Search results for "chromosome"

showing 10 items of 1175 documents

Meiosis in translocation heterozygotes in the mosquito Culex pipiens.

1971

Adult Culex pipiens males irradiated with both X-rays and neutrons were crossed to untreated females and F1-egg rafts were checked for dominant lethality. F1-progenies were outcrossed with normal individuals in order to obtain lines with inherited semisterility. From a total of 120 lines that showed a certain amount of sterility 12 lines were studied cytologically. 10 lines showed reciprocal chromosome exchanges.—At late pachytene and diplotene cross configurations with large asynaptic regions at the center of the cross are obligatory. Bivalents, chains of three, chains of four, and ring configurations are present at metaphase and anaphase I. The different frequencies of the occurrence of s…

MaleHeterozygoteBiometryMitosisInterference (genetic)ChromosomesMeiosisCulex pipiensCentromereGeneticsAnimalsCrossing Over GeneticMetaphaseGenetics (clinical)Crosses GeneticAnaphaseGenes DominantGeneticsChromosome AberrationsNeutronsbiologyChromosomebiology.organism_classificationChiasmaRadiation EffectsCulexMeiosisInfertilityFemaleGenes LethalChromosoma
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A high incidence of meiotic silencing of unsynapsed chromatin is not associated with substantial pachytene loss in heterozygous male mice carrying mu…

2009

Meiosis is a complex type of cell division that involves homologous chromosome pairing, synapsis, recombination, and segregation. When any of these processes is altered, cellular checkpoints arrest meiosis progression and induce cell elimination. Meiotic impairment is particularly frequent in organisms bearing chromosomal translocations. When chromosomal translocations appear in heterozygosis, the chromosomes involved may not correctly complete synapsis, recombination, and/or segregation, thus promoting the activation of checkpoints that lead to the death of the meiocytes. In mammals and other organisms, the unsynapsed chromosomal regions are subject to a process called meiotic silencing of…

MaleHeterozygoteCancer ResearchDevelopmental Biology/Germ Cellslcsh:QH426-470BiologíaCell Biology/Cell Growth and DivisionChromosomal translocationMeiocyteBiologyTranslocation GeneticMiceMeiosisSpermatocytesGeneticsHomologous chromosomeAnimalsGene SilencingMolecular BiologyMetaphaseGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsSex ChromosomesAutosomeSynapsisChromosomeSynapsisChromatinGenetics and Genomics/Chromosome BiologyChromosome PairingMeiosislcsh:GeneticsEvolutionary Biology/Nuclear Structure and FunctionFemalePachytene StageResearch ArticlePLoS Genetics
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Frontotemporal dementia: the post-tau era.

2006

As scientists have begun to decipher the molecular genetic bases of hereditary frontotemporal dementia (FTD), it has become clear that the biology of these human neurodegenerative diseases has a complexity not previously suspected. FTD has been found to be linked to several chromosomal loci including those in chromosome 9, chromosome 17, and chromosome 3. The article by Guyant-Marechal et al. in this issue of Neurology reports the clinical, pathologic, and molecular characteristics of a form of FTD associated with inclusion body myopathy and Paget disease of the bone observed in members of two families and expands our knowledge on genetically determined FTD.1 The disorder is associated with…

MaleHeterozygoteMultiple Organ FailureDNA Mutational AnalysisChromosome 9Cell Cycle ProteinsChromosome Disorderstau ProteinsBiologyRisk AssessmentMyositis Inclusion BodyExonRisk FactorsValosin Containing ProteinmedicinePrevalenceHumansGenetic Predisposition to DiseaseGeneRetrospective StudiesGeneticsAdenosine TriphosphatasesIncidenceChromosomeSyndromeMiddle Agedmedicine.diseaseOsteitis DeformansPhenotypePedigreeChromosome 17 (human)Chromosome 3MutationDementiaFemaleNeurology (clinical)FranceFrontotemporal dementiaNeurology
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Delineation of a new chromosome 20q11.2 duplication syndrome including the ASXL1 gene.

2013

We report on three males with de novo overlapping 7.5, 9.8, and 10 Mb duplication of chromosome 20q11.2. Together with another patient previously published in the literature with overlapping 20q11 microduplication, we show that such patients display common clinical features including metopic ridging/trigonocephaly, developmental delay, epicanthal folds, and short hands. The duplication comprised the ASXL1 gene, in which de novo heterozygous nonsense or truncating mutations have recently been reported in patients with Borhing-Opitz syndrome. Because of craniofacial features in common with Borhing-Opitz syndrome, in particular metopic ridging/trigonocephaly, we suggest that duplication of ASX…

MaleHeterozygotemedia_common.quotation_subjectDevelopmental DisabilitiesNonsenseChromosomes Human Pair 20TrigonocephalyTrisomyBiologymedicine.disease_causeCraniosynostosesPregnancyIntellectual DisabilityGene duplicationGeneticsmedicineHumansCraniofacialChildGenetics (clinical)media_commonGeneticsMutationMosaicismChromosomeInfantHeterozygote advantageSyndromemedicine.diseasePhenotypeRepressor ProteinsChild PreschoolMutationFemaleHand Deformities CongenitalAmerican journal of medical genetics. Part A
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Emerging genetic patterns of the european neolithic: Perspectives from a late neolithic bell beaker burial site in Germany

2011

The transition from hunting and gathering to agriculture in Europe is associated with demographic changes that may have shifted the human gene pool of the region as a result of an influx of Neolithic farmers from the Near East. However, the genetic composition of populations after the earliest Neolithic, when a diverse mosaic of societies that had been fully engaged in agriculture for some time appeared in central Europe, is poorly known. At this period during the Late Neolithic (ca. 2,8002,000 BC), regionally distinctive burial patterns associated with two different cultural groups emerge, Bell Beaker and Corded Ware, and may reflect differences in how these societies were organized. Ancie…

MaleHuman Y-chromosome DNA haplogroupPopulation geneticsDNA MitochondrialWhite PeopleHaplogroupAnthropology Physical03 medical and health sciencesBeakerCultural EvolutionGermanyHumansCemeteries0601 history and archaeologySociocultural evolutionHistory AncientMesolithic030304 developmental biology0303 health sciences060102 archaeology06 humanities and the artsEmigration and ImmigrationArchaeologyGeographyAncient DNAHaplotypesAnthropologyGene poolAnatomyAmerican Journal of Physical Anthropology
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Stage-specific chromosomal association of Drosophila dMBD2/3 during genome activation.

2002

The Drosophila gene dMBD2/3 encodes a protein with significant homologies to the mammalian methyl-DNA binding proteins MBD2 and MBD3. These proteins are essential components of chromatin complexes involved in epigenetic gene regulation. Because the available in vitro data on dMBD2/3 are conflicting we have started an in vivo characterization of dMBD2/3. We detected expression of two isoforms specifically during embryonic development. Staining of whole embryos combined with high-resolution confocal microscopy revealed a highly regulated spatial distribution. During the syncytial blastoderm stage, dMBD2/3 formed speckles that localized to the cytoplasm. Shortly after, during the cellular blas…

MaleImmunoblottingBiologyY chromosomeGenomeChromosomesSpermatocytesY ChromosomeGeneticsAnimalsDrosophila ProteinsEpigeneticsGeneGenetics (clinical)Regulation of gene expressionMicroscopy ConfocalGene Expression Regulation DevelopmentalMolecular biologyChromatinCell biologyDNA-Binding ProteinsCytoplasmDrosophilaFemaleBlastodermProtein BindingChromosoma
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Human Y-chromosome variation in the Western Mediterranean area: Implications for the peopling of the region

2001

Y-chromosome variation was analyzed in a sample of 1127 males from the Western Mediterranean area by surveying 16 biallelic and 4 multiallelic sites. Some populations from Northeastern Europe and the Middle East were also studied for comparison. All Y-chromosome haplotypes were included in a parsimonious genealogic tree consisting of 17 haplogroups, several of which displayed distinct geographic specificities. One of the haplogroups, HG9.2, has some features that are compatible with a spread into Europe from the Near East during the Neolithic period. However, the current distribution of this haplogroup would suggest that the Neolithic gene pool had a major impact in the eastern and central …

MaleImmunologyMediterranean BasinHaplogroupGene flowMiddle Eastwest mediterranean basinAfrica NorthernY ChromosomeGenetic variationHumansImmunology and Allergyy-chromosome polymorphismsAllelesRecombination GeneticGeneticsPolymorphism GeneticMiddle EastMediterranean Regioneuropean populationsy-chromosome haplogroupsHaplotypeGenetic VariationGeneral MedicinehumanitiesEuropeGeographyHaplotypesEvolutionary biologyMultivariate AnalysisPeriod (geology)Gene poolgeographic locationseuropean populations; west mediterranean basin; y-chromosome haplogroups; y-chromosome polymorphismsMicrosatellite Repeats
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Genome-wide analysis of multi-ancestry cohorts identifies new loci influencing intraocular pressure and susceptibility to glaucoma

2014

Elevated intraocular pressure (IOP) is an important risk factor in developing glaucoma, and variability in IOP might herald glaucomatous development or progression. We report the results of a genome-wide association study meta-analysis of 18 population cohorts from the International Glaucoma Genetics Consortium (IGGC), comprising 35,296 multi-ancestry participants for IOP. We confirm genetic association of known loci for IOP and primary open-angle glaucoma (POAG) and identify four new IOP-associated loci located on chromosome 3q25.31 within the FNDC3B gene (P = 4.19 x 10(-8) for rs6445055), two on chromosome 9 (P = 2.80 x 10(-11) for rs2472493 near ABCA1 and P = 6.39 x 10(-11) for rs8176693…

MaleIntraocular pressuregenetic structuresGlaucomaGenome-wide association studyCohort Studies0302 clinical medicinePolymorphism (computer science)Risk FactorsPOPULATIONGeneticsAged 80 and overRISK0303 health scienceseducation.field_of_studyCOMMON VARIANTSASSOCIATIONMiddle AgedFemaleTRIALChromosomes Human Pair 3OPEN-ANGLE GLAUCOMAChromosomes Human Pair 9Glaucoma Open-AngleATP Binding Cassette Transporter 1AdultEXPRESSIONmedicine.medical_specialtyOpen angle glaucomaGenotypePopulationChromosome 9BiologyPolymorphism Single NucleotideArticleABO Blood-Group System03 medical and health sciencesYoung AdultMeta-Analysis as TopicOphthalmologyGeneticsmedicineHumansGenetic Predisposition to DiseaseeducationCENTRAL CORNEAL THICKNESSIntraocular PressureMETAANALYSIS030304 developmental biologyGenetic associationAgedChromosomes Human Pair 11Glaucomamedicine.diseaseeye diseasesFibronectinsREDUCTIONGenetic Loci030221 ophthalmology & optometrysense organsGenome-Wide Association StudyNature Genetics
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Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion

1996

International audience; Friedreich's ataxia (FRDA) is an autosomal recessive, degenerative disease that involves the central and peripheral nervous systems and the heart. A gene, X25, was identified in the critical region for the FRDA locus on chromosome 9q13. This gene encodes a 210-amino acid protein, frataxin, that has homologs in distant species such as Caenorhabditis elegans and yeast. A few FRDA patients were found to have point mutations in X25, but the majority were homozygous for an unstable GAA trinucleotide expansion in the first X25 intron.

MaleIron-sulfur cluster assemblyPolymerase Chain Reaction0302 clinical medicineTrinucleotide RepeatsIron-Binding ProteinsGenetics0303 health sciencesMultidisciplinaryAutosomal recessive cerebellar ataxiaPedigree3. Good healthFemalemedicine.symptomChromosomes Human Pair 9HumanPair 9Heterozygotecongenital hereditary and neonatal diseases and abnormalitiesAtaxiaMolecular Sequence DataGenes RecessiveLocus (genetics)BiologyChromosomes03 medical and health sciencesGene mappingAlleles; Amino Acid Sequence; Base Sequence; Chromosomes Human Pair 9; DNA Primers; Female; Friedreich Ataxia; Genes Recessive; Heterozygote; Humans; Male; Molecular Sequence Data; Pedigree; Point Mutation; Polymerase Chain Reaction; Proteins; Sequence Alignment; Introns; Iron-Binding Proteins; Trinucleotide RepeatsmedicineRecessiveHumansPoint MutationAmino Acid SequenceAlleleAllelesDNA Primers030304 developmental biologyBase SequencePoint mutationProteins[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologymedicine.diseaseMolecular biologyIntronsGenes[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsFriedreich AtaxiaFrataxinbiology.proteinSequence Alignment030217 neurology & neurosurgeryScience
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Killer cell immunoglobulin-like receptor genes in Latvian patients with type 1 diabetes mellitus and healthy controls.

2004

T1DM is very common in Sweden and is positively associated with HLA class II genes. Approximately 89% of the newly diagnosed patients carry the high-risk HLA DR4-DQ8 and DR3-DQ2. The remaining 11% develop T1DM without them. This can be due to involvement of other genes and environmental factors. Natural killer (NK) cells of the innate immune system are important in antiviral and antitumor immunity. They are implicated in the etiology of autoimmune T1DM. Human NK cells express killer cell immunoglobulin-like receptors (KIR) that belong to the polymorphic multigene family in chromosome 19q3.4. They modulate NK cell response by interacting with HLA class I. In addition, polymorphic MICA in HLA…

MaleKiller-cell immunoglobulin-like receptorHuman leukocyte antigenBiologyGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of ScienceGene FrequencyReceptors KIRimmune system diseasesPolymorphism (computer science)HumansAlleleReceptors ImmunologicReceptorAllele frequencyAllelesInnate immune systemPolymorphism GeneticGeneral NeuroscienceHistocompatibility Antigens Class Inutritional and metabolic diseasesAcquired immune systemLatviaKiller Cells NaturalDiabetes Mellitus Type 1Gene Expression RegulationCase-Control StudiesReceptors KIR2DL2ImmunologyFemaleChromosomes Human Pair 19Microsatellite RepeatsAnnals of the New York Academy of Sciences
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