Search results for "chronic inflammation"

showing 5 items of 25 documents

Magnesium, Oxidative Stress, and Aging Muscle

2014

Abstract Magnesium (Mg) deficiency, aside from having a negative impact on the energy production pathways required by the mitochondria to generate ATP, also reduces the threshold antioxidant capacity of the aging organism and its resistance to free-radical damage. Mg acts as an antioxidant against free radical damage of the mitochondria. Chronic Mg deficiency results in excessive production of oxygen-derived free radicals and low-grade inflammation. Chronic inflammation and oxidative stress have been identified as pathogenic factors in several age-related conditions. Aging is often associated with Mg inadequacy, muscle loss, and sarcopenia. Although the importance of magnesium as a determin…

chemistry.chemical_classificationReactive oxygen speciesmedicine.medical_specialtySettore MED/09 - Medicina InternaAntioxidantbusiness.industryMagnesiummedicine.medical_treatmentchemistry.chemical_elementInflammationMitochondrionmedicine.diseasemedicine.disease_causeEndocrinologychemistrySarcopeniaInternal medicinemedicinemedicine.symptombusinessMagnesium Aging Mg Deficiency Muscle Oxidative stress Chronic inflammation Sarcopenia Free radical Reactive oxygen species Muscle performance IonsOxidative stressFree-radical theory of aging
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Regulation of T-Cell Immune Responses by Pro-Resolving Lipid Mediators

2021

Both the initiation and the resolution of inflammatory responses are governed by the sequential activation, migration, and control/suppression of immune cells at the site of injury. Bioactive lipids play a major role in the fine-tuning of this dynamic process in a timely manner. During inflammation and its resolution, polymorphonuclear cells (PMNs) and macrophages switch from producing pro-inflammatory prostaglandins and leukotrienes to specialized pro-resolving lipid mediators (SPMs), namely, lipoxins, resolvins, protectins, and maresins, which are operative at the local level to limit further inflammation and tissue injury and restore homeostasis. Accumulating evidences expand now the rol…

chronic inflammationT cellT-LymphocytesImmunologyInflammationReviewBiologymedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunity03 medical and health sciencesImmunomodulating Agents0302 clinical medicineImmune systemmedicineHumansImmunology and Allergy030304 developmental biology0303 health sciencestherapyEffectorautoimmunityresolutionT cellLipid signalingadaptive immunityRC581-607Acquired immune systemCellular Reprogramming3. Good healthLipoxinsmedicine.anatomical_structureEicosapentaenoic AcidImmunologyspecialized pro-resolving lipid mediators (SPMs)Eicosanoidsmedicine.symptomImmunologic diseases. AllergyInflammation MediatorsHomeostasis030215 immunologyFrontiers in Immunology
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MAGNESIUM IN AGING

2011

magnesium aging oxidative stress chronic inflammationSettore MED/09 - Medicina Interna
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Magnesium and aging.

2009

Over the past decades, the clinical relevance and biological significance of magnesium (Mg) have been documented. Deficiency in Mg, aside from having a negative impact on the energy production pathway required by mitochondria to generate ATP, also reduces the threshold antioxidant capacity of the aging organism and its resistance to free-radical damage. Mg also acts as an antioxidant against free radical damage of the mitochondria. Chronic inflammation and oxidative stress have both been identified as pathogenic factors in aging and in several age-related diseases. Chronic Mg deficiency results in excessive production of oxygen-derived free radicals and low grade inflammation. Aging is very…

magnesium aging oxidative stress chronic inflammationSettore MED/09 - Medicina Internamagnesium aging inflammation oxidative stress
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Treatment with a CO-releasing molecule (CORM-3) reduces joint inflammation and erosion in murine collagen-induced arthritis.

2008

Contains fulltext : 70589.pdf (Publisher’s version ) (Closed access) OBJECTIVE: CO-releasing molecules (CO-RMs) are a novel class of anti-inflammatory agents. We have examined the possible therapeutic effects of CORM-3 in collagen-induced arthritis (CIA). METHODS: Arthritis was induced in DBA-1/J mice by type II collagen. Animals were treated with CORM-3 (5 and 10 mg/kg/day, intraperitoneally) or the inactive compound iCORM-3 (10 mg/kg/day, intraperitoneally) unable to release CO, from days 22 to 31. Production of anti-type II collagen antibodies, cytokines and cartilage olimeric matrix protein (COMP) was evaluated by enzyme-linked immunosorbent assay, and prostaglandin E(2) (PGE(2)) by rad…

musculoskeletal diseasesmedicine.medical_treatmentImmunologyAnti-Inflammatory AgentsDrug Evaluation PreclinicalType II collagenArthritisInflammationPharmacologyAuto-immunity transplantation and immunotherapy [N4i 4]DinoprostoneGeneral Biochemistry Genetics and Molecular BiologyMiceRheumatologyOrganometallic CompoundsPerception and Action [DCN 1]medicineAnimalsImmunology and AllergyChronic inflammation and autoimmunity [UMCN 4.2]Dose-Response Relationship Drugbiologybusiness.industryRANK LigandInterleukinIntercellular Adhesion Molecule-1medicine.diseaseArthritis ExperimentalPathogenesis and modulation of inflammation [N4i 1]Cellular infiltrationCyclooxygenase 2Mice Inbred DBARANKLImmunologybiology.proteinCytokinesTumor necrosis factor alphaMicrobial pathogenesis and host defense [UMCN 4.1]Inflammation Mediatorsmedicine.symptombusinessInfection and autoimmunity [NCMLS 1]Heme Oxygenase-1Immunity infection and tissue repair [NCMLS 1]Prostaglandin E
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