Search results for "cerebral cortex"

showing 10 items of 529 documents

Attention to pain is processed at multiple cortical sites in man.

2004

Painful cutaneous laser stimuli evoked potentials (LEPs) were recorded over the primary somatosensory (SI), parasylvian, and medial frontal (MF) cortex areas in a patient with subdural electrode grids located over these areas for surgical treatment of epilepsy. The amplitudes of the negative (N2*) and positive (P2**) LEP peaks over SI, parasylvian, and MF cortex were enhanced by attention to (counting stimuli), in comparison with distraction from the stimulus (reading for comprehension). Late positive deflections following the P2** peak (late potential—LP) were recorded over MF and from the lateral premotor regions during attention but not during distraction. These findings suggest that att…

Adultmedicine.medical_specialtyNeurologyLaser-Evoked PotentialsPainPrefrontal CortexStimulus (physiology)AudiologySomatosensory systemCentral nervous system diseaseSeizuresDistractionNeural PathwaysmedicineReaction TimeHumansAttentionEvoked PotentialsCerebral CortexAfferent PathwaysBrain MappingGeneral NeuroscienceLasersMotor CortexSomatosensory Cortexmedicine.diseaseNociceptionSomatosensory evoked potentialFemalePsychologyNeuroscienceExperimental brain research
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Cortical stimulation and reflex excitability of spinal cord neurones in man.

1995

The H reflex technique was used to evaluate the influence exerted by cortical conditioning on the excitability of the alpha-motoneurone pool and on IA interneuronal activity (reciprocal inhibition). In ten subjects at absolute rest electrical and magnetic stimulation of the motor cortex was transcranially applied during flexor carpi radialis H reflex eliciting and in conditions of reciprocal inhibition induced by radial nerve stimulation. The time courses showed that at intensities below motor threshold, electrical brain conditioning induced an increase in the amplitude of the test reflex when the cortical shock was given 4 ms after the test H reflex. On the contrary, reciprocal inhibition …

Adultmedicine.medical_treatmentConditioning ClassicalWithdrawal reflexStimulationH-ReflexMagneticsMedicineHumansBiological PsychiatryCerebral CortexMotor Neuronsbusiness.industryReciprocal inhibitionElectric StimulationTranscranial magnetic stimulationPsychiatry and Mental healthElectrophysiologymedicine.anatomical_structurenervous systemNeurologySpinal CordReflexNeurology (clinical)H-reflexbusinessNeuroscienceMotor cortexJournal of neural transmission. General section
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Pattern of brain destruction in Parkinson's and Alzheimer's diseases

1996

Alzheimer's disease (AD) and Parkinson's disease (PD) are the most common age-related degenerative disorders of the human brain. Both diseases involve multiple neuronal systems and are the consequences of cytoskeletal abnormalities which gradually develop in only a small number of neuronal types. In AD, susceptible neurons produce neurofibrillary tangles (NFTs) and neuropil threads (NTs), while in PD, they develop Lewy bodies (LBs) and Lewy neurites (LNs). The specific lesional pattern of both illnesses accrues slowly over time and remains remarkably consistent across cases. In AD, six developmental stages can be distinguished on account of the predictable manner in which the neurofibrillar…

AgingPathologymedicine.medical_specialtyParkinson's diseaseModels NeurologicalLimbic systemAlzheimer DiseaseLimbic SystemmedicineHumansBiological PsychiatryNeocortexLewy bodyBrainParkinson DiseaseNeurofibrillary tangleHuman brainmedicine.diseasePsychiatry and Mental healthmedicine.anatomical_structurenervous systemNeurologyCerebral cortexLewy neuriteNeurology (clinical)PsychologyNeuroscienceJournal of Neural Transmission
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Cognitive deficits in aged rats correlate with levels of l-arginine, not with nNOS expression or 3,4-DAP-evoked transmitter release in the frontopari…

2005

Aging is associated with altered neurotransmitter function in the brain. In this study, we measured release parameters for acetylcholine (ACh), norepinephrine and serotonin in the frontoparietal cortex of young and aged rats. We also determined cortical amino acid concentrations and nitric oxide (NO) synthase function. Prior to sacrifice, the rats had been tested for Morris water-maze performance. In aged, compared with young rats, we observed a reduction in both uptake of choline and acetylcholine release. Serotonin release and L-arginine concentrations (a precursor of NO) showed an aging-related increase; however, L-citrulline/L-arginine ratios were decreased in aged rats. Moreover, while…

AgingSerotoninmedicine.medical_specialtyArginineNerve Tissue ProteinsNitric Oxide Synthase Type IArginineNitric oxideNorepinephrinechemistry.chemical_compoundNeurochemicalParietal LobeInternal medicineCortex (anatomy)medicineAnimalsCholineRats Long-EvansPharmacology (medical)4-AminopyridineNeurotransmitterBiological PsychiatryCerebral CortexPharmacologyNeurotransmitter AgentsAcetylcholineFrontal LobeRatsPsychiatry and Mental healthEndocrinologymedicine.anatomical_structureGene Expression RegulationNeurologychemistryFemaleNeurology (clinical)SerotoninAmifampridineNitric Oxide SynthaseCognition DisordersAcetylcholinemedicine.drugEuropean Neuropsychopharmacology
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Cellular Distribution and Expression of Cortical Acetylcholine Receptors in Aging and Alzheimer's Disease

1991

Ligand binding studies show marked reductions of nicotinic, but not of muscarinic binding sites in Alzheimer's disease. Using monoclonal antibodies we studied immunohistochemically the expression of the respective receptor proteins in the frontal cortex of middle-aged (55 +/- 5 yr) controls, age-matched controls (73 +/- 6 yr), and patients with Alzheimer's disease (74 +/- 5 yr). Density of nicotinic cholinoceptive neurons was 8000/mm3 for middle-aged controls and 4000/mm3 for age-matched controls, but only 900/mm3 in Alzheimer's brains (p less than 0.0001). Densities of muscarinic cholinoceptive and of Nissl-stained neurons were not significantly different between the groups, pointing to a …

Agingmedicine.medical_specialtyCORTEXGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of ScienceAlzheimer DiseaseInternal medicineMuscarinic acetylcholine receptormedicineHumansReceptors CholinergicBinding siteReceptorNEURONSAcetylcholine receptorCerebral CortexChemistryGeneral Neurosciencemedicine.diseaseCHOLINOCEPTORSCortex (botany)medicine.anatomical_structureEndocrinologyNicotinic agonistCerebral cortexAlzheimer's disease
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ADAM-10 over-expression increases cortical synaptogenesis.

2006

Cortical cholinergic, glutamatergic and GABAergic terminals become upregulated during early stages of the transgenic amyloid pathology. Abundant evidence suggests that sAPP alpha, the product of the non-amyloidogenic alpha-secretase pathway, is neurotrophic both in vitro and when exogenously applied in vivo. The disintegrin metalloprotease ADAM-10 has been shown to have alpha-secretase activity in vivo. To determine whether sAPP alpha has an endogenous biological influence on cortical presynaptic boutons in vivo, we quantified cortical cholinergic, glutamatergic and GABAergic presynaptic bouton densities in either ADAM-10 moderate expressing (ADAM-10 mo) transgenic mice, which moderately ov…

Agingmedicine.medical_specialtySynaptogenesisPresynaptic TerminalsAlpha (ethology)Mice TransgenicBiologyReceptors Metabotropic GlutamateGlutamatergicADAM10 ProteinMiceReceptors GABAInternal medicinemedicineAnimalsHumansReceptors CholinergicCerebral CortexGeneral NeuroscienceGene Expression Regulation DevelopmentalMembrane Proteinscarbohydrates (lipids)ADAM Proteinsmedicine.anatomical_structureEndocrinologyCerebral cortexSynaptic plasticitySynapsesbiology.proteinGABAergicCholinergicCattleNeurology (clinical)Geriatrics and GerontologyAmyloid Precursor Protein SecretasesDevelopmental BiologyNeurotrophinNeurobiology of aging
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Cortical responses of infants with and without a genetic risk for dyslexia

1999

We studied auditory event-related potentials (ERP) in newborns and 6-month-old infants, about half of whom had a familial risk for dyslexia. Syllables varying in vowel duration were presented in an oddball paradigm, in which ERPs to deviating stimuli are assumed to reflect automatic change detection in the brain. The ERPs of newborns had slow positive deflections typical of their age, but significant stimulus and group effects were found only by the age of 6 months. In both groups, the responses to the deviant /ka/ were more positive than those to the standard /kaa/ stimuli, contrary to the findings of adult ERPs to duration changes. The results also suggested differences in brain activatio…

Agingmedicine.medical_specialtygenetic structuresmedia_common.quotation_subjectStimulus (physiology)Audiologybehavioral disciplines and activitiesDevelopmental psychologyDyslexiaGroup differencesPhoneticsReference ValuesRisk FactorsPerceptionmedicineHumansLanguage disorderGenetic riskmedia_commonCerebral CortexGeneral NeuroscienceInfant NewbornDyslexiaInfantPhoneticsmedicine.diseaseIncreased riskAcoustic StimulationEvoked Potentials AuditoryPsychologypsychological phenomena and processesNeuroReport
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Neuronal nicotinic receptors in synaptic functions in humans and rats: physiological and clinical relevance.

2000

The present report describes the participation of nicotinic receptors (nAChRs) in controlling the excitability of local neuronal circuitries in the rat hippocampus and in the human cerebral cortex. The patch-clamp technique was used to record responses triggered by the non-selective agonist ACh and the alpha7-nAChR-selective agonist choline in interneurons of human cerebral cortical and rat hippocampal slices. Evidence is provided that functional alpha7- and alpha4beta2-like nAChRs are present on somatodendritic and/or preterminal/terminal regions of interneurons in the CA1 field of the rat hippocampus and in the human cerebral cortex and that activation of the different nAChR subtypes pres…

AgonistInterneuronmedicine.drug_classCentral nervous systemHippocampusBiologyHippocampal formationReceptors NicotinicHippocampusSynaptic TransmissionMembrane PotentialsRats Sprague-DawleyBehavioral NeuroscienceAlzheimer DiseaseInterneuronsCulture Techniquesmental disordersmedicineAnimalsHumansReceptorgamma-Aminobutyric AcidCerebral CortexNeuronsBrain Mappingmusculoskeletal neural and ocular physiologyBrainRatsmedicine.anatomical_structurenervous systemCerebral cortexSchizophreniasense organsNeuroscienceAcetylcholinemedicine.drugBehavioural brain research
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Postnatal alterations of the inhibitory synaptic responses recorded from cortical pyramidal neurons in the Lis1/sLis1 mutant mouse

2006

Mutations in the mouse Lis1 gene produce severe alterations in the developing cortex. We have examined some electrophysiological responses of cortical pyramidal neurons during the early postnatal development of Lis/sLis1 mutant mice. In P7 and P30 Lis1/sLis1 neurons we detected a lower frequency and slower decay phase of mIPSCs, and at P30 the mIPSCs amplitude and the action potential duration were reduced. Zolpidem (an agonist of GABAA receptors containing the alpha1 subunit) neither modified the amplitude nor the decay time of mIPSCs at P7 in Lis1/sLis1 neurons, whereas it increased the decay time at P30. The levels of GABAA receptor alpha1 subunit mRNA were reduced in the Lis1/sLis1 brai…

Agonistmedicine.medical_specialtyZolpidemPyridinesmedicine.drug_classAction PotentialsIn Vitro TechniquesBiologyInhibitory postsynaptic potentialMiceCellular and Molecular NeuroscienceInternal medicinemedicineAnimalsReceptorGABA AgonistsMolecular BiologyCerebral CortexReverse Transcriptase Polymerase Chain ReactionGABAA receptorPyramidal CellsAge FactorsGene Expression Regulation DevelopmentalCell BiologyElectric StimulationMice Mutant StrainsCortex (botany)ZolpidemElectrophysiologymedicine.anatomical_structureEndocrinologyAnimals NewbornInhibitory Postsynaptic Potentialsnervous systemCerebral cortex1-Alkyl-2-acetylglycerophosphocholine EsteraseMicrotubule-Associated Proteinsmedicine.drugMolecular and Cellular Neuroscience
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Design and synthesis of new trehalose-conjugated pentapeptides as inhibitors of Aβ(1-42) fibrillogenesis and toxicity

2009

Aggregation of the amyloid A? peptide and its accumulation into insoluble deposits (plaques) are believed to be the main cause of neuronal dysfunction associated with Alzheimer's disease (AD); small molecules that can interfere with the A? amyloid fibril formation are therefore of interest for a potential therapeutic strategy. Three new trehalose-conjugated peptides of the well known ?-sheet breaker peptide iA?5p,were synthesized. The disaccharide was covalently attached to different sites of the LPFFD peptide chain, i.e. at the N-terminus, C-terminus or at the Asp side chain. CD spectroscopy in different solvents was used to assess changes in the peptide conformation of these compounds. Th…

AmyloidCell SurvivalPeptideMicroscopy Atomic ForceBiochemistryMass Spectrometrychemistry.chemical_compoundbeta-sheet breaker peptideStructural BiologySFMmental disordersDrug DiscoveryAnimalsbeta-sheet breaker peptidesMolecular BiologyCells CulturedChromatography High Pressure LiquidtrehaloseCerebral CortexPharmacologychemistry.chemical_classificationthioflavin Tbeta-amyloidOrganic ChemistryP3 peptideFibrillogenesisGeneral MedicineTrehaloseSmall moleculeGlycopeptideNeuronal culturesRatsPeptide Conformationneuronal cultureBiochemistrychemistryMolecular MedicineAmyloid-betaPeptidesJournal of Peptide Science
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