Search results for "campus"

showing 10 items of 667 documents

Impaired hippocampal neuroligin-2 function by chronic stress or synthetic peptide treatment is linked to social deficits and increased aggression.

2014

Neuroligins (NLGNs) are cell adhesion molecules that are important for proper synaptic formation and functioning, and are critical regulators of the balance between neural excitation/inhibition (E/I). Mutations in NLGNs have been linked to psychiatric disorders in humans involving social dysfunction and are related to similar abnormalities in animal models. Chronic stress increases the likelihood for affective disorders and has been shown to induce changes in neural structure and function in different brain regions, with the hippocampus being highly vulnerable to stress. Previous studies have shown evidence of chronic stress-induced changes in the neural E/I balance in the hippocampus. Ther…

MaleRestraint PhysicalhippocampusmoodCell Adhesion Molecules NeuronalNeurexinstress disordersHippocampusPoison controlNeuroliginNerve Tissue ProteinsReceptors Cell Surfacebehavioral scienceHippocampal formationneuropharmacologyHippocampussocial behaviorRats Sprague-DawleystressmedicineNeuritesAnimalsChronic stressRats WistarSocial BehaviorCells CulturedPharmacologyNeuronsAggressionaggressionneuropeptideschronic restraint stressOrgan SizeanxietyRatsAggressionsociabilityPsychiatry and Mental healthChronic DiseaseOriginal Articleneuroliginmedicine.symptomPsychologyCorticosteronePeptidesNeuroscienceStress PsychologicalSocial behaviorNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Chronic restraint stress and chronic corticosterone treatment modulate differentially the expression of molecules related to structural plasticity in…

2004

Stress and stress-related hormones induce structural changes in neurons of the adult CNS. Neurons in the hippocampus, the amygdala and the prefrontal cortex undergo neurite remodeling after chronic stress. In the hippocampus some of these effects can be mimicked with chronic administration of adrenal steroids. These changes in neuronal structure may be mediated by certain molecules related to plastic events such as the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). The expression of PSA-NCAM persists in the adult hippocampus and it is up-regulated after chronic stress. The piriform cortex also displays considerable levels of PSA-NCAM during adulthood and indirect evide…

MaleRestraint Physicalmedicine.medical_specialtyDoublecortin ProteinHippocampusNeural Cell Adhesion Molecule L1AmygdalaRats Sprague-Dawleychemistry.chemical_compoundCorticosteroneStress PhysiologicalInternal medicinePiriform cortexmedicineAnimalsChronic stressOlfactory memoryPrefrontal cortexCerebral CortexNeuronal PlasticitybiologyGeneral NeuroscienceDoublecortinRatsmedicine.anatomical_structureEndocrinologynervous systemchemistryGene Expression RegulationChronic Diseasebiology.proteinSialic AcidsCorticosteroneNeuroscienceNeuroscience
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Role(s) of the 5-HT2C receptor in the development of maximal dentate activation in the hippocampus of anesthetized rats.

2014

Aims: Substantial evidence indicates that 5-HT2C receptors are involved in the control of neuronal network excitability and in seizure pathophysiology. Here, we have addressed the relatively unexplored relationship between temporal lobe epilepsy (TLE), the most frequent type of intractable epilepsy, and 5-HT2CRs. Methods: In this study, we investigated this issue using a model of partial complex (limbic) seizures in urethane-anesthetized rat, based on the phenomenon of maximal dentate activation (MDA) using 5-HT2C compounds, electrophysiology, immunohistochemistry, and western blotting techniques. Results: The 5-HT2C agonists mCPP (1 mg/kg, i.p) and lorcaserin (3 mg/kg, i.p), but not RO60-0…

MaleSerotonin receptorsmedicine.medical_specialtyGlutamate decarboxylaseDentate gyrus; Depression; GABA; Memory; Serotonergic2c drugs; Serotonin receptors; Temporal lobe epilepsyHippocampusHippocampal formationDentate gyruEpileptogenesisSettore BIO/09 - FisiologiaHippocampusDentate gyrus; Depression; GABA; Memory;LorcaserinRats Sprague-DawleySerotonergic 2c drugGABAEpilepsyMemoryDentate gyrus; Depression; GABA; Memory; Serotonin receptors; Temporal lobe epilepsyPhysiology (medical)Internal medicinemedicineReceptor Serotonin 5-HT2CAnimalsDentate gyrusPharmacology (medical)Serotonin receptorTemporal lobe epilepsyPharmacologyDepressionChemistryDentate gyrusDentate gyrus;Depression; GABA; Memory; Serotonergic2c drugs; Serotonin receptors; Temporal lobe epilepsyOriginal Articlesmedicine.diseaseRats5-HT2C receptorPsychiatry and Mental healthEndocrinologyDepression MentalEpilepsy Temporal LobeDentate GyrusSerotonergic2c drugAnesthetics IntravenousSerotonin 5-HT2 Receptor Agonistsmedicine.drug
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Acute behavioural and neurotoxic effects of MDMA plus cocaine in adolescent mice.

2008

The poly-drug pattern is the most common among those observed in MDMA users, with cocaine being a frequently associated drug. This study evaluates the acute effects of MDMA (5, 10 and 20 mg/kg), alone or in combination with cocaine (25 mg/kg), on motor activity, anxiety (elevated plus maze and social interaction test), memory and brain monoamines in adolescent mice, Both drugs, administered alone or concurrently, produced hyperactivity and a decrease in social contacts. However, an anxiolytic effect, studied by means of the elevated plus maze and expressed as an increase in the time spent on the open arms, was observed only in those animals treated with cocaine and MDMA. The passive avoidan…

MaleSerotoninElevated plus mazeMDMAmedicine.drug_classDopamineN-Methyl-34-methylenedioxyamphetamineStriatumPharmacologyAnxietyMotor ActivityToxicologyAnxiolyticHippocampusCellular and Molecular NeuroscienceMiceSerotonin AgentsDevelopmental NeuroscienceCocaineDopaminemental disordersmedicineAvoidance LearningAnimalsBiogenic MonoaminesInterpersonal RelationsBrain ChemistryCerebral CortexBehavior AnimalMDMACortex (botany)NeostriatumSocial behaviourAnxietyNeurotoxicity SyndromesSerotoninmedicine.symptomElevated plus mazePsychologypsychological phenomena and processesmedicine.drugNeurotoxicology and teratology
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Lateral habenula and hippocampal units: electrophysiological and iontophoretic study

1995

In previous works we studied, on cats, the effects of lateral habenula (LH) stimulation on hippocampal units. In particular, the results showed an excitation or an inhibition in relation to the stimulation frequency (0.5-3.0 Hz or 5.0-20 Hz, respectively). All the LH stimulation effects were antagonised by iontophoretic intrahippocampal application of methysergide (MS). In this series of experiments it was possible to demonstrate, on rats, that LH stimulation causes an excitatory effect in a major number of hippocampal units in relation to the frequency increase. The inhibitory effect by iontophoretic serotonine application and the reversible blockade of habenular modulation after iontophor…

MaleSerotoninN-MethylaspartateMethysergideStimulationHippocampal formationHippocampusMicromanipulationDorsal raphe nucleusThalamusmedicineAnimalsRats WistarNeuronsAnalysis of VarianceRapheMethysergideChemistryGeneral NeuroscienceIontophoresisElectric StimulationRatsHabenulaExcitatory postsynaptic potentialRaphe NucleiRaphe nucleiNeurosciencemedicine.drugBrain Research Bulletin
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Electrophysiological and Iontophoretic Aspects of the Habenular Influence on Hippocampal Neurones

1995

In previous experimental studies, carried out on cats, we demonstrated that electrical stimulation of lateral habenula (LH) at 0.5-3.0 Hz or 5-20 Hz had a double effect (low frequency-excitation; high frequency-inhibition) on the spontaneous firing rate of single hippocampal neurones. Our results, in agreement with similar case studies, allowed us to hypothesise that in the habenular modulation of the hippocampus the raphe nucleus is probably involved. In fact, all the effects of LH stimulation were antagonised by the iontophoretic intrahippocampal application of methysergide. In the present series of experiments, performed on rats, it was possible to demonstrate that LH stimulation at 1-10…

MaleSerotoninN-MethylaspartatePhysiologyMethysergideHippocampusStimulationHippocampal formationHippocampusPhysiology (medical)medicineAnimalsRats WistarNeuronsIontophoresisMethysergideChemistryGeneral MedicineIontophoresisElectric StimulationCochleaRatsElectrophysiologyElectrophysiologyRaphe NucleiNMDA receptorRaphe nucleiNeurosciencemedicine.drugArchives of Physiology and Biochemistry
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Adolescent pre-exposure to ethanol or MDMA prolongs the conditioned rewarding effects of MDMA

2011

Adolescents often take ethanol (EtOH) in combination with MDMA (3,4-methylenedioxymethylamphetamine). In the present work we studied the effect of repeated intermittent adolescent pre-exposure to both drugs on the behavioral and neurochemical effects of MDMA in mice. Sixteen days after pre-treatment, the rewarding and reinstating effects of MDMA in the conditioned place preference (CPP) paradigm were evaluated, along with the levels of biogenic amines, basal motor activity and corticosterone response to different challenges. Pre-exposure to EtOH, MDMA or EtOH+MDMA did not affect the CPP induced by 10mg/kg of MDMA. However, adolescent exposure to EtOH or MDMA increased the duration of the co…

MaleSerotoninmedicine.medical_specialtyDopamineN-Methyl-34-methylenedioxyamphetaminePoison controlExperimental and Cognitive PsychologyStriatumMotor ActivityChoice BehaviorHippocampusDrug Administration ScheduleExtinction PsychologicalMiceBehavioral Neurosciencechemistry.chemical_compoundNeurochemicalRewardCorticosteroneInternal medicineConditioning Psychologicalmental disordersAnimals Outbred StrainsmedicineAnimalsDrug InteractionsCerebral CortexEthanolIllicit DrugsMDMAExtinction (psychology)Hydroxyindoleacetic AcidCorpus StriatumConditioned place preferenceMonoamine neurotransmitterEndocrinologychemistryAnesthesia34-Dihydroxyphenylacetic AcidCorticosteronePsychologypsychological phenomena and processesmedicine.drugPhysiology & Behavior
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Effect of intermittent exposure to ethanol and MDMA during adolescence on learning and memory in adult mice

2012

Abstract Background Heavy binge drinking is increasingly frequent among adolescents, and consumption of 3,4-methylenedioxymethamphetamine (MDMA) is often combined with ethanol (EtOH). The long-lasting effects of intermittent exposure to EtOH and MDMA during adolescence on learning and memory were evaluated in adult mice using the Hebb-Williams maze. Methods Adolescent OF1 mice were exposed to EtOH (1.25 g/kg) on two consecutive days at 48-h intervals over a 14-day period (from PD 29 to 42). MDMA (10 or 20 mg/kg) was injected twice daily at 4-h intervals over two consecutive days, and this schedule was repeated six days later (PD 33, 34, 41 and 42), resulting in a total of eight injections. …

MaleSerotoninmedicine.medical_specialtyMDMA34-Dihydroxyphenylacetic acidDopamineN-Methyl-34-methylenedioxyamphetamineCognitive NeuroscienceBinge drinkingStriatumHippocampuslcsh:RC346-429MiceBehavioral Neurosciencechemistry.chemical_compoundSerotonin AgentsMemoryDopamineSerotonin AgentsInternal medicinemental disordersmedicineAnimalsLearningHippocampus (mythology)Maze Learninglcsh:Neurology. Diseases of the nervous systemBiological PsychiatryBehavior AnimalEthanolResearchMDMAGeneral MedicineHydroxyindoleacetic AcidCorpus StriatumEndocrinologychemistryAnesthesia34-Dihydroxyphenylacetic AcidHebb Williams mazeSerotoninPsychologypsychological phenomena and processesmedicine.drugBehavioral and Brain Functions
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Presynaptic effects of anandamide and WIN55,212-2 on glutamatergic nerve endings isolated from rat hippocampus

2006

We examined the effects of the endocannabinoide-anandamide (AEA), the synthetic cannabinoid, WIN55,212-2, and the active phorbol ester, 4-beta-phorbol 12-myristate 13-acetate (4-beta-PMA), on the release of [(3)H]d-Aspartate ([(3)H]d-ASP) from rat hippocampal synaptosomes. Release was evoked with three different stimuli: (1) KCl-induced membrane depolarization, which activates voltage-dependent Ca(2+) channels and causes limited neurotransmitter exocytosis, presumably from ready-releasable vesicles docked in the active zone; (2) exposure to the Ca(2+) ionophore-A23187, which causes more extensive transmitter release, presumably from intracellular reserve vesicles; and (3) K(+) channel block…

MaleSettore BIO/14 - FARMACOLOGIAPolyunsaturated AlkamideshippocampusMorpholinesmedicine.medical_treatmentPresynaptic TerminalsArachidonic AcidsNaphthalenesExocytosisCellular and Molecular Neurosciencechemistry.chemical_compoundGlutamatesglutamate releasemedicineAnimalsanandamideActive zoneRats WistarNeurotransmitterCannabinoidCalcimycinProtein kinase CSynaptosomeArachidonic AcidChemistrysynaptosomesDepolarizationCell BiologyAnandamideHippocampal synaptosomeCalcium Channel BlockersBenzoxazinesRatsBiochemistryBiophysicsTetradecanoylphorbol AcetateCannabinoidCapsaicinEndocannabinoidsNeurochemistry International
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Depletion of polysialic acid from neural cell adhesion molecule (PSA-NCAM) increases CA3 dendritic arborization and increases vulnerability to excito…

2012

Chronic immobilization stress (CIS) shortens apical dendritic trees of CA3 pyramidal neurons in the hippocampus of the male rat, and dendritic length may be a determinant of vulnerability to stress. Expression of the polysialylated form of neural cell adhesion molecule (PSA-NCAM) in the hippocampal formation is increased by stress, while PSA removal by Endo-neuraminidase-N (endo-N) is known to cause the mossy fibers to defasciculate and synapse ectopically in their CA3 target area. We show here that enzymatic removal of PSA produced a remarkable expansion of dendritic arbors of CA3 pyramidal neurons, with a lesser effect in CA1. This expansion eclipsed the CIS-induced shortening of CA3 dend…

MaleSilver StainingKainic acidExcitotoxicityHippocampusBiologyHippocampal formationmedicine.disease_causeReceptors N-Methyl-D-AspartateArticleBody Mass IndexRats Sprague-DawleySynapsechemistry.chemical_compoundDevelopmental NeuroscienceExcitatory Amino Acid AgonistsmedicineAnimalsOrganic ChemicalsReceptorNeural Cell Adhesion MoleculesAnalysis of VarianceKainic AcidPolysialic acidPyramidal CellsMetalloendopeptidasesDendritesFluoresceinsCA3 Region HippocampalRatsCell biologyDisease Models AnimalGene Expression Regulationnervous systemNeurologychemistryNerve DegenerationSialic AcidsNeural cell adhesion moleculeNeuroscienceStress PsychologicalExperimental Neurology
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