Search results for "cell damage"

showing 10 items of 38 documents

Evaluation of the Role of Candida albicans Agglutinin-Like Sequence (Als) Proteins in Human Oral Epithelial Cell Interactions

2012

The fungus C. albicans uses adhesins to interact with human epithelial surfaces in the processes of colonization and pathogenesis. The C. albicans ALS (agglutinin-like sequence) gene family encodes eight large cell-surface glycoproteins (Als1-Als7 and Als9) that have adhesive function. This study utilized C. albicans Δals mutant strains to investigate the role of the Als family in oral epithelial cell adhesion and damage, cytokine induction and activation of a MAPK-based (MKP1/c-Fos) signaling pathway that discriminates between yeast and hyphae. Of the eight Δals mutants tested, only the Δals3 strain showed significant reductions in oral epithelial cell adhesion and damage, and cytokine pro…

MAPK/ERK pathwaySciencemedicine.medical_treatmentImmunologyBlotting WesternMycologyMicrobiologyFungal ProteinsMolecular Cell BiologymedicineGeneticsHumansPhosphorylationCandida albicansCell damageBiologyMultidisciplinarybiologyQRImmunityMouth MucosaDual Specificity Phosphatase 1Epithelial Cellsbiology.organism_classificationmedicine.diseaseCorpus albicansSignaling CascadesCell biologyBacterial adhesinCytokineImmune SystemMedicineCytokinesSignal transductionCellular TypesCandidalysinCell Adhesion MoleculesProto-Oncogene Proteins c-fosResearch ArticleDevelopmental BiologySignal Transduction
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Zinc chelation during non-lesioning overexcitation results in neuronal death in the mouse hippocampus

2003

In the hippocampus, chelatable zinc is accumulated in vesicles of glutamatergic presynaptic terminals, abounding specially in the mossy fibers, from where it is released with activity and can exert a powerful inhibitory action upon N-methyl-D-aspartate receptors. Zinc is therefore in a strategic situation to control overexcitation at the zinc-rich excitatory synapses, and consequently zinc removal during high activity might result in excitotoxic neuronal damage. We analyzed the effect of zinc chelation with sodium dietyldithiocarbamate under overexcitation conditions induced by non-lesioning doses of kainic acid in the mouse hippocampus, to get insight into the role of zinc under overexcita…

MaleKainic acidSodiumchemistry.chemical_elementAMPA receptorPharmacologyInhibitory postsynaptic potentialHippocampusMicechemistry.chemical_compoundSeizuresmedicineAnimalsPremovement neuronal activityCell damageChelating AgentsNeuronsKainic AcidCell DeathGeneral NeuroscienceGlutamate receptormedicine.diseaseZincnervous systemBiochemistrychemistryNMDA receptorDitiocarbNeuroscience
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Neural overexcitation and implication of NMDA and AMPA receptors in a mouse model of temporal lobe epilepsy implying zinc chelation.

2006

Summary: Purpose: Zinc chelation with diethyldithiocarbamate (DEDTC) during nondamaging kainic acid administration enhances excitotoxicity to the level of cell damage. The objective of this work was to study the developing of the lesion in this model of temporal lobe epilepsy and the implications of the different types of glutamate receptors. Methods: The antagonist of the N-methyl-d-aspartate (NMDA) receptor MK-801, and the antagonist of the α-amino-3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA) receptor GYKI52466, were used concomitantly with intraperitoneal administration of kainic acid (15 mg/kg) followed by DEDTC (150 mg/kg) in mouse. The animals were killed at different times from 4 …

MaleKainic acidmedicine.medical_specialtyExcitotoxicityHippocampusKainate receptorHSP72 Heat-Shock ProteinsAMPA receptorBiologymedicine.disease_causeHippocampusReceptors N-Methyl-D-AspartateSynaptic Transmissionchemistry.chemical_compoundBenzodiazepinesMiceReceptors Kainic AcidInternal medicinemedicineAnimalsReceptors AMPACell damageChelating AgentsKainic AcidCell DeathGlutamate receptormedicine.diseaseDisease Models AnimalZincEndocrinologyNeuroprotective Agentsnervous systemNeurologychemistryEpilepsy Temporal LobeNMDA receptorNeurology (clinical)Dizocilpine MaleateDitiocarbProto-Oncogene Proteins c-fosEpilepsia
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Glutathione protects metastatic melanoma cells against oxidative stress in the murine hepatic microvasculature.

1998

Calcein-labeled B16 melanoma (B16M) cells were injected intraportally, and in vivo video microscopy was used to study the distribution and damage of cancer cells arrested in the liver microvasculature over a period of 4 hours. The contribution of glutathione (GSH)-dependent antioxidant machinery to the possible oxidative stress-resistance mechanism of B16M cell was determined by in vitro incubation with the selective inhibitor of GSH synthesis L-buthionine (S,R)-sulphoximine (BSO) before B16M cell injection in untreated and 0.5-mg/kg lipopolysaccharide (LPS)-treated mice. In addition, untreated and LPS-treated isolated syngeneic hepatic sinusoidal endothelial cells (HSE) were used to determ…

MalePathologymedicine.medical_specialtyCellMelanoma ExperimentalVideo RecordingVideo microscopyBiologymedicine.disease_causeAndrologychemistry.chemical_compoundMiceIn vivomedicineCell AdhesionTumor Cells CulturedAnimalsEnzyme InhibitorsNeoplasm MetastasisCell damageButhionine SulfoximineHepatologyMicrocirculationLiver NeoplasmsCell PolarityGlutathionemedicine.diseaseGlutathioneMice Inbred C57BLOxidative Stressmedicine.anatomical_structurechemistryLiverCancer cellOxidative stressIntracellularHepatology (Baltimore, Md.)
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Moderate controlled cortical contusion in pigs: effects on multi-parametric neuromonitoring and clinical relevance.

2004

Over the last decade, routine neuromonitoring of ICP and CPP has been extended with new on-line techniques such as microdialysis, tissue oxygen (ptiO(2)), acid-base balance (ptiCO(2), pH) and CBF measurements, which so far have not lead to clear-cut therapy approaches in the neurointensive care unit. This is partially due to the complex pathophysiology following a wide-range of brain injuries, and the lack of suitable animal models allowing simultaneous, clinically relevant neuromonitoring under controlled conditions. Therefore, a controlled cortical impact (CCI) model in large animals (pig) has been developed. After placement of microdialysis, ptiO(2), temperature and ICP catheters, an uni…

MalePathologymedicine.medical_specialtyMicrodialysisIntracranial PressureSwineGlutamic AcidBrain EdemaBody TemperatureCentral nervous system diseaseText miningOxygen ConsumptionPyruvic AcidmedicineAnimalsClinical significanceLactic AcidCell damageMonitoring Physiologicbusiness.industryGlutamate receptormedicine.diseasePathophysiologyDisease Models Animalmedicine.anatomical_structureGlucoseCerebral cortexAnesthesiaBrain InjuriesNeurology (clinical)businessJournal of neurotrauma
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Toxicity of used orthodontic archwires assessed by three-dimensional cell culture

2006

The aim of the present study was to determine whether used orthodontic wires made of different materials cause toxicity and loss of viability on three-dimensional (3D) cell cultures. Three types of orthodontic wires, stainless steel, Nitinol, and TMA (n = 9) which had been used clinically in fixed appliances for a period of 1 month, were retrieved at random from five patients. Both upper and lower archwires were collected and subjected to two different protocols: to assess toxicity, two pieces of each wire were placed on 3D cell cultures (reconstituted human epithelium); to investigate the possibility of cell damage, the 3-(4,5-dimethylthiazol-2-yl)2,5-diphenyl tetrazolium bromide (MTT) ass…

Malemedicine.medical_specialty3D cell culturesAdolescentH&E stainBiocompatible MaterialsHaematoxylinEpitheliumAndrologychemistry.chemical_compoundToxicity TestsAlloysOrthodontic WiresmedicineHumansMTT assayChildCell damageCells CulturedEosinChemistryStainless Steelmedicine.diseaseSurgeryStainingToxicitycytotoxicityFemaleFormazanorthodontics
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Unbaffled, Stirred Bioreactors for Animal Cell Cultivation

2017

One of the main features of animal cell bioreactors is that the cultured cells lack a strong membrane and are therefore more prone to shear damage. It is widely accepted that animal cell damage in aerated bioreactors is mainly related to burst bubbles at the air–liquid interface. A viable alternative to sparged bioreactors, aimed at minimizing cell damage, may be represented by uncovered, unbaffled, stirred tanks, which are able to provide sufficient mass transfer through the deep free surface vortex that takes place under agitation. As a consequence the need for bubble formation and subsequent bursting accompanied by cell damage is conveniently avoided. In this chapter, mass transfer perfo…

Mass transfer coefficientMass transfer coefficientMaterials scienceSettore ING-IND/25 - Impianti ChimiciMixing timeBioreactortechnology industry and agricultureMixing (process engineering)Mechanical engineeringMechanicsequipment and suppliesmedicine.diseaseImpellerkLaUnbaffled tanksMixingFree surfaceMass transferMultiphase reactorBioreactormedicineFree surface shapeLiquid bubbleCell damageAnimal cell culture
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Development of a nanostructured sensor for monitoring oxidative stress in living cells

2018

Oxidative burden is elevated in the lung of COPD patients and is associated with aging and chronic inflammation. When overcoming physiological levels, reactive oxygen species (ROS) cause cell damage and sustain inflammation. Both lung epithelium and alveolar macrophages contribute to ROS generation. Currently, ROS generation is measured using fluorescent probes and colorimetric/fluorimetric assays. We present an amperometric nanostructured sensor for real-time detection of hydrogen peroxide (H2O2) released by living cells. The H2O2 sensing performance was evaluated through the current vs time response of platinum rod at a working potential of −0.45 V vs saturated calomel electrode acting as…

Nigericin02 engineering and technology010402 general chemistrymedicine.disease_cause01 natural sciencesFlow cytometrychemistry.chemical_compoundIn vivoelectrochemical sensors hydrogen peroxide COPD ROSmedicineHydrogen peroxideCell damagechemistry.chemical_classificationReactive oxygen speciesmedicine.diagnostic_testbusiness.industryCell growth021001 nanoscience & nanotechnologymedicine.disease0104 chemical sciencesSettore ING-IND/23 - Chimica Fisica ApplicatachemistryBiophysics0210 nano-technologybusinessOxidative stress
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Swelling, Acidosis, and Irreversible Damage of Glial Cells from Exposure to Arachidonic Acid in vitro

1994

Swelling and damage of C6 glioma cells and of primary cultured astrocytes were analyzed in vitro during incubation with arachidonic acid (AA; 20:4). The cells were suspended in a physiological medium supplemented with AA at concentrations of 0.001–1.0 m M. Cell swelling was quantified by flow cytometry with hydrodynamic focusing. Flow cytometry was also utilized for assessment of cell viability by exclusion of the fluorescent dye propidium iodide and for measurement of the intracellular pH (pHi) by 2′,7′-bis-(2-carboxyethyl)−5(and −6)carboxyfluorescein. Administration of AA caused an immediate dose-dependent swelling of C6 glioma cells, even at a concentration of 0.01 m M. At this level cel…

Pathologymedicine.medical_specialtyCell SurvivalLinoleic acidIntracellular pHBiologychemistry.chemical_compoundTumor Cells CulturedmedicineLactic AcidViability assayPropidium iodideCell damageArachidonic AcidFatty AcidsSodiumHydrogen-Ion Concentrationmedicine.diseaseMolecular biologyNeurologychemistryCell cultureAstrocytesLactatesSteroidsArachidonic acidNeurology (clinical)Swellingmedicine.symptomAcidosisCardiology and Cardiovascular MedicineNeurogliaJournal of Cerebral Blood Flow & Metabolism
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Apoptosis in liver disease.

2006

The description of the morphological hallmarks of programmed cell death, apoptosis, in 1972 by Kerr, Wyllie and Currie started a field of research that revolutionized our understanding of cellular proliferation, tissue homeostasis and pathophysiology of many diseases. In the following years, a series of proteins involved in signaling and intracellular death pathways were identified and 30 years later the Noble Prize for physiology and medicine was awarded to S. Brenner, H. R. Horvitz and J. E. Sulston for their discoveries related to describing the mechanisms of cell death (apoptosis). The delineation of the signaling pathways that mediate apoptosis changed the paradigms of understanding in…

Programmed cell deathHepatologyLiver DiseasesIntrinsic apoptosisApoptosisBiologymedicine.diseaseCell biologyApoptosismedicineAnimalsHumansSignal transductionCell damageTissue homeostasisIntracellularDeath domainLiver international : official journal of the International Association for the Study of the Liver
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