Search results for "chromosome"

showing 10 items of 1175 documents

A Novel Risk Locus at 6p21.3 for Epstein-Barr Virus-Positive Hodgkin Lymphoma

2015

Abstract Background: A proportion of the genetic variants involved in susceptibility to Hodgkin lymphoma differ by the tumor's Epstein–Barr virus (EBV) status, particularly within the MHC region. Methods: We have conducted an SNP imputation study of the MHC region, considering tumor EBV status in 1,200 classical Hodgkin lymphoma (cHL) cases and 5,726 control subjects of European origin. Notable findings were genotyped in an independent study population of 468 cHL cases and 551 controls. Results: We identified and subsequently replicated a novel association between a common genetic variant rs6457715 and cHL. Although strongly associated with EBV-positive cHL [OR, 2.33; 95% confidence interva…

MaleEpstein-Barr Virus InfectionsEpidemiologyGenome-wide association studySUSCEPTIBILITYDISEASEMajor Histocompatibility Complex0302 clinical medicineNodular sclerosishemic and lymphatic diseasespolycyclic compoundsNetherlandsAged 80 and over0303 health scienceseducation.field_of_study[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyfood and beveragesMiddle AgedHodgkin Disease3. Good healthOncology030220 oncology & carcinogenesisUrological cancers Radboud Institute for Health Sciences [Radboudumc 15]Chromosomes Human Pair 6FemaleINFECTIOUS-MONONUCLEOSISSUBTYPEAdultAdolescentPopulationLocus (genetics)macromolecular substancesBiologyScandinavian and Nordic CountriesPolymorphism Single NucleotideSEQUENCE03 medical and health sciencesYoung AdultEBVmedicineSNPHumansGenetic Predisposition to DiseaseGENOME-WIDE ASSOCIATIONeducationEpstein–Barr virus infection030304 developmental biologyAgedCase-control studyEpstein-Barr Virus Positivemedicine.diseaseCase-Control StudiesImmunology[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyCancer Epidemiology Biomarkers & Prevention
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Distribution of heterochromatin on the mitotic chromosomes of Musca domestica L. in relation to the activity of male-determining factors

1998

In the housefly, male sex is determined by a dominant factor, M, located either on the Y, on the X, or on any of the five autosomes. M factors on autosome I and on fragments of the Y chromosome show incomplete expressivity, whereas M factors on the other autosomes are fully expressive. To test whether these differences might be caused by heterochromatin-dependent position effects, we studied the distribution of heterochromatin on the mitotic chromosomes by C-banding and by fluorescence in situ hybridization of DNA fragments amplified from microdissected mitotic chromosomes. Our results show a correlation between the chromosomal position of M and the strength of its male-determining activity…

MaleEuchromatinHeterochromatinMitosisBiologyY chromosomePolymerase Chain ReactionChromosomeschemistry.chemical_compoundHeterochromatinHousefliesGeneticsmedicineAnimalsMitosisIn Situ Hybridization FluorescenceGenetics (clinical)GeneticsB chromosomeSex ChromosomesAutosomemedicine.diagnostic_testGene Expression Regulation DevelopmentalSex Determination ProcessesMolecular biologyChromosome BandingchemistryDNA Transposable ElementsFemaleDNAFluorescence in situ hybridizationChromosoma
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Y-SNPs Do Not Indicate Hybridisation between European Aurochs and Domestic Cattle

2008

BackgroundPrevious genetic studies of modern and ancient mitochondrial DNA have confirmed the Near Eastern origin of early European domestic cattle. However, these studies were not able to test whether hybridisation with male aurochs occurred post-domestication. To address this issue, Götherström and colleagues (2005) investigated the frequencies of two Y-chromosomal haplotypes in extant bulls. They found a significant influence of wild aurochs males on domestic populations thus challenging the common view on early domestication and Neolithic stock-rearing. To test their hypothesis, we applied these Y-markers on Neolithic bone specimens from various European archaeological sites.Methods and…

MaleEvolutionary Biology/PaleontologyMitochondrial DNAScienceEvolutionary Biology/Evolutionary EcologyBiologyY chromosomePolymorphism Single NucleotideHaplogroupEvolutionary Biology/Animal GeneticsGene FrequencyY ChromosomeAnimalsDomesticationAllele frequencyHistory AncientPhylogenyGeneticsMultidisciplinaryEvolutionary Biology/Evolutionary and Comparative GeneticsQHaplotypeRAurochsbiology.organism_classificationEuropeGenetics PopulationAncient DNAHaplotypesEvolutionary biologyHybridization GeneticMedicineCattleResearch ArticlePLoS ONE
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ALDH1A3 Mutations Cause Recessive Anophthalmia and Microphthalmia

2013

Anophthalmia and microphthalmia (A/M) are early-eye-development anomalies resulting in absent or small ocular globes, respectively. A/M anomalies occur in syndromic or nonsyndromic forms. They are genetically heterogeneous, some mutations in some genes being responsible for both anophthalmia and microphthalmia. Using a combination of homozygosity mapping, exome sequencing, and Sanger sequencing, we identified homozygosity for one splice-site and two missense mutations in the gene encoding the A3 isoform of the aldehyde dehydrogenase 1 (ALDH1A3) in three consanguineous families segregating A/M with occasional orbital cystic, neurological, and cardiac anomalies. ALDH1A3 is a key enzyme in the…

MaleGenetic LinkageRetinoic acidGenes RecessiveBiologymedicine.disease_causeMicrophthalmiachemistry.chemical_compoundsymbols.namesakeChromosome SegregationReportmedicineGeneticsFood and NutritionHumansMicrophthalmosMissense mutationGenetics(clinical)Genetics (clinical)Exome sequencingSanger sequencingGeneticsMutationAnophthalmiaHomozygoteAnophthalmosExonsSequence Analysis DNAAldehyde DehydrogenaseDisease gene identificationmedicine.diseaseAldehyde OxidoreductasesMolecular biologyIntronseye diseasesPedigreeHEK293 CellschemistryAlimentation et NutritionMutationsymbolsFemaleMutant Proteinssense organsThe American Journal of Human Genetics
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Genetic linkage of autosomal dominant progressive supranuclear palsy to 1q31.1

2005

Progressive supranuclear palsy (PSP) is a disorder of unknown pathogenesis. Familial clusters of PSP have been reported related to mutations of protein tau. We report the linkage of a large Spanish family with typical autosomal dominant PSP to a new locus in chromosome 1. Four members of this family had typical PSP, confirmed by neuropathology in one case. At least five ancestors had similar disease. Other members of the family have incomplete phenotypes. The power of the linkage analysis was increased by detecting presymptomatic individuals with 18F-fluoro-dopa and 18F-deoxyglucose positron emission tomography. We screened the human genome with 340 polymorphic markers and we enriched the a…

MaleGenetic LinkageTau proteinLocus (genetics)NeuropathologyProgressive supranuclear palsyGenetic linkagemedicineHumansAgedBrain ChemistryGeneticsbiologyPutamenChromosomeDNAMiddle Agedmedicine.diseaseeye diseasesDihydroxyphenylalaninePedigreeChromosome 17 (human)GlucosePhenotypeNeurologyChromosomes Human Pair 1Genetic markerPositron-Emission Tomographybiology.proteinFemaleSupranuclear Palsy ProgressiveNeurology (clinical)Caudate NucleusLod ScoreRadiopharmaceuticalsAnnals of Neurology
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A high-density SNP linkage scan with 142 combined subtype ADHD sib pairs identifies linkage regions on chromosomes 9 and 16

2008

As part of the International Multi-centre ADHD Gene (IMAGE) project we have completed an affected sibling pair study of 142 narrowly defined DSM-IV combined type ADHD proband-sibling pairs. We found suggestive linkage on chromosomes 9 and 16 with non-parametric multipoint peak LOD scores of 2.13 and 3.1 respectively. There have been several previous ADHD linkage scans. The UCLA study (Fisher et al. 2002; Ogdie et al. 2004; Ogdie et al. 2003), the Dutch study (Bakker et al. 2003), the German study (Hebebrand et al. 2006) and the MGH Study (Faraone et al., submitted) applied the affected sib pair (ASP) strategy; the Columbian study used extended pedigrees ascertained from a population isolate…

MaleGenetics and epigenetic pathways of disease [NCMLS 6]GENOMEWIDE SCANMedizin2804 Cellular and Molecular NeuroscienceCHILDRENComorbidityNeuroinformatics [DCN 3]Severity of Illness IndexDevelopmental psychology2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]HETEROGENEITYIsraelChildGeneticsObserver Variation0303 health sciencesATTENTION-DEFICIT/HYPERACTIVITY DISORDERPSYCHIATRIC-DISORDERSDOPAMINE TRANSPORTER GENEASSOCIATION10058 Department of Child and Adolescent PsychiatryEuropePsychiatry and Mental healthFemalePsychologyChromosomes Human Pair 9linkageFunctional Neurogenomics [DCN 2]GenotypeDEFICIT HYPERACTIVITY DISORDER610 Medicine & healthPolymorphism Single NucleotideMental health [NCEBP 9]Genetic determinismWhite PeopleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]Genetic linkage1312 Molecular BiologymedicineSNPAttention deficit hyperactivity disorderHumansADHDSiblingMolecular Biology030304 developmental biologyLinkage (software)SiblingsChromosomemedicine.diseaseSib pairsUnited Statesaffected sib pairsGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with HyperactivityCONDUCT DISORDERLod ScoreDISEQUILIBRIUM030217 neurology & neurosurgeryChromosomes Human Pair 16
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A novel DFNB1 deletion allele supports the existence of a distant cis-regulatory region that controls GJB2 and GJB6 expression

2010

Contains fulltext : 87760_1.pdf (author's version ) (Open Access) Contains fulltext : 87760_2.pdf (Publisher’s version ) (Closed access) Eleven affected members of a large German-American family segregating recessively inherited, congenital, non-syndromic sensorineural hearing loss (SNHL) were found to be homozygous for the common 35delG mutation of GJB2, the gene encoding the gap junction protein Connexin 26. Surprisingly, four additional family members with bilateral profound SNHL carried only a single 35delG mutation. Previously, we demonstrated reduced expression of both GJB2 and GJB6 mRNA from the allele carried in trans with that bearing the 35delG mutation in these four persons. Usin…

MaleGenetics and epigenetic pathways of disease [NCMLS 6][SDV]Life Sciences [q-bio]PenetranceMESH: Base SequenceRegulatory Sequences Nucleic Acidsensorineural hearing lossConnexinsMESH: GenotypeMESH: Hearing Loss Sensorineural/diagnosisMESH: PenetranceGenotypeCopy-number variationGenetics (clinical)Sequence DeletionGeneticsComparative Genomic Hybridization0303 health sciencesMESH: Genetic TestingMESH: Gene Expression Regulation*030305 genetics & heredityPenetranceGJB2PedigreeConnexin 26MESH: Sequence Deletion*MESH: Hearing Loss Sensorineural/geneticsFemaleChromosome DeletionFunctional Neurogenomics [DCN 2]GJB6GenotypeMESH: PedigreeMESH: Chromosome DeletionHearing Loss SensorineuralMolecular Sequence Dataconnexin 26connexin 30DFNB1gene expression regulationGJB2GJB6sensorineural hearing losssequence deletionBiologyMESH: Connexin 30MESH: Connexins/genetics*MESH: Sequence Homology Nucleic AcidArticleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesMonoallelic MutationGJB6MESH: Connexin 26Sequence Homology Nucleic AcidConnexin 30otorhinolaryngologic diseasesGeneticsHumansGenetic TestingAlleleGeneMESH: Regulatory Sequences Nucleic Acid/genetics*AllelesDFNB1030304 developmental biologyFamily HealthMESH: HumansMESH: Molecular Sequence DataBase SequenceChromosomes Human Pair 13MESH: AllelesBreakpointMESH: MaleMESH: Comparative Genomic HybridizationGene Expression RegulationMESH: Family Healthbiology.proteinHuman medicineMESH: Chromosomes Human Pair 13/geneticsMESH: FemaleClinical Genetics
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Esr, a second locus in the house mouse controlling esterase-5

1982

Electrophoretic variation characterized by the presence (ES-5B+) or absence (ES-5B−) of esterase-5B in the plasma of the house mouse has been observed. It is suggested that the expression of esterase-5B is controlled by an autosomal locus, Esr, linked to Ldr-1 on chromosome 6, in addition to the presumptive structural locus Es-5, which is located on chromosome 8. A gene order of Lyt-3-Esr-Ldr-1 was determined by two crosses.

MaleGeneticsGenetic LinkageEsterasesChromosome MappingGenes RecessiveLocus (genetics)General MedicineBiologybiology.organism_classificationBiochemistryMolecular biologyEsteraseHouse mouseMiceGene Expression RegulationGenesGenes RegulatorGeneticsAnimalsFemaleStructural locusMolecular BiologyGeneEcology Evolution Behavior and SystematicsBiochemical Genetics
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Evidence for male XO sex-chromosome system in Pentodon bidens punctatum (Coleoptera Scarabaeoidea: Scarabaeidae) with X-linked 18S-28S rDNA clusters

2004

In scarab beetle species of the genus Pentodon, the lack of analysis of sex chromosomes in females along with the poor characterization of sex chromosomes in the males, prevented all previous investigations from conclusively stating sex determination system. In this study, somatic chromosomes from females and spermatogonial chromosomes from males of Pentodon bidens punctatum (Coleoptera: Scarabaeoidea: Scarabaeidae) from Sicily have been analyzed using non-differential Giemsa staining. Two modal numbers of chromosomes were obtained: 2n = 20 and 19 in females and males, respectively. This finding along with other karyological characteristics such as the occurrence of one unpaired, heterotypi…

MaleGeneticsSex ChromosomesbiologyHeterochromatinZoologyChromosomeKaryotypeGeneral MedicineScarabaeoideabiology.organism_classificationChromosome BandingColeopteraHeterochromatinKaryotypingGeneticsSex-determination systemAnimalsConstitutive heterochromatinFemaleMolecular BiologyRibosomal DNAIn Situ Hybridization FluorescenceX chromosomeGenes & Genetic Systems
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Rare sex chromosome aneuploidies in humans: Report of six patients with 48,XXYY, 49,XXXXY, and 48,XXXX karyotypes

1999

MaleGeneticsbusiness.industryMental DisordersChromosomeKaryotypemedicine.disease48 XXXXKaryotypingHumansMedicineFemalebusinessSex Chromosome AberrationsGenetics (clinical)American Journal of Medical Genetics
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