Search results for "cancer cell"

showing 10 items of 756 documents

Data set of the protein expression profiles of Luminal A, Claudin-low and overexpressing HER2+ breast cancer cell lines by iTRAQ labelling and tandem…

2015

Breast cancer is the most common and the leading cause of mortality in women worldwide. There is a dire necessity of the identification of novel molecules useful in diagnosis and prognosis. In this work we determined the differentially expression profiles of four breast cancer cell lines compared to a control cell line. We identified 1020 polypeptides labelled with iTRAQ with more than 95% in confidence. We analysed the common proteins in all breast cancer cell lines through IPA software (IPA core and Biomarkers). In addition, we selected the specific overexpressed and subexpressed proteins of the different molecular classes of breast cancer cell lines, and classified them according to prot…

MultidisciplinaryQuantitative proteomicsLuminal aBiologyTandem mass spectrometryBioinformaticsClaudin-Lowmedicine.diseaselcsh:Computer applications to medicine. Medical informaticsProtein expressionBreast cancerBreast cancer cell lineLabellingCancer researchmedicinelcsh:R858-859.7lcsh:Science (General)skin and connective tissue diseaseslcsh:Q1-390Data ArticleData in Brief
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Cancer growth dynamics: stochastic models and noise induced effects

2009

In the framework of the Michaelis‐Menten (MM) reaction kinetics, we analyze the cancer growth dynamics in the presence of the immune response. We found the coexistence of noise enhanced stability (NES) and resonant activation (RA) phenomena which act in an opposite way with respect to the extinction of the tumor. The role of the stochastic resonance (SR) in the case of weak cancer therapy has been analyzed. The evolutionary dynamics of a system of cancerous cells in a model of chronic myeloid leukemia (CML) is investigated by a Monte Carlo approach. We analyzed the effects of a targeted therapy on the evolutionary dynamics of normal, first‐mutant and cancerous cell populations. We show how …

Mutation rateStochastic modellingmedicine.medical_treatmentMyeloid leukemiaCancerStochastic resonance (sensory neurobiology)BiologyBioinformaticsmedicine.diseaseTargeted therapyCancer cellCancer researchmedicineEvolutionary dynamics
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Routes to cell death in animal and plant kingdoms: from classic apoptosis to alternative ways to die—a review

2018

Programmed cell death is fundamental for multicellular organisms either in animal or plant kingdom. Classic apoptosis, which represents the best studied form of cell death, is dependent on caspase protease activity in animals. These proteases are not present in plants, where caspase-like activities, including metacaspases, are involved in the execution of plant cell death. Beyond apoptosis, various non-apoptotic forms of cell death also exist, including autophagy, necroptosis, pyroptosis, and ferroptosis. These types of cell death can be activated independently of apoptosis and sometimes occur when apoptosis is inhibited. Non-apoptotic forms of cell death are best characterized in animals, …

Necroptosi0301 basic medicineProteasesProgrammed cell deathNecroptosisCancer cellAnimal and plant cell death03 medical and health sciencesComparative death pathwaySettore BIO/10 - BiochimicaAutophagySettore BIO/04 - Fisiologia VegetaleCaspaseGeneral Environmental SciencebiologyAutophagyPyroptosiPyroptosisApoptosifood and beveragesCell biologyMulticellular organism030104 developmental biologyApoptosisbiology.proteinGeneral Earth and Planetary SciencesGeneral Agricultural and Biological SciencesFerroptosiRendiconti Lincei. Scienze Fisiche e Naturali
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Extracellular Membrane Vesicles as Vehicles for Brain Cell-to-Cell Interactions in Physiological as well as Pathological Conditions.

2015

Extracellular vesicles are involved in a great variety of physiological events occurring in the nervous system, such as cross talk among neurons and glial cells in synapse development and function, integrated neuronal plasticity, neuronal-glial metabolic exchanges, and synthesis and dynamic renewal of myelin. Many of these EV-mediated processes depend on the exchange of proteins, mRNAs, and noncoding RNAs, including miRNAs, which occurs among glial and neuronal cells. In addition, production and exchange of EVs can be modified under pathological conditions, such as brain cancer and neurodegeneration. Like other cancer cells, brain tumours can use EVs to secrete factors, which allow escaping…

Nervous systemectosomeCelllcsh:MedicineReview ArticleBiologyhorizontal transfer of pathological propertieGeneral Biochemistry Genetics and Molecular BiologySynapseExtracellular VesiclesMyelinextracellular membrane vesicles (EVs); ectosomes; exosomes; brain cancer; neuronal-glial unconventional cross-talk pathways; horizontal transfer of pathological properties; extracellular spreading of protein aggregates.Settore BIO/10 - BiochimicamedicineexosomeHumansSecretionextracellular membrane vesicles (EVs)Settore BIO/06 - Anatomia Comparata E CitologiaTransport Vesiclesbrain cancerNeuronsMembranesNeuronal PlasticityGeneral Immunology and Microbiologylcsh:RNeurodegenerationBrainBiological TransportGeneral Medicinemedicine.diseaseextracellular spreading of protein aggregates.Cell biologyMicroRNAsmedicine.anatomical_structureSynapsesCancer cellNeurogliaNeuroglianeuronal-glial unconventional cross-talk pathway
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Non-local multiscale approach for the impact of go or grow hypothesis on tumour-viruses interactions

2021

International audience; We propose and study computationally a novel non-local multiscale moving boundary mathematical model for tumour and oncolytic virus (OV) interactions when we consider the go or grow hypothesis for cancer dynamics. This spatio-temporal model focuses on two cancer cell phenotypes that can be infected with the OV or remain uninfected, and which can either move in response to the extracellular-matrix (ECM) density or proliferate. The interactions between cancer cells, those among cancer cells and ECM, and those among cells and OV occur at the macroscale. At the micro-scale, we focus on the interactions between cells and matrix degrading enzymes (MDEs) that impact the mov…

Non-local cell adhesion[SDV]Life Sciences [q-bio]Multiscale cancer modellingBiologyMatrix (biology)Models BiologicalVirusMigration-proliferation dichotomyExtracellular matrix03 medical and health sciences0302 clinical medicineNeoplasmsmedicineQA1-939HumansNeoplasm Invasiveness[NLIN]Nonlinear Sciences [physics][MATH]Mathematics [math]030304 developmental biology0303 health sciencesApplied MathematicsCancerGo or grow hypothesisGeneral Medicinemedicine.diseasePhenotypeExtracellular MatrixCell biologyOncolytic virusOncolytic VirusesComputational MathematicsViral replication030220 oncology & carcinogenesisModeling and SimulationTumour-oncolytic viruses interactionsCancer cellOncogenic VirusesGeneral Agricultural and Biological SciencesTP248.13-248.65MathematicsBiotechnology
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Gatekeeper of pluripotency: A common Oct4 transcriptional network operates in mouse eggs and embryonic stem cells

2011

Abstract Background Oct4 is a key factor of an expanded transcriptional network (Oct4-TN) that governs pluripotency and self-renewal in embryonic stem cells (ESCs) and in the inner cell mass from which ESCs are derived. A pending question is whether the establishment of the Oct4-TN initiates during oogenesis or after fertilisation. To this regard, recent evidence has shown that Oct4 controls a poorly known Oct4-TN central to the acquisition of the mouse egg developmental competence. The aim of this study was to investigate the identity and extension of this maternal Oct4-TN, as much as whether its presence is circumscribed to the egg or maintained beyond fertilisation. Results By comparing …

Octamer Transcription Factor-3lcsh:QH426-470lcsh:BiotechnologycellsGene regulatory networkDown-RegulationBiologyTranscriptomeMicelcsh:TP248.13-248.65GeneticsInner cell massAnimalsGene Regulatory NetworksEmbryonic Stem Cellsreproductive and urinary physiologyOligonucleotide Array Sequence AnalysisGeneticsGene Expression ProfilingfungiEmbryoEmbryonic stem cellGene expression profilinglcsh:GeneticsMultigene FamilyCancer cellembryonic structuresOocytesFemalebiological phenomena cell phenomena and immunityFunction and Dysfunction of the Nervous SystemOctamer Transcription Factor-3Research ArticleBiotechnologyBMC Genomics
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Oncogene Addiction in Solid Tumors

2015

The term “oncogenic addiction” refers to the phenomenon by which tumor cells become completely dependent on a single pathway, derived from the activation of a specific oncogene, for their survival and proliferation. The clinical relevance of oncogene addiction paradigm is highlighted by a growing number of examples that demonstrate the efficacy of several therapeutic agents that target specific oncogenes in various cancer types. This chapter aims to summarize the recent evidences concerning the concept of oncogene addiction and describes molecular mechanisms that could explain this phenomenon.

OncogeneCombination therapybusiness.industrymedicine.medical_treatmentCancerOncogenic Addictionmedicine.disease_causeOncogene Addictionmedicine.diseaseTargeted therapyCancer cellCancer researchmedicinebusinessCarcinogenesis
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The Detection of Androgen Receptor Splice Variant 7 in Plasma-derived Exosomal RNA Strongly Predicts Resistance to Hormonal Therapy in Metastatic Pro…

2017

Abstract Background The androgen receptor splice variant 7 (AR-V7) is associated with resistance to hormonal therapy in castration-resistant prostate cancer (CRPC). Due to limitations of the methods available for AR-V7 analysis, the identification of a reliable detection method may facilitate the use of this biomarker in clinical practice. Objective To confirm AR-V7 as a predictor of resistance to hormonal therapy and develop a new approach to assess AR-V7 by highly sensitive digital droplet polymerase chain reaction (ddPCR) in plasma-derived exosomal RNA. Design, setting, and participants Plasma samples were collected from 36 CRPC patients before they began second-line hormonal treatment. …

Oncology0301 basic medicineMaleResistanceExosomeschemistry.chemical_compoundProstate cancer0302 clinical medicineProtein IsoformsNeoplasm MetastasisReceptorAged 80 and overProstate cancerMiddle AgedProstatic Neoplasms Castration-ResistantReceptors Androgen030220 oncology & carcinogenesisBenzamidesAdenocarcinomaBiomarker (medicine)Hormonal therapyAR-V7; Digital droplet PCR; Exosomes; Hormonal therapy; Pharmacogenetics; Prostate cancer; Resistance; UrologyAndrostenesHormonal therapymedicine.medical_specialtyAntineoplastic Agents Hormonalmedicine.drug_classUrologyCastration resistantAdenocarcinomaDisease-Free Survival03 medical and health sciencesSDG 3 - Good Health and Well-beingInternal medicineNitrilesPhenylthiohydantoinmedicineEnzalutamideHumansAgedDigital droplet PCRPlasma derivedbusiness.industryRNAAndrogen Receptor Splice Variant 7medicine.diseaseAndrogenEndocrinology030104 developmental biologychemistryPharmacogeneticsDrug Resistance NeoplasmCancer cellCancer researchRNAAR-V7businessPharmacogenetics
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In the literature: February 2018

2018

Non-invasive liquid biopsies may transform the management of patients with cancer. Circulating tumour DNA (ctDNA) derived from cancer cells can be identified in most patients with advanced cancer, representing a potential non-invasive source of tumour DNA. The analysis of ctDNA may be useful to genotype the cancer, to select treatment options and to monitor response to treatment. ctDNA is often at a low level in plasma, requiring highly sensitive and accurate assays for ctDNA analysis. ctDNA was detected and profiled together with primary tumour DNA obtained from surgical specimens in 40 patients with lung cancer with localised disease who were diagnosed at stages I–III and operated on with…

OncologyCancer Researchmedicine.medical_specialtyLungbusiness.industryliteratureCancerDiseaseNewsmedicine.diseasemedicine.disease_causeMinimal residual diseasemedicine.anatomical_structureOncologyInternal medicineCancer cellGenotypemedicine1506KRASbusinessLung cancerESMO Open
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Microregional expression of glucose transporter-1 and oxygenation status: lack of correlation in locally advanced cervical cancers.

2005

Abstract Purpose: Glucose transporter-1 (GLUT-1), a target gene of hypoxia-inducible factor-1, has been considered a candidate endogenous marker of tumor hypoxia. Expression of GLUT-1 may also serve as an indicator for the induction of the transcriptional response to hypoxia, which has been linked to enhanced proliferation, resistance to therapy, and metastatic propagation of cancer cells. Overexpression of GLUT-1 has been shown to correlate with poor prognosis in several tumor entities, among them cancers of the uterine cervix. The validity of these hypotheses is investigated. Experimental Design: The expression of GLUT-1 was assessed in 80 biopsies of Eppendorf oxygenation measurement tra…

OncologyCancer Researchmedicine.medical_specialtyPathologyMonosaccharide Transport ProteinsUterine Cervical NeoplasmsBiologyInternal medicinemedicineHumansSurvival analysisNeoplasm StagingProportional Hazards ModelsCervical cancerGlucose Transporter Type 1Tumor hypoxiaProportional hazards modelGlucose transporterHypoxia (medical)Middle Agedmedicine.diseaseImmunohistochemistrySurvival AnalysisOxygenOncologyCancer cellMultivariate AnalysisImmunohistochemistryFemalemedicine.symptomClinical cancer research : an official journal of the American Association for Cancer Research
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