Search results for "binding proteins"

showing 10 items of 911 documents

Cognitive correlates in amyotrophic lateral sclerosis: a population-based study in Italy.

2014

Background There is less data available regarding the characteristics of cognitive impairment in patients with amyotrophic lateral sclerosis (ALS) in a population-based series. Methodology Patients with ALS incident in Piemonte, Italy, between 2009 and 2011 underwent an extensive neuropsychological battery. Cognitive status was classified as follows: normal cognition, frontotemporal dementia (ALS-FTD), executive cognitive impairment (ALS-ECI), non-executive cognitive impairment (ALS-NECI), behavioural impairment (ALS-Bi), non-classifiable cognitive impairment. We also assessed 127 age-matched and gender-matched controls identified through patients’ general practitioners. Results Out of the …

Malemedicine.medical_specialtyPediatricsNeuromuscular diseasePopulationNeuropsychological TestsSuperoxide Dismutase-1Risk FactorsmedicineDementiaHumansEPIDEMIOLOGYAmyotrophic lateral sclerosisPsychiatryeducationCognitive reserveAgededucation.field_of_studyC9orf72 ProteinSuperoxide DismutaseDEMENTIAAmyotrophic Lateral SclerosisProteinsCognitionmedicine.diseaseSurvival AnalysisALS DEMENTIA EPIDEMIOLOGYDNA-Binding ProteinsPsychiatry and Mental healthItalyCase-Control StudiesMutationSurgeryFemaleSettore MED/26 - NeurologiaNeurology (clinical)ALSPsychologyCognition DisordersMotor neurone diseaseFrontotemporal dementia
researchProduct

Up‐regulation of the α‐secretase ADAM10 by retinoic acid receptors and acitretin

2009

Late-onset Alzheimer's disease is often connected with nutritional misbalance, such as enhanced cholesterol intake, deficiency in polyunsaturated fatty acids, or hypovitaminosis. The alpha-secretase ADAM10 has been found to be regulated by retinoic acid, the bioreactive metabolite of vitamin A. Here we show that retinoids induce gene expression of ADAM10 and alpha-secretase activity by nonpermissive retinoid acid receptor/retinoid X receptor (RAR/RXR) heterodimers, whereby alpha- and beta-isotypes of RAR play a major role. However, ligands of other RXR binding partners, such as the vitamin D receptor, do not stimulate alpha-secretase activity. On the basis of these findings, we examined the…

Malemedicine.medical_specialtyReceptors Retinoic AcidReceptors Cytoplasmic and NuclearMice TransgenicTretinoinRetinoic acid receptor betaRetinoid X receptorBiologyBiochemistryCell LineAcitretinADAM10 ProteinAmyloid beta-Protein PrecursorMiceKeratolytic AgentsAlzheimer DiseaseInternal medicineGeneticsmedicineAnimalsHumansPromoter Regions GeneticMolecular BiologyLiver X ReceptorsReceptors Thyroid HormoneMolecular StructureRetinoid X receptor alphaMembrane ProteinsOrphan Nuclear ReceptorsRetinoid X receptor gammaAcitretinUp-RegulationDNA-Binding ProteinsPPAR gammaADAM ProteinsRetinoic acid receptorRetinoid X ReceptorsEndocrinologyGene Expression RegulationRetinoic acid receptor alphaReceptors CalcitriolAmyloid Precursor Protein SecretasesRetinoid X receptor betaBiotechnologymedicine.drugThe FASEB Journal
researchProduct

The predictive value of heart-type fatty acid-binding protein is independent from symptom duration in normotensive patients with pulmonary embolism.

2013

Heart-type fatty acid-binding protein (H-FABP) is a useful biomarker for risk stratification of patients with pulmonary embolism (PE). In patients with acute myocardial infarction, H-FABP plasma concentrations rise after 30 minutes and return to normal within 20-24 hours. We tested whether the predictive value of H-FABP is affected by the duration of symptoms prior to diagnosis in patients with PE.We prospectively studied 257 consecutive normotensive patients with confirmed symptomatic PE.Patients with acute (24 hours; n=150) symptom onset presented more often with syncope (28.7% vs. 6.5%; p0.001) compared to patients with symptoms ≥ 24 hours (n=107); other baseline characteristics, comorbi…

Malemedicine.medical_specialtyRenal function030204 cardiovascular system & hematologyFatty Acid-Binding Proteins03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumans030212 general & internal medicineMyocardial infarctionProspective StudiesAgedbusiness.industryArea under the curveHematologyOdds ratioMiddle Agedmedicine.diseasePrognosisConfidence interval3. Good healthPulmonary embolismHeart-type fatty acid binding proteinCardiologyBiomarker (medicine)lipids (amino acids peptides and proteins)FemalebusinessPulmonary EmbolismFatty Acid Binding Protein 3Thrombosis research
researchProduct

The impact of ovarian stimulation on the expression of candidate reprogramming genes in mouse preimplantation embryos.

2012

Ovarian stimulation with gonadotrophins is an integral part of assisted reproductive technologies in human subfertility/infertility treatment. Recent findings have associated ovarian stimulation with the increased incidence of imprinting disorders in humans as well as defects in genome-wide methylation reprogramming and, in particular, imprinting in mice. Here, we present the first study that determined the impact of ovarian stimulation on the expression of developmentally important reprogramming genes <i>(Apex1, Lig1, Lig3, Mbd2, Mbd3, Mbd4, </i>and<i> Polb)</i> in single early mouse morula embryos (16-cell stage). Using absolute quantification of mRNA by quantitati…

Malemedicine.medical_specialtyTime FactorsGonadotropins EquineDown-RegulationStimulationReproductive technologyBiologyChorionic GonadotropinMBD4AndrologyMiceOogenesisOvulation InductionInternal medicineGeneticsmedicineDNA-(Apurinic or Apyrimidinic Site) LyaseAnimalsHumansHorsesRNA MessengerMolecular BiologyGenetics (clinical)GametogenesisDNA Polymerase betaRegulation of gene expressionReverse Transcriptase Polymerase Chain ReactionEmbryogenesisGene Expression Regulation DevelopmentalEmbryoDNA-Binding ProteinsMice Inbred C57BLEndocrinologyBlastocystMicroscopy Fluorescenceembryonic structuresFemaleReprogrammingTranscription FactorsCytogenetic and genome research
researchProduct

The effect of RAAS blockade on markers of renal tubular damage in diabetic nephropathy: u-NGAL, u-KIM1 and u-LFABP.

2012

Blockade of the renin-angiotensin-aldosterone system (RAAS) affects both the glomerulus and tubules. We aimed to investigate the effect of irbesartan on the tubular markers: urinary (u) neutrophil gelatinase associated protein (NGAL), Kidney injury molecule 1 (KIM1) and liver-fatty acid-binding protein (LFABP).A substudy of a double-masked, randomized, cross-over study including 52 patients with type 2 diabetes, hypertension and microalbuminuria. After 2 months washout of all antihypertensive medication except bendroflumethiazid, patients were treated in random order with irbesartan 300, 600 and 900 mg for 2 months.Urinary tubular markers at baseline and after each treatment period (ELISA),…

Malemedicine.medical_specialtyUrinary systemClinical BiochemistryUrologyRenal functionEnzyme-Linked Immunosorbent AssayType 2 diabetesurologic and male genital diseasesFatty Acid-Binding ProteinsDiabetic nephropathyRenin-Angiotensin SystemIrbesartanLipocalin-2Internal medicineDiabetes mellitusProto-Oncogene ProteinsmedicineHumansDiabetic NephropathiesHepatitis A Virus Cellular Receptor 1AgedMembrane Glycoproteinsurogenital systembusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseLipocalinsEndocrinologyKidney TubulesAlbuminuriaReceptors VirusMicroalbuminuriaFemalemedicine.symptombusinessmedicine.drugAcute-Phase ProteinsScandinavian journal of clinical and laboratory investigation
researchProduct

Protection against 2,4,6-trinitrobenzenesulphonic acid-induced colonic inflammation in mice by the marine products bolinaquinone and petrosaspongioli…

2005

Proinflammatory mediators, namely eicosanoids, reactive oxygen and nitrogen species and cytokines, are clearly involved in the pathogenesis of intestinal bowel disease. bolinaquinone (BQ) and petrosaspongiolide M (PT), two marine products with potent anti-inflammatory action, have been shown to control the production of mediators in acute and chronic inflammatory processes. Hence, we have tested here the hypothesis that BQ and PT could ameliorate inflammation and oxidative stress parameters in 2,4,6-trinitrobenzenesulphonic acid (TNBS)-induced colitis in Balb/c mice. BQ and PT were given orally in doses of 10 or 20mg/kg/day. Treatment of the animals with BQ or PT at the highest dose signifi…

Malemedicine.medical_treatmentAnti-Inflammatory AgentsNitric Oxide Synthase Type IIInflammationNerve Tissue ProteinsPharmacologymedicine.disease_causeBiochemistryProinflammatory cytokinechemistry.chemical_compoundMiceSynaptotagminsDysideamedicineAnimalsOleanolic AcidPharmacologyMice Inbred BALB CMembrane GlycoproteinsbiologySuperoxideNitrotyrosineCalcium-Binding ProteinsInterleukinMembrane ProteinsColitisInflammatory Bowel DiseasesImmunohistochemistryNitric oxide synthasechemistryBiochemistryTrinitrobenzenesulfonic AcidCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesSynaptotagmin IHeme Oxygenase (Decyclizing)biology.proteinmedicine.symptomNitric Oxide SynthaseSesquiterpenesOxidative stressHeme Oxygenase-1Prostaglandin EInterleukin-1Biochemical pharmacology
researchProduct

Neuroimmune and Mu-Opioid Receptor Alterations in the Mesocorticolimbic System in a Sex-Dependent Inflammatory Pain-Induced Alcohol Relapse-Like Rat …

2021

Evidence concerning the role of alcohol-induced neuroinflammation in alcohol intake and relapse has increased in the last few years. It is also proven that mu-opioid receptors (MORs) mediate the reinforcing properties of alcohol and, interestingly, previous research suggests that neuroinflammation and MORs could be related. Our objective is to study neuroinflammatory states and microglial activation, together with adaptations on MOR expression in the mesocorticolimbic system (MCLS) during the abstinence and relapse phases. To do so, we have used a sex-dependent rat model of complete Freund’s adjuvant (CFA)-induced alcohol deprivation effect (ADE). Firstly, our results confirm that only CFA-…

Malemedicine.medical_treatmentFreund's AdjuvantReceptors Opioid mualcohol deprivation effectNitric Oxide Synthase Type IImicroglianeuroinflammationRats Sprague-DawleyRecurrenceLimbic SystemImmunology and AllergypainPhosphorylationReceptormedia_commonMicrogliaAlcohol AbstinencealcoholMicrofilament ProteinsNF-kappa BBrief Research ReportInterleukin 10AlcoholismCytokinemedicine.anatomical_structureCytokinesFemaleμ-opioid receptorInflammation Mediatorsmedicine.medical_specialtyNeuroimmunomodulationmedia_common.quotation_subjectImmunologyPrefrontal CortexSex FactorsDownregulation and upregulationInternal medicinemedicineAnimalsNeuroinflammationbusiness.industryCalcium-Binding ProteinsAbstinenceRC581-607EndocrinologyCyclooxygenase 2mu-opioid receptorImmunologic diseases. AllergybusinessFrontiers in Immunology
researchProduct

Differential expression of suppressors of cytokine signaling-1, -2, and -3 in the rat hippocampus after seizure: implications for neuromodulation by …

2003

Numerous studies have investigated the expression of various cytokine families in the CNS after brain injury. The gp130 or interleukin (IL)-6-type cytokines have received a great deal of focus, and it is clear that they exhibit an acute and robust upregulation in various brain injury models. We are interested to determine, however, whether endogenously expressed cytokines in the CNS act in a direct neuromodulatory manner. In an accompanying study, we examined the expression of five gp130 cytokines and their receptors in the lithium-pilocarpine model of status epilepticus. We follow up that study here by trying to determine if gp130 signal transduction occurs in hippocampal principal neurons…

Malemedicine.medical_treatmentPopulationSuppressor of Cytokine Signaling ProteinsHippocampal formationBiologyNeuroprotectionHippocampusRats Sprague-DawleySuppressor of Cytokine Signaling 1 ProteinSeizuresmedicineAnimalsRNA MessengerReceptors Cytokineeducationeducation.field_of_studyGeneral NeuroscienceInterleukinGlycoprotein 130RatsDNA-Binding ProteinsRepressor ProteinsCytokineGene Expression RegulationSuppressor of Cytokine Signaling 3 ProteinTrans-ActivatorsCytokinesSignal transductionCytokine receptorCarrier ProteinsNeuroscienceSignal TransductionTranscription FactorsNeuroscience
researchProduct

FABP4 plasma levels are increased in familial combined hyperlipidemia

2010

The lipid profile of familial combined hyperlipidemia (FCHL) shares some characteristics with atherogenic dyslipidemia seen in diabetes, metabolic syndrome, and obesity. Adipocyte fatty acid-binding protein 4 (FABP4) appears to be a determinant of atherogenic dyslipidemia. We examined relationships between FABP4 plasma concentrations, dyslipidemia, and metabolic variables in patients with FCHL. We studied 273 unrelated FCHL patients and 118 control subjects. FABP4 was higher in FCHL than controls, with mean levels of 21.8 (10.1) microg/l and 19.2 (9.2) microg/l, respectively (adjusted P= 0.012). In FCHL, FABP4 correlated to body mass index (BMI), waist circumference, insulin levels, and hom…

Maleobesitymedicine.medical_specialtyMagnetic Resonance Spectroscopymedicine.medical_treatmentHyperlipidemia Familial CombinedQD415-436Fatty Acid-Binding ProteinsBiochemistryCohort StudiesInsulin resistanceEndocrinologyinsulin resistanceInternal medicineDiabetes mellitusmedicineHumansmedicine.diagnostic_testbusiness.industryInsulindyslipidemiaHypertriglyceridemiaCell BiologyMiddle AgedLipid Metabolismmedicine.diseaseEndocrinologyFCHLFemaleMetabolic syndromePatient-Oriented and Epidemiological ResearchLipid profilebusinessBody mass indexDyslipidemiaJournal of Lipid Research
researchProduct

Rac1 GTPase, a multifunctional player in the regulation of genotoxic stress response

2013

The Ras-related C3 botulinum toxin substrate 1 (Rac1) belongs to the Ras-homologous (Rho) family of small GTPases, which transduce signals from the outside to the inside of a cell. Rac1 becomes activated upon ligand binding of a variety of receptors, including receptor tyrosine kinases and heterotrimeric G-protein-coupled receptors. After GTP loading by guanine exchange factors (GEFs), GTP-bound Rac1 engages numerous effector proteins, thereby eventually regulating cell motility and adhesion, cell cycle progression through G1, mitosis and meiosis, as well as cell death and metastasis.1 Besides, Rac1 adjusts cellular responses to genotoxic agents, such as UV light and alkylating agents, by r…

Malerac1 GTP-Binding Proteintopoisomerase IIAgingRHOADNA repairDNA damagep38 mitogen-activated protein kinasesApoptosisRAC1Editorials: Cell Cycle FeaturesDNA damage responseReceptor tyrosine kinasechemical carcinogenesisHistonesMiceTransforming Growth Factor betaRho GTPasesAnimalsMolecular BiologyTranscription factoranthracyclinesMice KnockoutbiologyKinaseNeuropeptidesConnective Tissue Growth FactorHMG-CoA reductase inhibitors (statins)Cell BiologyFibrosisgenotoxic stressActinsrac GTP-Binding ProteinsCell biologyOxidative Stressnormal tissue damageGene Expression RegulationLiverBiochemistryDoxorubicinGamma Raysbiology.proteinFemaleDNA DamageMutagensSignal TransductionDevelopmental BiologyCell Cycle
researchProduct