Search results for "catenin"

showing 10 items of 83 documents

Wnt1 Promotes Cementum and Alveolar Bone Growth in a Time-Dependent Manner

2021

The WNT/β-catenin signaling pathway plays a central role in the biology of the periodontium, yet the function of specific extracellular WNT ligands remains poorly understood. By using a Wnt1-inducible transgenic mouse model targeting Col1a1-expressing alveolar osteoblasts, odontoblasts, and cementoblasts, we demonstrate that the WNT ligand WNT1 is a strong promoter of cementum and alveolar bone formation in vivo. We induced Wnt1 expression for 1, 3, or 9 wk in Wnt1Tg mice and analyzed them at the age of 6 wk and 12 wk. Micro–computed tomography (CT) analyses of the mandibles revealed a 1.8-fold increased bone volume after 1 and 3 wk of Wnt1 expression and a 3-fold increased bone volume aft…

0301 basic medicineanimal structuresCementoblastmineralized tissue/development03 medical and health sciences0302 clinical medicinestomatognathic systemmedicineCementumGeneral DentistryDental alveolusperiodontal ligament (PDL)Chemistrybone biologyWnt signaling pathwayResearch ReportsPeriodontiumBiologicalCementogenesisCell biologycementogenesis030104 developmental biologyOdontoblastmedicine.anatomical_structure030220 oncology & carcinogenesisembryonic structuresPulp (tooth)signal transductionWnt/β-catenin signalingJournal of Dental Research
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Alterations in Tight- and Adherens-Junction Proteins Related to Glaucoma Mimicked in the Organotypically Cultivated Mouse Retina Under Elevated Press…

2020

Purpose To scrutinize alterations in cellular interactions and cell signaling in the glaucomatous retina, mouse retinal explants were exposed to elevated pressure. Methods Retinal explants were prepared from C57bl6 mice and cultivated in a pressure chamber under normotensive (atmospheric pressure + 0 mm Hg), moderately elevated (30 mm Hg), and highly elevated (60 mm Hg) pressure conditions. The expression levels of proteins involved in the formation of tight junctions (zonula occludens 1 [ZO-1], occludin, and claudin-5) and adherens junctions (VE-cadherin and β-catenin) and in cell-signaling cascades (Cdc42 and activated Cdc42 kinase 1 [ACK1]), as well as the expression levels of the growth…

0301 basic medicineelevated pressureBlotting WesternVimentinReal-Time Polymerase Chain ReactionOccludinRetinaTight JunctionsAdherens junctionMice03 medical and health scienceschemistry.chemical_compoundOrgan Culture Techniques0302 clinical medicineAntigens CDOccludinmedicinecell signalingAnimalscell contactsEye Proteinscdc42 GTP-Binding ProteinReceptorretina explantsmousebeta CateninRetinabiologyTight junctionGlial fibrillary acidic proteinChemistryGlaucomaRetinalAdherens JunctionsProtein-Tyrosine KinasesCadherinsImmunohistochemistryCell biologyMice Inbred C57BLAtmospheric Pressure030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisZonula Occludens-1 Proteinbiology.proteinInvestigative Opthalmology & Visual Science
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Diseño por Topología molecular y ensayos farmacológicos de nuevos inhibidores frente a WNT/β-CATENINA Y PI3K/Akt/mTOR

2015

El cáncer colorrectal y el de próstata causan una elevadísima tasa de muerte a nivel mundial. Por lo que, cualquier estrategia para detener o al menos reducir su desarrollo y progresión sería una importante contribución en la investigación actual. Por todo ello, el objetivo principal de la presente Tesis es la identificación de nuevos agentes quimiopreventivos frente al cáncer, que actúen modulando dos de las vías más implicadas en su desarrollo: PI3K/Akt/mTOR y Wnt/β-catenina. Hoy en día, existen diferentes enfoques para el descubrimiento de nuevos hits and leads, pero esta Tesis se centra en la metodología de la Topología Molecular, que ya ha demostrado su extraordinaria eficacia en este …

239001Cáncer de próstataQSARAktTopología molecularCáncer colorrectalDiseño de fármacos320990beta-catenina
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Topical application of the Wnt/β-catenin activator methyl vanillate increases hair count and hair mass index in women with androgenetic alopecia

2016

Activation of the WNT/β-catenin pathway has emerged as a potential therapeutic target in androgenetic alopecia (AGA). Methyl vanillate (MV) - a safe plant-derived ingredient - has been recently shown to activate the WNT/β-catenin signaling. Objectives Two distinct substudies were conducted. First, we designed a 6-month, uncontrolled, open-label clinical study to investigate whether topically applied MV may increase hair count and hair mass index (HMI) in female AGA. Second, we conducted a molecular study on the effect of MV on WNT10B mRNA expression in scalp biopsies of women with AGA. A total of 20 Caucasian women (age range: 25-57 years) with AGA (Sinclair grade 1-2) were included. The re…

Adult0301 basic medicinemedicine.medical_specialtyGene ExpressionPilot ProjectsDermatologyGenética humanaAdministration Cutaneous03 medical and health sciencesMolecular levelProto-Oncogene ProteinsInternal medicinemedicineHumansMass indexRNA MessengerAdverse effectWnt Signaling Pathwaybeta CateninVanillic AcidActivator (genetics)business.industryWnt signaling pathwayAlopeciaWNT/β-cateninMiddle AgedWnt Proteins030104 developmental biologyEndocrinologymedicine.anatomical_structureMethyl vanillateCateninScalpFemalebusinessHair
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Inhibition of tumor cell proliferation in human uterine leiomyomas by vitamin D via Wnt/β-catenin pathway.

2018

To assess the effect of vitamin D (VitD) on human uterine leiomyomas through Wnt/β-catenin pathway inhibition, apoptosis induction, and cell growth arrest.A prospective study comparing leiomyoma vs. myometrium tissues. Paired design study comparing human uterine leiomyoma primary (HULP) cells treated with or without VitD.University hospital.Human uterine leiomyoma and myometrium were collected from women (aged 35-52 years) without hormonal treatment.Samples were collected from women undergoing surgery due to symptomatic uterine leiomyoma pathology.Uterine leiomyoma and myometrium tissues were analyzed by western blot (WB) to determine proliferation, Wnt/β-catenin, and apoptosis pathways. HU…

AdultAntineoplastic AgentsApoptosisTumor Cells CulturedMedicineHumansVitamin DWnt Signaling PathwayCell ProliferationUterine leiomyomaLeiomyomabusiness.industryCell growthWnt signaling pathwayMyometriumObstetrics and GynecologyCell cycleMiddle Agedmusculoskeletal systemmedicine.diseasefemale genital diseases and pregnancy complicationsGene Expression Regulation NeoplasticLeiomyomaReproductive MedicineApoptosisCateninUterine NeoplasmsCancer researchFemalebusinessApoptosis Regulatory ProteinsFertility and sterility
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Beta-catenin and surviving expression in keratocystic odontogenic tumor (KCOT). A comparative immunohistochemical study in primary, recurrent and nev…

2013

Aim: To determine the epithelial expression of ß-catenin and survivin in sporadic (primary, and recurrent) and nevoid basal cell carcinoma syndrome (NBCCS) keratocystic odontogenic tumour (KCOT) in order to assess activation of the ß-catenin pathway and evidence of apoptotic inhibition, processes that may contribute to the known differences in their biological behaviour. Materials and Methods: Sections from 40 cases of KCOT (19 sporadic/primary; 9 sporadic/recurrent and 12 NBCCS-associated) were immunohistochemically stained for ß-catenin and survivin. The extent and intensity of immunoreactivity within the lining epithelium was assessed, using semi-quantitative scales, independently by two…

AdultMaleHistologyAdolescentSurvivin:6 - Ciencias aplicadas::61 - Medicina::616 - Patología. Medicina clínica. Oncología [CDU]2734Settore MED/50 - Scienze Tecniche Mediche ApplicateApoptosisOdontogenic Tumorsß-cateninNBCCSInhibitor of Apoptosis ProteinsSettore MED/29 - Chirurgia MaxillofaccialeYoung AdultRecurrenceSettore MED/28 - Malattie OdontostomatologicheHumansbeta CateninCell CycleNBCCS ß-catenin Survivin KCOTBasal Cell Nevus SyndromeMiddle AgedImmunohistochemistryGene Expression Regulation Neoplasticstomatognathic diseasesß-catenin; KCOT; NBCCS; Survivin; 2734; HistologyKCOTKeratinsFemale
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Gluten affects epithelial differentiation-associated genes in small intestinal mucosa of coeliac patients

2007

Summary In coeliac disease gluten induces an immunological reaction in genetically susceptible patients, and influences on epithelial cell proliferation and differentiation in the small-bowel mucosa. Our aim was to find novel genes which operate similarly in epithelial proliferation and differentiation in an epithelial cell differentiation model and in coeliac disease patient small-bowel mucosal biopsy samples. The combination of cDNA microarray data originating from a three-dimensional T84 epithelial cell differentiation model and small-bowel mucosal biopsy samples from untreated and treated coeliac disease patients and healthy controls resulted in 30 genes whose mRNA expression was simila…

AdultTranslational StudiesGlutensDuodenummedicine.medical_treatmentCellular differentiationBiopsyImmunologyBiologyCoeliac diseaseGrowth factor receptorIntestinal mucosaGene expressionmedicineImmunology and AllergyHumansRNA MessengerIntestinal MucosaImmunity Mucosalbeta CateninEpithelial cell differentiationAgedOligonucleotide Array Sequence AnalysisReverse Transcriptase Polymerase Chain ReactionGrowth factorGene Expression ProfilingCell DifferentiationEpithelial CellsMiddle Agedmedicine.diseaseSmall intestinedigestive system diseasesWiskott-Aldrich Syndrome Protein FamilyErbB ReceptorsCeliac Diseasemedicine.anatomical_structureGene Expression RegulationImmunology
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Could beta-catenin regulate the expression of proteolitic enzymes during angiogenesis?

2009

During angiogenesis several modifications occurs at endothelial cell plasma membrane level as: MT-MMPs and serine integral membrane proteases (SIMPs) over-expression. Moreover, when endothelial cells during angiogenesis acquired a mesenchymal phenotype the cell-cell interactions mediate by cadherins are lost and β-catenin, a stabilizer of interaction occurring between cadherins and cytoscheleton, can translocate to the nucleus where acts as transcription factor in association to TCF/LEF. Our experiments were focalized to the expression/activation of proteolytic enzymes when cell-cell contacts are perturbed, by mechanical or site specific perturbations. We have analyzed the mRNA, proteins an…

AngiogenesiECM remodelingBeta-cateninSettore BIO/10 - BiochimicaCell migrationProtease
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GSK-3? Can Regulate the Sensitivity of MIA-PaCa-2 Pancreatic and MCF-7 Breast Cancer Cells to Chemotherapeutic Drugs, Targeted Therapeutics and Nutra…

2021

Glycogen synthase kinase-3 (GSK-3) is a regulator of signaling pathways. KRas is frequently mutated in pancreatic cancers. The growth of certain pancreatic cancers is KRas-dependent and can be suppressed by GSK-3 inhibitors, documenting a link between KRas and GSK-3. To further elucidate the roles of GSK-3β in drug-resistance, we transfected KRas-dependent MIA-PaCa-2 pancreatic cells with wild-type (WT) and kinase-dead (KD) forms of GSK-3β. Transfection of MIA-PaCa-2 cells with WT-GSK-3β increased their resistance to various chemotherapeutic drugs and certain small molecule inhibitors. Transfection of cells with KD-GSK-3β often increased therapeutic sensitivity. An exception was observed wi…

Berberineendocrine system diseasesmedicine.medical_treatmentRegulatormedicine.disease_causeDeoxycytidinePiperazinesTargeted therapychemotherapeutic drugsTargeted therapyNitrophenolsBreast cancerGSK-3BGlycolysisMolecular Targeted TherapyNeoplasm Metastasistargeted therapy;lcsh:QH301-705.5Tumor Stem Cell AssaySulfonamidesTumorbiologyChemistryGeneral MedicineTransfectionMetforminDisease ProgressionMCF-7 CellsFemaleKRASNutraceuticalsFluorouracilSignal transductionGlycolysisSignal TransductionBCL2bcl-X ProteinAntineoplastic AgentsBreast Neoplasmsmacromolecular substancesAdenocarcinomaArticleCell LineInhibitory Concentration 50Cell Line TumorThiadiazolesmedicineDiabetes MellitusKRasHumansGlycogen synthaseProtein Kinase InhibitorsCell ProliferationChemotherapeu-tic drugsGlycogen Synthase Kinase 3 betaGSK-3βAdenylate KinaseBiphenyl Compoundsnutraceuticals;PDACβ-cateninGemcitabine?-cateninMalariaPancreatic Neoplasmslcsh:Biology (General)MCF-7DoxorubicinDietary SupplementsCancer researchbiology.protein
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Wnt activity defines colon cancer stem cells and is regulated by the microenvironment.

2010

Despite the presence of mutations in APC or beta-catenin, which are believed to activate the Wnt signalling cascade constitutively, most colorectal cancers show cellular heterogeneity when beta-catenin localization is analysed, indicating a more complex regulation of Wnt signalling. We explored this heterogeneity with a Wnt reporter construct and observed that high Wnt activity functionally designates the colon cancer stem cell (CSC) population. In adenocarcinomas, high activity of the Wnt pathway is observed preferentially in tumour cells located close to stromal myofibroblasts, indicating that Wnt activity and cancer stemness may be regulated by extrinsic cues. In agreement with this noti…

Beta-cateninColorectal cancerTransplantation HeterologousMice NudeBiologyMiceCancer stem cellParacrine CommunicationmedicineAnimalsHumansAPC microenvironmentbeta CateninHepatocyte Growth FactorWnt signaling pathwayLRP6LRP5Cell BiologyNeoplasms ExperimentalFibroblastsmedicine.diseaseCoculture TechniquesCell biologyNeoplasm ProteinsWnt ProteinsColonic Neoplasmsbiology.proteinNeoplastic Stem CellsHepatocyte growth factorStem cellmedicine.drugSignal Transduction
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