Search results for "brain"

showing 10 items of 3997 documents

Exercise and lactate production:implication in fatigue and in brain signaling

2016

More than two hundred years after its discovery, lactate still remains an intriguing molecule. Considered for a long time as a waste product of metabolism and the culprit behind muscular fatigue, it was then recognized as an important fuel for many cells. In particular, in the nervous system, it has been proposed that lactate, released by astrocytes in response to neuronal activation, is taken up by neurons, oxidized to pyruvate and used for synthesizing acetyl-CoA to be used for the tricarboxylic acid cycle. More recently, in addition to this metabolic role, the discovery of a specific receptor prompted a reconsideration of its role, and lactate is now seen as a sort of hormone, even invol…

lactate transporterbrain metabolismlactic acidlactate receptorexercise and lactatelactic acid; brain metabolism; lactate transporters; blood-brain barrier; lactate receptors; exercise and lactateblood-brain barrier
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Cerebrospinal fluid glucose and lactate concentrations after traumatic brain injury

2021

El estudio del metabolismo cerebral mediante el uso de la microdiálisis en el paciente con traumatismo craneoencefálico (TCE) ha puesto de manifiesto que un descenso a nivel cerebral en la concentración de glucosa o un ascenso de los niveles de lactato son marcadores de alteraciones en el metabolismo cerebral. Aun así, la microdiálisis no es una técnica de uso rutinario en la práctica clínica habitual en el momento actual y existen pocos datos y estudios acerca de las concentraciones de glucosa y lactato en líquido cefalorraquídeo (LCR) de estos pacientes. Este estudio analiza la relación entre los niveles de glucosa y lactato en LCR y el pronóstico de los pacientes tras el TCE. In patients…

lactatemetabolismo cerebralbrain metabolismceerebrospinal fluidpronóstico tceUNESCO::CIENCIAS MÉDICAStraumatismo craneoencefálicotce:CIENCIAS MÉDICAS [UNESCO]glucosalactato
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Healthy mitochondria for stroke cells.

2018

Stroke is a debilitating disease that remains as a significant unmet need. Although our understanding of the disease pathology has advanced over the years, treatment options for stroke are limited. Recent studies have implicated the important role of healthy mitochondria in neuroprotection against stroke. Under the stroke pathological condition, transfer of healthy mitochondria is observed from astrocytes to ischemic neurons. However, without additional therapeutic intervention, such astrocyte-to-neuron transfer of mitochondria may not sufficiently afford a robust and stable therapeutic effect against the devastating primary insult and progressive neurodegeneration associated with stroke. W…

lcsh:Diseases of the circulatory (Cardiovascular) systemAginglcsh:Medical technologyneuronsReview ArticleDiseaseMitochondrion010402 general chemistryBioinformatics01 natural sciencesNeuroprotectionstem cellsmedicineStrokePathological010405 organic chemistrybusiness.industryNeurosciencesGeneral Medicinemedicine.diseaseStem Cell Researchstrokeneuronendothelial cells0104 chemical sciencesReview articleBrain Disordersstem cellTransplantationmitochondriaStrokelcsh:R855-855.5lcsh:RC666-701AstrocytesNeurologicalendothelial cellStem cellAstrocytebusiness
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Responsiveness to anti-PD-1 and anti-CTLA-4 immune checkpoint blockade in SB28 and GL261 mouse glioma models.

2018

Immune checkpoint blockade (ICB) is currently evaluated in patients with glioblastoma (GBM), based on encouraging clinical data in other cancers, and results from studies with the methylcholanthrene-induced GL261 mouse glioma. In this paper, we describe a novel model faithfully recapitulating some key human GBM characteristics, including low mutational load, a factor reported as a prognostic indicator of ICB response. Consistent with this observation, SB28 is completely resistant to ICB, contrasting with treatment sensitivity of the more highly mutated GL261. Moreover, SB28 shows features of a poorly immunogenic tumor, with low MHC-I expression and modest CD8(+) T-cell infiltration, suggest…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentGL261ImmunologyOncology and CarcinogenesisMajor histocompatibility complexMalignancylcsh:RC254-282Mutational loadVaccine Related03 medical and health sciences0302 clinical medicineRare DiseasesGliomamedicineImmunology and Allergyddc:576.5sb28mutational loadCancerddc:616biologybusiness.industryBrief ReportSB28glioblastomaNeurosciencesImmunotherapyimmune checkpoint blockadelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseImmune checkpointBlockadeBrain DisordersBrain Cancer030104 developmental biologyOncologygl261030220 oncology & carcinogenesisCancer researchbiology.proteinImmunizationlcsh:RC581-607businessGlioblastomaCD8Immune checkpoint blockadeGlioblastoma
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Interleukin 10 restores lipopolysaccharide-induced alterations in synaptic plasticity probed by repetitive magnetic stimulation

2020

Systemic inflammation is associated with alterations in complex brain functions such as learning and memory. However, diagnostic approaches to functionally assess and quantify inflammation-associated alterations in synaptic plasticity are not well-established. In previous work, we demonstrated that bacterial lipopolysaccharide (LPS)-induced systemic inflammation alters the ability of hippocampal neurons to express synaptic plasticity, i.e., the long-term potentiation (LTP) of excitatory neurotransmission. Here, we tested whether synaptic plasticity induced by repetitive magnetic stimulation (rMS), a non-invasive brain stimulation technique used in clinical practice, is affected by LPS-induc…

lcsh:Immunologic diseases. AllergyLipopolysaccharides0301 basic medicinenon-invasive brain stimulationInterleukin-1betaImmunologyTNFα-reporter mouseMice TransgenicStimulationNeurotransmissionHippocampusSynaptic TransmissionneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineGenes Reportertranscranial magnetic stimulationAnimalsImmunology and Allergyddc:610NeuroinflammationOriginal ResearchInflammationNeuronsNeuronal Plasticitysynaptic plasticityInterleukin-6Tumor Necrosis Factor-alphaChemistryLong-term potentiationInterleukin-10Mice Inbred C57BLOrganoids030104 developmental biologyBrain stimulationSynaptic plasticityExcitatory postsynaptic potentialTumor necrosis factor alphaMicrogliainterleukin 10lcsh:RC581-607Neuroscience030217 neurology & neurosurgery
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The NG2 Proteoglycan Protects Oligodendrocyte Precursor Cells against Oxidative Stress via Interaction with OMI/HtrA2.

2015

The NG2 proteoglycan is characteristically expressed by oligodendrocyte progenitor cells (OPC) and also by aggressive brain tumours highly resistant to chemo- and radiation therapy. Oligodendrocyte-lineage cells are particularly sensitive to stress resulting in cell death in white matter after hypoxic or ischemic insults of premature infants and destruction of OPC in some types of Multiple Sclerosis lesions. Here we show that the NG2 proteoglycan binds OMI/HtrA2, a mitochondrial serine protease which is released from damaged mitochondria into the cytosol in response to stress. In the cytosol, OMI/HtrA2 initiates apoptosis by proteolytic degradation of anti-apoptotic factors. OPC in which NG…

lcsh:MedicineApoptosisdrug effects [Cytosol]HTRA2 protein humangenetics [RNA Small Interfering]genetics [Serine Endopeptidases]genetics [Glioblastoma]570 Life sciencespathology [Glioblastoma]MiceCytosolCerebellumpathology [Cerebellum]RNA Small Interferinglcsh:Sciencemetabolism [Antigens]Mice Knockoutchondroitin sulfate proteoglycan 4metabolism [Proteoglycans]Brain NeoplasmsSerine Endopeptidasesdrug effects [Mitochondria]metabolism [Cerebellum]High-Temperature Requirement A Serine Peptidase 2Mitochondriametabolism [Brain Neoplasms]Gene Expression Regulation Neoplasticpharmacology [Antibodies Neutralizing]genetics [Mitochondrial Proteins]Proteoglycans570 BiowissenschaftenResearch ArticleProtein BindingSignal Transductionpathology [Brain Neoplasms]Primary Cell Culturedrug effects [Cerebellum]drug effects [Apoptosis]metabolism [Mitochondrial Proteins]Mitochondrial Proteinsantagonists & inhibitors [Proteoglycans]pharmacology [Hydrogen Peroxide]genetics [Antigens]Cell Line Tumormetabolism [Serine Endopeptidases]AnimalsHumansddc:610metabolism [RNA Small Interfering]Antigenslcsh:RHtra2 protein mouseHydrogen Peroxidemetabolism [Mitochondria]Antibodies Neutralizinggenetics [Proteoglycans]genetics [Brain Neoplasms]Mice Inbred C57BLOxidative Stressnervous systemlcsh:Qmetabolism [Cytosol]Glioblastomametabolism [Glioblastoma]
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Mismatch negativity (MMN) in freely-moving rats with several experimental controls.

2014

Mismatch negativity (MMN) is a scalp-recorded electrical potential that occurs in humans in response to an auditory stimulus that defies previously established patterns of regularity. MMN amplitude is reduced in people with schizophrenia. In this study, we aimed to develop a robust and replicable rat model of MMN, as a platform for a more thorough understanding of the neurobiology underlying MMN. One of the major concerns for animal models of MMN is whether the rodent brain is capable of producing a human-like MMN, which is not a consequence of neural adaptation to repetitive stimuli. We therefore tested several methods that have been used to control for adaptation and differential exogenou…

lcsh:MedicineMismatch negativityNeurophysiologyBiologyStimulus (physiology)ElectroencephalographyAuditory cortexResearch and Analysis Methodsbehavioral disciplines and activitiesModel OrganismsEvent-related potentialmedicineAnimalsHumanslcsh:ScienceOddball paradigmta515Auditory CortexBrain MappingMultidisciplinarymedicine.diagnostic_testlcsh:RNeural adaptationSkullBiology and Life SciencesElectroencephalographyAnimal ModelsSensory SystemsFrontal LobeRatsmedicine.anatomical_structureFrontal lobeAcoustic StimulationBrain ElectrophysiologyAuditory SystemModels AnimalEvoked Potentials Auditorylcsh:QNeuroscienceResearch ArticleNeurosciencePloS one
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FETAL GROWTH RESTRICTION: A GROWTH PATTERN WITH FETAL, NEONATAL AND LONG-TERM CONSEQUENCES

2019

Fetal growth restriction (FGR) or intrauterine growth restriction (IUGR) are the terms used for a fetus which has not attained its full growth potential for gestational age. FGR is a multifactorial syndrome responsible for increased fetal and neonatal morbidity and mortality as well as long term adverse outcomes involving auxological, metabolic, organic and functional domains. Clinicians distinguish early and late onset FGR, in relation to specific fetal anthropometric parameters related to the possible primary etiology and to different patterns of placental and maternal cardiovascular pathologies. Delivery of an early onset FGR or growth impaired newborn with congenital pathology should be…

lcsh:R5-920fetal programmingFetal growth restrictionponderal indextwinsdevelopmental impairmentlcsh:Medicine (General)brain sparingEuromediterranean Biomedical Journal
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Serratia marcescens infection or hypoxic-ischaemic encephalopathy in neonates: Is magnetic resonance imaging a problem-solving tool?

2017

To the Editor: We read with great interest the retrospective case series by A Madide and J Smith,[1] describing brain ultrasound (US) findings of neonates with Serratia marcescens hospital-acquired infections. S. marcescens is a Gram-negative organism that frequently colonises neonatal intensive care units (NICUs). This bacterium can cause severe brain infections in neonates, with irreversible neurological damage and long-term neurodevelopmental impairment. The authors conclude that in babies with acute onset of the illness, US scans allow doctors to highlight pathological changes in the brain immediately before the microbiological diagnosis of infection, and to follow the evolution of thos…

lcsh:R5-920medicine.medical_specialtymedicine.diagnostic_testbiologybusiness.industrylcsh:Rlcsh:MedicineMagnetic resonance imagingGeneral Medicinebiology.organism_classificationHypoxic ischaemic encephalopathyAcute onsetNeurological DamageIntensive careBrain ultrasoundSerratia marcescensmedicinelcsh:Medicine (General)Intensive care medicinebusinessPathologicalSouth African medical journal = Suid-Afrikaanse tydskrif vir geneeskunde
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RNA-Binding Proteins as Epigenetic Regulators of Brain Functions and Their Involvement in Neurodegeneration.

2022

A central aspect of nervous system development and function is the post-transcriptional regulation of mRNA fate, which implies time- and site-dependent translation, in response to cues originating from cell-to-cell crosstalk. Such events are fundamental for the establishment of brain cell asymmetry, as well as of long-lasting modifications of synapses (long-term potentiation: LTP), responsible for learning, memory, and higher cognitive functions. Post-transcriptional regulation is in turn dependent on RNA-binding proteins that, by recognizing and binding brief RNA sequences, base modifications, or secondary/tertiary structures, are able to control maturation, localization, stability, and tr…

learningsynaptic plasticityOrganic ChemistryneurodegenerationRNA-Binding ProteinsBrainGeneral MedicineCatalysisComputer Science ApplicationsmemoryInorganic ChemistryIntrinsically Disordered ProteinsGene Expression RegulationSettore BIO/10 - BiochimicaRNA-binding proteins (RBPs)Settore MED/26 - NeurologiaNervous System Physiological PhenomenaRNA Messengerpost-transcriptional regulation of gene expressionSettore BIO/06 - Anatomia Comparata E CitologiaPhysical and Theoretical ChemistryEVsMolecular Biologyintrinsically disordered regions (IDRs)SpectroscopyInternational journal of molecular sciences
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