Search results for "choline"

showing 10 items of 1138 documents

Uptake and metabolism of [3H]choline by the rat phrenic nerve-hemidiaphragm preparation

1987

A whole nerve-muscle preparation (about 160 mg) or an end-plate preparation (about 25 mg) of the rat phrenic nerve-hemidiaphragm were incubated with [3H]choline, to investigate choline uptake and choline metabolism. Choline uptake was measured from the disappearance of choline from the incubation medium during the loading period and from the retention of tritium in the tissue after the loading and washout period. Based on the results obtained with both methods the end-plate preparation takes up three times as much choline than the whole nerve-muscle preparation or a small muscle strip that was cut outside the end-plate region and had a similar size as the end-plate preparation. Choline upta…

Malemedicine.medical_specialtyMetaboliteDiaphragmNeuromuscular JunctionPhospholipidIn Vitro TechniquesBiologyMotor EndplateNeuromuscular junctionCholinechemistry.chemical_compoundPhosphatidylcholineInternal medicinemedicineAnimalsCholinePhrenic nervePharmacologyRats Inbred StrainsHemicholinium 3General MedicineMetabolismMuscle DenervationRatsPhrenic NerveLysophosphatidylcholinemedicine.anatomical_structureEndocrinologychemistryNaunyn-Schmiedeberg's Archives of Pharmacology
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Silibinin modulates lipid homeostasis and inhibits nuclear factor kappa B activation in experimental nonalcoholic steatohepatitis.

2012

Nonalcoholic steatohepatitis (NASH) is associated with increased liver-related mortality. Disturbances in hepatic lipid homeostasis trigger oxidative stress and inflammation (ie, lipotoxicity), leading to the progression of NASH. This study aimed at identifying whether silibinin may influence the molecular events of lipotoxicity in a mouse model of NASH. Eight-week-old db/db mice were fed a methionine-choline deficient (MCD) diet for 4 weeks and treated daily with silibinin (20 mg/kg intraperitoneally) or vehicle. Liver expression and enzyme activity of stearoyl-CoA desaturase-1 and acyl-CoA oxidase, and expression of liver fatty acid-binding protein were assessed. Hepatic levels of reactiv…

Malemedicine.medical_specialtyMice ObeseSilibininmedicine.disease_causeAntioxidantsTranslational Research BiomedicalMicechemistry.chemical_compoundMethionineNon-alcoholic Fatty Liver DiseasePhysiology (medical)Internal medicineNonalcoholic fatty liver diseasemedicineTBARSAnimalsHomeostasisNASH MCD Silibinin lipotoxicity.Reactive nitrogen speciesLiver injurychemistry.chemical_classificationReactive oxygen speciesAnti-Inflammatory Agents Non-SteroidalBiochemistry (medical)NF-kappa BPublic Health Environmental and Occupational HealthGeneral MedicineLipid Metabolismmedicine.diseaseCholine DeficiencyFatty LiverDisease Models AnimalOxidative StressEndocrinologyLiverchemistryLipotoxicitySilybinOxidative stressSilymarin
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A homeostatic mechanism counteracting K+-evoked choline release in adult brain

2002

Choline (Ch) is an essential nutrient as the biosynthetic precursor of acetylcholine (ACh) and phospholipids. Under resting conditions, the intracellular accumulation of Ch (above 10-fold), which is positively charged, is governed by the membrane potential and follows the Nernst equation. Accordingly, in synaptosomes from adult rats during depolarization, we observed a linear relationship between release of free cytoplasmic Ch and KCl concentration (2.7-120 mm). The K(+) -evoked Ch release was Ca(2+) -independent and did not originate from ACh or phospholipid hydrolysis. In superfused brain slices of adult rats, however, a K(+) -induced Ch efflux was absent. Also, under in vivo conditions, …

Malemedicine.medical_specialtyMicrodialysisMicrodialysisIn Vitro TechniquesHippocampusBiochemistryCholineCellular and Molecular Neurosciencechemistry.chemical_compoundInternal medicinePotassium Channel BlockersmedicineExtracellularAnimalsHomeostasisCholine4-AminopyridineRats WistarNeurotransmitterBrain ChemistrySynaptosomeMembrane potentialDose-Response Relationship DrugBrainBiological TransportDepolarizationHemicholinium 3RatsEndocrinologychemistryPotassiumExtracellular SpaceAcetylcholineSynaptosomesmedicine.drugJournal of Neurochemistry
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Presynaptic nicotine receptors mediating a positive feed-back on transmitter release from the rat phrenic nerve.

1986

The effects of 1,1-dimethyl-4-phenylpiperazinium (DMPP) and of nicotine receptor antagonists on [3H]acetylcholine release from the rat phrenic nerve preincubated with [3H]choline were investigated in the absence and presence of cholinesterase inhibitors (presynaptic effects). Additionally, the effects of hexamethonium and tubocurarine on the muscle contraction of the indirectly stimulated diaphragm were examined (postsynaptic effects). DMPP (1-30 microM) increased (76-92%), whereas hexamethonium (0.001-1 mM) and tubocurarine (1-10 microM) decreased (52-60%) the release of [3H]acetylcholine following a train of 100 pulses at 5 Hz. The release caused by a longer train (750 pulses at 5 Hz) was…

Malemedicine.medical_specialtyMotor nerveTubocurarineHexamethonium CompoundsIn Vitro TechniquesReceptors NicotinicNeuromuscular junctionFeedbackchemistry.chemical_compoundPostsynaptic potentialInternal medicinemedicineAnimalsCholinesterasePhrenic nervePharmacologyNeurotransmitter AgentsbiologyRats Inbred StrainsGeneral Medicinemusculoskeletal systemElectric StimulationRatsPhrenic NerveEndocrinologymedicine.anatomical_structurechemistrybiology.proteinHexamethoniummedicine.symptomDimethylphenylpiperazinium IodideAcetylcholineMuscle contractionmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Opposite role played by GABAA and GABAB receptors in the modulation of peristaltic activity in mouse distal colon.

2014

We investigated the role of GABA on intestinal motility using as model the murine distal colon. Effects induced by GABA receptors recruitment were examined in whole colonic segments and isolated circular muscle preparations to analyze their influence on peristaltic reflex and on spontaneous and neurally-evoked contractions. Using a modified Trendelenburg set-up, rhythmic peristaltic contractions were evoked by gradual distension of the colonic segments. Spontaneous and neurally-evoked mechanical activity of circular muscle strips were recorded in vitro as changes in isometric tension. GABA, at low concentrations (10-50 µM), potentiated peristaltic activity and the neural cholinergic contrac…

Malemedicine.medical_specialtyMouseColonGABAB receptorGABAA-rho receptorMicechemistry.chemical_compoundPhaclofenInternal medicinemedicineAnimalsPeristaltic activityCholinergic contractiongamma-Aminobutyric AcidPharmacologyGABAA receptorGABAA receptorBicucullineReceptors GABA-AElectric StimulationMice Inbred C57BLEndocrinologyGABA AgentsReceptors GABA-Bnervous systemchemistryMuscimolPeristalsisHexamethoniumDistal colonMuscle Contractionmedicine.drugGABAB receptor
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Beta-adrenoceptors mediate inhibition of [3H]-acetylcholine release from the isolated rat and guinea-pig trachea: role of the airway mucosa and prost…

1994

1. Rat or guinea pig isolated tracheae were labelled with [3H]-choline to measure evoked tritium outflow, which reflects neuronal release of [3H]-acetylcholine. Tritium outflow was evoked either by electrical stimulation of the extrinsic vagal nerve (rat tracheae) or by 27 mM potassium (guinea pig tracheae). 2. In rat tracheae isoprenaline (0.01, 0.1 microM) inhibited evoked [3H]-acetylcholine release, whereas beta 2-adrenoceptor-selective agonists (fenoterol, formoterol, salbutamol) were ineffective. 3. The inhibitory effect of isoprenaline was abolished under the following conditions: (i) presence of propranolol (1 microM) or of the beta 1-selective antagonist CGP 20712 A (0.1 microM); (i…

Malemedicine.medical_specialtyNeuroeffectorAdrenergic beta-AntagonistsGuinea PigsIndomethacinProstaglandinStimulationPropranololIn Vitro TechniquesCholineGuinea pigRats Sprague-Dawleychemistry.chemical_compoundIsoprenalineInternal medicineReceptors Adrenergic betamedicineAnimalsPharmacologyArachidonic AcidMucous MembranebiologyChemistryIsoproterenolMuscle Smoothrespiratory systemAdrenergic beta-AgonistsAcetylcholineRatsTracheaEndocrinologybiology.proteinProstaglandinsFemaleCyclooxygenaseAcetylcholinemedicine.drugResearch ArticleBritish journal of pharmacology
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Nicotine receptors do not modulate the 3H-noradrenaline release from the isolated rat heart evoked by sympathetic nerve stimulation.

1982

Isolated rat hearts with the right sympathetic nerves attached were perfused at a constant flow rate of 7 ml/min with Tyrode's solution. (-)-3H-Noradrenaline (final concentration 10–13.9 nM) was infused for 10 min to label the noradrenaline stores. After wash-out the sympathetic nerves were stimulated electrically (3 Hz, 180 impulses, 1 ms, 20–30 mA) three times (S1–S3) at intervals of 15 min. 3H-Noradrenaline and its metabolites were determined by liquid scintillation counting according to Graefe et al. (1973). Both, nicotine 50 μM and p-aminophenethyltrimethylammonium (PAPETA) 30 μM, enhanced the 3H-noradrenaline overflow in the absence of nerve stimulation. The effect of PAPETA was bipha…

Malemedicine.medical_specialtyNicotineSympathetic Nervous SystemSympathetic nerveStimulationIn Vitro TechniquesReceptors NicotinicTritiumReuptakeMethoxyhydroxyphenylglycol3h noradrenalineNicotinechemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsReceptors CholinergicReceptorPharmacologyNeuronsMyocardiumHeartRats Inbred StrainsGeneral MedicineRat heartElectric StimulationRatsQuaternary Ammonium CompoundsEndocrinologychemistryMandelic AcidsHexamethoniumFemalemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Differential effects of calcium channel antagonists (omega-conotoxin GVIA, nifedipine, verapamil) on the electrically-evoked release of [3H]acetylcho…

1990

Electrically-evoked release of [3H]acetylcholine from autonomic neurons (myenteric plexus), motoneurons (phrenic nerve) and the central nervous system (neocortex) was investigated in the presence and absence of the calcium channel antagonists omega-conotoxin GVIA, nifedipine and verapamil, whereby the same species (rat) was used in all experiments. Release of [3H]acetylcholine was measured after incubation of the tissue with [3H]choline. omega-Conotoxin GVIA markedly reduced (70%) the evoked release of [3H]acetylcholine from the myenteric plexus of the small intestine (IC50: 0.7 nmol/l) with a similar potency at 3 and 10 Hz stimulation. An increase in the extracellular calcium concentration…

Malemedicine.medical_specialtyNifedipinechemistry.chemical_elementMollusk VenomsMyenteric PlexusCalciumAutonomic Nervous Systemcomplex mixturesNifedipineomega-Conotoxin GVIAInternal medicinemedicineAnimalsMyenteric plexusPhrenic nervePharmacologyCerebral CortexMotor NeuronsVoltage-dependent calcium channelCalcium channelRats Inbred StrainsGeneral MedicineCalcium Channel BlockersAcetylcholineElectric StimulationRatsPhrenic NerveEndocrinologynervous systemchemistryVerapamilAnesthesiaVerapamilFemaleAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Modulation by NO of acetylcholine release in the ileum of wild-type and NOS gene knockout mice.

2002

Nitric oxide (NO) inhibits the release of acetylcholine and cholinergic contractions in the small intestine of several species, but no information is available about the mouse ileum. This study examines the effects of NO on the electrically evoked release of [3H]acetylcholine and smooth muscle contraction in myenteric plexus-longitudinal muscle preparations of wild-type mice and of neuronal NO synthase (nNOS) and endothelial NOS (eNOS) knockout mice. The NOS inhibitor N G-nitro-l-arginine (l-NNA) and the guanylyl cyclase inhibitor 1 H-[1,2,4]oxadiazolo[4,3-α]quinoxalin-1-one (ODQ) concentration dependently increased the evoked [3H]acetylcholine release and cholinergic contractions in prepa…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIPhysiologyNitric Oxide Synthase Type IIIleumNitric Oxide Synthase Type IBiologyIn Vitro TechniquesNitric OxideNitroarginineNitric oxidechemistry.chemical_compoundMiceIleumPhysiology (medical)Internal medicineQuinoxalinesmedicineAnimalsNitric Oxide DonorsEnzyme InhibitorsGene knockoutMice KnockoutOxadiazolesHepatologyPenicillamineGastroenterologyNitric Oxide Synthase Type IIISmall intestineAcetylcholineElectric StimulationNitric oxide synthaseEndocrinologymedicine.anatomical_structurechemistrybiology.proteinCholinergicNitric Oxide SynthaseGastrointestinal MotilityAcetylcholinemedicine.drugAmerican journal of physiology. Gastrointestinal and liver physiology
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Oxidative Inhibition of the Mitochondrial Aldehyde Dehydrogenase Promotes Nitroglycerin Tolerance in Human Blood Vessels

2007

Objectives We tested the hypothesis of whether an inhibition of the nitroglycerin (GTN) bioactivating enzyme mitochondrial aldehyde dehydrogenase (ALDH-2) contributes to GTN tolerance in human blood vessels. Background The hemodynamic effects of GTN are rapidly blunted by the development of tolerance, a phenomenon associated with increased formation of reactive oxygen species (ROS). Recent studies suggest that ROS-induced inhibition of ALDH-2 accounts for tolerance in animal models. Methods Segments of surgically removed arteria mammaria and vena saphena from patients undergoing coronary bypass surgery were used to examine the vascular responsiveness to GTN and the endothelium-dependent vas…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIVasodilator AgentsMyocardial InfarctionAldehyde dehydrogenaseVasodilationPharmacologyDrug Administration ScheduleTissue Culture TechniquesNitroglycerinIn vivoEnosmedicineHumansSaphenous VeinEndothelial dysfunctionMammary ArteriesAgedbiologybusiness.industryAldehyde Dehydrogenase MitochondrialDrug ToleranceAldehyde Dehydrogenasemedicine.diseasebiology.organism_classificationAcetylcholineSurgeryOxidative Stressmedicine.anatomical_structureCirculatory systemcardiovascular systembiology.proteinFemaleAnimal studiesbusinessCardiology and Cardiovascular Medicinecirculatory and respiratory physiologyBlood vesselJournal of the American College of Cardiology
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