Search results for "cachexia"

showing 10 items of 51 documents

Cancer-Related Cachexia: The Vicious Circle between Inflammatory Cytokines, Skeletal Muscle, Lipid Metabolism and the Possible Role of Physical Train…

2022

Cachexia is a multifactorial and multi-organ syndrome that is a major cause of morbidity and mortality in late-stage chronic diseases. The main clinical features of cancer-related cachexia are chronic inflammation, wasting of skeletal muscle and adipose tissue, insulin resistance, anorexia, and impaired myogenesis. A multimodal treatment has been suggested to approach the multifactorial genesis of cachexia. In this context, physical exercise has been found to have a general effect on maintaining homeostasis in a healthy life, involving multiple organs and their metabolism. The purpose of this review is to present the evidence for the relationship between inflammatory cytokines, skeletal mus…

CachexiaInflammatory cytokineOrganic ChemistrySkeletal muscleGeneral MedicineCancer‐related cachexiaLipid MetabolismCatalysisComputer Science ApplicationsInorganic Chemistrycancer-related cachexiaPhysical trainingNeoplasmsQuality of LifeCytokinesHumansPhysical and Theoretical ChemistryMuscle SkeletalMolecular BiologyExerciseSpectroscopyInternational journal of molecular sciences
researchProduct

Cachexia: a therapeutic approach beyond cytokine antagonism

2002

Cachexia is seen in a number of chronic diseases, and it is always associated with a poor prognosis. Irrespective of etiology, the development of cachexia appears to share a common pathophysiological pathway. This includes induction of proteasome-dependent myofibril-degradation, which is thought to be secondary to stimulation by enhanced levels of pro-inflammatory cytokines. Elevation of tumor necrosis factor-alpha (TNFalpha) and other plasma cytokines has been demonstrated in many conditions associated with cachexia. Despite improved pathophysiological understanding, a specific treatment for cachexia has not yet been established. Whilst direct TNFalpha antagonism has therapeutic appeal, th…

CachexiaTumor Necrosis Factor-alphabusiness.industrymedicine.medical_treatmentNF-kappa BImmunosuppressionNF-κBmedicine.diseaseCachexiaTranscription Factor AP-1PathogenesisTherapeutic approachchemistry.chemical_compoundTreatment OutcomeCytokinechemistryImmunologyCytokinesHumansMedicineTumor necrosis factor alphaCardiology and Cardiovascular MedicineAntagonismbusinessImmunosuppressive AgentsInternational Journal of Cardiology
researchProduct

Activin-A, myostatin and interleukin-6 in cancer associated cachexia

2017

Cachexia is a muscle wasting condition associated with multiple different chronic illnesses, such as cancer, diabetes and AIDS. In cancer, approximately 80% of patients with advanced disease have symptoms of muscle wasting, and around 25% of cancer mortality concerns cachexia. Elevated serum levels of different cytokines and TGF-β protein family members, such as Interleukin-6, Myostatin and Activin-A, have been observed in cachetic patients and test animals. However, the mechanistic role and the relative contribution of these molecules to muscle loss in the syndrome have not yet been fully elucidated. In this thesis, the gene-expression levels of Activin-A, Myostatin and Interleukin-6 was a…

CachexiamyostatiiniInterleukin-6interleukiinitaktiviini-aActivin-AsyöpätauditkakeksiaC2C12proteiinitMyostatinmusculoskeletal systemhormones hormone substitutes and hormone antagonists
researchProduct

Host-related factors and cancer: Malnutrition and non-Hodgkin lymphoma

2022

Assessment of host-related factors is a crucial aspect in the comprehensive management of cancer patients. A distinct nutritional disturbance linked to cancer has been recognized to be associated with negative outcomes. However, compared to solid tumors, only a limited number of studies have looked specifically at nutritional issues in the field of lymphoma. The aim of this review is to integrate the current knowledge on interactions between malnutrition and lymphoma and address most relevant and pertinent questions. We first provide a literature review on the mutual biological relationship between malnutrition and lymphoma. Next, we explore the overlap between malnutrition, sarcopenia, cac…

Cancer ResearchSarcopeniaCachexiaFrailtyLymphoma Non-Hodgkinnon-Hodgkin lymphomaMalnutritionHematologyGeneral MedicineDiffuse large B-cell lymphomaOncologyNutritional statusNeoplasmsHumansCancer metabolic syndrome
researchProduct

Role of different endurance training programs on cancer cachexia:pointing particular attention to the gender and age differences Macaluso

2013

Evidence from recent publications indicates that repeated exercise may enhance the quality of life of cancer patients (Maddocks et al., 2012). Regular physical activity may attenuate the adverse effects of cancer therapy, prevent or reverse cachexia and improve survival, although not all the patients are able or willing to undertake programs currently being offered. The aims of this study were to analyze: i) the effects of a progressive endurance exercise (progressive Training, pTR) on survival and cachexia in sedentary (SED) mice inoculated (I) with a fresh fragment of solid C26 tumor [SED-I-pTR; SED-I-SED]; ii) the effect of different protocols of endurance exercise (Trained for 30 min, T…

Endurance Training cancer cachexia
researchProduct

New Molecular Targets and Lifestyle Interventions to Delay Aging Sarcopenia

2014

The term sarcopenia was originally created to refer age-related loss of muscle mass with consequent loss of strength (Morley et al., 2001). There are now four international definitions of sarcopenia (Cruz-Jentoft et al., 2010; Muscaritoli et al., 2010; Morley et al., 2011). In essence they all agree, requiring a measure of walking capability [either low gait speed or a limited endurance (distance) in a 6-min walk], together with an appendicular lean mass of <2 SDs of a sex and ethnically corrected normal level for individuals 20–30 years old. Sarcopenia is a prevalent health problem among the elderly. On average, 5–13 and 11–50% of people aged 60−70 years and ≥80 years, respectively suffer …

Gerontologymuscle atrophyAgingmedicine.medical_specialtySignaling pathwaysCognitive NeuroscienceFrailty syndromeEnvejecimientoSaludfrailtyCachexialcsh:RC321-571Internal medicinemedicineSenescence factorsPharmacological Targetslcsh:Neurosciences. Biological psychiatry. Neuropsychiatrybusiness.industryPublic healthTestosterone (patch)Opinion Articlemedicine.diseaseMuscle atrophysignaling pathwayssenescence factorsSarcopeniaLean body massMolecular targetsMuscle atrophymedicine.symptombusinesspharmaceutical targetsNeuroscienceFrontiers in Aging Neuroscience
researchProduct

Prospective randomised trial of two dose levels of megestrol acetate in the management of anorexia-cachexia syndrome in patients with metastatic canc…

1996

Two doses of megestrol acetate (MA) have been prospectively compared in a random fashion as treatment for cancer-related anorexia-cachexia syndrome (ACS) in 122 patients with progressive soft tissue sarcoma, colorectal, lung, head and neck and renal cancer resistant to systemic chemotherapy. After 30 days of MA, 55% of patients receiving MA at 160 mg day-1 reported an increase in appetite, 27% of patients no variation and 18% complained of a decrease in appetite. Patients treated with MA at 320 mg day-1 reported an increase in appetite in 68% of cases, a stabilisation in 20% of cases and a decrease in 12%. Although an increase in appetite was more frequently observed in patients receiving M…

MaleCancer Researchmedicine.medical_specialtyCachexiamedia_common.quotation_subjectAppetiteAnorexiaGastroenterologyCachexiachemistry.chemical_compoundWeight lossNeoplasmsInternal medicinemedicineHumansProspective StudiesNeoplasm MetastasisProspective cohort studyAgedmedia_commonDose-Response Relationship DrugPerformance statusbusiness.industryMegestrol AcetateBody WeightAppetiteMegestrolSyndromeMiddle Agedmedicine.diseaseAnorexiaSurgeryOncologychemistryMegestrolMegestrol acetateFemalemedicine.symptombusinessResearch Articlemedicine.drugBritish Journal of Cancer
researchProduct

Muscle NAD+ depletion and Serpina3n as molecular determinants of murine cancer cachexia—the effects of blocking myostatin and activins

2020

Objective Cancer cachexia and muscle loss are associated with increased morbidity and mortality. In preclinical animal models, blocking activin receptor (ACVR) ligands has improved survival and prevented muscle wasting in cancer cachexia without an effect on tumour growth. However, the underlying mechanisms are poorly understood. This study aimed to identify cancer cachexia and soluble ACVR (sACVR) administration-evoked changes in muscle proteome. Methods Healthy and C26 tumour-bearing (TB) mice were treated with recombinant sACVR. The sACVR or PBS control were administered either prior to the tumour formation or by continued administration before and after tumour formation. Muscles were an…

MaleEXPRESSIONActivin receptor; APR; C26; Cancer cachexia; Nrk2; OXPHOSlcsh:Internal medicineCachexiaREVERSALActivin ReceptorsMETABOLISMactivin receptorOxidative PhosphorylationCell Line TumorAnimalsMuscle Skeletallcsh:RC31-1245aineenvaihduntaSerpinslihassolut318 Medical biotechnologyNrk2Cancer cachexiaMyostatinNADOXPHOSMUSCULAR-DYSTROPHYActivinsMitochondriaActivin receptorDisease Models AnimalMuscular AtrophyMICESIRTUINSOriginal ArticlesyöpätauditproteiinitC26lihassurkastumasairaudetAPRAcute-Phase Proteinscancer cachexia
researchProduct

Treating cachexia using soluble ACVR2B improves survival, alters mTOR localization, and attenuates liver and spleen responses.

2018

Background Cancer cachexia increases morbidity and mortality, and blocking of activin receptor ligands has improved survival in experimental cancer. However, the underlying mechanisms have not yet been fully uncovered. Methods The effects of blocking activin receptor type 2 (ACVR2) ligands on both muscle and non‐muscle tissues were investigated in a preclinical model of cancer cachexia using a recombinant soluble ACVR2B (sACVR2B‐Fc). Treatment with sACVR2B‐Fc was applied either only before the tumour formation or with continued treatment both before and after tumour formation. The potential roles of muscle and non‐muscle tissues in cancer cachexia were investigated in order to understand th…

MaleTUMOR-BEARING MICElcsh:Diseases of the musculoskeletal systemCachexiaprotein synthesisActivin Receptors Type IIMDSCphysical activityAcute phase responseKaplan-Meier EstimateACTIVATIONActivinMiceNeoplasmsOrthopedics and Sports MedicineTOR Serine-Threonine Kinasesactivinlcsh:Human anatomyII RECEPTORSRecombinant ProteinsProtein TransportLivermyostatinPROTEIN-SYNTHESISSKELETAL-MUSCLECytokinessyöpätauditInflammation MediatorsACUTE-PHASE RESPONSE3122 CancersINHIBITIONlcsh:QM1-695acute phase responsePhysiology (medical)Cell Line TumorAnimalsHumansMuscle SkeletalActivin; Acute phase response; MDSC; Myostatin; Physical activity; Protein synthesis; Orthopedics and Sports Medicine; Physiology (medical)Physical activityMyeloid-Derived Suppressor CellsMyostatinXenograft Model Antitumor AssaysDisease Models AnimalACTIVIN-APHYSICAL-ACTIVITY3121 General medicine internal medicine and other clinical medicineproteiinitEXPERIMENTAL CANCER CACHEXIAlcsh:RC925-935Protein synthesislihassurkastumasairaudetBiomarkersSpleenJournal of cachexia, sarcopenia and muscle
researchProduct

Hypothalamic eIF2 alpha signaling regulates food intake

2014

International audience; The reversible phosphorylation of the a subunit of eukaryotic initiation factor 2 (eIF2 alpha) is a highly conserved signal implicated in the cellular adaptation to numerous stresses such as the one caused by amino acid limitation. In response to dietary amino acid deficiency, the brain-specific activation of the eIF2 alpha kinase GCN2 leads to food intake inhibition. We report here that GCN2 is rapidly activated in the mediobasal hypothalamus (MBH) after consumption of a leucine-deficient diet. Furthermore, knockdown of GCN2 in this particular area shows that MBH GCN2 activity controls the onset of the aversive response. Importantly, pharmacological experiments demo…

Male[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEukaryotic Initiation Factor-2neuronsEatingMicepiriform cortex0302 clinical medicineGene Knockdown Techniquesarcuate nucleusamino-acid deficiency;arcuate nucleus;translational control;energy homeostasis;piriform cortex;cancer cachexia;protein-intake;transfer-rna;mechanism;neuronsPhosphorylationlcsh:QH301-705.52. Zero hungerchemistry.chemical_classification0303 health sciencesGene knockdownalimentationtranslational controlamino-acid deficiencyEukaryotic Initiation Factor-2Amino acidtransfer-rnaGene Knockdown TechniquesAlimentation et NutritionPhosphorylation[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Signal transductionmedicine.symptomSignal Transductioncancer cachexiamedicine.medical_specialtyCellular adaptationHypothalamusmechanismAnorexiaBiologyProtein Serine-Threonine KinasesGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health sciencesLeucineInternal medicinemedicineFood and NutritionAnimalsenergy homeostasis030304 developmental biologyNeurosciencesArcuate Nucleus of Hypothalamusprotein-intakeMice Inbred C57BL[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologychemistrylcsh:Biology (General)Neurons and Cognition[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
researchProduct