatal disorder: Evidence from late gestation brain overgrowth.

2018

This retrospective study aimed to specify the critical period for atypical brain development in individuals with autism spectrum disorder (ASD) using prenatal and postnatal head growth parameters. The sample consisted of 80 Caucasian, unrelated, idiopathic patients with ASD born after 1995. Fetal ultrasound parameters (head circumference [HC], abdominal circumference, and femur length) were obtained during the second and third trimesters of gestation. HC at birth and postnatal parameters at 12 and 24 months of age were also collected. Head overgrowth, assessed by HC, was highlighted during the second (20-26 weeks of amenorrhea) and third (28-36 weeks of amenorrhea) trimesters. Normal growth…

AdultMaleNeuroscience (all)ultrasoundAutism Spectrum DisorderCephalometryInfant NewbornBrainInfantbrain growthneurodevelopmental disorderUltrasonography PrenatalPregnancyChild Preschoolhead circumferenceHumansFemaleNeurology (clinical)ChildGenetics (clinical)Retrospective StudiesAutism research : official journal of the International Society for Autism Research
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Highly resilient coping entails better perceived health, high social support and low morning cortisol levels in parents of children with autism spect…

2013

The negative consequences of caring for people with developmental disabilities have been widely described. However, the ability to bounce back from the stress derived from care situations has been less studied. Those caregivers who have shown this ability are considered as resilient. This study aims to evaluate the relationship between resilience and self-reported health and cortisol awakening response (CAR) in a sample of caregivers of people with autism spectrum disorders (ASD). It also aims to evaluate the role of social support as a mediator in the association between resilience and health. Caregivers with higher resilience show better perceived health, lower morning cortisol levels, an…

AdultMaleParentsCoping (psychology)Cortisol awakening responseAdolescentHydrocortisoneHealth Statusmedia_common.quotation_subjectProtective factorLife Change EventsSocial supportSurveys and QuestionnairesAdaptation PsychologicalDevelopmental and Educational PsychologymedicineHumansChronic stressChildSalivamedia_commonSocial SupportMiddle AgedResilience Psychologicalmedicine.diseaseClinical PsychologyChild Development Disorders PervasiveAutism spectrum disorderAutismFemalePsychological resiliencePsychologyStress PsychologicalClinical psychologyResearch in Developmental Disabilities
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