Search results for "brain edema"

showing 10 items of 56 documents

Moderate controlled cortical contusion in pigs: effects on multi-parametric neuromonitoring and clinical relevance.

2004

Over the last decade, routine neuromonitoring of ICP and CPP has been extended with new on-line techniques such as microdialysis, tissue oxygen (ptiO(2)), acid-base balance (ptiCO(2), pH) and CBF measurements, which so far have not lead to clear-cut therapy approaches in the neurointensive care unit. This is partially due to the complex pathophysiology following a wide-range of brain injuries, and the lack of suitable animal models allowing simultaneous, clinically relevant neuromonitoring under controlled conditions. Therefore, a controlled cortical impact (CCI) model in large animals (pig) has been developed. After placement of microdialysis, ptiO(2), temperature and ICP catheters, an uni…

MalePathologymedicine.medical_specialtyMicrodialysisIntracranial PressureSwineGlutamic AcidBrain EdemaBody TemperatureCentral nervous system diseaseText miningOxygen ConsumptionPyruvic AcidmedicineAnimalsClinical significanceLactic AcidCell damageMonitoring Physiologicbusiness.industryGlutamate receptormedicine.diseasePathophysiologyDisease Models Animalmedicine.anatomical_structureGlucoseCerebral cortexAnesthesiaBrain InjuriesNeurology (clinical)businessJournal of neurotrauma
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Photodynamic therapy within edematous brain tissue: Considerations on sensitizer dose and time point of laser irradiation

1996

Photosensitizer is known to spread with vasogenic edema fluid arising from a cerebral lesion (Neurosurg 33:1075-1082, 1993), which may be essential for sensitizing malignant cells outside the main tumor mass. The present experiments seek to elucidate whether resultant necrosis of perifocal brain tissue after laser irradiation follows a corresponding time pattern and whether damage depends on the photosensitizer dose. Male Wistar rats were anaesthetized with chloralhydrate for venous cannulation, craniotomy and focal cold lesion in order to induce vasogenic edema. Simultaneously, Photofrin II (PF II) was administered at a dose of 5 mg kg-1. The animals were re-anaesthetized after either 4, 1…

MalePathologymedicine.medical_specialtyTime FactorsNecrosismedicine.medical_treatmentBiophysicsBrain EdemaPhotodynamic therapyBlood–brain barrierLesionmedicineAnimalsRadiology Nuclear Medicine and imagingPhotosensitizerRats WistarCraniotomySensitizationRadiationDose-Response Relationship DrugRadiological and Ultrasound TechnologyBrain NeoplasmsChemistryRatsmedicine.anatomical_structurePhotochemotherapyDihematoporphyrin EtherLaser Therapymedicine.symptomPerfusionJournal of Photochemistry and Photobiology B: Biology
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Kinetics of Photofrin II in perifocal brain edema.

1993

Photodynamic therapy is under intense investigation as a possible adjuvant for the treatment of malignant tumors of the central nervous system. It relies on the fact that photosensitizers are selectively taken up or retained by malignant tissue. However, most brain tumors are accompanied by substantial vasogenic edema as a consequence of blood-brain barrier disruption within the tumor, leading to extravasation and propagation of plasma constituents into the surrounding brain tissue. Systemically administered photosensitizers may enter healthy tissue together with the edema fluid, possibly leading to sensitization of tissues outside the tumor. To test this hypothesis, vasogenic edema was ind…

MalePathologymedicine.medical_specialtymedicine.medical_treatmentCentral nervous systemPhotodynamic therapyBrain EdemaCerebral edemaLesionchemistry.chemical_compoundEdemamedicineAnimalsPhotosensitizerTissue DistributionRats WistarFluorescein isothiocyanatebusiness.industryBrain Neoplasmsmedicine.diseaseExtravasationRatsCold TemperatureDisease Models Animalmedicine.anatomical_structurechemistryMicroscopy FluorescencePhotochemotherapySurgeryDihematoporphyrin EtherNeurology (clinical)medicine.symptombusinessFluorescein-5-isothiocyanateExtravasation of Diagnostic and Therapeutic MaterialsNeurosurgery
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Inhibition of Proteasomal Glucocorticoid Receptor Degradation Restores Dexamethasone-Mediated Stabilization of the Blood–Brain Barrier After Traumati…

2013

To establish the molecular background for glucocorticoid insensitivity, that is, failure to reduce edema formation and to protect blood-brain barrier integrity after acute traumatic brain injury.Controlled animal study.University research laboratory.Male C57Bl/6N mice.Mechanical brain lesion by controlled cortical impact.Our study demonstrates that 1) proteasomal glucocorticoid receptor degradation is established in brain endothelial cells after traumatic brain injury as a form of posttranslational glucocorticoid receptor modification; 2) inhibition of the proteasomal degradation pathway with bortezomib (0.2 mg/kg) in combination with the glucocorticoid dexamethasone (10 mg/kg) by subcutane…

MaleProteasome Endopeptidase ComplexTraumatic brain injuryBlotting WesternBrain EdemaPharmacologyReal-Time Polymerase Chain ReactionCritical Care and Intensive Care MedicineBlood–brain barrierSensitivity and SpecificityDexamethasoneStatistics NonparametricBortezomibMiceRandom AllocationReceptors GlucocorticoidGlucocorticoid receptorReference ValuesmedicineAnimalsRNA MessengerReceptorDexamethasonebusiness.industryBortezomibmedicine.diseaseBoronic AcidsImmunohistochemistryMice Inbred C57BLBlotDisease Models Animalmedicine.anatomical_structureBlood-Brain BarrierBrain InjuriesPyrazinesMultivariate AnalysisBlood Gas AnalysisbusinessGlucocorticoidmedicine.drugCritical Care Medicine
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Neuroprotection by erythropoietin administration after experimental traumatic brain injury.

2007

A large body of evidence indicates that the hormone erythropoietin (EPO) exerts beneficial effects in the central nervous system (CNS). To date, EPO's effect has been assessed in several experimental models of brain and spinal cord injury. This study was conducted to validate whether treatment with recombinant human EPO (rHuEPO) would limit the extent of injury following experimental TBI. Experimental TBI was induced in rats by a cryogenic injury model. rHuEPO or placebo was injected intraperitoneally immediately after the injury and then every 8 h until 2 or 14 days. Forty-eight hours after injury brain water content, an indicator of brain edema, was measured with the wet-dry method and bl…

MaleTime FactorsBrain EdemaFunctional LateralityRats Sprague-Dawleychemistry.chemical_compoundTraumatic brain injuryMedicineAnalysis of Variance Animals Blood-Brain Barrier; drug effects Brain Edema; drug therapy/etiology Brain Infarction; drug therapy/etiology Brain Injuries; complications/drug therapy Disease Models; Animal Erythropoietin; administration /&/ dosage Evans Blue; diagnostic use Functional Laterality Humans Male Neurologic Examination Neuroprotective Agents; administration /&/ dosage Rats Rats; Sprague-Dawley Reaction Time; drug effects Recombinant Proteins Time Factorsadministration /&/ dosageSpinal cord injuryEvans BlueNeurologic ExaminationGeneral Neuroscienceexperimental models of brain and spinal cord injuryExtravasationNeuroprotectionRecombinant Proteinsmedicine.anatomical_structureNeuroprotective AgentsBlood-Brain BarrierAnesthesiadiagnostic usemedicine.drugEvans BlueBrain InfarctionTraumatic brain injuryCentral nervous systemrecombinant human EPO (rHuEPO)PlaceboNeuroprotectionReaction TimeAnimalsHumansMolecular BiologyErythropoietinAnalysis of VarianceNeuroscience (all)business.industryAnimaldrug therapy/etiologymedicine.diseaseRatsDisease Models AnimalchemistryErythropoietindrug effectsBrain InjuriesDisease Modelsrecombinant human EPO (rHuEPO); experimental models of brain and spinal cord injury; NeuroprotectionNeurology (clinical)Sprague-Dawleybusinesscomplications/drug therapyDevelopmental BiologyBrain research
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Photochemically induced, graded cerebral infarction in the mouse by laser irradiation evolution of brain edema

1992

Cerebral infarction was produced in mice by intravenous injection of a photosensitive dye, rose bengal (10 mg/kg), and by focal illumination of the intact skull surface for 3 min with a laser source, operating at 570 nm with power levels of 2, 5, 10, and 20 mW. Location of infarction was made using 2,3,5-triphenyltetrazolium chloride and the time course of edema in the irradiated cerebral hemisphere was evaluated by measuring water, sodium, and potassium content 4, 24, and 72 hr after irradiation. With power levels of 2 and 5 mW, the infarct was restricted to the cortex, whereas with power levels of 10 and 20 mW, it extended to subcortical regions. Increases in water and sodium and decrease…

MaleTime FactorsPhotochemistryPotassiumSodiumInfarctionchemistry.chemical_elementBrain EdemaToxicologyMicechemistry.chemical_compoundCortex (anatomy)EdemaRose bengalAnimalsMedicineIrradiationBrain ChemistryCerebral CortexPharmacologyRose Bengalbusiness.industryCerebral infarctionLasersCerebral InfarctionWater-Electrolyte Balancemedicine.diseaseDisease Models Animalmedicine.anatomical_structurechemistrySpectrophotometryAnesthesiaInjections Intravenousmedicine.symptombusinessBiomedical engineeringJournal of Pharmacological and Toxicological Methods
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Toxicological investigations in a fatal and non-fatal accident due to hydrogen sulphide (H2S)poisoning

2019

Hydrogen sulphide (H2S) is one of the most toxic natural gas and represents a not rare cause of fatal events in workplaces. We report here a serious accidental poisoning by hydrogen sulphide inhalation involving six sailors. Three of them died while the other three survived and were transported to the emergency room. No greenish discolouration of the body, that could be a feature of these type of deaths, was observed at autopsy. Given that blood and/or urine H2S detection does not allow to discriminate if it is related to inhalation or to putrefactive processes, the determination of thiosulphate, H2S main metabolite, is decisive. The succession of fatal events reported here can be rebuilt b…

MaleTime FactorsPoison controlAutopsyFatal accidentBrain EdemaUrine01 natural scienceschemistry.chemical_compound0302 clinical medicineThiosulfateHydrogen sulphideMedicineHydrogen SulfideThiosulfateAir PollutantsOccupational accidentInhalationFatal poisoningMiddle AgedPulmonary edemaFatal poisoning; Hydrogen sulphide; Non-fatal poisoning; Occupational accident; Thiosulphate; Administration Inhalation; Adult; Air Pollutants; Brain Edema; Emphysema; Humans; Hydrogen Sulfide; Hyperemia; Italy; Male; Middle Aged; Military Personnel; Pulmonary Edema; Thiosulfates; Time Factors; Accidents OccupationalOccupationalMilitary PersonnelInhalationItalyAir PollutantAnesthesiaAdministrationNon-fatal poisoningHumanAdultTime FactorThiosulphateThiosulfatesHyperemiaPulmonary EdemaHydrogen sulphidePathology and Forensic Medicine03 medical and health sciencesAdministration InhalationHumansAccidents Occupational030216 legal & forensic medicineEmphysemabusiness.industry010401 analytical chemistrymedicine.disease0104 chemical scienceschemistryAccidentsbusinessLaw
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Cranioplasty with autologous bone flaps cryopreserved with Dimethylsulphoxide : does tissue processing matter

2021

Este artículo se encuentra disponible en la siguiente URL: https://www.sciencedirect.com/science/article/abs/pii/S1878875021001625?via%3Dihub En este artículo de investigación también participan: Dolores Ocete, Lucas Aranda, Ana Melero, Antonio J. Guillot, Nuria Yagüe y Carlos Botella. Este es el pre-print del siguiente artículo: Mirabet, V., García, D., Roca, A., Quiroz, A. R., Antón, J., Rodríguez-Cadarso, M., Ocete, D., Aranda, L., Melero, A., Guillot, A. J., Yagüe, N., Guillén, I. & Botella, C. (2021). Cranioplasty with autologous bone flaps cryopreserved with Dimethylsulphoxide: does tissue processing matter. World Neurosurgery, vol. 149 (may.), pp. e582?e591, que se ha publicado de fo…

MaleTime Factorsmedicine.medical_treatmentBrain EdemaSurgical Flaps0302 clinical medicineCryoprotective AgentsPostoperative ComplicationsHuesos - Crioconservación.Brain Injuries TraumaticAutograftsAutologous bone flapMiddle AgedCranioplastyResorptionAnti-Bacterial AgentsStrokeCryopreservacion of organs tissues etc.030220 oncology & carcinogenesisTissue bankVancomycinDecompressive craniectomyFemalemedicine.drugCrioconservación de órganos tejidos etc.Adultmedicine.medical_specialtyDecompressive CraniectomyAdolescentDecompressive craniectomyCráneo - Cirugía.CranioplastySkull - Surgery.03 medical and health sciencesYoung AdultmedicineHumansSurgical Wound InfectionDimethyl SulfoxideBones - Cryopreservacion.Bone ResorptionCryopreservationbusiness.industryBone storageSkullPostoperative complicationBone processingPlastic Surgery Proceduresmedicine.diseaseSurgeryHydrocephalusSurgeryNeurology (clinical)businessComplication030217 neurology & neurosurgery
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A new model of thromboembolic stroke in the posterior circulation of the rat

2006

The prognosis of vertebrobasilar occlusion is grave and therapeutic options are limited. The aim of the present study was to develop a new model of embolic hindbrain ischemia in the rat that closely resembles the clinical situation and that can be used to study pathophysiology and treatment options. After thoracotomy in 20 male Wistar rats, 15 animals received an injection of in vitro prepared autologous blood clots into the left vertebral artery. Five animals without clot injection served as controls. Neurological deficits were assessed in all animals 2 h after embolism. After 2 h, five animals were sacrificed to measure cerebral blood flow (CBF) by iodo-antipyridine autoradiography, and t…

Malemedicine.medical_specialtyCerebellumIschemiaBrain EdemaThromboembolic strokeThromboembolismInternal medicineOcclusionmedicineAnimalsRats WistarStrokebusiness.industryGeneral NeuroscienceCerebral InfarctionBlood flowmedicine.diseaseImmunohistochemistryRatsStrokeDisease Models Animalmedicine.anatomical_structureIntracranial EmbolismEmbolismCerebral blood flowCerebrovascular CirculationAnesthesiaCardiologyAutoradiographybusinessJournal of Neuroscience Methods
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Minimally Invasive Management of Spontaneous Supratentorial Intracerebral Lobar Hemorrhages by a “Homemade” Endoscopic Strategy: The Evangelical Doct…

2019

Background Although the incidence of intracerebral hemorrhage (ICH) has appeared to be increasing over the years, its prognosis remains dismal. No consensus has yet been reached regarding the management of ICH; however, minimally invasive surgery should limit, if not avoid, intraoperative parenchymal damage. Therefore, we have presented a novel, modified “homemade” approach aimed to shorten the operative time and minimize the corticectomy and brain manipulation. Methods From 2008 to 2017, 53 patients (32 men and 21 women; mean age, 63.8 years) were admitted to our neurosurgery department and surgically treated for a lobar ICH. A modified suction tube, coupled with the endoscope light source…

Malemedicine.medical_specialtyEndoscopeFamous PersonsSettore MED/27 - NEUROCHIRURGIACerebral hemorrhageIntracranial hypertension03 medical and health sciences0302 clinical medicineHematomaMinimally invasive surgerymedicineHumansMinimally Invasive Surgical ProceduresClot evacuationStrokeAgedIntracerebral hemorrhagemedicine.diagnostic_testbusiness.industryFamous PersonGlasgow Coma ScaleDisease ManagementEndoscopyMinimally Invasive Surgical ProcedureMiddle Agedmedicine.diseaseLoupeSurgeryEndoscopyStroke030220 oncology & carcinogenesisBrain edemaNeuroendoscopySurgeryFemaleNeurology (clinical)Neurosurgerybusiness030217 neurology & neurosurgeryHuman
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