Search results for "bioinformatics"

showing 10 items of 1632 documents

A de novo microdeletion of SEMA5A in a boy with autism spectrum disorder and intellectual disability.

2016

AbstractSemaphorins are a large family of secreted and membrane-associated proteins necessary for wiring of the brain. Semaphorin 5A (SEMA5A) acts as a bifunctional guidance cue, exerting both attractive and inhibitory effects on developing axons. Previous studies have suggested that SEMA5A could be a susceptibility gene for autism spectrum disorders (ASDs). We first identified a de novo translocation t(5;22)(p15.3;q11.21) in a patient with ASD and intellectual disability (ID). At the translocation breakpoint on chromosome 5, we observed a 861-kb deletion encompassing the end of the SEMA5A gene. We delineated the breakpoint by NGS and observed that no gene was disrupted on chromosome 22. We…

Male0301 basic medicinemedicine.medical_specialtyAutism Spectrum DisorderChromosomes Human Pair 22Translocation BreakpointNerve Tissue ProteinsSemaphorinsBiology[SDV.GEN.GH] Life Sciences [q-bio]/Genetics/Human geneticsBioinformaticsArticleTranslocation GeneticautismeChromosome Breakpoints03 medical and health sciencesSemaphorin[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyIntellectual Disabilitymental disordersIntellectual disabilityGeneticsmedicineHumans[ SDV.GEN.GH ] Life Sciences [q-bio]/Genetics/Human geneticsChildGenetics (clinical)Genetics[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyNeurosciencesMembrane Proteinsmedicine.disease030104 developmental biology[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsAutism spectrum disorderNeurons and CognitionPaternal InheritancecerveauChromosomes Human Pair 5AutismMedical geneticsChromosome DeletionmicrodélétionhumainChromosome 22[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyGenetic screen
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Bigger is not better: cortisol-induced cardiac growth and dysfunction in salmonids

2015

This is a Published Manuscript of an article published by Company of Biologists in Journal of Experimental Biology, available online: http://www.biologists.com/ Stress and elevated cortisol levels are associated with pathological heart growth and cardiovascular disease in humans and other mammals. We recently established a link between heritable variation in post-stress cortisol production and cardiac growth also in salmonid fish. A conserved stimulatory effect of the otherwise catabolic steroid hormone cortisol is likely implied, but has to date not been established experimentally. Furthermore, whereas cardiac growth is associated with failure of the mammalian heart, pathological cardiac h…

Male0301 basic medicinemedicine.medical_specialtyendocrine systemHydrocortisonePhysiologyGene ExpressionHeart failureCardiac performance030204 cardiovascular system & hematologyAquatic ScienceBiologyBioinformatics03 medical and health sciences0302 clinical medicineSDG 3 - Good Health and Well-beingInternal medicinemedicineAnimalsChronic stressCardiac OutputMolecular BiologySwimmingEcology Evolution Behavior and SystematicsVentricular RemodelingHeartStroke VolumeHypertrophyMyocardial hypertrophymedicine.diseasePeer reviewRainbow trout030104 developmental biologyEndocrinologyOncorhynchus mykissInsect ScienceHeart failureMyocardial hypertrophy/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingExperimental biologyChronic stressFemaleAnimal Science and ZoologyRainbow trout
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Patients experiencing statin-induced myalgia exhibit a unique program of skeletal muscle gene expression following statin re-challenge

2017

Statins, the 3-hydroxy-3-methyl-glutaryl (HMG)-CoA reductase inhibitors, are widely prescribed for treatment of hypercholesterolemia. Although statins are generally well tolerated, up to ten percent of statin-treated patients experience myalgia symptoms, defined as muscle pain without elevated creatinine phosphokinase (CPK) levels. Myalgia is the most frequent reason for discontinuation of statin therapy. The mechanisms underlying statin myalgia are not clearly understood. To elucidate changes in gene expression associated with statin myalgia, we compared profiles of gene expression in skeletal muscle biopsies from patients with statin myalgia who were undergoing statin re-challenge (cases)…

Male0301 basic medicinemyalgiaGene Expressionlcsh:MedicineApoptosis030204 cardiovascular system & hematologyPathology and Laboratory MedicineBioinformaticsBiochemistry0302 clinical medicineMedicine and Health SciencesGene Regulatory Networkslcsh:ScienceMusculoskeletal SystemEnergy-Producing OrganellesMyositisRegulation of gene expressionMultidisciplinaryCell DeathbiologyMusclesDrugsMiddle AgedMitochondriaCell ProcessesHMG-CoA reductaseFemalelipids (amino acids peptides and proteins)AnatomyCellular Structures and Organellesmedicine.symptomResearch ArticleSenescencemedicine.medical_specialtyStatinmedicine.drug_classPainBioenergeticsPolymorphism Single Nucleotide03 medical and health sciencesSigns and SymptomsDiagnostic MedicineInternal medicineGeneticsmedicineHumansGene Regulationcardiovascular diseasesMuscle SkeletalAgedPharmacologybusiness.industrylcsh:RStatinsBiology and Life SciencesComputational Biologynutritional and metabolic diseasesMyalgiaCell Biologymedicine.disease030104 developmental biologyEndocrinologyGene Expression RegulationSkeletal MusclesLeukocytes Mononuclearbiology.proteinProtein prenylationlcsh:QHydroxymethylglutaryl-CoA Reductase InhibitorsSLCO1B1businessPLOS ONE
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Exome sequencing of a family with lone, autosomal dominant atrial flutter identifies a rare variation in ABCB4 significantly enriched in cases

2015

Background Lone atrial flutter (AFL) and atrial fibrillation (AF) are common and sometimes consequential cardiac conduction disorders with a strong heritability, as underlined by recent genome-wide association studies that identified genetic modifiers. Follow-up family-based genetic analysis also identified Mendelian transmission of disease alleles. Three affected members were exome-sequenced for the identification of potential causative mutations, which were subsequently validated by direct sequencing in the other 3 affected members. Taqman assay was then used to confirm the role of any mutation in an independent population of sporadic lone AFL/AF cases. Results The family cluster analysis…

MaleATP Binding Cassette Transporter Subfamily BDNA Mutational AnalysisPopulationMutation MissenseSNPGenome-wide association studySingle-nucleotide polymorphismAtrial flutterBiologyBioinformaticsPolymorphism Single NucleotideDNA Mutational AnalysiExome-sequencingGeneticCardiac conductionGeneticsHumansGenetics(clinical)ExomeAlleleeducationExomeATP-binding cassette B4 (ABCB4)Genetics (clinical)Exome sequencingAgedGenetic associationAged 80 and overGeneticseducation.field_of_studyP-GlycoproteinAtrial fibrillationPedigreeFemaleHumanGenome-Wide Association StudyResearch ArticleSNPs
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On versus off-hour care of patients with acute coronary syndrome and persistent ST-segment elevation in certified German chest pain units

2016

BACKGROUND Regional healthcare projects improve the off-hour care of patients with acute coronary syndromes and persistent ST-segment elevation myocardial infarction (STEMI). To analyse differences in quality of care between on and off-hour care of STEMI patients admitted to certified German chest pain units. METHODS A total of 1107 STEMI patients from the German chest pain unit registry were enrolled. Analyses comprised critical time intervals (symptoms to first medical contact (FMC), FMC to admission, symptoms to admission, symptoms to balloon, FMC to balloon, door to balloon times) and major adverse cardiac and cerebrovascular events at follow-up. RESULTS 54.8% of patients were admitted …

MaleAcute coronary syndromePercutaneousmedicine.medical_treatmentMedizin030204 cardiovascular system & hematologyCritical Care and Intensive Care MedicineBioinformaticsBalloonChest painTime-to-Treatment03 medical and health sciences0302 clinical medicineAfter-Hours CareGermanyMyocardial RevascularizationmedicineHumansST segment030212 general & internal medicineMyocardial infarctionAcute Coronary SyndromeAngioplasty Balloon CoronaryAgedbusiness.industryPercutaneous coronary interventionGeneral MedicineMiddle Agedmedicine.diseaseAnesthesiaDoor-to-balloonST Elevation Myocardial InfarctionFemalemedicine.symptomCardiology and Cardiovascular Medicinebusiness
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Redefining the MED13L syndrome

2015

Congenital cardiac and neurodevelopmental deficits have been recently linked to the mediator complex subunit 13-like protein MED13L, a subunit of the CDK8-associated mediator complex that functions in transcriptional regulation through DNA-binding transcription factors and RNA polymerase II. Heterozygous MED13L variants cause transposition of the great arteries and intellectual disability (ID). Here, we report eight patients with predominantly novel MED13L variants who lack such complex congenital heart malformations. Rather, they depict a syndromic form of ID characterized by facial dysmorphism, ID, speech impairment, motor developmental delay with muscular hypotonia and behavioral difficu…

MaleAdolescentHeart malformationTransposition of Great VesselsRNA polymerase IIBioinformaticsArticleMediatorIntellectual DisabilityIntellectual disabilityGeneticsmedicineTranscriptional regulationHumansAbnormalities MultipleChildTranscription factorGenetics (clinical)GeneticsScience & TechnologyMediator ComplexbiologyMuscular hypotoniaSyndromemedicine.diseasePhenotypeChild PreschoolMutationbiology.proteinMuscle HypotoniaFemaleNeurocognitiveEuropean Journal of Human Genetics
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Is the Mean Blood Leukocyte Telomere Length a Predictor for Sporadic Thoracic Aortic Aneurysm? Data from a Preliminary Study

2012

Telomeres have been postulated as a universal clock that shortens in parallel with cellular aging. They are specialized DNA-protein structures at the ends of chromosome with remarkable functions--preventing their recognition as double-stranded DNA breaks, protecting their recombination and degradation, and avoiding a DNA damage cellular response. Telomere shortening is currently considered the best aging marker, but is also a predictor for age-related diseases, including cardiovascular diseases. Biological age clearly seems to be a better predictor of vascular risk rather than chronological age. This concept is supported by key assumptions that peripheral blood leukocyte telomere content ac…

MaleAgingPathologymedicine.medical_specialtyThoracicBiological ageVascular riskBiologyBioinformaticsThoracic aortic aneurysmGeneticLeukocytesmedicineHumansSettore MED/05 - Patologia ClinicaAged; Aging; Aortic Aneurysm Thoracic; Case-Control Studies; Cellular Senescence; DNA; DNA Damage; Female; Humans; Leukocytes; Male; Middle Aged; Recombination Genetic; Telomere; Vascular DiseasesVascular DiseasesCellular Senescencevascular ageingAgedRecombination GenetictelomereAortic Aneurysm ThoracicVascular diseaseChromosomeSettore MED/23 - Chirurgia CardiacaDNAMiddle Agedmedicine.diseaseRecombinationPeripheral bloodAortic AneurysmTAATelomereCellular AgingCase-Control StudiesFemaleGeriatrics and GerontologyDNA DamageRejuvenation Research
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Gut microbiota disturbance during antibiotic therapy: a multi-omic approach

2014

It is known that the gastrointestinal tract (GIT) microbiota responds to different antibiotics in different ways and that while some antibiotics do not induce disturbances of the community, others drastically influence the richness, diversity, and prevalence of bacterial taxa. However, the metabolic consequences thereof, independent of the degree of the community shifts, are not clearly understood. In a recent article, we used an integrative OMICS approach to provide new insights into the metabolic shifts caused by antibiotic disturbance. The study presented here further suggests that specific bacterial lineage blooms occurring at defined stages of antibiotic intervention are mostly associa…

MaleAnabolismAntibioticsGene ExpressionGut floraGastrointestinal FunctionBioinformaticsprokaryotesFecesRNA Ribosomal 16SSystems and Synthetic Biologyhuman fecal microbiota1506MetaproteomicGut Microbiotadatabase2. Zero hungerlong-term impactsGastrointestinal tract0303 health sciencesSysteem en Synthetische Biologiehuman intestinal microbiotabiologyMicrobiotaGastroenterologyBiodiversity3. Good healthAnti-Bacterial AgentsBacterial Typing TechniquesArticle AddendumRNA BacterialInfectious DiseasesMetabolomecommunitymetaproteomicsHuman gut microbiotaMicrobiology (medical)DNA BacterialDisturbance (geology)medicine.drug_classperturbationMetabolomicbeta-LactamsMicrobiologyMicrobiologyresistance03 medical and health sciencesMetagenomicAntibiotic therapyMetabolomemedicineHumans030304 developmental biologyAgedVLAGBacteria030306 microbiologyGene Expression ProfilingColonic MicrofloraAkkermansiaAntibiotic therapybiology.organism_classificationGastrointestinal TractEnterococcusGene Expression RegulationMetaproteomicsMetatranscriptomicGastrointestinal functionmetabolismMeta-Analysis
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Co-registering kinematics and evoked related potentials during visually guided reach-to-grasp movements.

2013

Background:In non-human primates grasp-related sensorimotor transformations are accomplished in a circuit involving the anterior intraparietal sulcus (area AIP) and both the ventral and the dorsal sectors of the premotor cortex (vPMC and dPMC, respectively). Although a human homologue of such a circuit has been identified, the time course of activation of these cortical areas and how such activity relates to specific kinematic events has yet to be investigated.Methodology/Principal Findings:We combined kinematic and event-related potential techniques to explicitly test how activity within human grasping-related brain areas is modulated in time. Subjects were requested to reach towards and g…

MaleAnatomy and PhysiologyVisual Systemlcsh:MedicineKinematicsBioinformaticsBrain mappingParietal Lobelcsh:ScienceBrain MappingMultidisciplinaryHand StrengthMedicine (all)StatisticsParietal lobeMotor CortexSensory SystemsBiomechanical PhenomenaElectrophysiologymedicine.anatomical_structureMedicineFemaleMotor cortexHumanResearch ArticleAdultCognitive NeuroscienceMovementNeurophysiologyIntraparietal sulcusBiologyStimulus (physiology)BiostatisticsNeurological SystemPremotor cortexMotor ReactionsmedicineHumansStatistical MethodsBiologyMotor SystemsBiochemistry Genetics and Molecular Biology (all)Settore M-PSI/02 - Psicobiologia E Psicologia Fisiologicalcsh:RIndex fingerEvoked Potentials MotorHandbody regionsAgricultural and Biological Sciences (all)lcsh:QNeuroscienceMathematicsPsychomotor PerformanceNeurosciencePLoS ONE
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Association of estrogen receptor α gene with Alzheimer's disease: A case-control study

2006

Abstract Recent experimental data have offered the biological background to study the estrogen receptor (ER) alpha gene as a candidate gene for AD. Genetic association studies proposed ERalpha PvuII and XbaI gene polymorphisms as susceptibility factors for AD, although subsequent studies did not replicate this finding. To verify this association in a Caucasian Italian sample, we conducted a case-control study in a dataset of 172 clinic-based probable AD cases and 172 age- and sex-matched controls. Possible interaction between ERalpha polymorphisms and sex, age at onset of AD or apolipoprotein E (APOE) was examined. The xx-genotype of the XbaI polymorphism was associated with the risk of dev…

MaleApolipoprotein EOncologymedicine.medical_specialtyCandidate geneGenotypeEstrogen receptorNeuropsychological TestsBioinformaticsRisk AssessmentApolipoproteins ESex FactorsGene FrequencyAlzheimer DiseasePolymorphism (computer science)Internal medicineOdds RatiomedicineHumansRisk factorAllelesAgedGenetic associationAged 80 and overPolymorphism Geneticbusiness.industryGeneral NeuroscienceAge FactorsEstrogen Receptor alphaCase-control studyDNAGeneral MedicineMiddle AgedPsychiatry and Mental healthClinical PsychologyItalyCase-Control StudiesSettore MED/26 - NeurologiaFemaleGeriatrics and GerontologypolymorphismsbusinessEstrogen receptor alphaAPOEestrogen receptor
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